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I Tested 100,000 People's DNA. This Diet Will Kill You - Gary Brecka

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I Tested 100,000 People's DNA. This Diet Will Kill You - Gary Brecka

Transcript

2819 segments

0:00

No one really tells people that have

0:02

anxiety what it is. And this is why very

0:05

often people don't have a specific

0:07

trigger they can point to. So, they're

0:08

trying to pin it on their outside

0:09

environment, but the truth is that they

0:11

are deficient. Usually in

0:15

Gary Brecka.

0:16

He's a human biologist.

0:17

Who spent 20 years working in life

0:19

insurance predicting when people are

0:21

going to die to the nearest month.

0:23

And now he's on a mission to extend your

0:25

life. A couple of days ago someone did a

0:27

swab inside of my mouth. What was that

0:30

test and why did I do it? You did it to

0:32

look at whether your parents gave you a

0:33

gene mutation. And it's one of the most

0:35

overlooked things in all of modern

0:36

medicine because it's this deficiency

0:38

that leads to some of the most common

0:40

elements that we suffer from. Mental

0:42

illness, ADHD, OCD, manic depression,

0:45

bipolar, sleep disorders, very severe

0:47

gut issues. I mean, there are so many

0:49

that don't seem to be fixable with

0:51

conventional therapies or dietary

0:52

changes because very often disease is

0:54

not happening to us, it's happening

0:56

within us. And I'm not going to stop

0:58

getting the message out to the masses

1:01

because

1:02

I just think about all the times I could

1:03

have

1:05

made a real material change in

1:06

somebody's life and I didn't have the

1:08

opportunity to do it and felt like I was

1:11

you know, sitting behind a thick glass

1:13

wall just watching blind people walk

1:15

into traffic.

1:18

Now I got a chance to make a difference.

1:21

So, what are like the simple things that

1:23

we can be doing to prevent us even

1:24

getting these chronic diseases? So,

1:26

there's five things that I would commit

1:28

to doing on a regular basis. Number one

1:30

is upon waking, I would

1:34

I wanted to invite in Dr. Carrie Saida

1:36

who's going to give me those results of

1:37

my test. I want to know there's any sort

1:39

of health implications that I should be

1:40

aware of. Okay.

1:41

So, that right there is an issue.

1:46

Congratulations Diary of a CEO gang.

1:47

We've made some progress. 63% of you

1:50

that listen to this podcast regularly

1:52

don't subscribe, which is down from 69%.

1:56

Our goal is 50%. So, if you've ever

2:00

liked any of the videos we've posted, if

2:01

you like this channel, can you do me a

2:03

quick favor and hit the subscribe

2:04

button. It helps this channel more than

2:05

you know and the bigger the channel gets

2:07

as you've seen, the bigger the guests

2:09

get. Thank you and enjoy this episode.

2:15

Gary.

2:16

Steven. Good to see you back.

2:19

Don't throw me off. It's good to have

2:21

you I was saying

2:22

I knew I'd do your game off there.

2:24

Steven.

2:26

Great to be back, man. It really is.

2:28

If someone's just clicked on this

2:29

podcast

2:31

and they're wondering why they should

2:32

stay and listen. You're going to direct

2:34

the conversation. I'm going to go where

2:35

you want to go. I'm going to follow my

2:36

curiosity and ask questions. But what

2:38

are they going to get from this

2:39

conversation today if you are at the

2:40

driving wheel?

2:42

I'm just data on how to live a

2:43

healthier, happier, longer life and

2:45

maybe answers to some of the most um

2:48

pesky health related challenges that

2:51

they're having and I and when I say

2:53

pesky health related challenges, I mean,

2:54

everybody has these little tiny anchors

2:56

off their stern, right? Weight gain,

2:58

water retention, brain fog, lack of

3:00

focus and concentration, poor waking

3:02

energy, lack of deep sleep.

3:04

And it's shocking how many of these

3:07

conditions have

3:09

a common repository. I mean, they like

3:10

the hub of the wheel where they all

3:12

meet. All these individual spokes to

3:13

come together to common hub of

3:16

methylation. And they and and

3:18

methylation is essentially nutrient

3:19

deficiencies in the human body.

3:22

And

3:23

I don't usually start off with this

3:25

analogy, but I'll start off with an

3:26

analogy.

3:27

Um when I was in grad school, first of

3:29

all, I'm a human biologist. I'm not a

3:30

physician. I My undergraduate degrees

3:32

are in biology. My post-graduate degrees

3:34

are in human biology.

3:35

But when I was in my second four years

3:37

of of grad school getting human biology

3:40

degree, I had to I had to take all these

3:41

plant botany courses,

3:43

which I hated cuz I was like I wanted to

3:45

study like anatomy and physiology in

3:46

human beings, but I'm studying algae and

3:49

and but the one thing that stood out to

3:51

me about plant physiology was

3:53

let's say you have a a palm leaf that's

3:55

rotting in a palm tree and you call a

3:57

true arborist, a true botanist out to

3:59

your house

4:00

and they see that that leaf is rotting

4:02

in the tree. They won't touch that leaf.

4:03

They will core test the soil.

4:05

And then they'll say, "You know what,

4:06

Steven, there's no nitrogen in the

4:07

soil." And they'll add nitrogen to the

4:10

soil and the leaf will heal.

4:12

Only we've stopped thinking about human

4:14

beings this way. We've lost a lot of

4:16

faith in humanity and mankind, the

4:17

body's ability for this to heal this.

4:20

And we believe very often, and this is

4:23

true in some cases, that disease and

4:25

pathology are happening to us,

4:27

not something that happens within us.

4:30

And if you go back to the tree analogy,

4:31

you know, you could put it anything you

4:33

wanted on that soil, right? You could

4:34

supplement for the sake of

4:36

supplementing. And I think a lot of

4:37

people get lost in this realm where

4:40

well, I heard NMN is good and

4:41

resveratrol is good and CoQ10 and St.

4:43

John's Wort and ashwagandha and vitamin

4:44

C and I should take a multivitamin and

4:46

you know, and pretty soon you have this

4:48

paralysis of analysis because you're

4:50

supplementing for the sake of

4:51

supplementing.

4:52

And in the tree's case, if you didn't

4:54

find the nitrogen, the leaf never would

4:56

have healed. And and the reason why most

4:58

of us supplement for the sake of

4:59

supplementing is that we don't have

5:00

data. We just don't get data on our

5:02

bodies. You know, when when when I bring

5:05

and you you run into a lot of a lot more

5:07

young entrepreneurs than I do, but when

5:08

I run into them and I'll bring them up

5:10

sometimes when I'm doing a stage talk

5:12

and you can question them um about their

5:15

priorities. Like, what's the most

5:17

important thing to you? My health. Um

5:19

how important is health to you? Oh, it's

5:21

the number one priority I have. And I

5:23

say, well, come come on up here and

5:24

let's let's talk about, you know, how

5:26

much you're you're prioritizing your

5:28

health and you say, um you know, what

5:31

kind of business do you have? A

5:31

marketing agency. What does your

5:33

business earn on a monthly basis?

5:35

$148,000

5:36

a month. What's your net income? $38,250

5:40

a month. How many employees do you have?

5:41

16.

5:42

What's your hemoglobin A1C?

5:46

Blank.

5:47

Right? Where are your testosterone

5:49

levels?

5:50

Um how about your triglycerides?

5:53

Do you ever look at your C-reactive

5:55

protein? And their face is just blank.

5:57

And we have more data on our businesses

5:59

than we do on our temple.

6:02

And you know, I I I actually saw you on

6:04

a stage talk, I believe it was. Um

6:06

and you talked about how you could take

6:08

anything away from me in my life. You

6:10

know, you you you referred to your dog

6:12

and your girlfriend.

6:14

Yeah. So,

6:15

hopefully you still have the dog and the

6:17

girlfriend, but Yeah. you said, you

6:18

know, if you took my girlfriend away,

6:20

you took my dog away. I don't want I

6:22

don't want to get you in a fight with

6:23

your girlfriend. So, let's talk about

6:23

the dog. So, you took my dog away, I'd

6:25

still have my business. I still have my

6:27

life, right? But if you took away my

6:29

health,

6:31

I'd lose everything.

6:32

Right? And I think most of us don't

6:35

realize the importance of it until it is

6:37

taken away. And so, recognizing that

6:39

that the temple is the most important

6:40

vehicle that we have,

6:42

I just really encourage people to get

6:44

data, basic data on their body so they

6:46

have some kind of road map so they are

6:49

supplementing for deficiency, not just

6:51

the sake of of of supplementing and that

6:53

they're getting the most out of their

6:55

body because that's what they're going

6:57

to get That's how they're going to get

6:57

the most out of their business.

6:59

You know what I mean? Just

7:00

picking up these little tiny anchors

7:02

that are nibbling away at productivity.

7:04

You know, people that suffer from ADD

7:06

and ADHD don't really realize that

7:10

ADD and ADHD very often are not

7:12

attention deficit disorders or attention

7:14

deficit hyperactivity disorders. They're

7:15

actually attention overload disorders.

7:18

And we characterize people that have ADD

7:21

as as not being able to pay attention,

7:23

but the truth is they they don't lack

7:25

the ability to pay attention. They lack

7:26

the ability to pay attention to so many

7:27

things.

7:29

And if we understand that this is an

7:31

overactive mind,

7:33

not a mind that's trying to pay

7:34

attention to too many uh too many

7:36

things, then

7:38

we can go about quieting the mind and

7:40

not stimulating the central nervous

7:43

system to match that pace of the mind,

7:45

which is kind of what Adderall and

7:46

Five-Ams and and um

7:48

uh amphetamines do when you when you you

7:51

when you take them for ADD and ADHD. So,

7:53

if we understood that

7:56

as normal or as good as we think we

7:58

feel, we have no idea how good normal

8:00

feels until we find the missing raw

8:02

material in our body and we put it back.

8:04

You want to see magic happen in human

8:06

beings?

8:07

Find the raw material that's missing and

8:09

put it back in their body. And by raw

8:11

material, I mean simple things. You

8:13

know, I mean, depending on who you talk

8:14

to, there's 72 minerals. I think 16 of

8:18

those are essential minerals. There are

8:20

there are two essential fatty acids.

8:21

There are eight essential amino acids.

8:24

It is astounding how many people are

8:26

clinically deficient in some of those

8:28

basics.

8:29

And then they go searching in all of the

8:32

esoteric super supplements and red light

8:34

therapy and and NAD boosting supplements

8:38

and they're actually just missing that

8:39

one of those raw materials. Basic

8:42

essential amino acids, basic fatty

8:44

acids, and basic minerals. And that's

8:46

where all human beings should start. And

8:48

then from there, we should do some

8:51

testing, biomarkers in the blood. In my

8:53

opinion, every human being should do a

8:55

genetic methylation test, the very same

8:56

test that you did.

8:58

Whether they do it through me or not, a

9:00

genetic methylation test is a test

9:03

you'll do once in your lifetime and it

9:05

will tell you exactly what raw materials

9:07

your body can convert into the usable

9:10

form and what it can't.

9:12

Because in human beings, just like in

9:14

that tree analogy, when you have a

9:16

deficiency, you get the expression of

9:18

disease.

9:19

You know, when we say you talk about

9:20

deficiencies,

9:21

Mhm. it brings me back to something that

9:23

I think we talked about briefly last

9:25

time, which is it makes me feel like

9:27

humans are being born broken.

9:30

Is that true? Because if I've got if I'm

9:31

deficient in something that my body

9:33

needs, then does that not mean that my

9:35

body was born broken? It's not that it's

9:36

born broken, it's just not functioning

9:38

optimally, right? And and all of us have

9:42

um

9:42

genetic snips. We have these that are

9:44

called single nucleotide polymorphisms.

9:46

We have these

9:47

um our genes which are coding for

9:49

enzymes to conduct these different

9:51

activities in the body. And what is

9:53

astounding about human beings is is how

9:56

beautifully intricate the human body is.

9:58

We take one raw material, we put it into

10:00

a physiological process, and then we

10:02

take the waste product from that

10:05

process and we feed another process and

10:07

on and on. So, for example, we'll take

10:11

um folate from green leafy vegetables.

10:13

We'll convert that into methylfolate.

10:16

Methylfolate becomes one of the most

10:17

prevalent nutrients in the human body.

10:19

It helps to downregulate an inflammatory

10:22

amino acid called homocysteine, which

10:24

then turns into something called

10:26

methionine, which then goes up to the

10:27

brain and helps to quiet the mind. So,

10:30

you start with this green leafy

10:31

vegetable and it winds its way all the

10:33

way up to helping you sleep. And it's

10:35

not that the spinach leaf is helping you

10:37

sleep, it's what the spinach leaf has

10:39

become that's helping you sleep. And

10:41

this sequence of events is called

10:42

methylation.

10:44

And what's astounding about methylation

10:46

is that

10:47

in many cases when it's broken,

10:50

while we can't fix the gene, we can just

10:52

supplement for its function.

10:54

So, the most common gene mutation in the

10:55

world, which we talked about last time

10:56

on your podcast, MTHFR,

10:59

affectionately called the [ __ ]

11:00

gene, um 44% of the population has this

11:02

gene mutation. I talk about it all the

11:04

time and it's a simple inability to to

11:07

to to convert folic acid and its

11:10

derivatives um folate into the usable

11:13

form called methylfolate. Well, it's

11:15

very easy to supplement with

11:17

methylfolate and very inexpensive, I

11:19

might add, to supplement with

11:20

methylfolate. And deficiencies in

11:22

methylfolate are are linked to all kinds

11:25

of conditions, including neural tube

11:27

defects, because it's not folic acid

11:29

that prevents neural tube defects, it's

11:30

methylfolate that prevents neural tube

11:32

defects. It's what the body converts it

11:34

into, the usable form. And so, when we

11:36

look at um

11:38

methylation in the human body, we get an

11:40

exact roadmap of what we need to

11:42

supplement with, so that we're not

11:44

wandering around just supplementing for

11:47

the sake of just supplementing.

11:49

You mentioned the entrepreneurs that you

11:50

meet that you bring up on stage and you

11:51

ask them various questions and then you

11:52

ask them about sort of biomarkers in

11:54

their body. What are the simple

11:57

biomarkers you think that everyone needs

11:59

to understand because listen, I'm not a

12:01

chemist, I'm not going to be a

12:01

biologist. So, if there's a couple of

12:03

them, I can probably get my head around

12:05

them and stay on top of them, but I

12:06

can't stay on top of everything. Yeah,

12:08

so there there's I would say three.

12:10

Okay. Number one is um what's called

12:12

glycemic profile, which is a a check of

12:15

how well your insulin and sugar

12:18

metabolism is. Yep. And it has three

12:20

markers, glucose,

12:22

hemoglobin A1C, 3-month average of your

12:24

blood sugar, and insulin.

12:26

So, definitely your glycemic profile,

12:28

because blood sugar, I promise you, is

12:30

the root of all evil. So, first I would

12:32

I would do your glycemic profile.

12:33

Second, I would do um

12:35

your hormones. Can I just check on the

12:37

glycemic profile? That's basically my

12:39

relationship with

12:41

sugar. That's your relationship with

12:42

sugar. And it's it's also your

12:44

relationship with insulin, because, you

12:46

know, very often even people that don't

12:48

eat high amounts of refined sugar and

12:50

Ben & Jerry's ice cream every night have

12:52

issues with insulin sensitivity.

12:54

And so, as insulin

12:56

rises, it causes a whole myriad of

13:00

conditions. It's one of the hallmarks of

13:01

something called metabolic syndrome,

13:02

which we're seeing in younger and

13:03

younger populations. And it's generally

13:06

easy to catch early on. You have three

13:08

markers that look at how well you're

13:10

regulating your blood sugar, glucose,

13:11

which is a measure of your the amount of

13:13

sugar in your blood right now,

13:15

hemoglobin A1C, which is essentially a

13:17

3-month average of your blood sugar, and

13:19

then you have your insulin.

13:21

And the higher your insulin relative to

13:23

your blood sugar, the more insulin

13:25

resistant you are, right? So, the more

13:27

insulin it takes to drop your blood

13:29

sugar, the more resistant you are to

13:30

insulin. This is an early warning sign

13:32

of metabolic syndrome.

13:34

But it's not just the metabolic

13:35

syndrome, it's that when insulin rises,

13:37

there's a whole downstream cascade of

13:39

events, because insulin's not just

13:40

responsible for helping us metabolize

13:43

sugar, it's also responsible for

13:45

blocking other forms of energy use in

13:47

the body, one of which is fatty acid

13:49

metabolism. And so, generally people

13:51

that have very high insulin have very

13:53

high blood triglycerides. They have high

13:56

blood fat.

13:57

And high blood fat and elevated

13:59

cholesterol are other markers for

14:01

cardiovascular disease. So, by actually

14:03

bringing down one biomarker, you have a

14:06

positive effect all the way downstream.

14:09

And I would say if you're only going to

14:10

look at three things, I would look at um

14:12

your glycemic profile, your your blood

14:15

sugar, and your insulin, and your

14:16

hemoglobin A1C.

14:18

Hormone panel.

14:19

Okay. Um so, looking at your hormones

14:22

and then specifically looking at what

14:24

contributes to healthy hormone

14:26

production, DHEA and a protein called

14:29

SHBG.

14:31

And then I would look at basic nutrient

14:33

deficiencies,

14:34

um vitamin D3, magnesium, potassium, and

14:39

um vitamin B12, which are on most blood

14:41

panels. And that is a great place to

14:43

start to get the basics. How am I

14:45

regulating my blood sugar? Are my

14:47

hormones balanced? What nutrient

14:49

deficiencies do I have? And then the

14:51

second piece of information I would get,

14:53

which you only do once in your entire

14:54

lifetime, is is a methylation test. And

14:56

these are these are widely available,

14:59

you know, all over the world. I'm sure

15:00

they're very easy to get in the UK. And

15:02

you want to look at five genes, MTHFR,

15:05

MTR, MTRR,

15:08

AHCY,

15:09

and COMT. And just to be clear, so I I

15:12

can dumb this down for myself here,

15:14

those five genes

15:16

relate to how my body processes the

15:18

things I put into it at different stages

15:21

in that sort of processing line. Yes, I

15:23

mean, to to take for example that gene

15:25

at the top, COMT.

15:27

Um if you look at what COMT does, it

15:29

stands for catechol-O-methyltransferase.

15:32

So, it's a fancy way of saying it

15:33

transfers a methyl group from the

15:35

category of neurotransmitters called

15:37

catecholamines. And that's a huge

15:39

mouthful, but essentially what this

15:40

means is these four neurotransmitters

15:43

that are called catecholamines are

15:45

responsible for our fight-or-flight

15:47

response. So, for example, if you drove

15:49

home tonight and you got out of your car

15:52

and somebody was standing in front of

15:53

you with a knife. Mhm. Right? You would

15:55

instantly have a fight-or-flight

15:57

response. Your pupils would dilate, your

15:58

heart rate would increase, your

15:59

extremities would flood with blood. You

16:01

would begin to have a fight-or-flight

16:02

response based on that stimulus. But you

16:04

could also be laying in your bed at

16:06

night

16:07

and you could start thinking about

16:09

getting eaten by a shark.

16:11

And you could have the exact same

16:13

response,

16:14

because the brain doesn't really know

16:15

the difference between perception and

16:17

reality. The the similarity between

16:20

these two events, a very real fear and a

16:22

reaction to it, and an imagined fear, is

16:24

they both meet at

16:27

catecholamines. It's a rise in the same

16:29

class of neurotransmitters.

16:31

So, now some people are very slow to

16:34

break these down. And what are the

16:35

consequences of this? Well, if you've

16:37

ever had anxiety or know somebody that's

16:40

suffered from anxiety, no one really

16:42

tells people that have anxiety what it

16:44

is. They describe the feeling. So, they

16:48

say it's a fear of the future. It's

16:50

it's a sense of impending doom. It's a

16:51

sense of anxiousness.

16:53

But what is it? Physiologically, what's

16:55

going on in the body? Well, very often

16:57

it is a rise in catecholamines, the same

17:01

class of neurotransmitters that are that

17:03

are involved in a fight-or-flight

17:04

response. And this is why very often

17:06

people that suffer from anxiety don't

17:08

have a specific trigger they can point

17:10

to. They could be on a podcast like you

17:12

and I are going doing right now, and all

17:14

of a sudden as those catecholamines

17:16

rise, they get that sensation of

17:18

anxiety. And they're trying to pin it on

17:20

their outside environment. They're

17:21

trying to look for a cluster of symptoms

17:23

outside their body. But this is because

17:25

they are deficient, usually, in a

17:27

complex of B vitamins, um a very

17:30

specific form of B12 called

17:31

methylcobalamin, methylfolate, these

17:34

methylated nutrients that that

17:35

downregulate these catecholamines. What

17:38

else could it be?

17:40

Well, I mean, there are true anxiety

17:42

disorders, right? Um and generally

17:44

people that have true anxiety actually

17:46

know what the trigger is. They're afraid

17:47

of heights, they walk to the edge of a

17:48

30th floor balcony, they have an anxiety

17:50

um attack. They have a fear of flights.

17:52

Is this different from So, someone's

17:54

been through a trauma in their life, say

17:55

they had an early trauma, and then

17:57

certain things in their adult life end

17:58

up triggering that. Mhm.

18:00

This is that's very different from what

18:02

you're saying here. So, some people that

18:04

have trauma and they wake up, for

18:06

example, consistently in the past,

18:08

right? Their first thought of the day is

18:10

about the pain that they are already

18:13

suffering from. Well, these thoughts,

18:15

fight-or-flight thoughts, these these

18:17

thoughts have a tendency to be

18:18

worst-case scenario, because they are

18:20

also highly related to catecholamines.

18:23

In fact, people that have that gene

18:25

mutation fall into one of two

18:26

categories. Think about it this way. If

18:29

catecholamines rise very fast,

18:31

you're a worrier. And the nickname for

18:33

that gene is either a worrier or a

18:36

warrior,

18:37

because as those catecholamines, they

18:39

call it fast COMT or slow COMT. So, just

18:42

looking at this one gene mutation, if

18:44

you're slow to break these

18:46

catecholamines down and they rise, what

18:48

are the consequences of that? Um I lay

18:51

down to go to sleep at night and I'm

18:52

body tired, but I'm mind awake. Right?

18:54

My mind is just clicking through the

18:56

day, thinking about the most innocuous

18:57

little thoughts.

18:58

Um I have a tendency when I consider

19:01

scenarios to go straight to worst-case

19:02

scenario. I'm an overthinker. Um I I am

19:07

prone to anxiousness and anxiety. I walk

19:09

around at a six instead of walking

19:11

around at a two. So, things that would

19:13

only move somebody from a zero to a two

19:16

take me from a six to an eight. Very

19:18

often people in their surrounding

19:19

environment will say the punishment

19:21

doesn't fit the crime, the way that they

19:23

react to certain situations. So, this

19:25

means that the mind is in awakened

19:27

state, a heightened state of alertness.

19:30

Think about a fight-or-flight response,

19:31

but not quite to that level, but they're

19:34

in a heightened state of alertness.

19:36

And this gives you that that feeling of

19:38

anxiety. Now, what's driving the

19:40

catecholamines can have different

19:41

drivers. It could be this gene mutation,

19:43

it could be trauma, it could be the

19:46

presence of a real fear, could be that

19:47

you're claustrophobic in stepping on a

19:49

crowded elevator. But for people that

19:52

have not isolated trauma in their life,

19:54

they have a tendency to consider worst

19:56

case scenario. They find that their mind

19:59

is very active at night, interrupts

20:00

their sleep. Or if they get up to go to

20:02

sleep at night and they go back to bed

20:03

and they can't fall asleep because their

20:05

mind is awake thinking about the most

20:07

innocuous little thoughts. They have a

20:08

tendency to be anxious. They have a

20:10

tendency to be a warrior. They have a

20:12

tendency to have feelings of anxiety

20:13

that are not tied to their outside

20:15

environment. Those are all hallmarks of

20:18

that gene mutation.

20:20

So can I view this as a predisposition?

20:22

I you know, I often wonder why we can

20:24

all be in the same situation, but we can

20:26

have entirely different experiences.

20:28

In the case of anxiety, some people as

20:29

you report just for some reason they're

20:32

just more anxious in the in the modern

20:33

world than some other people are. The

20:35

modern world has changed. We use screens

20:37

and we have notifications. We have all

20:38

this stimuli. They're struggling more in

20:40

the modern world than others. Um

20:43

What you're suggesting is that they

20:44

could have a predisposition

20:47

to

20:48

worry more because of these

20:50

catecholamines? Because of the

20:52

catecholamines, yes. Because of these

20:53

catecholamines, norepinephrine,

20:55

epinephrine, ephedrone, dopamine. One of

20:58

those we can also call adrenaline. And

21:00

so you have the main driver of behavior

21:02

and you have catecholamines. And we all

21:03

know what adrenaline does in the body.

21:05

So when these four neurotransmitters are

21:07

not down regulated,

21:10

right? Then our mind is awake and it is

21:12

very often fearful. Think about somebody

21:15

who has a sensation of impending doom or

21:19

anxiety without any any trigger. And and

21:23

and the other hallmark is they will have

21:24

had it on and off throughout their

21:25

entire lifetime. Even when they were a

21:27

child. When they were a child, they

21:28

might not have understood the complex

21:30

sensation of anxiety, but they had that

21:33

sensation. And then as they grew to be

21:35

an adult, they understood that this is

21:37

anxiety. I mean, when you when you're

21:38

when you're a child, you're just

21:41

fearful, right? I mean, you don't know

21:42

how to explain to your mom, "Hey, I'm

21:44

worried about something that is

21:46

might happen in the future, that's

21:48

probably not likely to happen, that's

21:49

never happened, but I'm still afraid

21:51

it's going to happen." It's a very

21:52

complex emotion, right? So

21:55

they've had it on and off their entire

21:56

lifetime. It's very hard for them to

21:58

point to the specific trigger that

22:00

causes it. The majority of the time if

22:02

they try to anti-anxiety medications,

22:04

they don't work. They just make them

22:05

feel like a zombie. So this is time to

22:07

look inside and make sure the body has

22:09

the raw material it needs to do its job,

22:11

which is the complex of B vitamins to

22:14

dismantle catecholamines. So if we went

22:16

out into the street now and I pulled in

22:17

a hundred people just off the street

22:18

that were walking past, Mhm. and we did

22:20

these three tests on them to look at

22:22

their glycemic profile, their hormone

22:24

panel, their nutrient deficiencies,

22:27

what are some of the most popular things

22:29

that

22:30

a random group of people off the street

22:31

would be missing that are central to

22:34

their high performance? Um so

22:37

let's let's take men and then we'll take

22:38

women so so we can be specific about

22:40

hormones. So

22:41

in 50% of that population, you would see

22:44

a clinical deficiency in vitamin D3. I

22:46

have to say you've actually you actually

22:47

run a lot of tests every single month.

22:49

Yes. Tens of thousands. We run 20,000 a

22:52

month. We do about 20,000 gene tests a

22:54

month. I mean, so we so one of the

22:56

unique things about

22:58

the perspective that I come from is we

22:59

have voluminous amounts of data. You

23:01

know, we see

23:02

20,000 of these new

23:04

um

23:05

patients a a month testing for for

23:07

genetic methylation. And on a lot of

23:09

these patients, we also have blood work.

23:11

So we have a full what's called a CBC,

23:13

comprehensive metabolic panel, lipid

23:15

panel, hormone profile, a full thyroid

23:18

panel. We have their nutrient

23:20

deficiencies that I'm speaking of. Um we

23:22

have cholesterol, triglycerides. So we

23:24

have a pretty We have about 74

23:26

biomarkers on them. Then we also get

23:28

this genetic test. And then we look at

23:31

what happens to certain biomarkers on

23:33

average when you when you simply

23:35

supplement for a deficiency. So for

23:37

example, I'm not saying that every

23:38

person that has high blood pressure or

23:40

hypertension has this gene mutation, but

23:43

two of these genes are highly linked to

23:44

poor homocysteine metabolism. And there

23:47

have been plenty of peer-reviewed

23:48

studies. We can put the link to the one

23:50

below in the Journal of Hypertension,

23:52

which linked higher levels of urinary

23:54

catecholamines to an and urinary

23:57

homocysteine to

23:59

cardiovascular disease. Because what

24:01

happens is when you have a certain amino

24:04

acid in particular rise in the blood

24:05

called homocysteine.

24:07

As this amino acid rises, it has a

24:09

tendency to cause the vascular system to

24:11

constrict.

24:12

And if we make the pipes smaller in a

24:14

fixed system, pressure goes up. But

24:17

there's nothing wrong with the heart.

24:19

Right? And so think about the fact that

24:21

85% of all diagnosis of hypertension is

24:25

idiopathic. It's of unknown origin.

24:28

Well, of unknown origin means we can't

24:29

find anything wrong with the heart.

24:30

We've tested the heart, EKGs, EEGs,

24:33

stress tests,

24:34

dye contrast studies, cardiac cath, what

24:36

have you. But we we haven't looked at

24:38

the vascular system. We haven't looked

24:40

to see was there a simple nutrient

24:41

deficiency keeping this person from

24:43

breaking down homocysteine, which caused

24:46

the vascular system to constrict?

24:48

Because we know that there's a

24:48

correlation between this

24:52

amino acid homocysteine and its elevated

24:54

nature and and increased risk of

24:56

cardiovascular disease. So before we

24:58

actually went the routes of chemicals

24:59

and synthetics and pharmaceuticals, why

25:01

wouldn't we just test to see see if we

25:03

have an issue

25:05

um dismantling this amino acid, you

25:07

know, breaking this amino acid down into

25:09

or something called methionine.

25:11

And why don't we supplement for that

25:13

deficiency and see if by putting that

25:16

raw material back into the body and

25:18

bringing homocysteine metabolism into

25:19

normal, we can normalize this person. So

25:21

So back to your question about the

25:22

people in the street, you would see that

25:24

50% of them are clinically deficient in

25:26

vitamin D3, cholecalciferol, you know,

25:28

the sunshine vitamin. Um the darker

25:31

their complexion, the higher the

25:33

uh risk that they would be clinically

25:35

deficient in vitamin D3. And if if you

25:37

put that vitamin D3 at the center of a

25:39

hub of a wheel and looked at all of the

25:41

different spokes, it's one of the only

25:42

vitamins that human beings make on our

25:44

own. I have argued and and um people

25:47

have counter-argued, but I take the

25:49

position that it's arguably one of the

25:51

most if not the most important nutrient

25:54

in the human body. You need all You need

25:55

a lot of essential nutrients, but if you

25:57

really start isolating them, you know,

25:59

vitamin D3 is the only vitamin that

26:01

human beings make on our own. Um every

26:04

cell in the body has a receptor site for

26:05

vitamin D3. And when we're deficient in

26:07

this vitamin,

26:08

um this nutrient acts like a hormone

26:10

sometimes, it acts like a vitamin other

26:11

times. We make it from sunlight and

26:13

cholesterol. When it's deficient, we

26:16

have a compromised immune system. We

26:18

know that it leads to osteopenia,

26:20

osteoporosis. There are all kinds of

26:21

consequences that you wouldn't think

26:23

stem from a simple nutrient deficiency,

26:24

but they do. One that we get from going

26:26

outside in the sun. We get it from going

26:28

outside in the sun. We make it from

26:29

sunlight and cholesterol. Um and and you

26:32

know, if you look at you know, COVID

26:33

statistics, it was the second leading

26:34

cause of morbidity in COVID. Um and so

26:38

you if So first you would see that

26:39

they're D3 deficient. The majority of

26:40

them are also B12 deficient. If you look

26:42

at the vitamin B12, you'd see it's less

26:44

than 500.

26:45

Um the higher end of B12 is around 1250.

26:48

And then you would see

26:50

25 to 40% of that population would be

26:54

hormone deficient, meaning that their

26:56

hormones would be out of the optimal

26:58

range, but not because

27:01

they have an endocrine system issue per

27:05

se, generally because they have

27:07

especially in younger ages

27:09

nutrient deficiencies. Things like

27:11

elevated SHBG, sex hormone binding

27:14

globulin, deficiencies in DHEA, raw

27:17

materials that the body needs to

27:19

manufacture hormones. So a good hormone

27:21

panel will tell you not only what your

27:23

hormone levels are, but what some of the

27:25

nutrients are that are your body's using

27:27

to make those hormones. And again, by by

27:30

putting some of these raw materials,

27:32

very often DHEA, not all the time, but

27:34

very often putting DHEA and vitamin D3

27:36

alone back into men with deficient

27:39

levels of testosterone or deficient

27:41

levels of free testosterone or looking

27:43

at a protein that interrupts the

27:45

conversion of testosterone into free

27:47

testosterone called SHBG, by actually

27:50

just addressing these, you see that the

27:52

hormone levels rise back to the normal

27:54

range. They don't need to take hormones

27:56

from outside the body and shut their

27:57

production down. They need to put

27:58

nutrients and raw materials back into

28:00

the body so their body can produce

28:02

hormones on their own. And then if you

28:04

looked at at their glycemic control, you

28:07

would see a shockingly high

28:10

percentage of people that are

28:12

pre-diabetic.

28:14

It is an absolute pandemic right now

28:17

because the amount of processed foods we

28:18

we think that the pre-diabetes

28:21

um you know, is only because people that

28:23

are eating a ton of sugar. So they must

28:24

be drinking soda and eating chocolate

28:26

cake and Ben & Jerry's every night, but

28:28

that's actually not true. When we

28:29

overload the body with high glycemic

28:32

carbohydrates, even if they ate a lot of

28:35

white flour, white rice, white bread,

28:36

white pasta, white potatoes and fruit,

28:39

you know, they I'm not saying any of

28:41

those things are going to kill you, but

28:43

when we eat diets high in refined

28:44

carbohydrates, even things that we don't

28:46

consider to be sweets, it overpowers our

28:49

pancreas and our blood sugar gets out of

28:52

control. But wouldn't you want to know

28:53

that? Wouldn't you want to know that

28:56

um do I have some of these nutrient

28:58

deficiencies or hormone imbalances or

29:00

poor blood sugar control that could be

29:02

nibbling away at my performance? Am I

29:04

maybe one raw material, one methylated

29:07

multivitamin away or an amino acid away

29:10

from being in a state of being optimal?

29:12

Maybe even not having to deal with

29:14

little things like

29:16

um intermittent feelings of anxiousness

29:18

and anxiety or poor focus and

29:20

concentration or even mild states of

29:23

mood numbness. Remember that

29:26

nutrients,

29:27

amino acids for example,

29:29

in our gut become neurotransmitters.

29:32

Neurotransmitters form the basis of all

29:34

mood. They drive our emotion. They they

29:37

um they govern our behavior.

29:39

And so is it possible that an amino acid

29:42

like tryptophan or phenylalanine or

29:44

tyrosine, which becomes serotonin and

29:46

dopamine, that deficiencies in these

29:48

amino acids could lead to deficiencies

29:50

in hormones, which could actually lead

29:51

to deficiencies in neurotransmitters

29:53

that would be labeled a mental illness.

29:56

Yes.

29:58

So

29:59

again, I feel I feel like I'm eating

30:00

your face a little bit, but

30:03

but what I what I really mean to say is

30:04

that

30:05

you know, if

30:07

if we would get basic information, basic

30:09

data on the body, hormone balance,

30:11

um glycemic control, nutrient

30:13

deficiencies, if we would actually look

30:15

at what our body can convert into the

30:17

usable form and what it can't and

30:18

supplement for that deficiency,

30:20

you would see your body begin to thrive

30:22

in ways you never thought possible.

30:24

You work with a lot of high-profile

30:25

individuals as well. I do. What are some

30:28

of the high-profile names that you're

30:30

you have permission to share?

30:32

Well, clearly anyone that's shared their

30:33

their journey with me on on on the

30:35

podcast. Um

30:37

there's a great hit piece on me and in

30:38

the Daily Mail that listed I listed a

30:40

lot of them. Um but uh Dana White, um

30:44

Steve Harvey, uh Stephen A. Smith, um

30:47

Steve Aoki,

30:49

um

30:51

Kendall Jenner and and I were on a uh

30:53

one of her Hulu specials

30:55

uh together running some IVs. There are

30:57

a few others that will be

30:59

public here very shortly that have gone

31:01

on podcasts with me and I and it's I

31:03

don't necessarily want to be known as

31:05

like a celebrity biologist or working

31:07

with um just working with professional

31:09

athletes and A-listers. It's my message

31:12

is for

31:13

is actually for the non-woke biohacker.

31:16

Like I don't feel like my job is to sit

31:17

here and impress you with how smart I

31:18

am. I feel like my job is to put

31:20

information out to the masses that that

31:24

is

31:25

educational enough to inspire them to

31:28

make a change.

31:29

And and I think

31:31

all too often we you know, we're we're

31:33

we're all competing for eyeballs in this

31:35

in this space and we're trying to become

31:37

the biggest influencer and we we really

31:39

forget about the mission of speaking to

31:41

the masses and we just start speaking to

31:43

each other. Like we we want to get on

31:45

podcasts and stage talks and interviews

31:46

and impress people with how much we know

31:48

about the carboxylic acid cycle or you

31:51

know, electron transport chain or

31:52

something going on inside of the

31:53

mitochondria and and those minute

31:56

nuances are not what's going to impact

31:59

humanity.

32:00

A lot has changed since we last sat down

32:02

in your life.

32:03

It has. Yeah, it really has. I feel like

32:05

I feel like I live somebody else's life.

32:08

I really do. What's what's changed? I

32:10

mean, when I when I first reached out to

32:12

you, it was because I saw a clip on

32:14

YouTube which had 20,000 views. And that

32:17

clip on YouTube I found really

32:18

interesting. So I think I I personally

32:19

sent you a DM and said, "Hey, Gary, do

32:21

you want to come on my show?" which I,

32:23

to be honest,

32:24

never personally send a DM. Because

32:26

Yeah, because my team the way our system

32:28

works here is

32:29

the um they understand what I'm

32:31

interested in and curious about right

32:32

now. So they'll go out into the market

32:34

and try and find people for me. They'll

32:36

bring those people those people to me as

32:37

a pitch. They'll pitch the individuals

32:39

to me and then I have the say whether

32:41

I'm curious enough to sit down and have

32:43

the conversation right now.

32:44

it twice. All right. Yeah.

32:46

So in this case it was I I'd seen

32:47

something you'd you'd done online, I

32:49

don't know, more than a year ago now it

32:50

feels like and it was really compelling

32:52

to me. So I wanted to sit down with you.

32:53

Since then I've observed you're you've

32:55

kind of had this sort of meteoric rise

32:58

um on loads of different podcasts and

33:00

social media and your business has

33:02

exploded.

33:04

There is something different about you.

33:05

Mhm.

33:07

And the thing that's different about you

33:08

that strikes me is

33:11

you strike me as a man that has been

33:14

through some [ __ ] Oh, yeah. Frankly.

33:16

Yeah. Cuz cuz the

33:19

the Gary that I met the first time

33:20

versus this Gary slightly different and

33:22

it's the type of thing when someone's

33:23

been through some [ __ ]

33:24

Mhm.

33:25

And with all good things come the

33:27

opposite. Yeah. It's unavoidable.

33:30

Yeah, I mean you you you you go under a

33:32

level of scrutiny, you know, you start

33:34

off you're like so excited, you're like

33:35

I'm going to get the message out and God

33:37

God's blessed me with the ability to

33:39

take ultra-complicated information,

33:40

distill it down and get it to the masses

33:42

and then you realize that there are

33:43

people that are watching your videos

33:46

like a 3-hour podcast and they're

33:48

looking for the one gotcha moment.

33:51

Right, he said sodium chloride, not

33:52

sodium hydroxide. Scam artist,

33:54

charlatan, you know, he pretends to be a

33:56

doctor, he's not a doctor. I've never

33:57

pretended to be a doctor. You will not

33:59

find a a video, a stage talk, a podcast,

34:02

anything in the media where I've ever

34:03

represented that I'm a doctor. I go out

34:05

of my way to say that I'm not licensed

34:06

to practice medicine. So yeah, I have

34:08

become a little more gun-shy and a

34:10

little more guarded

34:11

um with what I say. It's an effort to be

34:13

more precise with what I'm saying. Um

34:17

but I'm not going to stop getting the

34:20

message out to the masses because

34:23

I know that this is God's calling for

34:25

me.

34:26

I know that.

34:28

Because I spent so many years of my life

34:31

not in service to humanity.

34:34

And I think a lot of people find

34:38

their purpose in their pleasure and I

34:40

found my purpose from my pain.

34:43

What pain?

34:47

You know, when when um

34:52

when I was doing

34:54

life expectancies and and and mortality

34:56

predictions,

34:57

um

34:59

we were sort of brainwashed to believe

35:00

that this was just data.

35:03

Right? You weren't responsible for it.

35:04

You didn't have anything

35:06

to do with this person.

35:08

I was on a mortality team and uh we were

35:11

charged with predicting the life

35:13

expectancy of people for um large life

35:16

insurance and investment companies.

35:18

So when you apply for a large life

35:20

insurance com policy, you know,

35:21

everybody's on an actuarial curve.

35:23

Right? So you're on one, I'm on one,

35:25

everybody listening this podcast is on

35:27

an actuarial curve. What happens is

35:29

when a life insurance company is getting

35:31

ready to put 10 million or 20 million or

35:33

50 million dollars worth of risk on your

35:35

life,

35:36

only one thing matters.

35:37

How many more months do you have left on

35:38

Earth?

35:40

And

35:41

the science of predicting that

35:43

mortality is a very accurate science. I

35:46

get a lot of flak about it, but if you

35:47

want to know how accurate life insurance

35:49

companies are at predicting death,

35:52

just look at what happened during the

35:54

2008-2009 financial services crisis.

35:57

We had

35:58

we had 364 banks fail. Not a single life

36:01

insurance company failed. A valid death

36:03

claim in the United States has never

36:05

failed to have been paid. They are some

36:06

of the most solvent institutions in the

36:08

world. There's not another financial

36:09

services enterprise anywhere on the

36:11

planet that would take that level of

36:12

risk on one variable.

36:15

I mean, you have an investment fund.

36:17

You wouldn't put that level of risk on a

36:18

single variable. Right? How many more

36:21

months does this person have left on

36:23

Earth? And they have data that no other

36:25

medical enterprise has. They have data

36:27

that no other collegiate university has.

36:29

Not even the government has. They know

36:30

the day, the date, the time, the

36:31

location, and the cause of death

36:34

for millions and millions of lives. So

36:37

they know what leads to early mortality.

36:40

And so How do they get your sort of

36:42

health biomarkers to overlap that with?

36:44

Well, first of all, they they do a blood

36:46

test on you. So if you've ever had a

36:47

large life I'm not talking about term

36:48

life insurance where you get a hundred

36:50

thousand dollar two hundred thousand or

36:51

even a million dollar term life

36:52

insurance policy. I'm talking about

36:54

permanent universal life or whole life

36:56

life insurance. Um also annuities. When

36:59

when you um there's something in in the

37:02

states called a a SPIA, single premium

37:04

immediate annuity where you give the

37:06

insurance company, for example, a check

37:08

for a million dollars.

37:09

They guarantee you an income stream for

37:11

life.

37:13

Well, how do you think they're

37:13

determining that income stream?

37:16

They They're predicting how many more

37:17

months you have left on Earth. And they

37:19

they use morbidity factors and

37:20

comorbidity factors and yes, they factor

37:22

into to your your recreational profile,

37:25

your demographic profile. It's not as

37:26

simple as a blood test or a gene test,

37:28

but essentially what you do is you start

37:30

on a curve in a pool of a thousand lives

37:33

that are similar to yours.

37:35

And and your life expectancy is the dead

37:38

center of that curve. So if your life

37:40

expectancy is 200 months, that means in

37:42

200 months you have the exact same

37:44

chance of being dead as you do of being

37:46

alive.

37:47

Now, what determines your increased

37:49

probability of death or your mortality

37:52

factors? Are you obese, diabetic,

37:54

anemic? Do you have cognitive decline?

37:56

Are you compliant with your medication?

37:58

You know, there are all of these

37:59

different debits.

38:01

And then there are certain debits that

38:03

we called comorbidities.

38:05

Right? So if you were hypertensive, that

38:06

was a debit. If you were if you were

38:08

diabetic, that was a debit. If you were

38:10

um obese, that was a deb- debit. But if

38:13

you were hypertensive, diabetic,

38:16

and obese,

38:17

it wasn't 1 + 1 + 1, it was 1 + 1 + 1 =

38:20

10. Right? These were massive

38:22

comorbidity factors.

38:24

My job was to read the medical record

38:26

and do the medical record extraction.

38:29

And we had

38:31

incredible data on on on these people.

38:33

You saw their trust um you saw their

38:35

wills, their trusts, their divorce

38:37

decrees. You knew that they were

38:38

treating their children differently in

38:39

their in their estate, um bank accounts,

38:42

brokerage accounts, tax returns, um and

38:45

their medical records. And you have

38:47

recent blood work on them. But when you

38:50

read a medical record on somebody,

38:51

there's more than just their height,

38:53

weight, and blood pressure and the

38:54

medications that they're on. You really

38:56

start to get a profile for a lot for the

38:58

for the person.

38:59

And a lot of times I felt like I was

39:02

really getting to know these people,

39:04

oddly, because I had so much personal

39:06

information on them.

39:08

And

39:10

you know, a lot of these people's

39:13

came alive to me.

39:16

I know that sounds very strange, but

39:18

when you're reading about

39:20

their repeated, you know, visits to the

39:22

doctor and they're constantly talking

39:24

about, you know, their grandchildren and

39:26

then all of a sudden you see in the

39:28

medical record where the husband passed

39:29

away and then you see the

39:31

antidepressants creeping in and you see

39:34

um their their weight change, their body

39:36

mass index change and you you actually

39:38

as you're going through years of their

39:40

medical record, you really get a real

39:42

profile for them.

39:44

And I started to

39:46

realize that there were human beings on

39:48

the other side of these spreadsheets.

39:51

And

39:52

there were cases where

39:54

I knew that if I could have picked up

39:56

the phone and just contacted that

39:59

patient,

40:00

I could have completely changed the

40:02

trajectory of their life.

40:04

And I was prohibited from doing so by

40:06

law. And

40:09

even at one point in my career, I was

40:11

threatened with prosecution for

40:13

threatening to call a patient and warn

40:15

them about um a a

40:18

a life-threatening potential

40:19

life-threatening drug interaction that

40:20

I'd spotted in the medical record

40:22

between

40:23

two physicians that had written

40:24

contraindicated scripts in something

40:26

called the MIB, the Medical Information

40:27

Bureau, hadn't

40:28

uh picked it up. And

40:31

the data that I had said that this was

40:32

going to lead to a thrombolytic event,

40:33

like a plugged a blood clot, a stroke,

40:35

you know, a heart attack, an embolism.

40:37

And um I remember

40:40

calling the human resources director and

40:42

just, you know, basically saying that

40:43

I'm going to contact this patient. And

40:46

and and being threatened with

40:47

prosecution. And I

40:51

I think about it a lot. And

40:55

I just think about all the times I could

40:56

have picked up the phone and just

40:59

made a real material change in

41:00

somebody's life. And I didn't have the

41:01

opportunity to do it. And

41:05

big part of my career felt like I was,

41:08

you know, sitting behind a thick glass

41:09

wall just watching blind people walk

41:11

into traffic.

41:13

And

41:14

so I wasn't in service to humanity. All

41:16

I wanted to do was be wealthy. I was

41:17

very unauthentic.

41:19

And then I just woke up one day and

41:21

said, "What the hell am I doing? I mean,

41:23

I

41:24

I have so much information. I'm a human

41:26

biologist. And I have

41:27

I I've been studying this database for

41:29

20 years.

41:31

I could help people live healthier,

41:32

happier, longer lives.

41:34

And

41:36

and I quit my career. And I went home

41:38

and told my

41:39

fiancée at the time, now my wife, that I

41:41

wanted to start a wellness firm.

41:44

And that was the the the genesis of of

41:46

my firm, Streamline. And part of the

41:48

trajectory that I'm on.

41:50

And so

41:51

It still sits with you?

41:53

Everyday.

41:55

Really? Oh, dude, it's it's

41:58

Well, it it sits with me in a good way

42:00

because

42:02

you know, whereas before

42:04

it's it's really hard to imagine, you

42:07

know, somebody coming into your office

42:08

and going, "Hey, you know, Gary, you

42:10

know, my god, remember the um

42:12

you know, the Mrs. Smith life expectancy

42:14

we we we did 13 years ago, you know, you

42:16

did this life expectancy was 188 months.

42:18

You predicted 188 months. She died in

42:21

184 months."

42:23

Oh my god, you did great job. It's

42:24

amazing.

42:26

The death claim just paid. And I'm like,

42:28

"Is it really amazing?"

42:30

You know, um

42:32

when you start to realize that was

42:34

somebody's, you know, it was like

42:35

somebody's sister or somebody's daughter

42:36

or somebody's mother,

42:38

you you you start to realize

42:40

that

42:41

I I allow myself to be brainwashed and

42:43

just think that it was data and and

42:45

forget that there was human beings on

42:46

the other side of the the spreadsheet.

42:48

And so now

42:51

I'm sorry, I'm getting emotional, but

42:52

but um

42:54

you know, now I wake up every day and I

42:55

look over my eyes and I go, "Fuck yeah,

42:57

you know, I got a chance to to make a

42:59

difference." And

43:01

and I talk about the research and and

43:04

the fact patterns that we saw in

43:06

predicting death. And I want to counter

43:08

those

43:09

so that we can extend life. Yeah, so we

43:12

can help people live longer, healthier,

43:15

happier lives.

43:18

So the

43:19

counter arguments that you've

43:20

experienced,

43:22

you know, you used the word counter

43:23

argument in hip hippies. What do those

43:25

sort of counter arguments tend to center

43:27

on as it relates to your work?

43:29

Obviously, you talked about the doctor

43:30

thing. I've definitely made some

43:31

mistakes, you know, I I I made a mistake

43:33

early in my career of of

43:35

quoting

43:37

articles and not and not research, which

43:39

I regret. And I and I've made the most

43:41

mistakes.

43:43

I think very often what I try to do is

43:44

is simplify the message. I talk, for

43:47

example, about a 2018 study. We should

43:49

put the link to this, um which was in

43:51

the journal of um

43:54

uh headaches and face pain. There's a

43:56

journal of headaches and face pain. I

43:58

want to say it was 2018.

44:00

There were 8,819 participants in this

44:02

meta-analysis. So a very large

44:05

um analysis.

44:07

And they found

44:09

a direct inverse relationship between

44:11

sodium intake and migraine headaches.

44:14

And meaning that as sodium levels went

44:16

up, migraine headaches went down.

44:18

Now, by no means am I telling everybody

44:20

that has a migraine headache that you

44:21

need to take a little bit of salt and

44:22

you're going to be fine.

44:23

What I'm saying is on your on your

44:25

comprehensive metabolic panel, you can

44:26

see your sodium level.

44:28

When your sodium level gets to a

44:30

critically low level, which believe it

44:31

or not quite a few people have, people

44:33

that regularly salt on their plate,

44:34

people that exercise and don't

44:35

remineralize with electrolytes, people

44:36

that drink um filtered bottled water in

44:39

an effort to filter out fluoride and

44:41

microplastics, but don't remineralize

44:43

their water, get nutrient deficient

44:45

sodium.

44:46

And you know, remembering that the brain

44:48

actually doesn't have any pain

44:49

receptors, but the covering of the brain

44:51

does. You know, something called the

44:53

dura.

44:54

And the dura hates two things. It hates

44:56

being stretched and hates being

44:57

contracted. And what what determines

44:59

whether or not it's stretching or

45:00

contracting is something called the

45:01

osmotic gradient, the movement of water

45:02

across the membrane.

45:04

And yes, it can be as simple as

45:07

supplementing with sodium. Um my

45:09

preference would be Baja Gold sea salt

45:11

or Celtic salt, um so that you get all

45:14

of the other trace minerals as well,

45:16

um to permanently put migraine headaches

45:18

in into remission. And then, you know,

45:20

out come all of the physicians saying

45:22

there's no evidence of that. Where are

45:23

the clinical trials on that? And the

45:25

other

45:26

the other

45:27

tool that I have in my chest is for 20

45:29

years I worked with one of the largest

45:31

databases in the world. And we're at the

45:33

point now where we see 20,000 new gene

45:35

tests a month. I don't know many clinics

45:37

that are that busy. So we have

45:38

voluminous amounts of data.

45:41

We see what happens when you have a high

45:43

homocysteine and you put them on you put

45:46

a patient on an amino acid called

45:47

trimethylglycine and the homocysteine

45:50

comes down. And then they go to their

45:51

doctor and their blood pressure is

45:53

normalized. Not once, not twice, not

45:55

anecdotally, thousands and thousands of

45:57

times. You see what happens to people

46:00

when you bring their hemoglobin A1c and

46:02

their insulin back down into the optimal

46:04

level and their triglycerides return to

46:05

normal and their risk for cardiovascular

46:07

disease declines. You see what happens

46:08

to C-reactive proteins when people take

46:11

simple things like silica clays um and

46:14

activated charcoals. And so

46:17

I want to keep getting the message out

46:19

that

46:20

very often disease is not happening to

46:22

us, it's happening within us. And very

46:24

often it's happening because

46:26

of deficiencies in the human body, not

46:28

pathology in the human body.

46:30

And you know, in in in in in the United

46:33

States we're we're by far the largest

46:35

spender of on health care. You know, we

46:36

spend 4 and 1/2 trillion dollars a year

46:38

on on health care in the US.

46:40

We have the highest infant mortality

46:42

rate. We have the highest maternal

46:44

mortality rate. Um even though we lead

46:47

the the world in flu vaccinations and

46:49

breast screen um and breast cancer

46:51

screening and colorectal screening, we

46:53

also lead the world in cancer. Um we're

46:56

ranked 52nd in life expectancy. We're

46:57

ranked 39th in in health care delivery.

47:00

Um we're one of the most obese nations

47:02

on the planet, twice the rate of obesity

47:04

of any other civilized um nation.

47:07

And yet modern medicine being

47:11

you know, medical error being the third

47:12

leading cause of death,

47:14

is where we're going to get information

47:16

on how we extend our life.

47:18

And I watched in medical records, I've

47:21

probably read thousands of times more

47:22

medical records than most physicians cuz

47:23

I read medical records all day, every

47:25

day, 6 days a week for for almost 20

47:27

years.

47:28

And I would see what would happen when

47:31

simple deficiencies would be mistaken

47:34

as a pathological condition. And I've

47:36

talked about these a lot. Um

47:40

you know, like clinical deficiencies in

47:41

in vitamin D3 for prolonged periods of

47:43

time eventually

47:45

present as rheumatoid arthritis-like

47:47

symptoms. People get joint aches and

47:50

pains and and stiff and sore ankles and

47:52

they have a hard time making a fist. And

47:53

and you know, when you're speaking to

47:55

the wrong physician, very often a doctor

47:56

will diagnose you based on your medical

47:59

history. Not before they do said rates

48:02

in rheumatoid arthritis, you know,

48:03

actual blood uh checks, they'll say,

48:05

"You know what, Stephen, you've got

48:06

rheumatoid arthritis." And they put you

48:08

on on things like corticosteroids. And

48:12

in the mortality space, we had data.

48:14

So we had data on all of these

48:16

pharmaceuticals. So we knew the

48:17

trajectory of of hormones and cell walls

48:20

and cell membranes and um production of

48:23

vitamin D3 when somebody took a statin

48:25

and reduced cholesterol. And we looked

48:27

at, you know, they the studies will look

48:29

at cholesterol in a complete vacuum. So

48:32

LDL cholesterol high, so that's bad.

48:34

Let's bring LDL cholesterol down with a

48:35

statin, so we decrease the risk of

48:38

cardiovascular disease. But then you

48:39

have a concomitant outcome where you

48:41

you're reducing the ability for the body

48:43

to make hormones and cell walls and cell

48:45

membranes. And so you buy yourself a

48:46

consequence downstream.

48:48

When really, if we go back to just

48:51

studying the physiology of the human

48:52

body, when we in in the mortality space,

48:55

I don't think I saw a single centenarian

48:58

once, and we processed hundreds of these

49:00

death claims. I don't think I saw a

49:01

single centenarian that at the time of

49:03

death did not have clinically elevated

49:05

levels of LDL cholesterol.

49:07

So begs the question, is

49:09

simply having high LDL cholesterol a

49:11

marker for

49:12

um longevity or is it a a marker for

49:15

cardiovascular disease that needs to be

49:17

intervened

49:18

with a chemical or a synthetic? And

49:22

and these corticosteroids that people

49:23

are put on, you know, very often they

49:25

they they're they're anti-inflammatory

49:27

in the beginning, but then they eat the

49:28

joint like a termite.

49:30

And

49:31

and so these were resulting in

49:33

voluminous amounts of joint replacements

49:38

so accurately that we would

49:40

we were able to predict that the course

49:43

of some of these medications would

49:44

result in a joint replacement in roughly

49:46

6 years.

49:47

And so we would artificially advance

49:49

people's age 6 years and we would

49:50

actually schedule the joint replacement

49:52

for them. And then we would reduce

49:54

what's called their ambulatory profile,

49:55

how well they ambulated, how well they

49:57

moved. And as we reduce their mobility,

49:59

we could bring in all of these diseases

50:01

that exacerbated with reduced mobility.

50:03

And in my mind, I'm just watching all

50:05

this happen and I wanted to call this

50:07

these people and say

50:08

I'm not qualified to do that cuz I'm not

50:10

licensed to practice medicine, but I I

50:12

wanted to call them and just say Mrs.

50:13

Jones, stop taking the corticosteroid.

50:15

Start supplementing vitamin D3. Get your

50:17

B12 level to here. Let's fix your

50:19

hormones because this is killing your

50:21

red blood cell count and this is what's

50:23

leading you to be so exhausted. And and

50:26

no one was looking at at these basic

50:29

nutrient deficiencies that we would see

50:31

run in blood work that would cause all

50:34

of these diseases to exacerbate and

50:37

people were succumbing much earlier

50:40

to death or to the loss of their health

50:43

span. How many records do you think you

50:45

saw in your time where you saw the full

50:47

picture?

50:48

I would be working on two or three of

50:50

these four of these cases at a time.

50:52

Some were shorter cases, some were

50:53

longer cases, thousands.

50:55

I mean thousands. And in the tail end of

50:57

my career, I started to manipulate the

51:00

um

51:01

record artificially just to see what

51:02

would happen to the life expectancy. I

51:04

would never submit that as a report, but

51:05

I would say, what if I fix the anemia?

51:08

What if I actually just corrected the D3

51:09

deficiency? What if um you know, I I and

51:13

I was able to

51:14

um take out the pre-diabetic condition

51:17

or reduce their hemoglobin A1C and you

51:19

would see the life expectancy jump.

51:22

Right? And so these are modifiable risk

51:24

factors and I think how many times you

51:27

know, I would be reading a medical

51:28

record and go, what? I know what this is

51:29

going to happen. This is just going to

51:30

get worse cuz this patient has anemia.

51:33

Like the classic treatment for some

51:35

anemias is folic acid, B12 and and iron.

51:38

And they would give them folic acid, B12

51:40

and iron and it wouldn't correct. Then

51:41

they give them folic acid, B12 and iron

51:42

and it wouldn't correct. Folic acid, B12

51:44

and iron wouldn't correct. And they

51:45

wouldn't realize that

51:46

um

51:47

that person can't process folic acid. If

51:49

they gave them methylfolate

51:52

methylcobalamin

51:54

and iron bisglycinate

51:56

the anemia would correct. But these are

51:57

all sort of symptoms of further upstream

52:00

issues, right? Like something that a

52:01

decision that someone has made in their

52:03

life typically

52:04

typically Mhm. that has caused them to

52:07

develop these conditions which far down

52:08

the stream, like the tree you talked

52:10

about with the bad leaf doctors then

52:13

point at the leaf and go, we need to fix

52:14

the leaf. Mhm. But it's down in the

52:16

roots somewhere. Mhm. So what are the

52:18

what are like the

52:20

societal and individual level things

52:23

that we can be doing

52:24

to prevent us even getting these chronic

52:26

diseases? Like the the simple simple

52:28

things.

52:28

I'll tell you the simplest thing that we

52:30

can do.

52:31

First I you know, we should think about

52:34

having an an invisible fence around us.

52:36

Right? Like a like a little force field.

52:38

And we should filter things before they

52:39

make it to the temple.

52:41

Um because

52:43

either we can filter things for the

52:45

temple or we can let the temple be the

52:46

filter. So you can drink tap water and

52:49

if you drink tap water, your body will

52:50

filter out the fluoride, the chlorine,

52:52

the microplastics, the pharmaceuticals

52:54

or you can filter your water before you

52:56

drink it. Right? And and take one toxic

52:59

load off your body. So what I would say

53:01

is

53:02

probably five things that I would

53:04

commit to doing on a regular basis.

53:06

Number one is upon waking, I would I

53:10

would drink a mineral mineralized water.

53:12

I would take 10 oz of water and I would

53:14

add either a Celtic sea salt or a Baja

53:16

gold salt to my water. The reason for

53:19

that is that most of us are deficient in

53:21

some or several of the trace minerals in

53:24

our body. The boring ones, boron,

53:26

manganese, molybdenum, selenium.

53:29

And stir it up and just whack it back.

53:31

The second thing I would do is

53:32

Wait, you know what I'm talking about

53:33

table salt here. No, no, no, not sodium

53:35

chloride. No, I'm talking about Baja

53:37

gold sea salt. That's probably the best

53:39

salt that

53:40

you can put in the human body because it

53:41

has all 91 trace minerals. It's tested

53:44

down to 250 parts per billion

53:47

for microplastics and glyphosate. Um

53:50

only about 75% of that sodium crystal is

53:52

actually sodium. The rest of them is all

53:53

of these trace minerals. You can get

53:55

very close to that with Celtic salt.

53:58

Right? And if you can't get Celtic salt,

54:00

then you could move to a pink Himalayan

54:01

sea salt. The problem with pink

54:02

Himalayan sea salt recently is that a

54:04

lot of it has um heavy metals cuz it's

54:07

coming out of China. So I would say the

54:08

best salt is Baja gold. A a great salt

54:12

is Celt Celtic salt.

54:14

And

54:15

a decent salt is pink Himalayan sea

54:17

salt. Forget table salt. I would just

54:19

get that permanently out of your life.

54:21

Okay, so number one, I have my Baja

54:22

Mineralized. Mineralized. Um and then

54:26

number two, I would I would take a DHA

54:28

EPA fish oil supplement or a a fatty

54:31

acid supplement with DHA or EPA oil. Um

54:36

an MCT oil. I'll take a fatty acid um

54:39

oil in the morning. An omega supplement.

54:41

An omega. Omega 3. An omega 3

54:44

supplement. And then I would develop a

54:46

morning routine that included the basics

54:47

from mother nature, sunlight, grounding

54:51

breath work cold shower. Okay, so I

54:53

wanted to zoom in here on grounding.

54:55

Mhm.

54:57

I'm a huge fan of grounding. My

54:58

girlfriend grounds and again, listen, my

55:01

girlfriend's much smarter than I am at

55:02

it transpires because everything she

55:03

says, I think I said this to you last

55:04

time. Everything she says to me

55:06

eventually I sit here with like a

55:07

neuroscientist a year later and turns

55:09

out she was absolutely right. I thought

55:10

she was a bit a little bit kooky for

55:12

thinking that getting outside in the

55:13

morning and putting her feet on the

55:14

ground were

55:15

at all beneficial, but I've been told

55:17

time and time again it is.

55:19

What is grounding and why does it help?

55:22

So we get three things from mother

55:24

nature, right? We get magnetism from the

55:26

earth, we get oxygen from the air, we

55:27

get light from the sun.

55:29

The further we get away from those

55:30

things, the sicker we become. Really?

55:32

Yes. So the magnetism

55:34

Absolutely.

55:34

piece of it sounds like like a spiritual

55:37

kooky stuff.

55:37

I you know, I mean probably

55:39

10,000 years ago they probably thought

55:40

the same thing about gravity, you know,

55:42

but um

55:43

but the earth has a low Gauss current.

55:46

Right? I mean we were meant to spend 85%

55:48

of our time outside. We spend 97% of our

55:50

time indoors now.

55:52

The truth is most of us are not getting

55:53

enough sun. We're not getting too much

55:55

sun. We're not getting enough sun.

55:57

And you know, because of the way we eat

55:59

and seed oils and everything that are

56:01

that are oxidizing in our skin, our

56:03

cancer rates are are are exploding, but

56:05

not because of our sun exposure, it's

56:07

because of our our diet. We could talk

56:08

about that later, but

56:10

when you touch the surface of the earth,

56:11

when bare feet touch a pair of soil,

56:13

grass, sand, we discharge into the

56:15

earth. And by that, I mean you actually

56:17

change the polarity in the body. And

56:19

this is measurable. In fact, if you want

56:21

to do a little experiment, um find find

56:23

somebody that has a microscope, a basic

56:25

microscope and get a slide and just take

56:28

a prick prick your finger and take a

56:29

drop of your blood and put it on that

56:30

slide, smear it around and look at it

56:32

under the microscope. I think I have a

56:34

video of this on my Instagram. And what

56:37

you'll see when you look at your blood

56:38

in real time is you'll see most of your

56:39

red blood cells are stuck together and

56:41

clumped up. Not clotted, but they're

56:43

attracted to each other. Because when

56:45

cells have the same charge, they repel.

56:47

When they repel, it increases the amount

56:49

of surface area that that cell has to

56:51

contact the outside environment. So now

56:53

it can exchange waste. It can eliminate

56:56

waste, detoxify, repair, can regenerate.

56:59

So imagine that you have bloodstream

57:01

full of red blood cells and they start

57:03

to get opposite charges.

57:05

So they attract. And when they attract,

57:07

they touch and everywhere that they

57:09

touch, that cell loses surface area to

57:11

exchange with the outside environment.

57:13

When you touch the surface of the earth

57:15

for a few minutes, you will repolarize

57:17

those. Prick your finger 10 minutes

57:19

after you come inside, put it back on

57:21

that same slide, look at your blood,

57:23

it's going to look like

57:25

eggs slithering around in a bowl of oil.

57:28

They will bump into each other and

57:29

they'll be sliding around, but they will

57:30

not be clumped together and stuck. So

57:33

what's going on then? May I must be

57:35

coming through my feet. The charge

57:36

coming through my feet. Yeah, so you're

57:38

actually discharging into the earth, you

57:39

know, you're exchanging um ions. It's a

57:41

low Gauss current. So like a magnet,

57:43

you're exchanging ions with the earth

57:45

and you're discharging. You're you're

57:47

you're grounding. What if I live on the

57:49

ground floor, do I still have to go

57:50

outside? Yes, you got to touch bare

57:51

dirt, soil, grass, sand.

57:54

Why can't I if I live on the ground

57:55

floor, why doesn't the floor on the

57:57

lower floor of the house Because they

57:59

they that insulates you from um from the

58:01

earth's magnetic field. There's usually

58:02

steel, concrete, wood. There's other

58:05

barriers, tile, asphalt. There are

58:07

things that actually prevent you from

58:08

actually contacting the surface of the

58:10

earth. You know, there there are there

58:11

are grounding mattresses that you plug

58:13

into the ground wire and then um that

58:16

ground wire, if you if you look at how,

58:19

you know, the grounding of a circuit

58:20

occurs, at some point is running

58:22

directly into the ground. There will be

58:24

a pole in the ground that is connected

58:27

usually by copper to that wire and

58:28

connected to your outlet to ground that

58:30

outlet. Can I just get some kind of mat

58:32

that does has the same charge?

58:34

you could get a PEMF mat. But again, you

58:36

know,

58:37

one of the things I get a lot of flack

58:38

of is is saying that you have to buy all

58:40

this expensive equipment. So there's two

58:42

ways to do it.

58:43

You can buy a pulse electromagnetic

58:45

field mat, a PEMF mat. I have one. Um

58:48

they cost about five grand. Um so if you

58:50

got five grand lying around, it's one of

58:51

the best investments you can make. You

58:53

put it in your bed, you go to sleep on

58:54

it, you you run it you run a low Gauss

58:56

current at night, it will help get you

58:58

into a deep sleep. You'll wake up

58:59

alkaline every morning. Um it will push

59:02

the electrosmog right out of your your

59:04

body um cuz PEMF gets rid of

59:06

electrosmog, 5G, Wi-Fi. When you say you

59:08

wake up alkaline every morning. So when

59:11

you change the um so the pH of the blood

59:13

is a it's a pretty narrow range. It's

59:15

about it's about 5/10 of a point. It's

59:17

about half a point. And it's a complete

59:20

fallacy that you can change the pH of

59:22

the blood by drinking alkaline water.

59:24

Alkaline water will actually not

59:26

actually change the pH of your blood. If

59:28

you want to change the pH of your blood

59:29

amongst other things, you you apply a

59:32

low Gauss current. pH stands for

59:34

potential hydrogen. It's a charge. And

59:36

so by running a low Gauss current

59:37

through the body or touching the surface

59:39

of the earth, you actually can move the

59:41

pH of the blood slightly. And that does

59:43

An alkaline state is a disease-free

59:45

state. The more acidic we get, the

59:47

sicker we become.

59:49

And so

59:50

um and so if we want to move the pH of

59:53

of the blood slightly, if we want to

59:55

wake up alkaline, if we want to run a

59:57

low Gauss current through our body, we

59:59

can either touch the surface of the

60:00

earth or wear a bio PMF mat. See, so

60:02

they've done tests where someone lays on

60:03

a PMF mat for a certain amount of time.

60:05

They then do a blood test and they find

60:07

that their blood is more alkaline.

60:09

Yes. Mhm. Yes, and that that separation

60:12

of blood cells, you can see instantly on

60:15

getting off of a PMF mat. Again, I've

60:16

got videos of me doing this to my

60:18

production manager on um you know, I'm

60:20

in my house breaking his finger, putting

60:22

it on the

60:23

on a slide, putting it on the uh the PMF

60:26

and actually looking at it afterwards.

60:27

The second thing I would do is I would

60:28

learn to do breath work. I use something

60:30

called a HyperMax, which is based on um

60:32

Dr. Van Arden and Dr. Otto Warburg's um

60:35

Nobel Prize

60:37

winning work and that is the it's called

60:38

multi-step oxygen therapy where you

60:40

actually take an oxygen concentrator,

60:42

you fill up a bag full of 900 L of 95%

60:45

O2 and you actually just breathe that

60:46

95% O2 for 10 to 12 minutes while you're

60:49

active on a treadmill.

60:51

But if you don't want to have an

60:52

EWOT, exercise with oxygen therapy

60:55

machine, you can learn to do breath

60:56

work, engage the auxiliary muscles of

60:59

respiration, get oxygen down into the

61:01

lobes of your lungs and out of the apex

61:03

of your lungs. One of the one of the

61:05

articles that I quoted that turned out

61:06

not to be a study and I still can't find

61:08

the reference for it was that

61:10

after age 35 90% of people will never

61:13

sprint again.

61:16

And again, I haven't been able to find

61:18

if that came from a clinical study or if

61:19

it was an an article, but

61:22

whether or not that's true, the vast

61:24

majority of people stop engaging their

61:27

auxiliary muscles of respiration.

61:30

You know, really exercising our

61:32

diaphragm, using the intercostal muscles

61:34

between our ribs, pushing air down into

61:37

lobes of our body. And as our posture

61:39

collapses and our CO2 rises, you know,

61:41

if you think about the expired air in

61:43

your body from the tip of your nose to

61:44

the tip of your mouth all the way down

61:46

your esophagus out your bronchioles into

61:49

the farthest reaches of your lungs,

61:50

that's all expired air. Until you get

61:53

the oxygen all the way down and out to

61:55

the edges of the lung, you're not

61:57

getting oxygen into the bloodstream. So

61:59

as we age and our posture collapses, our

62:01

respiratory rate gets more and more

62:03

shallow. We're essentially

62:04

hyperventilating carbon dioxide.

62:06

Right? And which is accelerating aging.

62:08

I mean, aging is

62:10

the presence of oxygen is the absence of

62:12

disease. And so by just learning how to

62:15

do breath work. So one, I would ground.

62:17

Two, I would I would learn to do breath

62:19

work. I do a Wim Hof style of breath

62:20

work. I do three rounds of 30 breaths

62:23

with an extended breath hold every

62:24

single morning. It is the one thing that

62:27

I never ever ever ever miss. Why? Ever.

62:32

Because

62:33

I make little promises to myself and I

62:35

try to keep them.

62:37

And I find that

62:40

I lose confidence in myself when I

62:41

consistently break really small promises

62:44

to myself.

62:45

Um and I think a lot of people do this.

62:48

And our bodies crave consistency.

62:51

And so You lose confidence in yourself.

62:54

You say, you know, I'm going to go to

62:55

bed at, you know,

62:57

10:30 tonight and you go to bed at 1:00

62:59

a.m. You know, and then you say I'm

63:01

going to work out and first thing in the

63:03

morning and you actually don't work out.

63:05

Or you get up in the morning you say, I

63:08

listened to that podcast, I'm going to

63:09

do what Gary said, I'm going to ground

63:10

and get some sunlight and I'm going to

63:11

do some breath work. And then you

63:12

actually don't do it. So these are

63:14

little internal promises that you make

63:15

to yourself.

63:17

And I feel like a lot of people

63:19

break these little promises to

63:21

themselves. If they're not making them

63:22

to their spouse or to their kids or to

63:24

their partners or, you know, they're

63:26

they're they're not the big promises

63:27

that everybody knows about. And I think

63:29

it nibbles away at our self-confidence

63:31

and our own ability to trust ourselves.

63:34

And so

63:35

I have a morning routine. Um I'm very

63:37

consistent with it. But the one thing

63:39

that is portable for me is the ability

63:41

to get outside and ground and do breath

63:44

work.

63:45

And I never ever ever miss.

63:48

I can't even tell you how many years

63:49

I've gone without missing a single

63:51

morning of breath work. The other thing

63:52

that it does for me because, you know,

63:54

human beings crave consistency. So if

63:56

within 30 minutes of waking every day,

63:58

no matter what time zone you're in,

64:00

you're doing three rounds of 30 breaths,

64:03

your body begins to zero in on that and

64:05

it begins to understand that that's the

64:07

morning. This is go time. And

64:11

so simple to do. You know, when I'm

64:12

here, I wake up. I might be at a

64:14

different time because I'm usually on

64:15

the East Coast, so I wake up earlier

64:16

here, but I go I open the door, I go out

64:19

on the balcony, I sit on the chair, it's

64:20

nice and cool outside, I face the sun

64:22

and I do three rounds of 30 breaths

64:24

every single day.

64:26

My partner brought me one of those big

64:28

red light panels for Christmas. It was

64:30

my Christmas present. And funnily

64:32

enough, guess what my Christmas present

64:34

to her was as well.

64:36

A [ __ ]

64:37

Yeah. You got a bed or did you get the

64:38

panels? The panels. It was so sweet.

64:40

like JuveXo or what did you get?

64:42

idea. What did you get? Okay. I've no

64:44

idea the brand. But it but she brought

64:45

me one. It's like a small one. And then

64:48

I was like, "Babe, open your present."

64:50

And then I opened them and she opened

64:51

hers and hers was like a big one. She's

64:53

like literally half the size of me, so

64:55

it was quite sweet. So we swapped. Um

64:57

but but we now both use it. It's a bit

64:58

of a routine in the morning. We both

64:59

wake up.

65:00

Amazing.

65:00

and sit down by it. And I'm not really

65:03

sure what's it's doing. I've just kind

65:05

of noticed it's doing

65:06

positive things. I've done a little bit

65:07

of my own research on it and how to use

65:09

it to make sure I'm not like killing

65:10

myself somehow. But um

65:13

what is it doing and why should everyone

65:14

consider getting one? So it's it's

65:17

referred to in the literature as

65:18

photobiomodulation, photobiomodulation.

65:21

So if you want to look up any of the

65:22

clinical studies, put photobiomodulation

65:25

and then put and dementia and

65:27

Alzheimer's and skin and um inflammation

65:31

and and the studies will come up. But

65:33

basically, different nanometers of of of

65:36

light have different effects in the

65:38

body.

65:39

And um so they are um well researched

65:44

and and publicized to reduce

65:46

inflammation,

65:47

um increase microvascular circulation,

65:49

so the smallest of the capillaries in

65:51

our body are affected by light. Um they

65:55

have a very specific effect in the

65:56

mitochondria, the powerhouse of the

65:58

cell.

65:59

So if you actually went through the wall

66:00

of a cell and into the cytoplasm and

66:01

found the mitochondria and you went into

66:03

the mitochondria, you'd see that there's

66:04

a motor in there that's spinning around.

66:06

It's called the Krebs cycle.

66:08

And this motor, when it spits out energy

66:11

called ATP, um

66:13

it you know, essentially it has two

66:14

choices every time it makes a

66:16

revolution, right? It it can either

66:18

create two units of energy or it creates

66:20

36 units of energy. It's either 16 times

66:23

more efficient or 16 times less

66:25

efficient. And what determines that is

66:28

whether or not oxygen enters that cycle.

66:31

So one of the things that red light does

66:33

is it goes through the wall of the

66:34

mitochondria and it kicks out a gas

66:36

called mitochondrial nitric oxide and

66:38

forces oxygen to dock.

66:41

So when you get into a red light therapy

66:42

bed or use red light therapy panels, one

66:44

of the things that's happening is you're

66:45

essentially forcing oxygen into the

66:47

mitochondria. You're forcing the oxygen

66:49

to use mitochondria and release a gas

66:51

called mitochondrial nitric oxide. This

66:53

is also measurable, by the way. You can

66:55

get saliva nitric oxide strips, you can

66:57

put it in your mouth and before you got

66:59

in a red light therapy bed, and you

67:00

could look at the saliva

67:02

um the amount of nitric oxide in your

67:04

saliva and you'd see it's like a pale

67:05

kind of yellowish pink.

67:07

Then you get in one of those red light

67:08

therapy beds for 20 minutes and about 10

67:10

minutes after you get out, test it

67:12

again, you'll see that your nitric oxide

67:13

levels are through the roof. That's a

67:15

positive sign that the mitochondria has

67:17

thrown this gas out and brought oxygen

67:19

in.

67:20

And it's to imagine what happens in a

67:21

cell when you give it 16 times the

67:25

amount of energy. So imagine upstaging

67:27

trillions of cells to allow them to

67:29

eliminate waste, repair, detoxify,

67:31

regenerate just by using light. It also

67:34

has a very positive effect on collagen,

67:36

elastin, fibrin. Um it's known to

67:39

improve angiogenesis, the new blood

67:41

vessel formation. Um I was I was on the

67:44

uh Joe Rogan's podcast a few months ago

67:46

and um he ended up buying one of these

67:48

red light um beds uh from me and we

67:52

installed it in his house and

67:54

he told me about four or five weeks ago

67:56

that he's no longer in wearing readers

67:58

anymore. Like his his eyesight has

68:00

improved that much. And he said he's

68:02

starting to really notice the changes in

68:03

his skin. So photobiomodulation is very

68:06

real and it absolutely works. But um

68:10

you know, without people having to think

68:11

that they have to spend that kind of

68:12

money on a red light therapy bed, you

68:14

can also just expose your skin to

68:15

sunlight, especially during first light,

68:17

the first 45 minutes of the day when

68:19

there's no UVA, there's no UVB, there's

68:21

high amounts of healthy blue light. Um

68:23

you can still generate vitamin D3. Let

68:25

me just run that back so I'm clear. Um

68:27

on the point about Rogan's eyesight, uh

68:30

I did some I was looking through some

68:31

research about the impact of red light

68:34

on eyesight and it said that it's good

68:35

for eyesight and so Incredibly good for

68:37

eyesight.

68:38

Cuz I was wondering whether I should be

68:39

looking at this thing while it's on.

68:41

Yes. And then I went online, Googled it,

68:43

had a rummage around and it said you can

68:45

look at it, you can stare at it.

68:46

You can. Yeah. You can. Because

68:48

remember, there's no UVA, there's no

68:49

UVB. Um and and and some of the

68:54

marginal information that comes out

68:55

about red light being damaging to you,

68:57

you have to remember that red light is a

68:59

spectrum. Infrared, for example, is a

69:00

spectrum. Most most red light therapy

69:03

beds run from 600

69:05

um nanometers to about 1,000 nanometers

69:07

wavelength of light.

69:09

As you get above that, you're you're in

69:11

the infrared spectrum, but you're going

69:13

all the way to 1,100, maybe even above.

69:17

So so in other words, when you say

69:18

infrared light, this is a non-visible

69:20

spectrum of light.

69:22

But there's a there's a broad number of

69:24

wavelengths, right? So an infrared

69:28

um

69:28

red light bed will have infrared light,

69:31

but it will be very low in the spectrum,

69:33

so it doesn't create heat. It doesn't

69:35

excite a chromophore that creates

69:36

vibration and makes you sweat. When you

69:38

get an infrared sauna, you're getting

69:40

very high into those wavelengths. You're

69:42

exciting a different chromophore in the

69:44

body and your water water to be specific

69:46

and it vibrates and creates heat and you

69:48

start to sweat. So, you don't sweat in a

69:50

red light therapy bed even though it's

69:52

infrared, low in the spectrum, but you

69:54

do sweat in an infrared sauna um even

69:57

though it's infrared light, it's high in

69:59

the spectrum. So,

70:02

the infrared light and the red light

70:03

that comes from red um red light beds

70:06

and red light panels and face masks is

70:07

incredibly beneficial for you. I mean,

70:09

I've

70:10

I would be scared to even tell you all

70:11

the the positive outcomes that we've

70:14

seen people that regularly use red light

70:16

therapy

70:17

because you know, you can't really make

70:19

medical claims around them, but I can

70:21

tell you firsthand we have seen just

70:23

astounding things that people would

70:24

probably consider miracles with red

70:27

light therapy. You mentioned the first

70:28

45 minutes of sunlight. Mhm. First

70:31

light. Uh cuz I'm try I always try to

70:32

try and figure out the sort of

70:33

evolutionary backstory to red light and

70:36

where it came from in nature and why it

70:37

was good for us as humans and why we've

70:39

lost it.

70:41

Those are the three sort of questions I

70:42

Well, we're really photovoltaic beings.

70:44

I mean, we're very tied to the circadian

70:46

cycle of the sun. I mean,

70:48

light causes the body to behave in very

70:50

special ways. I mean, you know,

70:53

you probably heard that getting first

70:54

light can actually reset your circadian

70:56

cycle and do more for you to sleep that

70:58

night than probably just about any other

71:00

sleep habit. So, your sleep routine

71:02

really starts with your morning routine

71:04

and it has a an effect on cortisol

71:06

receptors. It has effect on dopamine I

71:08

mean, on on melatonin receptors.

71:10

Remember, cortisol is a hormone that

71:11

responds to light, right? I mean, um

71:14

when our light when our eyes are closed

71:15

and light is passing through our

71:17

eyelids, it has a tendency to raise our

71:18

cortisol levels, which is why they tell

71:20

you not to use blue light at night,

71:22

right? You're stimulating cortisol and

71:24

you're stimulating a waking hormone when

71:25

you actually are trying to go to sleep.

71:27

So, by getting first light, you're

71:29

you're you're telling the body that it's

71:32

that it's morning. You know, you're

71:34

you're raising cortisol, you're

71:35

downregulating your melatonin receptors,

71:37

you're getting healthy blue light into

71:38

your eyes, you're getting healthy light

71:40

onto your skin. There's no UVA, there's

71:42

no UVB, none of the damaging rays of of

71:45

the sun. And and in 15 or 20 minutes, if

71:48

if you stack them all together, you can

71:50

ground do breathwork and get sunlight. I

71:54

mean, just try it for 7 days.

71:55

What if I have the red light at night

71:57

time? Is that going to trick my body

71:59

into thinking it's the morning? No.

72:01

The red light won't won't do that. It's

72:02

completely different. It's not the it's

72:04

not the blue light spectrum that we're

72:05

talking about.

72:06

have red light anytime of day.

72:07

Dope. You can have red light anytime. In

72:08

fact, red light I find it very relaxing.

72:10

I sometimes will do my um my red light

72:12

bed right before bed. Sleep like a baby.

72:15

We've been doing that as well at home,

72:16

so I was just checking cuz I I did

72:18

Google to to see if it was something

72:20

that would wake me up, but no, yeah,

72:21

you're right. Blue light is the thing

72:22

that wakes us.

72:23

Um bit of a tangent, but I just saw you

72:26

have a gulp of that water. Mhm.

72:29

What is in that water?

72:30

Hydrogen water. Why hydrogen water? This

72:33

is a little hydrogen generator I get. I

72:36

don't know if you can just see that, but

72:37

there's

72:38

um

72:39

what it's doing is a there's a little

72:41

Pick it up and and you'll be able to see

72:42

it in the Yeah, a little electrolysis

72:43

pump down there and it's and it's

72:45

basically adding hydrogen gas to the

72:47

water. It's not much left in there, but

72:49

if you if you fill it with water, you

72:51

can see that.

72:52

I mean, it is

72:53

fascinating. I am so convinced that

72:55

hydrogen water is the best water that

72:56

you can put in the human body and

72:59

there's a there's a website called

73:01

hydrogenstudies.com

73:02

that has about 1,350

73:05

studies um on the site. You can go to

73:07

hydrogenstudies.com. When you get to

73:09

that site, you can actually search um by

73:12

human clinical trials or animal clinical

73:14

trials, so you could sort out and look

73:15

at human clinical trials and look at all

73:18

of the ways that hydrogen gas is used in

73:22

therapeutic treatments, reducing

73:23

inflammation, improving the absorption

73:25

of supplements, improving proving

73:27

athletic performance, delaying um the

73:31

uh addressing delayed onset muscle

73:32

soreness, reducing neural inflammation.

73:34

I mean, there are so many clinical

73:35

trials

73:37

proving the efficacy of hydrogen gas in

73:40

the body. And people do hydrogen gas

73:42

through a nasal cannulas, through ear

73:44

cannulas, through eye cannulas. You can

73:46

breathe hydrogen gas, but by drinking um

73:48

hydrogen water, you have a very positive

73:50

effect on inflammation in the body. When

73:52

you pump the hydrogen into that, doesn't

73:54

it just come out the top? No, it's

73:56

sealed. So, it's under pressure. So,

73:58

what it does is it forces the gas back

74:00

into the liquid. Okay. And so, the

74:02

liquid actually gets has a high part per

74:04

million concentration of hydrogen gas.

74:07

The colder the liquid, the more gas you

74:08

can dissolve. So, it takes about 5 hours

74:10

for it to dissipate from that. Um some

74:13

people use hydrogen H2 tablets. Um I

74:16

just use this

74:18

this

74:19

hydrogen bottle and I take it literally

74:21

everywhere I go. I notice when I don't

74:23

have it.

74:24

How many of you started thinking about

74:26

your long-term health when you hit 30?

74:27

For me, this was a wake-up moment of me

74:31

thinking to myself, okay, I probably

74:32

need to start paying a little bit more

74:33

attention now. I already felt a change

74:35

in myself when I hit 30 with things like

74:37

my metabolism, my energy levels. So,

74:40

this year is no different. Zoe, which is

74:42

a company I've invested in, but also a

74:44

company that are a sponsor of this

74:46

podcast, helps me to make smarter food

74:48

choices all based on their world-leading

74:50

science and my own test results. If I'm

74:53

ordering food, I know how to make my

74:55

takeaways so much smarter by adding

74:57

things like a side of vegetables to eat

74:59

first or choosing the option with the

75:01

most fiber. Zoe helps me to make that

75:04

choice. It guides me and coaches me.

75:05

It's my personalized nutrition coach

75:07

that I have on me 24/7. And to help you

75:10

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75:12

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75:14

guys 10% off when you join Zoe now. All

75:16

you've got to do is use code CEO10 at

75:19

the checkout when you sign up. Enjoy and

75:22

let me know how you get on.

75:24

One of the subjects that's been really

75:26

sort of pertinent culture at the moment

75:27

is this subject of Ozempic. Mhm.

75:30

You know, since we spoke, it's got even

75:32

more popular.

75:33

Um and it's everywhere. I looked

75:34

yesterday at the company that make

75:36

Ozempic and I believe if my

75:38

Apple stock app wasn't deceiving me, the

75:41

company's worth trillions now. Oh, I'm

75:42

sure. Yeah, it was Ozempic and so,

75:45

Ozempic is is um

75:47

a peptide called semaglutide. Um it's

75:49

GLP-1 inhibitor. There's a there's

75:51

another one called tirzepatide, which

75:53

actually did better in side-by-side

75:54

clinical trials than semaglutide and

75:56

that's the Wegovy version um or the

75:59

Mounjaro version. Um semaglutide I think

76:02

is Ozempic and Wegovy.

76:04

But,

76:06

these are great for for people that have

76:09

um that are type 2 diabetics or that are

76:12

morbidly obese and have issues with

76:14

cravings um and that have that have

76:19

either diabetes or or or

76:21

significant obesity. I think that they

76:24

become drugs for vanity

76:27

and what people are realizing now is all

76:29

of the issues with gastric emptying,

76:31

paralytic gut, um the fact that when you

76:35

start to Paralytic gut? Paralytic gut,

76:37

which is where you actually get

76:38

paralysis in in the gut. Because one of

76:40

the things that they slow is gastric

76:42

emptying. And so, if you slow the rate

76:44

of gastric emptying, very often contents

76:46

can putrefy

76:47

um in the gut.

76:49

And

76:50

it's not that I'm totally against these

76:52

these peptides. If you use these

76:53

peptides, you have to be in a weight

76:55

training program. So, you have to be um

76:58

doing resistance training because

77:00

a third of the weight that you lose, as

77:02

much as half of the weight that you lose

77:04

in some in some of the studies is lean

77:07

body mass.

77:08

So, if you're taking a semaglutide or

77:10

tirzepatide, then you want to make sure

77:11

that you're also on a I would

77:15

our clinical team would put you on a

77:16

peptide, a growth hormone peptide like

77:20

uh sermorelin, ipamorelin to muscle

77:23

protect and then also make sure that

77:24

you're on a a a good strength training

77:27

regimen because just taking these you

77:29

you don't get to spot remove fat. And

77:31

so, what happens is you start to

77:32

aggressively mobilize and metabolize fat

77:35

very often from the cheeks and from the

77:36

face and people are getting semaglutide

77:39

face or Wegovy face, they're saying now,

77:41

where like their cheeks get really

77:42

sunken in, their eyes, the fat pads

77:43

beneath their eyes um are metabolized,

77:46

their eyes start to look like they're

77:48

sunken in. So, if you're morbidly obese

77:50

or or have a significant amount of

77:53

weight to lose, you struggle with

77:55

cravings, you have you're either

77:57

severely pre-diabetic or you're

77:59

diabetic, I mean, they can be a

78:00

life-changing. But, for vanity purposes,

78:04

I I don't I I think there are a lot

78:06

better peptides and a lot better ways to

78:08

do it. What's your life like these days?

78:11

It's amazing.

78:12

You know, I

78:14

I think I was telling you before I got

78:15

on the show today that I feel like I

78:16

live somebody else's life.

78:19

I really do. I

78:21

I've I can't believe that I found

78:22

something that I would

78:24

like otherwise do for free and somehow

78:26

monetized it.

78:27

And

78:29

you must feel the same way, you know,

78:30

when you're when you're doing a podcast

78:32

and

78:33

you know, your your message starts to

78:35

resonate, the caliber caliber of people

78:37

that it attracts. I mean, the rooms that

78:39

you get to get in.

78:40

And

78:41

for me, I have a insatiable

78:45

level of intellectual curiosity. Like

78:47

I'm super super curious.

78:49

And the fact that I get to sit down with

78:51

people like yourself, but some of the

78:53

greatest minds um you know, in in the in

78:55

the world that are studying longevity,

78:58

anti-aging, biohacking, um cancer,

79:01

mortality, um sports performance, it's

79:05

it's just

79:06

I pinch myself.

79:08

I really do.

79:09

With all privilege comes our pain.

79:11

Yeah.

79:12

What's the pain?

79:15

Um You need to be honest with me here

79:17

cuz this is why we did start this

79:18

podcast many years ago.

79:20

Um

79:22

you know, for me um

79:25

the pain is as I've

79:28

as I've become more popular, I guess, um

79:31

and as the message has resonated

79:35

I've become a little more distant from

79:37

the

79:38

the folks that I initially

79:40

sought to serve and support.

79:43

I had a lot more individual reaction. I

79:45

mean uh interaction in the beginning.

79:48

And now I just

79:50

simply can't

79:51

interact with a number of people

79:55

that would like me to, that actually do

79:57

need me.

79:59

And I've turned my attention to trying

80:01

to

80:03

train and support the training of as as

80:05

many qualified people as I can so we can

80:08

really touch the masses.

80:10

And

80:12

I had no idea how much the message would

80:14

resonate.

80:15

And it resonated in a way that

80:18

overpowered my company.

80:21

And

80:22

you know, one of the worst things is

80:23

kind of collapsing under the weight of

80:24

your own success.

80:27

And that didn't quite happen, but

80:30

you know, the the message began to

80:31

resonate and there were so many people

80:34

coming to take our test and seek our

80:36

services that really really needed us

80:39

and I felt the burden of of that need.

80:42

Um

80:43

we were for a period of time unable to

80:45

respond. We were overwhelmed and that

80:47

turned to vitriol in some cases. Um

80:50

that's stabilized now, but um

80:54

you know, it's kind of the it's kind of

80:56

be careful what you ask for.

80:59

Because you might get it, but

81:01

by the same token I wouldn't I wouldn't

81:03

change a thing.

81:04

I feel like the most blessed person in

81:06

the world. What about the family impact

81:08

cuz you got kids and and all that? You

81:10

know,

81:11

that is the greatest blessing for me. My

81:13

my kids are landing at LAX in in within

81:17

a few hours. They're they're in the air

81:18

right now. Um

81:20

So I've got three children and the

81:22

oldest two work for me full-time. And my

81:25

daughter just graduated from nursing

81:27

school. She's starting her PhD in

81:28

nursing. My son's about 14 weeks behind

81:30

her. So they're both going for their

81:32

their PhDs in nursing. So they'll both

81:34

have their doctorate in nursing.

81:36

And

81:38

that is the greatest blessing in life is

81:41

when

81:42

your you see that your kids have a

81:46

passion because I feel like you can

81:48

teach your kids anything, but you can't

81:49

give them a passion.

81:51

And the and the fact that they think

81:53

enough about me and what I'm doing to

81:55

want to follow in my footsteps is

81:59

that is beyond

82:02

anything I can even put into words.

82:04

Because I travel with my kids. I

82:07

I see clients with my my my kids. You

82:10

know, my wife is also in in the business

82:13

and I think the pace of our life would

82:14

be a lot more difficult if I didn't have

82:16

my family around. I saw Dr. Peter Attia

82:18

talking on a podcast once and he

82:21

I'm paraphrasing so I don't know if I'm

82:23

getting right, but he said

82:25

you get 19 years with your kids.

82:28

The first 18

82:30

is from when they're born to when

82:31

they're 18 years old.

82:33

And then they're gone.

82:35

The last year

82:37

is spread out over the entirety of the

82:38

rest of their lifetime.

82:41

And I thought how sad.

82:43

Cuz I spend more time with my kids now

82:45

than I did

82:47

almost since the day they were born.

82:50

And they're just becoming these adults

82:52

that really inspire me.

82:54

And so I think that

82:57

of all the blessings that God has given

82:58

me, that's the biggest one.

83:02

There are a couple of other things that

83:03

I was really curious about um when I

83:05

knew I was going to speak to you today.

83:07

One of them was kind of what we were

83:08

talking about there with your family,

83:09

which is just like the role of

83:10

community. Mhm. Which we're clearly in a

83:13

bit of a loneliness epidemic.

83:15

And I Well, um you know, we knew in the

83:18

in the life expectancy space um

83:20

this is a material fact that if you

83:21

wanted to cut somebody's life expectancy

83:23

in half at any age

83:25

put them in isolation.

83:28

So if you put a human being in

83:29

isolation, you will cut their life

83:31

expectancy in half. How could you see

83:33

that in the data?

83:34

Because you would there there was

83:36

something we we call the broken heart

83:37

syndrome or caregiver syndrome um and

83:40

it's it's well documented in the

83:41

elderly. Um you know, when you have

83:44

a

83:45

companion that you've been with for 40

83:48

or 50 or 60 years and that companion

83:50

passes, very shortly thereafter the

83:53

second companion goes.

83:55

And I always thought that was a myth,

83:57

like a nice tale of heartbreak and love.

83:59

Well, we call it the broken heart

84:00

syndrome. It has nothing to do with a

84:02

broken heart, but I mean the the

84:03

emotional state, I mean the frequency in

84:06

their body changes. Um and when this

84:09

surrenders, this surrenders. The mind

84:12

and the body.

84:12

Mhm. When the mind surrenders, the body

84:14

surrenders and there's a lot of

84:16

emerging body of evidence that that's

84:18

actually putting some science behind the

84:20

the the theory that emotions can make us

84:22

sick and and I think everyone's believes

84:25

that in the in the stress can actually

84:28

um lead to pathology and lead to

84:30

disease, but um so you know that when we

84:33

isolate human beings, it's hard to

84:35

completely isolate them, but we know

84:36

when you isolate human beings that that

84:38

it

84:39

it has dramatic effect on life

84:40

expectancy. Some of the worst

84:42

science that we and research that we do

84:45

is when we study components of the body

84:48

or cells from the human body in

84:49

isolation. You take a cell out and you

84:51

put it in a Petri dish and you look how

84:53

it behaves in vitro and then you assume

84:55

when you put that cell back into the

84:56

body that it's going to behave that way

84:58

because cells exist in communities, too.

85:01

They exchange with their outside

85:02

environment. They eliminate waste. They

85:04

repair. They detoxify. They're they're

85:06

very active community. Um and so

85:11

um

85:12

you know, the impact of community has a

85:13

meaning all the way down to a cellular

85:15

level. They do animal studies on this

85:16

kind of thing, right? Oh yeah, no

85:18

question. On loneliness and On

85:21

loneliness and and and and isolation and

85:23

it and it has a dramatic effect on life

85:26

expectancy. It's been a while since I've

85:27

read an animal study, but we knew

85:30

that isolation

85:32

had a dramatic effect on mortality. So

85:35

if when when a loved one got moved into

85:38

an assisted care living facility or we

85:41

looked at the proximity of family

85:44

members to a mother or a father that had

85:46

just lost a grandmother or grandfather

85:49

that had just lost one or the other

85:50

spouse.

85:51

Um and you knew that

85:54

the family wasn't going to visit

85:55

frequently and then now that person was

85:57

in isolation. Um and when I mean

86:00

isolation, not completely isolated, but

86:02

they were isolated from daily activity.

86:04

That had dramatic effect on life

86:06

expectancy. It was it was a comorbidity

86:09

factor that we used.

86:11

And mainly in the elderly, but it would

86:13

happen in younger ages as well.

86:15

So I I think that community is

86:17

increasingly important for me. You know,

86:20

I remember when I

86:21

sold my company, my my partner Grant

86:23

Cardone at the time told me, he said,

86:25

"Your sphere is about to get a lot

86:26

smaller."

86:27

And I was like

86:28

that doesn't make sense.

86:30

Um

86:31

my sphere is about to get a lot larger.

86:33

And it was true. What he said was very

86:35

true. You know, my

86:37

I spend the majority of my time with my

86:40

kids.

86:41

Um they're working for me full-time. We

86:44

travel together. We see clients

86:45

together. Um we're in the hunt together.

86:48

Um they're big supporters of the

86:50

business. They caught the bug. They're

86:51

in school together. You know, my

86:52

youngest still still lives with me.

86:55

So my my circle has gotten so much

86:57

smaller even though you see

87:00

me out there with like Dana Whites and

87:02

you know, and and and celebrities and

87:04

athletes and those are those are the

87:05

flashbulb moments. But in my day-to-day

87:08

and week-to-week and month-to-month

87:10

I'm I'm intensely surrounded by my

87:12

family and a very small team that I have

87:15

a high level of trust in that is really

87:18

helping me continue to be in service.

87:20

You know, to

87:22

to the clients that I'm working with.

87:24

What about retirement and and purpose

87:25

and the role that plays in our

87:26

longevity?

87:28

It's been a while since I I used to have

87:29

the VBT, the variable basic table

87:31

memorized, but there is a probabilistic

87:34

factor

87:35

um

87:36

um for retirement and communal

87:39

interaction and

87:41

um I forget exactly what that the level

87:44

of impact was, but we had we had a

87:46

probabilistic model where we would use

87:47

this demographic data. Um but there is

87:50

no question that

87:52

mortality accelerates post-retirement. I

87:55

don't know that I've dealt enough into

87:56

the science to really accurately comment

87:58

on it, but it must have something to do

87:59

with the loss of the sense of purpose.

88:01

When you look at blue zones and and

88:03

centenarians, you know, one of the

88:06

one of the key themes even beyond the

88:08

diet because

88:10

you know, the diets were very different.

88:12

You know, Singapore has one of the

88:13

longest life expectancies on earth. They

88:15

eat the highest amount of meat. Sardinia

88:17

has very long life expectancies. They

88:19

eat they high amounts of bread, pasta,

88:22

and flour. Um you know, the the

88:24

Mediterranean blue zones eat high

88:26

amounts of of oils, fish, and fats.

88:29

But what was a common theme between all

88:31

of them was mobility into older ages

88:34

and a sense of purpose.

88:36

And there was no such thing as assisted

88:38

care living facilities where you

88:39

assisted care was when grandma and

88:40

grandpa moved back in with their kids

88:44

and live with the kids until the day

88:45

that they died. And maybe her purpose

88:46

was just to get vegetables that night

88:48

for

88:49

for dinner. Um

88:50

and grandpa's purpose was maybe to

88:52

continue to make belts for the a leather

88:54

smith down the road, but they they had a

88:55

sense of purpose. When you think back to

88:58

your your job in life insurance and

89:00

the the role that you had, is there any

89:02

parts of it that you look back on now

89:03

and you think about that industry that

89:04

are unethical? Because you can't reach

89:07

out because of law and privacy to these

89:09

people, as you've said. You know, that'd

89:11

be a violation of a variety of different

89:13

sort of policies and stuff. But is Is

89:15

anything else it within the the practice

89:18

of it that you find unethical?

89:20

Just the fact that you know

89:22

I wasn't allowed to have any contact

89:24

with the patient or the training

89:25

physician and I understand for good

89:27

reason because most of the people that

89:28

are doing this work are not licensed to

89:30

practice medicine. They don't want them

89:31

jumping Yeah.

89:32

into the practice of medicine. Um but

89:35

when you notice things that are obvious

89:37

and then maybe you know that a doctor

89:39

would have appreciated appreciated that

89:40

phone call. Oh my gosh, I didn't know

89:42

that she was on that other script. Thank

89:43

you for calling me. I mean wasn't to

89:46

besmirch them or or take over their

89:48

practice of medicine.

89:50

But I really wish that the database

89:54

would see the light of day.

89:56

The databases that are used in

89:58

predicting mortality in my opinion could

90:00

change the face of humanity.

90:02

I know why they won't because it would

90:04

upend modern medicine in a way in in my

90:06

opinion that would be catastrophic.

90:07

Destroy that business as well, wouldn't

90:09

it? Cuz they need people to die, really.

90:11

They do. Because they don't want to be

90:14

paying out. Well, you know, annuities um

90:18

need people to die. Life insurance wants

90:20

people to to live a little bit longer.

90:22

Okay. Oh, yeah, cuz the longer they

90:24

live, the more they pay. The longer they

90:26

live, the more they pay.

90:28

want

90:28

But annuities You've put down a deposit,

90:30

basically. So, they want that deposit

90:31

They guarantee me an income stream for

90:32

life. Wow. So, if you could kindly

90:35

expire tomorrow, that would be good for

90:37

me. And the same companies do both? Same

90:39

companies do both. Okay. Um there's

90:41

something called a life insurance life

90:43

annuity contract, a lilac. Where you

90:45

actually put an annuity and a life

90:46

insurance policy on the same life and

90:48

you can't lose.

90:50

As you know, we have a closing tradition

90:51

on this podcast where the last guest

90:52

leaves a question for the next guest not

90:53

knowing who they're going to be leaving

90:54

it for. Um The question that has been

90:57

left for you is this one.

90:59

I wish I knew who your last guest was.

91:02

Okay.

91:03

So,

91:05

they said they're quoting someone and it

91:07

says Gundalini said, "Be the change

91:12

that you want to see in the world."

91:16

What is the change you want to see?

91:22

God, it looks like Gundalini.

91:25

Gundalini.

91:26

You try to read this. Gandhi. Gandhi

91:29

said, "Be the change you want to see in

91:30

the world." What is the change you want

91:32

to see and how

91:37

will you be it? Wow. Well, I want to see

91:40

people live healthier, happier, longer

91:43

lives, more fulfilling lives. And

91:47

I will be that by continuing to get the

91:48

message out.

91:50

And that's why I'm here.

91:52

And that's what I wake up and do every

91:53

day.

91:55

And I can't hold a candle to Gandhi.

91:58

Um but I will spend the balance of my

91:59

adult lifetime continuing to get the

92:02

message out.

92:04

Gary, thank you so much. Um having

92:05

getting to know you on and off camera,

92:07

you're such a a genuine, true

92:10

lovely human. Thank you.

92:11

And your intent and your intentions are

92:13

so clear to me and so pure. So, you

92:15

know, I've had loads of people reach out

92:17

to me since our last conversation and

92:18

speak to the value that your advice has

92:20

had on their lives. Oh, man.

92:22

Thousands and thousands of people, I

92:23

mean

92:23

Dude, that makes me I mean, I looked at

92:25

the last conversation, I looked at my

92:27

emails around that time. I searched your

92:28

name and when I say thousands, I mean

92:30

thousands and thousands of people that

92:31

are reporting to have better lives,

92:33

happier lives because they listened to

92:35

that conversation. So,

92:36

So awesome. They probably won't be able

92:38

to reach you. So, on behalf of those

92:39

people, I wanted to say thank you so

92:40

much for doing what you do because it's

92:41

very important.

92:42

Oh, man. It's not always easy.

92:43

Yeah. But you know, it's an occupational

92:45

hazard. Yeah, it is. Putting yourself

92:47

out there in the world as I would know.

92:48

So, thank you so much, Gary. I

92:50

appreciate your time. Super welcome.

Interactive Summary

This episode features Gary Brecka, a human biologist, who explains his philosophy of optimizing health by addressing nutrient deficiencies and understanding body data. Brecka discusses the impact of genetic methylation, the importance of specific blood markers, and how lifestyle choices—including sunlight, grounding, and proper nutrition—can prevent chronic disease and improve longevity.

Suggested questions

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