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The Doctor That Got Banned For Speaking Out:“We've Been Lied To About Medication!” Dr Aseem Malhotra

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The Doctor That Got Banned For Speaking Out:“We've Been Lied To About Medication!” Dr Aseem Malhotra

Transcript

3673 segments

0:00

From everything I know now, I've

0:01

reluctantly come to the conclusion that

0:04

the COVID vaccine introduction has had a

0:07

catastrophic net negative effect on

0:10

society. And this is really most

0:11

disturbing and I've not been public

0:13

about this before, so I'm going to tell

0:14

you this for the first time.

0:15

Dr. Aim Malhatra is one of the UK's

0:18

leading celebrity heart doctors whose

0:20

influential research has sparked

0:22

important conversations about nutrition

0:24

and exercise and the healthare system.

0:25

The Times journalist called me and said,

0:27

"We've got [music] reports now of a 25%

0:29

increase in heart attacks in hospitals

0:31

in Scotland which are unexplained. What

0:33

do you think about the vaccine?" I was a

0:34

bit surprised. I said, "What do you

0:35

mean?" He said, "And to have that deep

0:37

knowledge and understanding gives me

0:38

very deep pain." And medical knowledge

0:41

is under commercial control. This

0:42

[music] is the root of the problem. The

0:44

system is psychopathic when it comes to

0:46

making money. And there [music] is so

0:47

much harm that comes from drug

0:49

prescriptions. One credible estimate

0:51

suggests the third most common cause of

0:53

death globally after heart disease

0:54

[music] and cancer is prescribed

0:56

medications. This is a dirty secret

0:58

within medicine. We should shift our

1:00

approach [music] to health from a

1:01

predominantly drug-based model within

1:03

healthcare to one which is more based

1:06

upon lifestyle. So what is that? The

1:08

first thing that needs to be done is by

1:10

2030 it's estimated that about 23.3

1:13

million people will die yearly because

1:15

of heart disease globally and there are

1:17

200 million people taking statins

1:17

[music] around the world. But the

1:19

increase in life expectancy with statins

1:22

over a 5year period, 4.2 days.

1:24

How do I prevent myself ending up on

1:26

either statins or having a heart attack?

1:29

This is really important information.

1:31

So,

1:34

quick one before we get back to this

1:35

episode. Just give me 30 seconds of your

1:37

time. Two things I wanted to say. The

1:39

first thing is a huge thank you for

1:41

listening and tuning into the show week

1:43

after week. It means the world to all of

1:44

us and this really is a dream that we

1:46

absolutely never had and couldn't have

1:48

imagined getting to this place. But

1:50

secondly, it's a dream where we feel

1:51

like we're only just getting started.

1:53

And if you enjoy what we do here, please

1:55

join the 24% of people who watch this

1:58

channel regularly and have hit the

2:00

subscribe button. Means more than I can

2:02

say. And if you hit that subscribe

2:04

button, here's a promise I'm going to

2:05

make to you. I'm going to do everything

2:07

in my power to make this show as good as

2:09

I can now and into the future. We're

2:12

going to deliver the guests that you

2:13

want me to speak to and we're going to

2:14

continue to keep doing all of the things

2:16

you love about this show. Thank you.

2:19

Thank you so much. Back to the episode.

2:22

[music]

2:24

Dr. Aim,

2:26

you're working on a documentary called

2:28

First Do No Farm, which will be out

2:30

shortly. Why did you choose the name for

2:34

your documentary, First Do No Farm?

2:36

Well, first and foremost, the credit to

2:38

that name actually goes to my

2:39

co-producer, Donal O'Neal, who's made a

2:41

number of health documentaries. Um, and

2:43

he's a former international athlete. Uh,

2:46

you know, and he used to work in

2:47

marketing and PR, so he's very good with

2:48

with slogans and sound bites. Um but it

2:51

it it totally resonated with with my

2:54

work and what I've been doing probably

2:55

for well over a decade which is to try

2:58

and shift um our approach to health from

3:01

a predominantly pharmacologicalbased

3:04

drug-based model within healthcare to

3:07

one which is more based upon lifestyle.

3:10

And that's because that's where the best

3:12

evidence is in terms of improving our

3:15

health but also in terms of managing the

3:18

health care system where there is so

3:20

much waste where drugs are

3:21

overprescribed and obviously first do

3:24

noarm means in the consultation room the

3:27

ideal scenario should be with every

3:30

patient that if there is a uh

3:34

non-drug-based way non-farmacological

3:36

way of managing their illness chronic

3:38

disease which is the big problem in

3:40

society right now on healthare systems.

3:42

Uh that should be the primary primary

3:44

approach. Um as well as highlighting

3:48

through the title that there is so much

3:50

harm that comes from drug prescriptions.

3:52

Just I mean this is something that even

3:55

brings gasps from audiences when I give

3:57

talks and lectures all around the world.

3:59

And whenever even I say it, I even think

4:01

bloody hell, how have we got to this

4:02

situation? That, you know, one credible

4:05

estimate suggests the third most common

4:07

cause of death globally after heart

4:08

disease and cancer is prescribed

4:11

medications. What your doctor prescribes

4:13

for you so-called appropriately.

4:16

mainly because the information which

4:20

doctors use to make decisions for

4:22

patients when it's prescribing drugs

4:24

invariably is based upon a gross

4:28

exaggeration on the safety and the

4:31

benefits of those drugs

4:33

and the phrase first do no harm which is

4:36

the original phrase that you've spanned

4:37

for the title where does that phrase

4:39

originate from

4:39

well actually it's one of the basic

4:41

principles of medical ethics you know as

4:43

doctors that's the something that's

4:45

almost we indoctr ated or ingrained with

4:47

whenever we you know practice medicine

4:50

treat patients um that should be the

4:52

that's at the forefront of our minds. It

4:54

should be at the forefront of our minds.

4:55

What what professionally what is your

4:58

professional title?

5:00

Um I'm a consultant cardiologist.

5:02

What does that mean if I

5:03

So I uh I special so I qualified as a

5:05

doctor um medical doctor in 2001. Um and

5:09

then after um becoming a um I I decided

5:13

after two or three years of doing my

5:15

sort of initial post-graduate basic

5:17

training in medicine to subsp

5:19

specialcialize in cardiology which is

5:21

basically everything to do with the

5:22

heart and then within that subsp

5:24

specialcialization of cardiology or

5:26

specialization of cardiology I trained

5:29

in interventional cardiology. Um in

5:33

layman's term Steve that means keyhole

5:35

heart surgery. So that's what I trained

5:37

to do.

5:37

[clears throat]

5:38

Um and I did that um uh you know up

5:40

until probably 2014 2015 and then I

5:44

shifted more towards a more holistic

5:46

approach to managing heart disease

5:48

especially looking at the science and

5:51

practicing the evidence base behind how

5:54

lifestyle changes can manage heart

5:56

disease and even potentially reverse it.

5:58

You must have seen a lot of hearts in

6:00

your time.

6:00

Yes. I I I think in terms of I was

6:03

thinking about this because you know up

6:05

until 2019 I was you know an NHS doctor.

6:08

We'll come back to later why what

6:10

happened in 2019 but and that's my

6:12

passion. Um but within the NHS which I

6:15

think trains doctors brilliantly um it's

6:19

a very high intense workload. Um and uh

6:23

just to give you perspective on that um

6:25

you know we have the highest uh capita

6:30

population per doctors in Europe. So

6:32

least number of doctors per uh

6:35

population in the country um and the

6:37

most number of doctors on night shift.

6:38

So it's quite intense and because of

6:40

that I was thinking back in terms of the

6:42

patients I would see on the wards and

6:44

the throughput and the people that I

6:45

would see you know in the what we call

6:47

cardiac catheter lab where we did the

6:49

diagnostic angagrams to visualize the

6:51

heart arteries and we put stances in

6:53

etc. Over my career I've probably

6:55

managed tens of thousands of patients.

6:58

Why did you choose to specialize in the

7:01

heart versus other parts of the sort of

7:04

medical um ecosystem that you could have

7:06

pursued? What was there something?

7:09

Yeah, I don't know if there's one answer

7:11

to that. I think one trigger very early

7:13

on in my life is um you know I grew up

7:16

in a medical family. Both my parents

7:18

were GPS and I had an older brother who

7:20

had Down syndrome

7:22

and uh which also meant he had a small

7:24

hole in his heart as well. And when I

7:27

was 11 and he was 13, shortly after his

7:30

birthday um he got a tummy bug. Uh

7:34

standard tummy bug. We didn't think

7:36

anything of it. Within 6 days, Steve um

7:38

he became breathless and rapidly

7:41

deteriorated. Got admitted to hospital

7:42

and had a cardiac arrest and died. And

7:45

later on it emerged his you know the

7:47

postmortem showed his heart was

7:48

massively enlarged and essentially had

7:50

something called viral myocarditis. It

7:52

can happen actually to anybody. You get

7:54

a a cold and in one in 10,000 people up

7:58

to one in 100,000 people. The body then

8:01

has what we call an autoimmune reaction.

8:03

instead of dealing with a cold on its

8:05

own, it then attacks the heart. And and

8:07

in a third of those patients, you can

8:09

basically who get that, they will

8:11

deteriorate and they will die. And so I

8:13

think for me that had such a profound

8:14

impact on my life. Um and with both

8:17

parents being doctors, of course, there

8:18

was a bit of there was no pressure from

8:20

my parents. They wanted me to, you know,

8:21

my dad wanted to be a cricketer. But,

8:23

um, I think that was implanted in my

8:26

mind that if I was to go into medicine,

8:29

I wanted to get involved in managing and

8:32

helping people, you know, or prevent

8:34

heart disease. So, I think that's where

8:36

it started from. It's interesting cuz

8:37

when we think about our health,

8:40

a lot of people think about the amount

8:41

of weight they have on them. They think,

8:43

you know, abdominal fat. They think

8:45

about how strong they are, their muscles

8:46

and things like that. Then a lot of the

8:47

other things are very superficial the

8:50

sort of measures of our health because

8:52

we've never really seen our heart and

8:54

it's similar to our brain because we've

8:55

never seen our heart or our brain we I

8:58

think we typically devalue the role that

8:59

they play in our over overall health but

9:01

when I was reading some stats around

9:03

heart disease and how how many people

9:07

die from heart disease I was shocked so

9:11

if we start there then can you give me a

9:13

sort of macro picture on why it's

9:15

important to keep our hearts healthy

9:17

and how um how many people are are dying

9:21

because of unhealthy hearts.

9:22

Yeah. So globally

9:25

um it's estimated and it's on the

9:27

increase by uh that certainly by 2030

9:30

it's estimated that about 23.3 million

9:32

people will die um yearly because of

9:36

heart disease globally right and that's

9:39

a huge number and and just to put it in

9:40

perspective it's the one of the leading

9:43

causes of premature death in European

9:45

men it is the leading cause of premature

9:47

death and uh and that's why I think it's

9:50

so important not just about people's

9:52

lives being cut short early. But also

9:54

there is an associated morbidity which

9:56

means the quality of life deterioration

9:58

that happens with people who are

10:00

diagnosed with heart disease that may

10:01

not be able to exercise very much

10:03

because they get pain in their chest

10:04

because of a blockage or they may

10:06

develop what we call heart failure where

10:07

the pump function of the heart is

10:09

affected [snorts] because of blockages

10:11

or because of a previous heart attack

10:12

they survived and therefore they can't

10:14

actually do the activities they want to

10:15

enjoy. And that's you know way bigger in

10:18

fact that's well sorry just as important

10:20

as as of course the the premature death

10:22

rate. So, no, it's a massive issue. And

10:23

it's interesting, Steve, that you you

10:24

mentioned um the image side where people

10:27

think about excess body fat and big

10:28

muscles and that kind of thing. And that

10:30

also plays into a certain culture and

10:32

mindset that I think detracts from us

10:35

actually addressing the root cause of

10:36

many problems with society today in our

10:38

health. Um which is not related to image

10:41

actually. It's about the the basics of

10:43

of uh reducing stress, about our social

10:45

interactions, etc.

10:47

um to some degree obviously what we eat

10:49

is linked to to how we look but um yeah

10:51

I mean absolutely I think it's not

10:53

something people really think about

10:55

enough um often until it's too late. How

10:58

does it impact women? Because we

11:00

mentioned it being the single biggest

11:02

killer of men, I think, in Europe, you

11:03

said. What about women?

11:05

It's um not as bad for women. And part

11:09

of the reason for that, Steve, is and

11:11

traditionally women on average will live

11:14

live up to 10 years longer than men,

11:16

right?

11:16

And the reason for that is that women

11:18

don't tend to develop heart disease um

11:20

at the same age as men. But after the

11:23

menopause, the rates of heart disease

11:25

actually start to catch up. So even

11:27

though women will live longer, it's

11:29

still a big issue with women as well.

11:31

Um, absolutely.

11:33

Your mother passed in November 2018

11:36

after a bout of sepsis.

11:38

The

11:40

interesting I read this quote you'd

11:41

said, I think it was on um on I news.

11:44

You said G you're a GP that had

11:46

dedicated 25 years of your life to the

11:47

NHS and ultimately you failed by it.

11:51

How were you failed by it?

11:57

In my mom's circumstance, what happened

11:59

was, you know, the system was under so

12:03

much strain. I mean, she had suffered

12:05

with a a debilitating rheumatoid and

12:07

osteoarthritis for many years.

12:09

Initially, I think a lot of her health

12:12

issues were rooted in her weight. I

12:14

mean, she was addicted to all to

12:15

processed food. She consumed a lot of

12:17

sugar. I grew up in a household where

12:18

there were just cakes and biscuits

12:20

everywhere. And, you know, she was a

12:21

very loving mom. Um, but I was addicted

12:24

to sugar probably because of that as

12:26

well for some time. But it affected her

12:28

health and I and and and I love my mom

12:30

very very dearly and it was

12:31

heartbreaking to watch her suffer slowly

12:33

over a number of years. But the way she

12:35

was failed by the NHS specifically is

12:37

[snorts] that um I had already been

12:40

campaigning for many many years in terms

12:41

of seeing how the system of the NHS was

12:43

being put under more pressure. a lot of

12:45

it by diet related disease and there

12:47

were so many opportunities for us to

12:50

that were being missed to actually

12:51

improve the system so that doctors could

12:54

provide quality care to every patient to

12:56

a good degree. When my mom was admitted

12:58

to hospital and she was sick with

13:00

infection in her spine, she'd become so

13:01

frail from her arthritis that her immune

13:04

system probably wasn't functioning

13:05

properly as well. because the hospital

13:07

was under so much strain because we had

13:09

not sorted out the root causes of the

13:10

pressure on the system. They missed a

13:12

heart attack for nine days. And it's

13:15

extraordinary, Steve. I remember I was a

13:17

they knew I was a cardiologist and it

13:19

was our local hospital. My dad was a

13:21

local GP and he was considered a medical

13:23

leader. I mean, he was loved and liked

13:25

and respected massively. So, even with

13:27

all of that, [snorts] um they did

13:30

everything they could to help her. But,

13:31

um she she went she became breathless

13:33

one day. They treated her for, you know,

13:35

um, uh, you know, for infection with

13:39

lots of fluid, etc. And, um, they

13:42

carried out a heart scan. It was

13:43

decided, let's do a heart scan on her.

13:44

But nobody actually looked at the result

13:47

of the heart scan and shared it with the

13:49

team because they were so busy they just

13:50

missed it. And I get sent the results 9

13:54

days later being asked, "What do you

13:57

think?" And I immediately noticed that

13:59

it was an echo. It was a heart scan

14:00

looking at the pump muscle of the heart

14:01

that part of the heart muscle wasn't

14:03

working well. I said, "She's had it.

14:04

You've missed a it was a a relatively

14:07

minor heart attack, but enough to cause

14:08

her to go into what we call heart

14:09

failure." And within 24 hours, fluid in

14:11

the lung, intensive care, went into a

14:14

coma, and that was it. So that was a was

14:17

a failing of the NHS, but not because of

14:21

the principles of the NHS, but because

14:22

the NHS um over the years, Steve, has

14:27

lost its um basic ability to care for

14:31

patients because the principles of the

14:33

NHS have not been upheld.

14:36

For anyone that isn't aware, the NHS

14:38

means National Health Service, which is

14:40

the health service and system um within

14:42

the UK. the the I mean just a few years

14:47

later your father passes in July 21 from

14:50

a sudden unexplained heart attack at 73

14:52

years old. Um

14:57

I mean so there's three members of your

14:59

family that have ultimately

15:02

um died as a result of or closely linked

15:05

to heart attacks. Your immediate family.

15:09

Yeah. Yeah. Um yeah to some degree. Yes.

15:13

All related to the heart. All related to

15:14

the heart.

15:18

Was your father's heart attack avoidable

15:21

in your view? If you think about the

15:22

lifestyle choices and things like that

15:24

that you you talked about your mother

15:25

there, was that avoidable any in any

15:27

sense of the word?

15:28

Yeah. Interesting. Um so there are two

15:30

components to my dad's um death. Um

15:34

strictly the the postmortem findings

15:37

didn't reveal a heart attack per se, but

15:39

for all intents and purposes it can be

15:40

seen that way. So, um, the first thing

15:43

that happened was, um, my dad, by the

15:45

way, was a very fit man for his age, 73

15:47

years old. Um, played badson every week.

15:50

Um, had a bit of high blood pressure,

15:53

but had got that better controlled after

15:54

quitting sugar, partly because of all my

15:56

campaigning. Uh, and he listened to me

15:58

and he was very into eating healthy

15:59

food. In fact, you know, I consider

16:02

myself to be extremely fit. I'm obsessed

16:04

with exercise. Um, you know, I played a

16:06

lot of competitive sport when I was

16:07

younger, partly because of him. And uh

16:11

it was very unexpected when he called me

16:13

up. It's coming up to his anniversary,

16:15

July 26, 2021.

16:18

Uh and he said, "I seem I've got some

16:20

chest discomfort." And uh I asked him,

16:23

you know, to describe it. In medicine,

16:25

80% of your diagnosis, if you're a good

16:27

doctor, comes from the conversation. So

16:29

the way he described his chest

16:30

discomfort was a central heaviness going

16:32

to his shoulders. So immediately think

16:33

this is heart. And I said, "You need to

16:35

call an ambulance." He was a bit

16:36

reluctant to call an ambulance for

16:37

whatever reason. And then I said, "Okay,

16:40

I was in London. He was in Manchester.

16:41

That's where I grew up. And I decided to

16:43

go and have a shower, get ready, and get

16:45

on the train." And he was ringing up his

16:48

uh neighbors who were both doctors. And

16:50

by the time I came out of the shower, I

16:52

called him back and there was no answer.

16:54

And I remember my heart just thinking, I

16:56

really hope he's not had a cardiac

16:57

arrest. It just I thought, "No, this is

16:58

this can't be can't be what's going on."

17:00

Kept ringing ringing. And eventually

17:04

um the neighbor who was a doctor who

17:05

knew me, she answered the phone and

17:07

said, "Seeam, your dad's had a cardiac

17:08

arrest. We're doing CPR in him." I know

17:11

from national data and I've even written

17:12

about this in the BMJ. The average

17:14

response time, Steve, for for for many

17:17

many years, one of the things uh that we

17:19

do have done very well in the NHS, some

17:21

of the best in the world, is our

17:23

treatment of heart attacks and the speed

17:24

of treatment and even cardiac arrests. I

17:26

knew the average response time was 8

17:28

minutes. I said, "The ambulance will be

17:29

here in 8 minutes." because of the

17:31

timing almost certainly he will have

17:32

what we call a shockable rhythm as in um

17:35

the heart is probably in in a having a

17:38

electrical disturbance and we'll be able

17:40

to shock him out of it more than 50%

17:42

chance he will survive this is I'm

17:44

thinking already like this

17:47

10 minutes pass by hasn't shown 20

17:49

minutes nothing 30 minutes um they get

17:53

there and I FaceTime them they attach a

17:55

cardiac monitor it's a flatline he's

17:58

gone nothing to be done and of course

17:59

that was, you know, at that stage I was

18:02

obviously devastated and I I wrote in

18:05

the eye newspaper about how I I screamed

18:06

louder than ever screamed in my life.

18:09

But to come back to that situation first

18:12

and foremost so the first thing is if

18:14

the ambulance had turned up on time

18:15

almost certainly it's likely he would

18:17

have survived. That's the first thing.

18:18

But the second thing in my mind is why

18:20

has he had a cardiac arrest in the first

18:21

place? He was a really healthy guy in in

18:23

my family as in on his side there was no

18:26

family history of heart disease. He had

18:28

high blood pressure. Okay. But that was

18:30

about it, right? And he was otherwise

18:31

pretty good.

18:33

And when the postmortm came back, there

18:36

were three major arteries of the heart.

18:38

Two of them had severe blockages. And I

18:41

thought, "This is odd because I knew his

18:44

lifestyle. This is my area of interest

18:46

and expertise, heart disease

18:48

specifically, how it progresses, how you

18:49

can reverse it, all that stuff, what

18:51

medications can do, what they can't do

18:53

as well." And I thought to myself, hold

18:56

on, I knew his scan from a few years

18:58

earlier. Something has happened in the

19:00

last 2 or 3 years that has caused a

19:03

rapid acceleration in the disease of the

19:08

heart, right? In terms of the blockages

19:09

getting very rapidly, you know, um, uh,

19:12

progressing. And I thought, this doesn't

19:14

this this doesn't make sense. And I

19:16

thought to myself, was he just severely

19:18

stressed? Like stress can do it. Very

19:20

severe stress can do it. He'd lost

19:23

obviously my mom a couple of years

19:25

earlier, but he was a very resilient

19:27

guy. He was a vice president of the

19:28

British Medical Association. He was

19:29

still involved in medical politics

19:30

during COVID. He was one of the faces on

19:32

the BBC during the COVID pandemic

19:34

talking about, you know, how we should

19:36

manage this, etc. So, he was still very

19:38

mentally active. He wasn't a recluse

19:39

sitting in the corner, you know, he was

19:41

still social. So, doesn't really fit.

19:44

And then a publication appeared in the

19:47

journal circulation which is considered

19:49

a ver the premier cardiology journal and

19:53

a cardiologist called Steven Gundry who

19:56

you may have heard of. He's done a lot

19:57

of work in lifestyle and lectins and

19:58

stuff. So he he had and he actually had

20:00

a very senior role in the American Heart

20:02

Association very eminent published

20:03

cardiologist. he published um an

20:06

abstract where he had looked at several

20:08

hundred of of his patients who had taken

20:12

the COVID vaccines specifically mRNA

20:15

vaccines so either Fiser or Madna two

20:17

doses and what had happened was within 8

20:21

weeks of taking those jabs

20:24

their baseline risk went from 11% chance

20:27

of a heart attack in 5 years to 25%

20:31

chance just within 8 weeks that is a

20:34

huge jump. Like just to give you

20:36

perspective, Steve, if I today decided

20:39

that I was gonna just consume junk food,

20:42

all my meals, right? I was going to

20:45

smoke 20 cigarettes a day. I was going

20:47

to stop exercising. I wouldn't get

20:49

anywhere close to increasing my risk

20:50

within 8 weeks. And the mechanism was

20:53

through inflammation. And I thought, ah,

20:55

I know heart disease is a chronic

20:57

inflammatory condition. Think it's a

20:58

lifestyle. So if this is even partially

21:01

true, it means that the co vaccines may

21:03

be causing inflammation around the heart

21:05

and that means that many people are

21:07

going to have an acceleration in heart

21:09

disease and that may explain what

21:11

happened to my dad. But that was only

21:12

one bit of data. Of course, a good

21:14

scientist knows, okay, it gets you

21:16

thinking. It's a hypothesis potentially.

21:18

Obviously, there's some data, but it's

21:20

not enough to be make it more than

21:22

likely at this stage. And within two

21:25

weeks, call it providence or whatever

21:27

else, um I got a phone call from uh

21:30

somebody who I know, I consider him uh

21:33

you know, one of the smartest

21:35

cardiologists, very high integrity from

21:37

very prestigious institution.

21:39

And he called me up and he said, "Asim,

21:42

I've got something to tell you. I'm very

21:43

upset." He said, "A group of researchers

21:46

that I'm linked to in this institution

21:49

um had accidentally found, and this is

21:52

nothing to do with blood tests or

21:53

cardiac risk, through an a very

21:55

high-tech imaging of the heart modality

21:57

they were using, that there was

22:00

inflammation of the arteries, which

22:02

would cause obviously potentially heart

22:03

attacks. That was there in the

22:05

vaccinated, but not there in the

22:07

unvaccinated. The lead researcher sat

22:10

the team down and he said, "Guys, I just

22:14

want to make this clear. We are not

22:15

going to explore these findings any

22:17

further because it may affect our

22:20

funding from the drug industry."

22:22

[snorts] Now, obviously, people hearing

22:24

this are going to think, "Wow, that is

22:26

sounds corrupt. Um, this is something

22:28

that has been a big problem within

22:30

medicine for a very, very long time,

22:31

which isn't getting an airing." But it

22:33

didn't surprise me of that. But it was

22:35

still quite shocking. But for me at that

22:36

stage, Steve, it was okay, at the very

22:39

least, I should ask the question. And I

22:42

was nervous about it. I had been someone

22:44

took the jab myself. I went to Good

22:47

Morning Britain and very early on when

22:48

they were just offering it to

22:50

high-risisk people to say that um I

22:53

think that this is probably safe and

22:55

people shouldn't be worried. And this

22:57

was again specifically to address people

22:59

from black and ethnic minority

23:01

communities because there was a lower

23:02

uptake amongst those communities. Partly

23:03

also because they often amongst some of

23:06

the most marginal is marginalized

23:08

members of society and they have less

23:10

trust in authority in government. So you

23:11

can see how the psychology plays in

23:13

there and I convinced a friend of mine

23:15

who's a film director Ginda Chada who

23:17

directed um Bendit like Beckham. You

23:19

might know some of her movies and we

23:20

went on together in Good Morning Britain

23:22

to say listen I think this and you know

23:23

this is fine. So I had all of that and

23:25

so I was in many ways I was

23:26

indoctrinated and and there is you know

23:29

and people don't like to admit they're

23:30

wrong or think they've got it wrong. So

23:32

for me to start turning or changing my

23:35

mind or asking the question um is not

23:37

necessarily an easy thing to do but I've

23:39

done that throughout my career. So you

23:40

know if anyone has the um character you

23:45

I know myself to be able to say listen

23:46

okay there's new evidence here or

23:47

whatever else I felt I could do that but

23:49

I was still nervous and I thought you

23:51

know this is not something that I think

23:53

the BBC are going to pick up on. I had a

23:56

good rapport um with GB News at that

23:58

point um and I still do and one of the

24:02

presenters Alexander Phillips was a

24:03

friend of mine and I called her up and

24:04

said listen um you know we were doing

24:07

she wanted me on anyway I was doing

24:09

regular health slots every few weeks I

24:11

said what I see what do you want to talk

24:12

about next week and I said I think let's

24:14

talk about this said okay go on GB news

24:18

I say you know the vaccine committee of

24:21

the country should look into this this

24:22

is what I know this is what I found

24:23

whistleblower all this stuff I my dad

24:25

died as well and this may be the reason

24:28

for his death. And I just asked that

24:30

question and of course I didn't expect

24:32

it to go viral. You know, it got

24:34

millions of views and it was getting

24:35

reach all over America and that kind of

24:37

thing. Strangely, by this point, Steve,

24:41

I don't know if you remember, but

24:42

Omicron wave had started and um we were

24:45

getting reports from South Africa, which

24:47

was really reassuring that from the

24:49

doctor that discovered it that this

24:51

doesn't seem to be more harmful than the

24:53

flu. Now, great. Okay, it's mutated.

24:55

It's a different strain. This is really

24:56

great news. And then Saja Javad around

24:59

the same time the secretary for health

25:01

had come out in parliament and said we

25:03

are now going to pass through

25:05

legislation that the COVID vaccine needs

25:08

to be mandated for healthcare workers

25:11

despite the fact that the British

25:12

Medical Association Medical Colleges

25:14

we've never in our country one thing

25:15

that's really good especially within the

25:17

medical establishment we don't believe

25:18

in mandating any drug. They do this in

25:20

America. They've never done this here.

25:22

And although they weren't very vocal

25:23

about it, they kind of were a bit

25:24

relatively quiet. They weren't

25:26

supporting it openly saying that we

25:27

should mandate. This was coming from

25:28

this political decision. I thought this

25:30

is very odd. At a stage now where we're

25:32

thinking there's serious harm, we know

25:34

it's not. And by that stage, 2021,

25:36

November, most people were understanding

25:38

now it wasn't stopping infection, right,

25:40

for most people, right? So I said, this

25:42

doesn't make any sense. So I then

25:44

started campaigning on this issue. And I

25:46

started campaigning on this issue. was

25:48

able to get into the mainstream news on

25:49

this particular issue because around

25:51

that time um the I newspaper had

25:55

published an investigation into the

25:57

delay that led to my dad's death of the

25:59

ambulance service right and I had also

26:03

got privy to knowledge in that you know

26:05

in that story that the deputy chief

26:08

nurse of NHS singing had called me up

26:09

and said there had been basically a

26:11

cover up by the government and the

26:12

department of health to stop people

26:14

knowing for months there were ambulance

26:15

delays doctors and members of public I

26:17

thought this is unacceptable. So I

26:18

exposed this. It became a massive story

26:20

BBC news and when the BBC presenter

26:22

saying Dr. Mahhatra what's going on

26:24

wrong with the NHS why is it failing why

26:26

is this happening? I said it's

26:28

multiffactorial but I said one of the

26:29

most important reasons is you failed we

26:32

failed for years to address the root

26:34

cause of what's driving stress on the

26:35

system taking on the excesses of big

26:38

food and big farmer. But I said, "Right

26:40

now, we've got um 80 to, you know,

26:44

100,000 NHS staff who are refusing to

26:46

have the COVID vaccine. This would be a

26:49

crisis. They're going to lose their

26:50

jobs. This is this mandate needs to be

26:52

overturned. It's not scientific. It's

26:53

not ethical." So, I got I got that into

26:55

the mainstream and ultimately we end up

26:57

overturning, right? The the the um the

26:59

mandate. Um but there was a backlash,

27:03

Steve, behind the scenes. And I've not

27:05

been public about this before, so I'm

27:06

going to tell you this for the first

27:07

time because I think it's time I I tell

27:09

this story. Shortly after me going on GB

27:12

News as a doctor who'd had the vaccine,

27:15

had been on Good Morning Britain to say

27:16

it was likely safe and effective to then

27:19

talking about we should maybe look into

27:21

this and maybe pause the situation

27:22

because of um these heart issues that

27:25

need to be investigated. I received an

27:28

email from the Royal College of

27:30

Physicians, this is the oldest medical

27:32

institution in the world, saying, "Dr.

27:34

Dr. Mahhatra um we have received a

27:36

number of anonymous complaints from

27:39

other fellows

27:41

that you uh in reference to my GB news

27:43

interview that you are spreading antiax

27:47

misinformation

27:49

and you've got four weeks to respond to

27:51

this and they were saying all the

27:52

different sanctions that could happen

27:54

because of me doing this right Steve at

27:57

that point I thought if we are going to

27:59

get a pause on this vaccine and really

28:01

investigate it it's because of such an

28:03

IND indoctrination because so many

28:05

people, billions of people around the

28:06

world have taken this and therefore the

28:09

the the battle to expose it is going to

28:12

be harder than anything I've ever done.

28:14

The only way in my only chance is to get

28:17

it published in a peer-reviewed journal

28:18

and then to get it into the news. I

28:21

spent 9 months at that stage literally

28:23

eating, breathing, sleeping, speaking to

28:25

two FISA whistleblowers, speaking to

28:28

eminent scientists in expertise I didn't

28:30

have around immunology and vaccine

28:31

development. Of course, I had the

28:34

understanding of cardiology, you know,

28:35

better than anybody in in this

28:37

particular field, in this particular

28:38

area. And when I did that research and

28:42

looked at it, I first of all concluded

28:45

that there absolutely needs to be a

28:47

suspension of the vaccine because um

28:50

what happened by the summer of 2022 and

28:53

this is actually the the most crucial

28:55

and important piece of data on its own

28:57

which should have been enough to suspend

28:58

it and actually suggest that it probably

29:01

shouldn't have been rolled out in the

29:02

first place is that those trials that

29:05

were done by Fiser and Madna which led

29:08

to the all the media reports 95 100%

29:11

effective um you know the approval by

29:14

the regulator the roll out the coercion

29:16

the mandates they were reanalyzed by

29:19

some very eminent scientists including

29:21

the associate editor the BMJ the one of

29:24

the world's top epidemiologists and they

29:26

published in the journal vaccine which

29:28

is the premier journal for vaccines and

29:30

they were able to get new data that was

29:33

made available on health Canada's

29:34

website and the FDA in America's website

29:37

and what they did in their reanalysis of

29:38

the original highquality clinical trials

29:41

is they found Steve you were more likely

29:45

to suffer serious harm from taking the

29:47

vaccine at a rate of 1 in 800 that meant

29:50

hospitalization disability or a

29:52

life-changing event than you were to be

29:54

hospitalized with COVID and this is

29:56

during the early phase right this is

29:58

during the most most lethal strain

30:00

for all age groups

30:01

yes well absolutely well they put all

30:03

age groups together so on average in all

30:06

age groups that's a very good question

30:08

um but what's missing is that actually

30:10

okay is there a benefit that's greater

30:13

than harm in certain age groups but we

30:15

can indirectly answer that in a second

30:17

so that was the original trial so on

30:18

average it was more harmful than

30:20

beneficial okay but even before talking

30:23

about all age groups Steve a 1 in 800

30:26

harm rate for a vaccine is completely

30:29

unacceptable in the sense that we have

30:31

pulled other vaccines in the past for

30:34

much less harm the swine flu vaccine was

30:36

suspended globally because it was found

30:39

to cause Gillian Barry syndrome a

30:41

debilitating neurological condition in

30:43

one in a 100,000 people. Rotovirus

30:46

vaccine was pulled in 1999 because it

30:48

was found to cause a form of bowel

30:49

obstruction in children at 1 in 10,000.

30:52

So you've already got a harm rate of

30:54

1800 irrespective of right. So that

30:56

first and foremost should be a red flag

30:58

to say hold on this is this is too much.

31:00

When they say harm rate how do they

31:01

what's the the range of definitions of

31:04

harm? Well, in this one they categorize

31:06

serious harm as it caused you to be

31:08

hospitalized.

31:09

Yeah.

31:10

It caused a disability.

31:12

Okay.

31:12

Or something that was life-changing.

31:13

Now, of course, that can incorporate

31:15

lots of different things, but of those,

31:17

and I spoke to the lead researcher I

31:18

know work with him on other things, um

31:21

40% of those serious harms were actually

31:23

related to clotting disorders like lung

31:25

clots, heart attacks, etc.

31:27

I want to make sure I'm super clear here

31:28

because I don't understand um the the

31:31

data you're citing. So you're saying

31:32

that they found that one in 800 people

31:34

would have serious harm or harm.

31:37

Uh serious harm.

31:38

Serious harm. Serious.

31:39

One in 800 people had

31:40

in the trials. One in 800. Yeah. One in

31:42

800. Serious harm. Right now just to

31:45

give you perspective so you can balance

31:47

it out just because this is important.

31:48

It's a question you've asked is really

31:50

important. um we didn't have any good

31:53

real world data at that point on

31:57

can we separate vaccinated from

31:59

unvaccinated to look at what the

32:02

hospitalization rate would be for COVID

32:04

for example in people who took the

32:06

vaccine versus the people that didn't

32:07

according to age group that data in the

32:10

whole world the only country to get make

32:12

that data available was the UK and they

32:14

did that in the beginning of uh 2023 so

32:18

January last year and What did that

32:21

show? After two doses of the FISA

32:23

vaccine, Steve, if you were over 70, so

32:26

this is the highest risk group, you had

32:28

to vaccinate 2,500 people to prevent one

32:34

person being hospitalized with COVID.

32:37

And this was with the different strain,

32:38

the omnicom strain, because the original

32:40

strain was

32:41

Yeah, it was they didn't Yeah, it was.

32:43

You're right. It was with the Omicron

32:44

strain. So that was still, but it gave

32:46

us a ballpark figure that even that. So

32:47

it's like it's like so say a patient

32:49

comes to me and says doc what are the

32:51

benefits of this drug as a prevention

32:52

whatever else and I say to them well if

32:54

you take this there's a one in 2,500

32:57

chance it will help you prevent you

32:58

being hospitalized I'll be honest with

33:00

you Steve I mean in medicine in all the

33:03

drugs I've used and all the data I know

33:05

about different medications and heart

33:07

disease etc that figure I mean it's a

33:10

very serious issue but that figure is a

33:11

joke I mean there's nothing of that of

33:13

such poor and then when you get under

33:15

the a when you get to people under the

33:16

age of say uh 50 you're talking about

33:19

having to vaccinate maybe several

33:21

hundred thousand to prevent one one

33:23

is that relevant for that the first

33:24

strain of co as well because or do we

33:26

not have the data on that because

33:28

we don't have that data on that we do in

33:30

my paper which I published actually we

33:31

did have some data on delta and if I

33:33

remember correctly the data on the over

33:35

70s the there's also problems with this

33:38

a little bit because it's not corrected

33:39

for other factors such as social

33:42

economics etc risk factors of you that

33:44

[snorts] might make people more

33:45

vulnerable So, if I remember correctly

33:47

from that paper, if you're over 70, that

33:49

was about 1 in 250. Delta was the worst

33:51

strain, actually. So, about 1 in 250.

33:53

Okay.

33:54

On the British Heart Foundation website,

33:55

I'm sure you've read this. Um,

33:57

just to read out what they say on there,

34:00

it says that up to one in 10,000 people

34:03

with the FISA vaccine might experience

34:06

are at risk of mo myocarditis or

34:09

pericarditis.

34:11

Have I pronounced that correctly? Yeah.

34:12

Up to one in 10,000 people for the Madna

34:15

vaccine. And uh it's not possible to

34:17

estimate other vaccines because they're

34:18

not frequently used in the UK. All three

34:21

of these COVID 19 vaccines are mRNA

34:24

vaccines designed to target the omnicon

34:26

omnicom strain. And at the top of this

34:28

it says the risk of myocarditis or

34:30

pericarditis after covid-19 vaccine is

34:32

very low. How do you respond to that? Do

34:35

you think you agree with that?

34:36

No, I don't agree with it at all. I

34:38

think there are a number of layers to

34:39

respond to this. I think the first thing

34:40

to say Steve is the British Art

34:43

Foundation with the greatest respect to

34:44

them and they do a lot of good work

34:45

overall is still part of the so-called

34:48

establishment which has been blinded for

34:51

years to actually even address so many

34:54

issues on health when it doesn't uh suit

34:58

the interests of big farmer and and and

35:00

I can say that categorically because I

35:02

know one of the the the chief advisor uh

35:06

uh to heart disease um for the British

35:08

Heart Foundation with the greatest

35:10

respect to him is a guy called professor

35:11

Rory Collins um University of Oxford and

35:15

they have said similar things when it

35:17

comes to statin drugs which we'll talk

35:18

about later um but that person the

35:21

people who advise them are people who

35:22

are heavily funded and linked to pharma

35:25

taking their institutions taking

35:26

hundreds of millions for example so

35:28

there's a huge bias there to start with

35:30

that's the first thing but it for me

35:33

what the British Heart Foundation are

35:34

not doing is actually countering and I

35:37

would love them to counter that cuz I'm

35:39

very open for the debate here is that

35:41

you've got a reanalysis of and they know

35:43

this the best way of determining serious

35:46

harm from any drug is actually looking

35:49

one of the best ways is the highest

35:50

quality level of evidence which is the

35:52

randomized control trials which is what

35:54

led to the approval when you've got an

35:56

independent reanalysis in a

35:57

peer-reviewed journal saying more harm

35:59

than good from the beginning that in

36:01

itself and and then we look at real

36:02

world data Steve there's so many other

36:04

bits of data that they are ignoring

36:06

basically to answer your question

36:08

they're ignoring ing lots of other data

36:10

which is very clear whether it's autopsy

36:12

data whether it's other studies that

36:13

came out of Israel that showed for

36:15

example this was published in a journal

36:17

called nature scientific reports and

36:19

again they ignore this they don't talk

36:20

about it so it's it's like hold on guys

36:22

this is you're ignoring you're not even

36:24

mentioning this data they showed and

36:26

this is really most disturbing in 2021

36:30

there was a 25% increase in heart

36:32

attacks and or cardiac arrests in people

36:36

aged between 16 and 39 which was

36:40

associated with the COVID vaccine but

36:42

not associated with COVID.

36:45

How do they how do they tease out I was

36:47

thinking about um you know the increase

36:49

in heart related conditions around the

36:52

pandemic and following the pandemic and

36:54

in in much of your work and I think in

36:55

this book I can't actually pronounce the

36:57

word pop diet. Pop diet,

36:58

the Popey diet. You talk about how these

37:00

other sort of lifestyle factors like

37:02

community, friendships, relationships,

37:04

stress, mental health, being sedentary,

37:06

sedentary, all of these things can

37:08

contribute to heart problems. So when I

37:10

think about the pandemic, I go, people

37:12

weren't seeing their friends. We were

37:13

stressed more than ever. People were

37:14

losing their jobs. They were furoughed.

37:16

Um they had mental health, you know, we

37:18

saw the mental health uh stats explode.

37:21

Yeah.

37:21

Um

37:22

all the factors there that are linked to

37:25

heart disease. So, how do we know that

37:27

it it wasn't th those factors of the

37:29

pandemic that caused an increase in

37:31

heart related issues and how do how can

37:33

we tease that out from the the vaccine?

37:35

Really good question. Um because

37:37

remember I also said that early on

37:39

before I realized the vaccine might be

37:40

playing a role. I actually thought that

37:42

was that was the most likely explanation

37:44

of the increased heart attacks, lockdown

37:45

stress, poor diet, etc.

37:47

You'd expect to see an increase in

37:48

you would you would and I think it has

37:49

played a role, Steve, for sure it has

37:51

played a role. But then when you look at

37:53

the quality of data to say how much of a

37:55

role that's played that it it's on a

38:00

different level when you look at the

38:02

vaccine when you look at the plausible

38:04

mechanism you look at the types of

38:05

people that are dying young people and

38:07

stuff like that it doesn't fit. Um I'll

38:10

be honest my personal view it is a

38:11

primary driver without any shadow of a

38:13

doubt in my mind and a personal view of

38:14

the excess deaths. As a scientist, I'll

38:17

say it's a likely significant

38:18

contributing factor, but probably the

38:20

most likely because another aspect to

38:24

all of this is what we call

38:24

pharmarmaccovigilance reports. So these

38:27

are reports that are done by members of

38:30

the public when they they have an

38:32

adverse reaction to any drug and it's

38:34

not easy to fill in. You fill in these

38:35

what we call yellow card scheme. You can

38:37

get it online and you send it off to

38:39

your doctor or to you know the

38:40

regulator. those reports and I've

38:43

throughout my career I've never seen the

38:45

extent like for example I'll give you an

38:47

example um after 9.7 million doses of

38:50

the Astrogenica vaccine which was

38:52

ultimately pulled right astrogenica of

38:54

course was also one of the co vaccines

38:56

there were 800,000 in this country

38:58

800,000 yellow card reports now some of

39:00

them um are not going to be serious

39:02

serious but people don't fill in a

39:05

yellow card report if you've had a bit

39:06

of a fever after having a vaccine

39:08

they've felt quite ill ill enough to

39:10

that's already and then within at it's

39:12

estimated maybe one in five of those

39:14

from other data would suggest serious

39:16

harm. So other data from reporting and

39:19

with the so with about I think 30

39:21

million doses if it was probably about

39:22

30 million doses of FISA we had about

39:25

500,000 yellow card reports in this

39:26

country right which is still a lot you

39:28

know that's one in 60 yellow card

39:30

reports now they're all not going to be

39:31

super serious hospitalization death

39:33

whatever else um but when you put all of

39:36

the data together Steve that it paints a

39:39

picture that makes it look as clear as a

39:42

day that anybody doubting you know it

39:46

should the the evidence should be this

39:48

is the primary cause of the excess

39:50

deaths until proven otherwise. That's

39:51

the level of evidence, Steve, but it's

39:53

just being ignored. It's being ignored

39:55

and I can talk about why it's being

39:56

ignored.

39:57

One of the things I've been sort of gaps

40:00

in my head that I've been keen to fill

40:01

is do you believe that if we hadn't have

40:06

introduced the vaccine, more or less

40:08

people would have survived CO? Because

40:10

I've got close friends of mine that got

40:11

COVID and I watched them go from very

40:14

healthy looking people to basically

40:15

skeletons. Good friend actually good

40:16

friend one of the CEOs of my company his

40:18

dad went from being a very healthy man

40:20

to being basically looking like a

40:22

skeleton and almost died. And then I've

40:24

got I know of other people that did die.

40:26

So I think in the grand scheme of things

40:28

when we think about um vaccines was it a

40:31

net positive that we had a vaccine?

40:34

There is from everything I know now I've

40:37

slowly and reluctantly come to the

40:40

conclusion that the COVID vaccine

40:43

introduction has had a catastrophic

40:47

overall net negative effect on the

40:51

population and society. And one of the

40:54

re and and let me just caveat this

40:56

because you've mentioned the fact that

40:58

people suffered from COVID and I'm not

41:00

denying that. I've got patients, Steve,

41:02

that I see that have had long CO that

41:04

weren't vaccinated, okay? And have

41:06

suffered quite badly.

41:08

Most of the most serious aspects of

41:10

COVID happened early on in 2020 and

41:15

predominantly affected the elderly.

41:16

We've got all of that data now that's

41:18

been reanalyzed by one of the world's

41:19

top scientists. And even looking back

41:22

now, essentially if you were under 70,

41:25

even from the beginning, your risk of

41:28

serious harm from COVID is in the

41:31

ballpark figure of the flu, right? And

41:33

even I actually was wrong. I wrote an

41:35

article in European scientist in Mar in

41:39

April 2020 because I actually initially

41:42

started making a lot of noise about why

41:44

we not talking about lifestyle with

41:45

COVID to help people mitigate, you know,

41:47

the immune improve their immune system.

41:49

And I said, you know, talking to um a

41:52

friend of mine who works in the busiest

41:54

DR in America in in New York who I've

41:56

done work with and he said, it seemed

41:58

this is I've never seen anything like

41:59

this is devastating. Some of my

42:00

colleagues are dying. So I have no doubt

42:02

that at the very beginning in the early

42:04

strain of the virus, it was really bad

42:06

especially for vulnerable people, people

42:08

with obesity, etc.

42:09

Cuz you know I I I remember getting CO I

42:11

was actually used to live on the top

42:12

floor of this building and I remember

42:14

I've never experienced anything quite as

42:16

bizarre as the symptom set that I had

42:18

when I got CO. The fact that at 3:00

42:19

a.m. in the morning, I don't take

42:20

medicine. So, fact [snorts] at 3:00 a.m.

42:22

in the morning, I'm lying flat on my

42:24

floor ordering ibuprofen on Uber Eats

42:26

because my back I just had the most

42:29

bizarre like back pain. And so, I was

42:32

having to like lie flat on the floor cuz

42:34

I couldn't even lie in bed. It was so

42:35

bad.

42:36

Just this weird set of symptoms that I'd

42:38

never had before. My my partner, she

42:39

lost her smell and taste. And

42:41

it was so unusual. It was so unusual. Um

42:44

there's not been a time in my lifetime

42:45

that people have lost their smell and

42:46

taste on mass. So when I when it when

42:49

you hear it compared to the flu, you go,

42:51

"This was not the flu. This is something

42:52

different."

42:53

Yes. No, the symptoms are very

42:54

different. And I think now it's accepted

42:57

um that um and we won't go into a lot of

42:59

detail, but I think one of the reasons

43:01

as well, it was human engineered. You

43:02

know, almost certainly the evidence

43:03

points to it being a lab leak, right? So

43:05

it had a very You're right. It was very

43:07

different to any other virus

43:08

that used to be a conspiracy theory.

43:11

I know, right?

43:11

Now it's not a conspiracy theory.

43:13

[laughter]

43:13

I know. I know. Exactly.

43:14

It's funny. Yeah. when you heard about

43:15

that that that lab in Wuhan that were

43:17

messing around with viruses [laughter]

43:18

and then we we decided to put the blame

43:20

on like a market store but I think now

43:21

the general consensus is that it

43:23

probably came from that lab in Wuhan.

43:25

Yeah. Absolutely. Yeah. Absolutely. And

43:26

I've spoken to in fact I I spent time

43:28

with actually the scientists that first

43:29

went public with it who identified it um

43:32

guy in America sorry in Australia. Um so

43:35

yeah that came from lab but so I think

43:38

it had these different strange things

43:39

loss of smell etc. But in terms of

43:40

serious illness, um it was there at the

43:43

beginning. Now when you when you look

43:44

back, I think essentially there were

43:47

vulnerable elderly but uh you know who

43:49

who suffered um especially people in

43:51

nursing homes with a lot of deaths

43:53

there. But there's so many other

43:55

components to this. So one is did we

43:57

institute the correct treatments? A lot

43:59

of people were killed because they

44:01

weren't managed properly in ITU, you

44:02

know, in terms of putting people

44:04

incubating them, putting on respirators

44:06

when they didn't need it. And that in

44:07

itself has a risk. um some of the wrong

44:09

treatments were given. There were other

44:11

treatments now that we look back that

44:13

probably would have been helpful things

44:14

like ivormectin which I know has been a

44:16

bit controversial but um was very it's a

44:18

very safer than paracetamol right so

44:20

first do no harm okay um but it may have

44:22

done some good and a lot of people and

44:24

doctors around the world that used it in

44:25

several thousand there's a doctor in

44:27

South Africa that used it in 14,000

44:28

patients including many elderly not a

44:30

single one died from co and this is

44:32

early on so all these things that we

44:34

missed we miss a lifestyle intervention

44:36

so all those things are there but by the

44:38

time you get to the end of 2020 the

44:39

beginning of 2021 on there are so many

44:41

things that happened Steve that you have

44:42

to think about before you introduce a

44:44

vaccine. One is what is the state of the

44:45

virus right now um and it had already

44:48

mutated to some degree and become less

44:50

lethal. There is natural immunity which

44:52

we know is very powerful right um and

44:56

but the issue with the vaccine is and

44:59

certainly it was probably there from the

45:01

beginning. We know that when one looks

45:03

at the original trials there was a and

45:06

this is what the drug companies have

45:07

doing for a long long period of time.

45:09

They will mislead people using

45:11

statistics about the benefits. So use

45:14

something called relative risk

45:15

reduction. Let me just explain this

45:17

because you can apply this to statins as

45:18

well. Is they presented the benefit as a

45:22

95% protection against infection.

45:24

Remember that figure 95% right? And it

45:27

was what we call relative risk

45:28

reduction. So

45:31

if you've got for example two groups in

45:33

a trial, say 100 in one trial in in in

45:36

in one group and 100 in another and

45:38

let's just say let's give you an example

45:40

of statins and you're you're following

45:42

them up over 5 years to look at a drug

45:44

to see if it benefits them in preventing

45:45

having a heart attack.

45:48

In one group they get the dummy pill

45:50

um and you follow them up over five

45:52

years. And in the people that got the

45:53

dummy pill, in fact you didn't do

45:54

anything different, two of them suffered

45:56

a heart attack.

45:58

In the other group, the other hundred

46:00

people that were followed up over five

46:01

years who got the pill, the the drug,

46:04

right, only one suffered a heart attack.

46:07

So you've reduced the heart attack risk

46:09

by 50%. Right? Two to one, right? 50%.

46:13

But you've only prevented one heart

46:15

attack. You've treated 100 people, but

46:16

you've prevented one heart attack out of

46:18

treating 100. Yeah. Does that make

46:19

sense? So that's a 1% absolute benefit.

46:22

In other words, when you explain that to

46:24

a patient, when I ask when I have engage

46:26

in sort of what we call informed

46:28

consent, shared decision-making, when

46:30

they ask me about drug, I'll say, "This

46:31

gives you a 1% chance if you take this

46:34

drug religiously of preventing a heart

46:35

attack." Now, you you apply that to the

46:37

original COVID vaccine trials, which by

46:39

the way have so many other problems with

46:41

them because even those trials were

46:44

conducted and analyzed and designed by

46:46

the drug industry. I mean this is one of

46:47

the biggest myths that needs to be

46:49

busted Steve out there for most doctors

46:51

um as well as members of the public

46:52

medical knowledge is under commercial

46:54

control but most people don't know that.

46:57

So what happens is they did the trial

46:59

but let's just talk about what the

47:00

results their their results showed us a

47:03

95% relative risk reduction against

47:05

infection. They didn't show any

47:07

reduction in uh co death by the way in

47:09

that trial right they just said prevent

47:11

from infection but we then presume it

47:13

may then prevent right uh reduce death

47:14

rates absolute risk reduction from

47:17

infection at the beginning was one was

47:21

0.84%.

47:24

One in 119

47:27

so that's how many people you need to

47:28

vaccinate to prevent one infection which

47:30

actually people were not told that. So

47:32

imagine you're thinking, should I take

47:33

this vaccine? Say, well Steve, there's a

47:35

one less than 1% chance that it's going

47:37

to prevent you getting infected. People

47:38

weren't told that.

47:39

But but that it then reduce my chance of

47:42

getting seriously ill. Right.

47:43

No, but we've then talked about that,

47:44

haven't we? Like as in when you look at

47:45

the data certainly beginning of 2023

47:48

that was looking over the previous year,

47:49

2022, you have to vaccinate 2,500 people

47:53

to prevent one person getting seriously

47:55

ill with COVID, right? If you're over 70

47:57

with the second with the other strain,

47:58

but

47:58

with the other strain and it may have

47:59

been better, Steve, you're right. It may

48:01

it probably was better but it still

48:02

numbers are still much smaller than what

48:04

people were led to believe. And and by

48:05

the way Steve the narrative at the

48:08

beginning they kept changing the

48:09

goalpost. Remember it wasn't about

48:10

preventing serious illness and death. It

48:11

was all about preventing infection. You

48:13

are not in America you've seen it all

48:15

over CNN Rachel Madau and she's saying

48:18

it so passionately. If you take this

48:21

vaccine you are not going to get COVID

48:23

and calling anyone who who questions it

48:26

being a science deny. I mean Jesus

48:28

Christ. So my last question on that

48:30

before I I say I was going to say is um

48:32

do you think there would have been less

48:36

deaths overall

48:38

if we hadn't have had a vaccine?

48:40

Yes.

48:41

You think there would have been less

48:41

deaths

48:42

by now when you look at it. So I think

48:44

over time so so where we are now

48:46

start

48:48

um

48:49

so if we hadn't have introduced the

48:51

vaccine for that first strain of co you

48:52

think there'd be less deaths.

48:54

Okay. If I was to I still think that

48:59

if okay this is very nuanced but

49:02

important.

49:04

If the vaccine had only been offered to

49:09

the high-risisk people at the beginning,

49:12

say the over 70s or people with multiple

49:14

risk factors, [snorts]

49:15

I think there is a case to be made,

49:18

right? I'm going to counter that in a

49:19

minute though. But there is a case to be

49:20

made that there was overall benefit

49:23

versus harm. But there's a problem. One,

49:26

there wasn't true informed consent,

49:29

right? Because those figures, those

49:30

numbers weren't given to pay people

49:31

about the prevention of infection, etc.,

49:33

Right. Um and two, if you have an

49:37

average serious harm rate of 1 in 800,

49:39

any scientist, even regulators would

49:42

have said, "Hold on a minute. This is

49:44

way too high. This is too risky." And

49:46

this is, by the way, Steve, only the

49:47

short-term. Because remember, this

49:49

vaccine didn't go through what other

49:50

vaccines have gone through, which is 5

49:52

to 10 years of safety testing.

49:54

[snorts]

49:54

So if you throw all those caveats in and

49:57

use informed consent, I can guarantee

50:00

you with all of my knowledge, expertise,

50:03

experience with patients when you engage

50:05

in these conversations, most of those

50:07

elderly people will probably still

50:09

refused it. But but but yes, I think

50:12

there is a case to be made that the the

50:14

benefits may have outweighed the harms

50:16

in those high-risisk people at the very

50:18

beginning in the short term. Absolutely.

50:20

You know, when I think about Rachel

50:22

Maddau and what she said on TV about,

50:24

you know, that it's going to stop the

50:25

spread of infection, etc., I can have a

50:30

degree of empathy

50:31

because if that's the information you're

50:33

being fed and you are a public-f facing

50:36

broadcaster

50:37

and it's being fed to you by scientists

50:40

and it's been fed to you by the NHS and

50:42

whoever else and you know, very credible

50:44

people that you've been raised to

50:46

believe and to trust. If you're a public

50:48

facing broadcaster, what else are you

50:50

going to say? You're not going to say

50:50

the opposite. You're not you can't sit

50:52

on the fence. Your job is to broadcast.

50:54

It's the news, right? So I I have and I

50:56

think you did the same. You said earlier

50:57

you went on Good Morning Britain or

50:59

something and

50:59

and I'm not blaming Rachel Mad here. I'm

51:01

just saying that the indoctrination that

51:03

came through the mainstream media was so

51:04

strong through people like Rachel Maddo.

51:06

And why is that important, Steve? I had

51:08

a conversation with the chairman of the

51:09

British Medical Association in December

51:11

2021 when I was campaigning to overturn

51:15

vaccine mandates for healthcare workers.

51:17

He had access to Saji Javid. I had a

51:19

previous uh rapport with Matt Hanok but

51:21

he had obviously left by then. Um and I

51:24

spoke to his name Shan Nagpaul and I

51:26

explained him everything I knew about

51:27

the vaccine after looking at data at

51:30

that point. I hadn't published at this

51:31

point but I went through it in in in in

51:32

a logical way. chairman of the BMA, by

51:35

the way, not just some random person.

51:37

And he said, "Asim,

51:39

no one appears to have critically

51:41

appraised the evidence on the vaccine as

51:42

well as you have from our two-hour chat.

51:44

Most of my colleagues

51:47

who are in senior policy, you know,

51:49

medical positions, establishment

51:50

positions are getting their information

51:52

on the benefits and harms of the vaccine

51:54

from the BBC."

51:56

It's it's super difficult.

51:57

Isn't that extraordinary, though? I

51:58

think it's really difficult because if I

52:01

if you're dealing with lots of people

52:02

dying on mass and it's happened very

52:05

very quickly and people are just

52:07

dropping dead and you're seeing you know

52:09

hospitals being overrun,

52:12

you've got to

52:14

you got to tread carefully with the

52:16

information you're putting out there.

52:17

So, if the scientific information comes

52:19

in early and maybe a little bit too too

52:20

soon before it's really been vetted and

52:22

triple checked, saying one thing

52:26

and you're desperate for answers, I can

52:28

I can see why a group of people would

52:29

say, "Okay, this is the best

52:30

information." And then to go against

52:32

that information could potentially cause

52:34

tons of harm. So, I can also imagine why

52:36

a group of people would be really slow

52:38

to then change their mind away from that

52:40

because you're dealing with like

52:42

Steve, I was that person, too. So, I'm

52:43

with you on that 100%. I think where I'm

52:45

taking this is the system if it had been

52:48

more transparent early on and this is

52:51

where I've been, you know, banging my,

52:54

you know, head against a brick wall to

52:55

some degree for about a decade. If there

52:58

was more transparency in the system, we

52:59

would have had better information even

53:01

from the beginning. But that information

53:02

was kept commercially confidential

53:05

because of the system that really is

53:08

geared towards uh supporting the

53:10

interests of big farmer not in the

53:12

interest of people's health and that and

53:13

and if that and that's the key point

53:15

here right this is we're looking back

53:17

over time think how did this happen how

53:19

do we allow this to happen we need to go

53:20

deeper say how do we stop this happening

53:22

again in the future so we have better

53:23

information that's all I'm saying

53:25

do you think it is malicious at like a

53:27

government level do you think

53:28

no

53:28

you don't think it is

53:29

not at all I um know many politicians uh

53:33

very senior people um cross party uh

53:36

some of them I call my friends uh many

53:38

of them come to me for medical advice

53:40

right um in fact one of them uh you know

53:42

he lost 100 pounds on my POPY diet with

53:44

Tom Watson the deputy leader of the

53:46

Labour party right

53:47

so um and and and these genuinely by and

53:50

large are decent people that want to do

53:51

the right thing but they are also fed

53:54

you know misinformation by lobbyists

53:56

they take as expert opinion or

53:58

information stuff that has been created

54:01

for the purposes of the interest of big

54:03

food or big farmer. you know, when I um

54:05

campaigned on you getting the sugar tax

54:07

introduced, you know, I wrote articles

54:09

in the BMJ and I started ranging every

54:11

newspaper and I remember I thought we're

54:13

going to win this because the front page

54:14

of the Daily Mail was sugar is a new

54:16

tobacco and that put pressure on the

54:19

then secretary Jeremy Hunt because the

54:20

male then decided they were going to go

54:22

right and of course you know that

54:24

government conservative government are

54:25

particularly you know influenced by what

54:27

the Daily Mail writes because they

54:29

traditionally one of their supporters

54:31

but there was a story around that time

54:34

where they exposed and it was on the

54:35

front page that government ministers in

54:37

terms of obesity strategy, how do we

54:40

solve the obesity epidemic had had 99

54:43

meetings with representatives of the

54:46

food industry and not a single meeting

54:48

with a public health doctor for example.

54:51

So I know how that hap that obviously

54:53

that the system should be more

54:54

transparent to make sure that they

54:56

understand that those those politicians

54:57

but many of them um were shocked you

55:00

know when I when I told them this

55:01

information they trust me um one of them

55:04

you know was a very a former government

55:06

minister who who said it seem you know

55:08

this is she's shocked by it but like now

55:10

understands it they they were captured

55:12

as well but we were all we were in a

55:13

state of fear Steve as well we let's not

55:15

underestimate that at the very beginning

55:16

we were all scared we didn't know what

55:17

we were dealing with and of course we

55:18

have to have empathy for ourselves when

55:20

you're in a state of fear right for all

55:23

be you know I think it was a big error

55:26

to some degree and everybody was scared

55:28

and I don't think it was malicious to

55:29

create that fear initially

55:31

um it stops us you know being being able

55:35

psychologically it inhibits your ability

55:37

to engage in critical thinking and all

55:39

of us were in that position is there a

55:41

risk now that if there was a deadly vac

55:44

deadly virus that broke out across the

55:45

world people are so scared of vaccines

55:48

now that they would not go and get it

55:52

because there something I was saying to

55:54

my friend the other day I was like we've

55:55

gotten to a point now where I think so

55:56

many people are skeptical about vaccines

55:59

that if if something does come from

56:00

another lab somewhere and it is really

56:03

fatal and the government stand on that

56:05

podium again and say hands face or

56:08

whatever it was that slogan and they say

56:10

um we need you to all go get this

56:12

vaccine

56:14

who's going to go get it

56:15

yeah no I I agree I don't like that

56:18

situation I don't want us to be in this

56:19

situ situation. But you're right, there

56:21

is

56:21

and there will be another pandemic.

56:22

There there is a big risk and um the way

56:25

around that is and this is what we're

56:28

taught as doctors,

56:29

right? It's one of the things that is

56:30

ingrained into us as a medical school um

56:34

is that when you make a mistake, you

56:37

tell the patient.

56:39

Patients are very forgiving if they

56:42

think and know that you acted from a

56:44

place of good intent cuz mistakes

56:47

happen, things go wrong. this is what we

56:49

need as a MIA culpa right I was willing

56:53

to do that I mean in some way I was

56:55

partly responsible certainly at the very

56:57

early stage to support a vaccine roll

56:59

out but I know that the most important

57:03

thing for me to do when new information

57:04

comes available and medicine again is

57:06

not an exact science it evolves

57:09

but let's play that out let's play that

57:10

out so if we do if we had a situation

57:12

where the I don't know the scientist

57:14

that said really positive things about

57:16

the vaccine that it was side effect free

57:18

or whatever They come out now and they

57:20

say, "We were totally wrong." And they

57:22

say, "We got it." So, so let's let's

57:24

play out that scenario. What would

57:25

happen the next day on social media is

57:28

everybody who was criticized or

57:30

critiqued or lost their job or was, I

57:32

don't know, in some way penalized for

57:35

their views that there might be side

57:36

effects that we're not talking about,

57:38

would immediately go to their their base

57:40

of their audience and say, "I told you

57:42

so." the conspiracy theorists on the

57:45

internet who are really extreme that

57:47

believe that there's a group of people

57:48

wearing like tin hats that have come up

57:50

with this idea, they would be empowered

57:53

and what you'd then have is a situation

57:55

where another vac another pandemic rolls

57:58

in from a far away land. those people

58:00

said, "Listen, you know, those people

58:02

that you see what I'm to say it it would

58:05

in my mind it would fuel the um the

58:09

narrative that vaccines are bad and less

58:12

like people would be less likely to take

58:13

them because we're not driven by facts,

58:16

stats, graphs, and figures. We're driven

58:17

by emotion."

58:18

Y

58:19

it's much more powerful

58:20

than just if a scientist standing there

58:22

and showing me a graph. It's how I feel

58:24

which matters the most. And if I felt

58:26

like I was betrayed and lied to, there's

58:28

no chance that I'd run down and get

58:30

another jab or something in my arm.

58:34

It's an uncomfortable truth that needs

58:35

facing though, Steve, because if we

58:36

don't face it, these problems are going

58:38

to carry on. We're not going to improve

58:39

the situation by ignoring it and

58:41

sweeping under the carpet. So, there

58:43

will be that, you're right, there will

58:44

be that backlash. I myself have had

58:46

that, right? I've had people, you know,

58:48

I I got heckled, you know, um a talk I

58:51

gave on this for the first time when I

58:53

spoke at it in London, um saying, "I was

58:55

part of it all. You're a liar." All this

58:57

stuff. Yeah, absolutely. I I got, you

58:59

know, exploiters were thrown at me. And

59:01

I I understand where that emotion comes

59:02

from. But at the end of the day, the

59:04

only way we can progress and evolve is

59:05

it just accepting. But that, you know,

59:09

because we want to then it's not just

59:10

about saying we got this wrong. It's

59:12

actually explaining to people and

59:13

saying, "Okay, we thought we were doing

59:15

the right thing. These are problems in

59:17

the system we weren't aware of. Most

59:18

people are not aware of this. We need to

59:20

resolve this and move forward with

59:21

greater transparency. And over time now,

59:24

okay, within the immediate aftermath, of

59:26

course, there's going to be that

59:27

emotional reaction. There's going to be

59:28

a backlash if there happen to be a

59:30

pandemic within a short space of time

59:32

after that admission. Yes, it may well

59:34

be that people aren't going to go and

59:35

take vaccines. But why should they,

59:37

Steve?

59:38

Vaccines are can save your life.

59:40

No, no, no. I'm talking about a new

59:41

anything new. I'm not talking about So,

59:42

yes, absolutely. Traditional vaccines. I

59:44

mean, I'm still a big subscriber and

59:46

supporter, let me just make this clear,

59:48

of traditional vaccines. In my paper

59:49

that I wrote, I said, you know,

59:50

estimates suggest vaccines have saved 4

59:52

to 5 million lives a year. And the

59:54

serious harm rate of vaccines, I think

59:56

there's, of course, there's going to be

59:58

nothing's completely safe. No drug is

60:00

pharmacist completely safe. But just in

60:02

terms of published data, right, probably

60:04

still an exaggeration, but still it

60:05

gives you it gives you a comparison.

60:07

Serious harm rate for traditional

60:08

vaccines, 1 to two per million.

60:11

All right. [snorts]

60:11

People aren't very people aren't very

60:13

smart, though. including me when it

60:15

comes to when I hear the word vaccine I

60:17

think all vaccines you think we don't

60:18

know what a vaccine is so it's just a

60:20

word it's like if you said to me dogs

60:23

are savaging one in 800 people you know

60:27

the average person isn't might not

60:28

[laughter] the average person might not

60:30

think if that's a chihuahua or like a

60:31

German shepherd we just heard dogs and

60:33

then there's going to be a fear of dogs

60:35

and I think the same with the vaccines

60:36

we don't know the difference between

60:37

different vaccines we just think they're

60:38

all the same so if you tell me that

60:39

vaccines are causing x y and zed I'm

60:42

going to go I don't Yeah. If someone

60:43

offers me a flu shot or whatever thing

60:45

or whatever, it's the trust that's been

60:47

eroded in the system and it's my trust

60:49

in the word vaccine that's been eroded.

60:51

Yeah. No. And it's it's unfortunate.

60:52

It's unfortunate that that that has

60:54

definitely showed. There has been a dent

60:55

and a change I think an uptake of things

60:57

like MMR

60:59

because of this.

61:00

It saved my life when I was a kid. I got

61:02

my all my family got malaria. We were in

61:03

Africa so

61:04

they all got pretty bad malaria and uh

61:07

so we yeah my pretty pretty serious as

61:09

well. I think I almost died of uh

61:11

malaria I hear from my mother.

61:13

Steve the thing is with doial vaccines

61:15

they went through many many years of

61:17

safety checks right and this is an

61:19

important thing people I think we

61:21

shouldn't underestimate people's

61:23

intelligence and their ability to

61:26

understand and forgive as long as we

61:28

communicate it in the right way. I have

61:30

these conversations all the time with my

61:31

patients. You know I give them numbers.

61:33

I talk through it. When I talk about

61:34

statin drugs for example I say I'm going

61:36

to give you these numbers but there are

61:38

lots of caveats here. one the data has

61:40

never been independently verified right

61:41

and I give them all this and I give them

61:43

alternatives etc I do this all the time

61:45

and patients want that they want more

61:46

information in a way that they can

61:48

understand and of course yes they want

61:50

to trust their doctor but again it's all

61:52

it comes down to ethics values intent

61:55

and doctors not admitting their mistakes

61:57

is a very very bad place to be

62:00

the chair of the clinical cardiology at

62:02

the University of Edinburgh professor

62:03

Mark Dwek commented that um on your

62:05

opinion saying the co vaccines the co

62:08

vaccine opinions you are misguided and

62:10

in fact dangerous. The vast majority of

62:11

cardiologists do not agree with your

62:13

views and they are not based on robust

62:16

science. Now, if you're someone

62:18

listening to this now, I've got your

62:20

opinion and I've got this guy's opinion,

62:22

the chair of the clinical cardiology at

62:24

the University of Edinburgh. I've got

62:25

the NHS saying that vaccines are safe

62:27

and extensively reviewed in both adults

62:29

and children and that the independent

62:30

medicines and health care products

62:32

regulatory agency is continually

62:35

monitoring the safety of COVID vaccines

62:37

and reports of side effects are very

62:39

rare. And then I've got your view. How

62:41

do I as someone that's hearing this in

62:43

this year and this in this year figure

62:45

out what to believe

62:48

because everyone's so compelling.

62:50

Everyone's got data.

62:51

Well, you just ultimately got to go with

62:52

your own intuition. Steve, who

62:54

my intuition is always going to side

62:55

with fear

62:56

because I'm a human being.

62:57

May maybe and and this stuff works. Um

63:00

I'll come on to this and this was a bit

63:01

of a hatchet job by the Guardian.

63:02

Interestingly, I've written 19 opeds for

63:05

the Guardian Observer newspaper over the

63:07

years, but you know how these how this

63:08

journalism works. They'll they'll move

63:10

on. Um a couple of things just couple of

63:12

facts to throw back at you first and

63:13

then what you've raised is really

63:15

interesting historically and something

63:16

that I've learned from this sort of

63:18

backlash. um Mark Dwek with the greatest

63:21

respect to him uh what wasn't disclosed

63:23

in the article is that he has taken

63:25

money from FISA he's he's he's you know

63:27

been funded by FISA and that's factual

63:29

you can look that up right that's one

63:30

thing so that's a bias um the more

63:33

important than that the MH which was

63:35

described as independent right yes is

63:38

not the British medical journal BMJ did

63:42

an investigation

63:44

published um in the summer of 2022 and I

63:47

presented this data uh on the on the on

63:49

the MH at the British Medical

63:51

Association annual conference where the

63:53

president of the BMA was there, the

63:55

chair of the BMA was there, right? And

63:56

they were gobsmacked and they didn't

63:59

they couldn't believe and this is why

64:00

this information is so important, these

64:02

facts so important. The MA even I was

64:04

shocked when I read this our medical

64:06

regulator in this country MH gets 86% of

64:10

its funding from big farmer which is a

64:12

huge bias. So they're not independent.

64:13

So those are those are the two facts

64:14

that should at least if I threw that

64:16

back you say hold on a minute then

64:17

should I believe all this now what was

64:19

the purpose of that article of course it

64:21

was to undermine my credibility I'm

64:22

exposing essentially something you know

64:26

it for all intents and purposes pretty

64:27

horrific you know reluctantly on the BBC

64:30

with 25 million views or whatever else

64:31

right um but people that inspire me have

64:35

been through far worse right and and I'm

64:37

an activist that want to expose

64:39

injustices right the likes of Mandela

64:42

Gandhi Martin Luther

64:44

One of the lessons from them and even in

64:45

public health advocacy as soon as your

64:47

work threatens an industry or an

64:49

ideological cabal you will be attacked

64:51

sometimes unrelentingly and viciously

64:53

and that was really a hatchet job.

64:54

Can I what about the the second part of

64:56

the quote that where he says that the

64:58

vast majority of cardiologists do not

65:00

agree with your views?

65:01

Um

65:01

is that true in your opinion? He's

65:03

giving his opinion, but actually um from

65:05

every okay every cardiologist I spoke to

65:09

has basically said you're going to do

65:11

doing great work but they won't speak

65:13

out and this is part of the problem

65:14

right is that people are turning a blind

65:16

eye. I had uh one cardiologist um who

65:21

met me in the street right these are I

65:23

have this happens all the time and he

65:25

said I read your paper he said I can

65:26

tell you now although they won't admit

65:28

it publicly all the cardiologists in our

65:31

department and they know you and they

65:33

and they they trust where you come from

65:35

and your integrity none of them are

65:37

having any more COVID vaccines because

65:39

of you they're on to it but Steve this

65:41

is a problem only a small minority of

65:42

people are willing to speak out I have a

65:44

platform where I'm able to articulate it

65:46

and and do so. But that is my duty and

65:48

responsibility. This is just a

65:50

reflection of uh what is ultimately a

65:54

big farmer tyranny. You know, I know

65:56

you've interviewed Jordan Peterson, who

65:57

I um admire greatly. Uh and and Jordan

66:01

Peterson says when you have something to

66:02

say, silence is a lie. [snorts] And you

66:05

know, tyranny tyranny emerges when

66:07

people are afraid to say what they

66:09

think. And when everybody essentially

66:12

lies all the time by being silent,

66:14

that's when the tyranny is complete.

66:16

This is exactly where we are. But I do

66:19

want to mention something if you don't

66:20

if you don't mind like I have to go

66:22

through this right myself and it's not

66:24

easy right you get all this stuff and

66:26

people checking credibility and I

66:27

remember when that that that Guardian

66:29

hit piece which was like the you know

66:30

the top story that day I actually felt

66:33

in many way feelings but I felt ah over

66:36

the target here right because Gandhi

66:38

said first they ignore you then they

66:40

laugh at you then they fight you then

66:43

you win. So the point is these things

66:46

work Steve but you know the truth is the

66:49

truth. So

66:50

what is the reality then of your of your

66:52

life going through that? Because you

66:54

know if you get attacked from all

66:55

angles, you've not got immediate family

66:57

there, you're you're reading stuff about

66:59

yourself online all the time, your

67:01

colleagues, etc. are turning on you.

67:02

What what's life like in if I'm a fly on

67:04

the wall in those hard moments? Um

67:08

very deep pain

67:11

on

67:13

to to to have the knowledge and deep an

67:17

understanding that in my view we are

67:21

dealing with one of the greatest likely

67:26

corporate crimes, medical mistakes,

67:27

damage to people's health. People are

67:29

suffering, people are dying, people have

67:31

got all sorts of problems because of

67:32

this vaccine. to have that deep

67:34

knowledge and understanding and to not

67:38

be able to see any great progress or

67:41

enough progress for this to be resolved

67:44

or to be improved. That suffering around

67:46

me gives me very deep pain more than

67:48

anything else.

67:52

Why?

67:54

I think inherently I'm just very

67:56

sensitive to that around me. That's just

67:57

the way I am. I think it's part of my

67:59

innate nature. I've always been like

68:01

that. You know, I had a I went for um

68:05

you know, on the positive side, there

68:07

are a lot of good friends around. You

68:08

know, I haven't got immediate emotional

68:09

support, if that makes sense, right

68:11

where I am, but there are hundreds of

68:13

thousands of people that support me

68:14

certainly who follow me on Twitter and I

68:16

people come and meet me in the street. I

68:18

randomly bumped into this doctor in the

68:20

street who I didn't know, an older

68:21

doctor quite well known. I won't name

68:23

him. Um and he said he he got really

68:26

emotional in the street and he met me

68:28

and he said, "Dr. Satra, thank you for

68:30

everything you are doing with all of

68:31

this. And I said, well, you know, I I

68:33

tried to be as humble with it. I said,

68:35

I'm just a medium for a message. I'm

68:36

doing the right thing. He said, no, what

68:37

you're doing is extremely brave and I'm

68:39

with you 100% and I know the vaccine

68:40

killed one of my colleagues, etc., etc.

68:42

And he was very honest. He said, I'm

68:43

just too frightened to speak up,

68:45

[snorts]

68:45

but that gives me fuel, right? And this

68:48

this happens constantly. So, I think

68:49

there is definitely something that I get

68:51

from that. So let me summarize your

68:53

position then because I want to move on

68:54

to I want to talk about statins and

68:55

heart disease and particularly high

68:56

cholesterol because I got told by my

68:58

doctor I have high high cholesterol. To

69:00

summarize your position is you believe

69:02

that vaccines themselves are

69:06

net good for the world.

69:09

Some of the greatest achievements in

69:10

medicine are traditional vaccines. No

69:12

doubt. You believe that the COVID

69:14

vaccine at the start when administered

69:16

to certain age groups that were most

69:18

vulnerable

69:20

there's an argument to say that it was

69:21

it was a net positive.

69:22

Yeah, I think there's an argument

69:24

and that you believe after sort of 2022

69:28

or 2021 when the variant changed to

69:32

another I think it was called omnicron

69:33

wasn't it? Um that at that point it

69:36

certainly became a net negative. Is that

69:38

your view? I would think probably

69:39

earlier Steph and the only reason I say

69:40

this is anecdotal evidence of course is

69:42

my dad had a cardiac arrest in the

69:44

summer of 2021 he was 73 right and he

69:47

could be considered in a high-risisk

69:48

group so I think that if one was to make

69:52

that case I would say well well it

69:54

depends what we're talking about as well

69:56

in terms of COVID yes right the net

69:58

benefit in terms of COVID and COVID

70:00

deaths but the problem is what's the

70:02

point in preventing someone getting

70:04

COVID if six months later they're going

70:05

to die of a cardiac arrest

70:07

you see what I mean So we've got to look

70:08

at it in that nuanced way. But yeah, I

70:10

think there is a case to be made that if

70:12

it was just given to certain high-risisk

70:13

groups overall there may have been a net

70:16

benefit at the beginning. But where we

70:18

are now and where it's continued and the

70:20

mandates and all the stuff that extended

70:22

it to many more people taking it and

70:24

that almost certainly was being fueled

70:25

by FISA and that's been shown that they

70:28

were you know giving money to grass

70:31

credible grassroots organizations in the

70:33

US to promote the mandates. Right? this

70:35

is after the data they received showing

70:37

that it wasn't preventing infection and

70:38

causing serious harm. Right? So this is

70:40

this is a problem with the system. I'm

70:41

not blaming individuals here. This is

70:43

something we'll talk about with statins

70:44

as well is that um the uh the system the

70:49

corporate capitalist system or the way

70:50

capitalism is is actually being

70:52

implemented is in its in many ways and

70:57

this has been diagnosed by a forensic

71:00

psychologist. uh the the corporation as

71:03

an institution is psychopathic when it

71:05

comes to making money. That means they

71:06

have callous unconcern for the safety of

71:08

others, repeated lying, conning others

71:11

for profit. And this is unfortunately

71:13

this is the root of the problem.

71:15

And you also believe that there is a

71:18

chance as well and it's likely that the

71:21

impacts of the lifestyle changes, the

71:23

lockdowns etc. The stress, the mental

71:25

health issues, the removing people from

71:27

being able to see their loved ones, the

71:28

sedentary lifestyles also contributed to

71:31

the rise in heart related conditions and

71:34

heart disease.

71:34

100%. It's played a role. A smaller

71:36

role, but it's played a role 100%.

71:38

So, I want to talk about heart disease

71:40

because I don't even know what heart

71:41

disease is. And also, I don't know what

71:43

a heart attack is. I think we kind of

71:44

all just assume we know, but what what

71:46

is a what is a heart what's heart

71:47

disease and what's a heart attack? So

71:49

heart disease uh the

71:52

conventional

71:54

description or explanation of heart

71:56

disease is disease that affects the

72:00

blood vessels of the heart essentially.

72:02

Okay, that's that's coronary artery

72:04

disease we call it.

72:05

That's what most people when they talk

72:06

about heart disease that's what they

72:07

that's what they mean. Um so that

72:09

disease that affects the blood vessels

72:13

leads to buildup of something called

72:16

plaque uh which is furring if you like.

72:19

Okay. Like a

72:20

plaque.

72:21

Plaque. Yeah. So plaque is furring of

72:22

the arteries like a blockage.

72:23

Okay.

72:24

Right. A blockage.

72:25

That blockage is made up of uh cells of

72:29

the immune system. It's made up of

72:30

cholesterol. Right. And over time that

72:34

those blockages can either suddenly uh

72:38

like a pimple building up and the pimple

72:40

getting bigger and bigger and bigger or

72:41

even a small pimple suddenly bursting

72:44

and the contents of that plaque, right?

72:47

The response to um

72:50

the blood having contact with the

72:53

contents of that plaque that has built

72:55

up over time suddenly causes a clot to

72:57

form. And if the clot blocks the whole

73:00

artery then during the blood supply is

73:03

completely cut off to the heart muscle.

73:05

So the the purpose of those blood

73:07

vessels is to supply the heart muscle

73:10

with blood so it can contract. So if you

73:13

have a blockage that is there for

73:16

several minutes completely cutting off

73:18

the blood supply depending on where it

73:19

is, it will cause that area of the heart

73:22

muscle to lose its oxygen supply and

73:25

nutrients and die and scar and that

73:28

leads to cell death. But of course the

73:30

heart muscle is quite big. So it could

73:31

be a very small heart attack, could be a

73:32

big heart attack. And then so so heart

73:35

attack specifically is death of any

73:37

region of the heart muscle because of a

73:39

blockage.

73:41

Okay, I'm going to try

73:42

the death of the cells.

73:44

I'm going to try and play this back to

73:45

you. Yeah,

73:45

correct me if I'm wrong. So, you get

73:46

this buildup in your artery.

73:48

Yeah.

73:49

But due to a bunch of factors I was

73:51

talking about,

73:52

the buildup explodes.

73:54

Yes.

73:55

Flows through the blood blocking the

73:56

artery at some point.

73:58

Yes.

73:58

And then that blockage in the artery

74:00

causes a cell in the heart, some areas

74:04

in the heart to die because they're not

74:06

getting oxygen.

74:06

Exactly. So, so you you're cut you've

74:08

basically cut off the blood supply. So

74:09

it's cutting off the blood supply to

74:11

whatever area because there are many

74:12

branches. It depends where the blockage

74:13

is. You cut off the blood supply. If

74:14

it's cut off for long enough,

74:16

those cells, every cell in our body

74:17

needs oxygen to survive, right? Then

74:19

that those that part of the heart muscle

74:21

will die and become scarred.

74:23

So it dies and becomes scarred in about

74:25

seven 7 8 minutes. You said

74:26

it can well it can take you know 15 to

74:29

20 minutes. It depends on different

74:31

factors but within minutes. Yes.

74:33

And how does that feel from minute one

74:35

till minute sort of 10?

74:36

So the classic symptoms of a heart

74:38

attack, right? that is a central what we

74:41

call crushing heaviness or pain that can

74:45

travel radiate we say in medical terms

74:47

to the neck the jaw it can go into the

74:51

shoulders um it can go into the back

74:53

that's the classic symptoms or down the

74:55

left arm

74:55

and you're conscious at this point

74:57

yes you're conscious so a cardiac arrest

74:59

just means the heart stopping ultimately

75:02

actually we all have a cardiac arrest

75:03

when we die the last thing to go is our

75:04

heart right but um one of the most

75:06

common causes of a cardiac arrest

75:08

certainly prematurely, not because of

75:10

old age, for example, is um and this is

75:14

random. So you can have a heart attack

75:16

and you can be conscious and have pain

75:17

and you get to hospital and you get

75:18

diagnosed and you might have a you know

75:20

a stent put in or you might be put on

75:21

blood thinners or whatever. Um in a

75:24

certain proportion of people um and it

75:27

doesn't depend on the size of the heart

75:28

attack. So um you can have a big area of

75:32

the heart muscle that's damaged or a

75:33

small area of the heart muscle that

75:34

damaged and it's random that it can

75:36

cause an interruption in the normal

75:38

electrical activity of the heart. The

75:40

heart has electrical circuit that allows

75:42

it to pump. Right? There's an electrical

75:43

circuit that allows it to pump in a

75:45

certain you know in a in a in a regular

75:47

rhythm. that circuit can get interrupted

75:51

and then the heart muscle that's pumping

75:52

like this right because it's pumping all

75:54

the blood around your body to your brain

75:56

everything else suddenly starts

75:57

quivering right uh and that's known as

76:00

ventricular fibrillation

76:02

that's when you see on all the you know

76:04

on movies or on TV shows and stuff like

76:06

that when they start shocking people and

76:07

that shows on the heart tracing as a

76:10

kind of squiggle like this

76:11

okay

76:12

right and that is what we call a

76:13

shockable rhythm if you deliver a shock

76:15

you know often 200 jewels right is is

76:18

delivered with a defibrillator that will

76:20

often restart the heart into a normal

76:22

rhythm and then the patient can be

76:24

managed and treated for example. So

76:25

that's but that quivering of the heart

76:27

causes basically the heart to stop

76:29

pumping blood around the body. The

76:31

heart's still moving but it's not enough

76:32

to pump blood around the body and if

76:34

that's going for a very long time Steve

76:36

not lot long you know for example in my

76:38

dad's case it may have been similar 20

76:39

or 30 minutes then eventually then you

76:42

know that will even stop and patient

76:44

dies. So, I've read some stats from a

76:47

different source that said in the UK,

76:48

one in eight men and one in 14 women die

76:51

from conory heart disease over the

76:53

course of their lifetime and that nearly

76:55

50% of all US adults have some type of

76:57

heart disease. Um, and before the

76:59

pandemic hit in 2019, 12 children died

77:02

every single week in the UK from cardiac

77:05

arrest. So, I mean, this is if

77:08

anything's going to kill me, it's

77:09

probably this.

77:10

Yes, most likely.

77:12

So, how do I stop it? [laughter] Right.

77:15

Um, how do you stop it? How do you even

77:17

reverse it?

77:18

How about some statins?

77:19

Yeah. So, okay,

77:20

let me just say this. My doctor, I got

77:22

my lab results back like 3 days ago,

77:24

and he told me a couple of things. One

77:26

thing he told me is, "You got load

77:27

vitamin D." I said, "Fine, yeah, get it.

77:28

I'm in a room all day." Uh, the second

77:31

thing he said is, "Your cholesterol is

77:32

high." He said, "Your bad cholesterol is

77:34

high." And I I don't really know what to

77:36

make of that, but he said, "Your your

77:38

bad cholesterol is high, and I need to

77:39

get that down."

77:41

Um, my father takes statins, which I

77:46

know a lot of people take statins. I

77:47

think it's like 200 million people are

77:48

taking statins globally. Um, so because

77:52

I have you here, what do you think of of

77:55

what the advice I was given, which is

77:57

just to get my bad cholesterol down and

77:59

how do I prevent myself ending up on

78:01

either statins or having a heart attack?

78:04

Okay. So on the statins issue and the

78:07

cholesterol issue traditionally Steve

78:10

for uh decades and even now the one of

78:15

the the primary focus within medicine

78:18

within cardiology to combat heart

78:21

disease was to get your so-called bad

78:22

cholesterol LDL as low as possible.

78:24

So LDL means bad cholesterol.

78:26

It's called low density lipoprotein

78:28

which is the bad stuff.

78:29

It's so well it's thought of

78:32

conventionally as a bad cholesterol.

78:33

Right. And the reason for that is is

78:37

that earlier studies that were done on

78:41

uh the correlation that was found

78:43

between high cholesterol and heart

78:44

disease [snorts]

78:45

um revealed that very high levels of

78:48

total cholesterol. There was a very high

78:52

um prevalence of heart disease. But

78:55

those levels of of cholesterol go and we

78:57

go we're talking about going back from

78:59

you know studies that started in 1948

79:02

that went over three decades where they

79:04

they found cholesterol being associated

79:06

with heart disease was only really there

79:08

at very very high levels. That's the

79:10

first thing.

79:12

The second part of it and why they

79:14

thought that getting it as low as

79:15

possible was the solution is that people

79:17

who had very low levels of cholesterol

79:20

tended not get heart disease. Right?

79:22

Total cholesterol less than four, LDL

79:26

less than two mill moles. Let's just say

79:27

that for argument sake, less than 2 mill

79:28

moles per liter. And by the way, just so

79:31

just so people understand this, you

79:33

would have got a red mark probably

79:34

saying that your LDL cholesterol is high

79:35

if it's more now the the guideline

79:37

suggests if it's more than three mill

79:39

moles per liter in your blood. The

79:41

measurement that is considered high, but

79:43

we we we'll we'll tear that we'll tease

79:44

that apart in a second. Now the thinking

79:47

was that okay if people with low

79:50

cholesterol are not getting heart

79:51

disease and people with very high levels

79:53

of cholesterol are getting heart disease

79:55

and that was total cholesterol above 10

79:57

for example right and LDL's above seven

80:00

or eight massively high right then or or

80:03

or more likely to get heart disease at

80:05

significant

80:07

numbers uh the thinking was that the

80:09

lower the better so all these drug

80:11

trials started but there's one thing

80:14

missing first and foremost most is that

80:16

most people's cholesterol Steve LDL is

80:19

genetic.

80:20

80% of your cholesterol because

80:22

cholesterol is a very So why have we got

80:23

it? It's a very vital molecule in the

80:24

body. It's without cholesterol we would

80:26

die. It's required for maintaining the

80:29

integrity of cells and cell membranes.

80:31

It's required for hormone production,

80:33

right? It has a role in the immune

80:34

system. So it's really important vital

80:37

molecule in the body. You can change the

80:39

profile of the cholesterol. There are

80:40

different components. There's something

80:42

called triglycerides which is a blood

80:43

fat and HDL so called good cholesterol

80:45

and LDL through dietary changes but

80:48

predominantly it's genetic initially

80:49

right so this is a thing so that does

80:52

that so so one could then question maybe

80:56

those people in those original studies

80:58

had genetically high cholesterol but

81:01

that doesn't mean the cholesterol was

81:03

the problem there may been something

81:04

else that hadn't been measured that we

81:06

hadn't discovered yet

81:07

that was genetic

81:08

that was causing the heart disease but

81:09

it happened to also beausing causing a

81:11

raised LDL. At the same time, people

81:14

with low cholesterol, they may have had

81:16

something else, right? They may have had

81:17

other factors that we now know actually

81:20

are linked to heart disease and it's

81:21

something to do with the cholesterol. So

81:22

that's the first thing. The next

81:24

question is um does lowering

81:26

cholesterol? So the question to you is

81:27

does lowering your LDL make any

81:28

difference? And for many many years

81:30

there was a mantra that was pushed by

81:33

the medical establishment that there was

81:36

a linear relationship. The lower your

81:37

LDL, the less likely you are to get

81:39

heart disease. In fact, there was a a a

81:42

commentary written by one of America's

81:44

most well-known eminent cardiologists.

81:46

He was the editor of the American

81:48

Journal of Cardiology. His name was

81:49

William Roberts. And he wrote an article

81:51

in 2011 to try and push the cholesterol

81:54

message further. So more people, most

81:55

more people take cholesterol drug

81:57

statins. And it was called it's the

81:59

cholesterol stupid. And there's a line

82:01

in that which I mentioned in my book

82:04

where he said you can be an obese

82:08

diabetic sedentary smoker

82:11

but as long as your cholesterol is low

82:13

enough in other words total LDL

82:14

cholesterol you will not develop heart

82:16

disease. Think about that for a second.

82:17

Now I'm just putting that to you Steve.

82:19

Now you hearing that what is does that

82:20

sound plausible to you?

82:22

No.

82:23

Right. It doesn't does it. It sounds a

82:24

bit strange doesn't it? Right. And I

82:26

thought this is odd.

82:29

I started looking into this in a lot of

82:30

detail probably 2010 partly because

82:34

by the end of the by by the late 90s the

82:37

people who had discovered the

82:39

relationship with very high LDL

82:41

cholesterol which is actually by the way

82:43

genetically linked to a condition called

82:45

familial hyper lipidmia affects one in

82:48

250 people right people's cholesterol is

82:50

skyhigh

82:52

um those people who got the Nobel prize

82:54

for this discovery said they predicted

82:57

the almost the eradication of heart

82:59

disease in the world by the early 2000s

83:02

because we had discovered these drugs

83:03

called statin drugs that lower

83:04

cholesterol and are shown through

83:06

clinical trials to prevent heart attacks

83:09

and strokes and death which we'll come

83:11

on to. Right? So you've got the

83:13

combination of that. But before statins

83:15

came onto the market and have been

83:17

prescribed now to 200 million people,

83:19

one of the things that isn't talked

83:20

about enough is that there were lots of

83:23

trials done. So you've got this

83:24

hypothesis now. We think we're right

83:25

here. We've discovered that very high

83:27

cholesterol in the population has got a

83:29

strong link to heart disease. And we've

83:31

discovered that very low cholesterol

83:32

doesn't give heart disease. But by the

83:34

way, what's interesting is for 95% of

83:36

the population in the middle, there was

83:38

no relationship with who was going to

83:39

develop heart disease and who wasn't

83:41

depending on the based upon their

83:43

cholesterol. Okay. But they thought,

83:46

let's develop these drugs to lower

83:47

cholesterol and we prevent heart attack.

83:48

So all the drug trials, Steve, before

83:50

statins came on the market did not show

83:52

any benefit. So, you've got the trial

83:54

where you've got someone with high

83:55

cholesterol and and another person with

83:56

high cholesterol. One person gets a

83:58

drug, the other person doesn't. You

83:59

follow them up. The person on the drug

84:01

massively lowers the cholesterol. No

84:02

prevention heart attacks over several

84:03

years. Like, hold on. What's going on

84:05

here? Maybe we did the trial wrong.

84:06

Let's try again. Let's try again. Let's

84:08

try again. Statins are produced. Okay. A

84:11

different type of cholesterol-lowering

84:12

drug. And suddenly you start seeing

84:15

benefits. Hearts. Oh, great. We've

84:16

solved it. Then we can make this case

84:18

that the lower cholesterol are better.

84:21

Two problems with that. The first one is

84:23

that statins actually have other

84:26

properties

84:28

other than lowering LDL cholesterol.

84:30

They are also anti-inflammatory and they

84:32

have anti- clotting properties.

84:35

What's accepted now is we know heart

84:37

disease is a problem is a a is a

84:39

clotting problem linked to chronic

84:40

inflammation. So it means the benefit of

84:43

statins which I will give you in a

84:45

second, right? is probably more likely

84:48

because of those properties because

84:49

other studies when we did other drugs on

84:51

cholesterol there was no benefit. Does

84:52

that make sense? Am I making sense now?

84:53

Yeah. So it's targeting the inflammation

84:55

and the clotting.

84:55

Exactly. So the question then is what is

84:58

the benefit of statins? So the first

85:00

thing to say and this is where the

85:01

controversy has happened um is that what

85:05

I'm about to tell you is again based

85:07

upon drug industry sponsored trials

85:10

where the raw data on those trials with

85:13

the benefits I'm going to tell you has

85:14

never been independently verified. So

85:17

what I say to patients and I'll say this

85:18

to you is what I'm about to tell you is

85:20

likely in my opinion an exaggerated

85:23

benefit but it's still a benefit. I'm

85:25

going to tell you if we trust the drug

85:27

companies completely, this is a benefit

85:29

you're going to get. Steve, if you've

85:30

not had a heart attack, forget about

85:32

high cholesterol for a second, right?

85:33

This applies to everybody. If you've not

85:35

had a heart attack

85:37

or you have not been diagnosed with

85:39

severe blockages, right? You've not got

85:41

that, right? And you would you would

85:42

know if you did because you'd get

85:43

symptoms of chest pain doing exercise.

85:46

Then the benefit of a statin for you

85:48

over a fiveyear period at best is one in

85:52

100 in preventing you having a nonfatal

85:55

heart attack, a nondisabling stroke but

85:59

will not prolong your life by one day.

86:01

So that's the first thing. Now when you

86:03

do studies where you give patient that

86:05

information in that transparent way more

86:09

than twothirds of them most of them will

86:10

and I my experience as well say doc

86:13

those odds don't sound great I want to

86:14

prevent heart disease but I don't fancy

86:15

taking and this is before by the way

86:16

we've talked about side effects where

86:18

the controversy has happened which I've

86:20

been heavily involved in is in my

86:23

experience with with patients and also

86:25

other data that's out there a large

86:28

proportion of patients will suffer

86:31

quality let me be clear Here it's not

86:32

about serious, right? Quality of life

86:35

limiting side effects, right? That means

86:37

most commonly fatigue, muscle pain, can

86:40

be erectile dysfunction, sleep

86:41

disturbance, but stuff that makes you

86:42

don't feel you don't feel good like this

86:43

is not a good way to live.

86:45

[snorts and gasps] The good news is it's

86:46

reversible usually within a couple weeks

86:48

of stopping the statin or reducing the

86:49

dose. Okay. So that controversy uh led

86:53

to me publishing an article in the

86:54

British Medical Journal in 2013 where me

86:56

and another and a Harvard doctor in a

86:58

separate article said we we believe that

87:00

the side effect rate is probably in the

87:02

order of one in five people 20 20% which

87:03

is quite high of those sorts of side

87:05

effects right [snorts] and that caused a

87:08

bit of backlash because the group of

87:11

researchers in Oxford who take money

87:12

from big farmer who write the guidelines

87:15

around the world said this is going to

87:17

scare people stop them taking statins

87:18

and people will die but that for the

87:21

low-risisk people, no one's going to die

87:22

because there's no benefit in mortality.

87:25

Now, for high-risisk people, those are

87:27

people who have had a heart attack.

87:29

Um, the benefit of a statin is is

87:32

better, Steve. Okay. So, let's say, for

87:34

example, some patient comes in, they've

87:35

had a heart attack, and I'm telling them

87:36

the benefit of the statin if they take

87:38

it religiously every day for five uh

87:41

five years because that's how long the

87:42

trials last where you can give them that

87:44

information. The benefit of preventing a

87:46

further heart attack is 1 in 39, about

87:49

2.5%. 1 in 39 and the benefit in

87:52

prolonging their life is 1 in 83.

87:56

Right? There's another way of looking at

87:59

the stats though based upon again

88:01

industry sponsored trials all of the

88:03

trials that have been done on statins

88:05

and people might ask a question. They

88:06

don't often ask this but they may ask

88:07

the question how much longer am I going

88:09

to live?

88:11

Right? Like okay let's just throw this

88:13

question to you. Say you've had a heart

88:15

attack. You survived a heart attack.

88:17

doctor says take this drug and and over

88:20

a fiveyear period, how much extra life

88:24

ex life life extension would you hope or

88:26

hope it would add over that five years?

88:27

You can, you know, you can do it over

88:29

whatever 20 years, but just say 5 years

88:30

to start with. How much longer would you

88:32

hope to live from taking that statin?

88:34

Okay, so over five years then I'd say um

88:37

maybe a year.

88:38

Okay.

88:38

25% more.

88:39

Okay, fine. That sounds that sounds

88:40

pretty good.

88:41

20% more.

88:42

Yeah. Okay.

88:43

Um the actual figure not disputed from

88:46

industry sponsored trials right

88:49

4.2 two days, just over four days.

88:52

Is that because they haven't done it

88:53

over a long enough period of time?

88:55

No. I mean, and then if you then

88:56

extrapolate it out, if you presume the

88:58

benefits are going to keep going on, you

88:59

could then argue, well, over 10 years

89:01

it'll be 8 days and over 20 years it'll

89:04

be 16 days or whatever, right? So, so

89:06

that's the information. So, this the

89:08

reason I'm I'm I'm telling you this,

89:10

Steve, is that

89:12

this information is important. I'm not

89:14

just a doctor. I'm a potential patient.

89:16

I ask myself, what would I want to know,

89:18

right? I am here to try and improve my

89:20

quality of life and my health. What is

89:22

important to me? What is important to

89:23

patients? This information is important

89:26

especially Steve because it's not that

89:28

I'm saying don't take statins. A lot of

89:29

people will say okay I'll take the drug

89:30

right and if they don't get side effects

89:32

why not you know it might be a bit of a

89:33

lottery but why not take the drug and

89:35

the people say that to me and that's

89:36

fine and if they get side effects but

89:38

the thing that's missing Steve is and

89:39

this is where we come on to the question

89:40

you asked me about how to prevent heart

89:42

disease. Is there an alternative that

89:45

doesn't give me side effects, improves

89:47

my quality of life, and likely is going

89:49

to help prevent stop me getting heart

89:51

disease? So, what is that?

89:53

Lifestyle changes.

89:55

Absolutely. So, 80% of heart disease is

89:58

linked to environment and lifestyle.

90:01

Okay. So what is the the core most

90:05

important biological phenomenon in the

90:08

body that leads to heart disease which

90:10

is not disputed but doesn't get it

90:12

doesn't get much of a um there's no

90:14

market for the message because there is

90:16

no you can't commodify this this is an

90:18

interesting thing right so but it's it's

90:20

really important information it's called

90:21

insulin resistance insulin is a very

90:24

important hormone in the body released

90:25

by the pancreas helps maintain blood

90:28

glucose within certain ranges so our

90:29

cells can function optimally all Right?

90:32

Over time, if your insulin is

90:35

chronically raised too high or if your

90:38

cells become resistant to them for

90:40

whatever reason, that is the most

90:42

important driver of the development of

90:44

these so-called plaques, these furring,

90:45

these blockages in the arteries.

90:47

[snorts]

90:48

So, what causes insulin resistance and

90:50

how can you combat it or how, you know,

90:52

how can you prevent and and potentially,

90:53

you know, stabilize and even reverse?

90:55

We'll go on to reversal as well because

90:56

that's a fascinating area. [gasps]

91:00

most important components

91:02

diet. Okay, I would say in big figure

91:07

terms now probably 50% of heart disease

91:12

around the world can be linked to poor

91:14

diet.

91:16

Okay, so let's start with diet then.

91:17

I've actually got a bag of sugar that

91:20

I've brought with me because um when I

91:22

think about insulin glucose bad diet,

91:24

the first thing my my brain thinks about

91:25

is sugar. So, I've got this bag of sugar

91:28

here that I brought with me. Um, and

91:30

I've got two Look at us. We've we've

91:33

written sugar on it and covered up the

91:34

brand because they didn't pay us. But,

91:36

if there is a sugar brand that would

91:37

like to do a brand deal, please get in

91:38

touch. [laughter]

91:39

We're big fans if we need to be. Um,

91:42

what I actually would like you to to

91:43

show me is how much sugar do I need to

91:48

function and to be healthy.

91:50

Okay. The amount of sugar that you need

91:51

to function and be healthy is zero.

91:54

There is no nutritional requirement

91:57

whatsoever of of of this what we call

92:00

added sugar or table sugar. None at all.

92:03

So I don't need any.

92:04

You don't need any. And the reason for

92:05

that is very briefly our bodies need

92:08

glucose to survive. But you don't need

92:10

any dietary source of glucose to

92:12

survive. Right? Because your people go

92:14

on what we call ketogenic diets and they

92:16

literally eliminate all starch as well

92:18

which also contains glucose um from

92:21

their diet. Your body will make it

92:23

itself from fat and protein. So in terms

92:26

of diet, there is no value from sugar

92:28

whatsoever. It can give you energy of

92:31

course, yes, but you can get energy from

92:32

other things, but there's no nutritional

92:33

value whatsoever. Zero. So from a from a

92:36

health perspective, there is zero

92:38

requirement for dietary sugar.

92:41

So when we say that Americans are

92:42

consuming a lot of sugar every day, are

92:45

we talking about added sugar? Yeah,

92:46

we're talking about so the way that so

92:49

the World Health Organization now

92:52

um after I was involved in this

92:54

campaign, you know, and there was a lot

92:55

of attention on it, they revise their

92:57

guidelines. So they recommend a maximum

92:59

limit for the average adult of actually

93:02

six teaspoons of either um so maximum

93:05

limit, okay, which you don't need it,

93:06

but maximum limit because after that

93:08

that's when the health consequences

93:09

start to accumulate. One,

93:12

two,

93:15

3,

93:18

4,

93:21

5,

93:23

six.

93:24

Okay. So, that's the maximum daily

93:26

limit.

93:26

Maximum daily limit. Okay. But just to

93:28

give you an example, that's not let's so

93:32

one sugary drink, we don't name any

93:34

brands, right? You would have to would

93:36

often a 330 mil let's say the most

93:39

famous sugary drink uh in the world

93:40

I have no idea who you're talking about

93:41

would have nine teaspoons on top nine

93:44

spoons in one of those right and that's

93:46

just in the 330 mil imagine going to the

93:47

cinema and having half a liter whatever

93:49

else right

93:50

Jesus so one can of beep um has nine

93:54

teaspoons of sugar in it which is

93:56

which is so four yeah absolutely so four

93:59

grams is one teaspoon

94:00

okay which is well above the daily

94:02

recommended daily maximum one chocolate

94:06

bar.

94:06

Yeah.

94:06

Like a standard size chocolate bar. What

94:08

we grew up with

94:09

about the same.

94:10

Leave leave chocolate out of it.

94:11

Right. [laughter] Well, listen, you

94:13

know, dark chocolate. I love chocolate,

94:14

right? Have you know um for children,

94:17

the US Department of Agriculture, for

94:19

the average 4 to 8year-old child, think

94:21

about kids as well. They're the most

94:22

vulnerable to a lot of this pro these

94:24

problems of sugar. They recommend a

94:26

maximum limit of three teaspoons.

94:29

Yeah. Now the average person in this

94:32

country in America is consuming at least

94:35

20 teaspoons. Now when we talk about

94:37

sugar we're talking about world health

94:39

they they the word they use is free

94:41

sugars. So it means this sort of sugar

94:42

added right to foods. Um but it also

94:45

includes fruit juice,

94:47

honey and syrups.

94:50

same effect on your body. The that I

94:53

fruit, whole fruit is fine, but if you

94:54

think about it, um a glass of orange

94:58

juice, for example, would usually have

95:00

the uh the juice of six oranges.

95:04

Okay?

95:05

You couldn't eat six oranges that

95:06

easily. You'd have one orange, for

95:07

example. And there's fiber, and the

95:10

fiber does has a counteracting effect on

95:12

terms of the way that the um the glucose

95:15

and fructose is affected absorbed in

95:17

your body. A lot of it's to do with the

95:18

rapid increase in the bloodstream of

95:20

blood glucose and and that causes huge

95:23

spikes in insulin. And the problem with

95:26

that over time, of course, is you get

95:28

chronically raised insulin and heart

95:29

disease. But also, interestingly, the

95:30

more rapidly you have a food that causes

95:32

a glucose spike, the more quickly you're

95:35

going to get a crash because insulin

95:36

goes up quite quickly and it causes to

95:38

come down and it drives hunger.

95:40

Yeah.

95:40

So, that's another problem. So, the food

95:43

industry, I think, knew for a long time.

95:44

That's why you know in America and it

95:46

may be similar here 70% of foods

95:48

purchased in supermarket in America will

95:50

have added sugar because they knew that

95:52

it increases the palatability and the

95:54

consumption. Of course they're there to

95:55

sell food not look after your health.

95:57

So you said 20 teaspoons is the average

96:00

for an American.

96:01

At least 20 is probably much higher.

96:02

I'm going to have I'm going to have to

96:03

ask you to show us that.

96:05

Yeah.

96:05

So I can see what that looks like in a

96:07

glass.

96:07

It's disgusting. That's disgusting when

96:10

you see it like that. And you don't you

96:12

don't realize that you're having that

96:13

much sugar in a day. But I mean,

96:16

technically that's what two cans of

96:19

fizzy drink.

96:20

Yeah. Yeah. But the thing is a lot of

96:22

the sugar that's being consumed are in

96:24

foods that people wouldn't think have

96:25

sugar in them. They're not the usual

96:27

junk food, your cakes and biscuits and

96:29

whatever else, you know. Um they're in

96:32

things even marketed as being healthy.

96:34

Low-fat foods, etc., low-fat yogurts.

96:36

So, could you just summarize what we

96:38

have here then? These two glasses.

96:39

Yeah. So this is the maximum daily

96:42

recommended limit by the World Health

96:44

Organization of sugar consumption after

96:46

which you then start to see increases

96:48

risks of disease. Um and this is what at

96:53

least I think what most people are

96:55

consuming at the moment.

96:59

Are you hopeful that there's something

97:00

we can do about this [snorts] at a

97:02

society level? Well, I think one

97:05

important step was the fact that we in,

97:06

you know, this levy on sugary drinks

97:08

definitely would have some um impact

97:11

partly based upon what we learned from

97:13

big tobacco. You know, Steve, you know,

97:16

50% of it's interesting, crazy when you

97:19

think about it now. In 1970, 50% of

97:22

adults in the UK were smokers, right?

97:24

We've got that down to about 17% now,

97:26

but massive reduction, right? And it

97:29

happened. Education was important, but

97:30

the real the real intervention that had

97:34

more

97:35

um impact than anything else was

97:38

actually taxation of cigarettes. And it

97:41

had twofold effect. Of course, you make

97:43

it a little bit more expensive. You

97:44

know, you know this with your business

97:46

background, right? It's going to reduce

97:48

the the consumption, but it was a way of

97:49

also increasing awareness that this is

97:51

harmful. So, I think that the sugar

97:54

stuff is ongoing and I think a lot of

97:56

people have awareness on it. I think now

97:58

which is linked to sugar the and I've

98:00

written about this we need to treat

98:02

ultrarocessed food which a lot of the

98:05

ultrarocessed food has added sugar as

98:07

the new tobacco

98:09

right this is more than 50% of the

98:11

calories in the UK and more than 60% of

98:13

the calories in the US is of people's

98:16

daily consumption is coming from

98:17

ultrarocessed foods essentially

98:19

described as if it comes out of a packet

98:23

industrially produced and you can read

98:25

more than five ingredients usually with

98:27

additives and preservatives, it's

98:29

ultrarocessed and best avoided. And all

98:31

of the research that's been done only

98:33

points in one direction. And I think

98:34

there are at least 32 different

98:36

conditions or diseases now that are

98:38

associated with the consumption of

98:39

ultrarocessed food. That is the major

98:41

issue. So because this is a

98:43

predominantly an environmental problem,

98:45

right? Our our our food our food

98:47

environment is saturated with this

98:48

stuff. Even hospitals, Steve, I mean I

98:50

the first campaign I got involved with

98:51

is trying to get this stuff out of

98:52

hospital. I was like, hold on. We're

98:54

treating patients with obesity related

98:55

conditions, yet we're serving them junk

98:57

food in hospitals. The staff, 50% of NHS

98:59

staff are overweight or obese.

99:00

What's the probability that if I'm, cuz

99:02

I want to talk about ultressed foods as

99:04

well as the shoes, what's the

99:05

probability that if I'm having that much

99:07

sugar a day, the big glass, that I'm

99:10

going to end up in your practice with

99:12

some kind of heart disease.

99:14

I I wouldn't be able to give you a

99:15

figure, but you're definitely

99:17

significantly increasing your risk.

99:18

Well, if that's the average American,

99:20

then what's the average American's

99:21

chance of ending up in I guess causation

99:24

needs to be established.

99:24

Yeah, of course. And it depends what

99:26

else they're having as well. But but

99:28

it's definitely putting them at

99:29

significant increased risk of type 2

99:30

diabetes. I think the figure now in

99:32

America, if I'm not wrong, is that a

99:35

third to a half of adult Americans now

99:38

are um pre-diabetic.

99:40

um the the broad so to answer your

99:43

question in in a better way because

99:46

we're already there unfortunately

99:48

um the way to

99:52

understand insulin resistance on a

99:54

personal level in terms of measurements

99:56

body measurements is something called

99:57

metabolic health and there are five

99:58

components which are very easy to

100:00

measure of metabolic health okay um it's

100:03

your waist circumference

100:04

right it's your blood triglycerides

100:07

which is a form of the cholesterol being

100:09

above 1.7 mill moles. Okay. Um, it's

100:12

your HDL cholesterol, the good

100:13

cholesterol being less than 1 mill.

100:16

It's being pre-diabetic. Okay. Having a

100:20

HBA1C of more than 5.7%.

100:23

Um, or being prehypertensive, so your

100:25

blood pressure is above 120 over 80

100:27

average. If you have um three of those

100:32

that are abnormal, you have something

100:33

called metabolic syndrome. puts you at

100:35

the highest risk of heart disease and at

100:36

least 50% if not more patients admitted

100:39

with heart attacks will have metabolic

100:40

syndrome. But all those five parameters

100:44

being in the normal range in America

100:47

only one in eight adults have all of

100:50

those in the normal range and only one

100:52

in four people aged between 20 and 40.

100:55

Think about that for a second.

100:57

So seven out of eight yes adults will

100:59

have abnormal metabolic health. If we

101:03

within a year, if we just shifted a

101:07

little bit people's eating habits and

101:08

let's say we eliminated ultrarocessed

101:10

food, we got people to eat more nuts and

101:12

seeds, oily fish, you know, whole fruit

101:15

and vegetables, it's been estimated that

101:18

you would half the death rates from

101:20

heart disease within globally within one

101:23

year from 20 million to 10 million.

101:24

You'd be out of business

101:27

as a heart doctor.

101:29

Um,

101:31

there's still be a lot of people to

101:32

treat. We got to help the other 10

101:33

million as well, right? But but I think

101:35

the other thing as well is um what it

101:37

does Steve though is it shifts the age.

101:40

So instead of someone having saying a

101:42

dying prematurely having a heart attack

101:44

at the age of say 65, you know, they'll

101:46

live to 85. So all we'll be doing as

101:49

doctors is we'll be managing older

101:50

people. But that's fine. You know, we've

101:52

improved longevity, improved quality of

101:53

life as well.

101:54

You refer to stress as a silent killer.

101:57

It's something we're not addressing, I

101:58

think, as a society. So, to give put it

102:00

in perspective, when you look at risk

102:02

factor for heart disease, risk factors

102:03

for heart disease, um the major ones are

102:06

smoking, type two diabetes, high blood

102:08

pressure. The risk of heart disease

102:11

linked to stress, chronic stress of more

102:14

than a moderate degree is actually the

102:16

same as being a smoker or having high

102:18

blood pressure or having type two

102:19

diabetes. And most people, I think, are

102:21

not managing it. And certainly of all

102:23

the patients I see, all the heart

102:25

patients I see, invariably all of them

102:29

have had over the preceding years before

102:31

their heart attack stress stress levels

102:34

that are quite high. What's the

102:36

mechanism?

102:37

So

102:39

from an evolutionary perspective,

102:43

if we were acute stress can be a

102:45

lifesaver, right? If we were running

102:47

away from a saber-tooth tiger, right?

102:50

Then what happens is the body produces

102:53

clotting factors and inflammatory

102:54

markers uh factors in the blood that if

102:58

we were attacked they help reduce the

103:00

risk of us bleeding to death. So imagine

103:02

now imagine that happening chronic

103:04

stress. You've got a lowgrade what we

103:05

call chronic inflammation and clotic

103:07

factors increase. And that's been shown

103:09

actually in proven in um a study that

103:11

was published in the Lancet a few years

103:12

ago where they did an MRI of the brain

103:16

of of young healthy people, adults, and

103:19

they followed them up but they looked at

103:22

the their subjective stress score. They

103:24

did like um questionnaires to measure

103:27

their stress levels subjectively. They

103:28

correlated that with looking at the

103:31

amygdala in the brain which is the

103:32

emotional center and then they looked at

103:35

clotting factors and they even looked at

103:36

heart attacks further down the line and

103:38

there was a perfect correlation with the

103:41

subjective stress score the amydala

103:42

lighting up the clotting factors in the

103:44

blood the inflammatory markers in the

103:45

blood and heart attack. So we we've got

103:46

the plausible mechanism. So if that's

103:48

the case Steve there must be a way of

103:51

curbing it and well how does one do

103:53

that? Well, the two most important

103:56

causes of stress in society are work and

103:58

relationship stress. So, those are

103:59

things people can potentially work on,

104:01

right? Potentially, but it's just be

104:03

aware of it. But one of the

104:04

interventions that I use with my

104:06

patients um is actually breath work and

104:08

meditation.

104:10

And one of the most extraordinary bits

104:11

of research that I came across

104:14

and it's in our documentary as well

104:16

because we go into a bit more detail on

104:17

it. I had to go and see it for myself to

104:19

believe it actually properly believe it.

104:21

um a cardiologist in India for more than

104:24

20 years has been um through a lifestyle

104:28

approach has been reversing heart

104:30

disease. So he did a study called the

104:32

Mount Abu that is a place in northern

104:33

India uh where he's got a huge community

104:36

in a hospital people come to see him

104:38

there and um he puts him through this

104:40

lifestyle plan which in this study was

104:43

that you know they're devout Hindus so

104:44

it was a high fiber vegetarian diet it

104:46

was two 30-minute brisk walks a day okay

104:48

and something called Raj yoga meditation

104:51

for 40 minutes

104:53

which is a form of breath work but it's

104:54

also a bit of a spiritual transformation

104:56

as well it's about understanding where

104:58

your anger comes from and all that kind

105:00

of So it goes quite deep. And anyway, he

105:02

followed these people up who were

105:04

diagnosed with at least 50 to 70%

105:07

blockages in their arteries. They had

105:09

heart disease. For some reason, they

105:11

didn't want to have a um a bypass

105:13

operation or a stent or they couldn't

105:15

afford it and they wanted this lifestyle

105:18

plan. So he put them on a lifestyle

105:19

plan. He repeated their angagrams after

105:20

2 years, right? He looked at the

105:22

arteries again and on the people that

105:24

had adeared to the plan, there was an

105:26

average reduction of the blockage of

105:27

20%.

105:29

70% became 50, 50 became 30. This is

105:32

unheard of. If you ask any cardiologist,

105:34

can heart disease reverse saying, "No,

105:35

I've never even seen it. It's not

105:36

possible, right?" But he showed this to

105:38

be the case. He then looked at his data

105:40

a bit in a bit more detail and said,

105:41

"Was it the diet? Was it the exercise,

105:44

the two or was it the stress reduction?"

105:46

The only of course they're all

105:48

important, but the only independent

105:49

factor for the reversal of the blockages

105:52

was 40 minutes of Raj Yoga meditation

105:54

per day.

105:57

Now I asked him, you know, he published

106:00

this data in what we call a um an

106:02

observational study. It wasn't what we

106:04

call a randomized trial where it was

106:05

just more rigorously done. And he said,

106:07

"Why have you not done the randomized

106:08

trial?" Said, "I have years ago." And he

106:12

showed me the data. And I was like,

106:12

"Wow, why didn't you publish this

106:14

anywhere? I couldn't get it published."

106:16

He invited and I could be wrong about

106:19

it. It was a it was senior it was a

106:21

senior cardiology society. can't

106:24

remember if the American Heart

106:25

Association but it was a scenist cardio

106:26

society in America

106:28

to um he invited them to India so he

106:30

could present his data he presented his

106:33

data to them and they basically thought

106:36

it was fascinating but in between the

106:38

lines they said to him you know we can't

106:41

publish this because it will affect our

106:42

stent business

106:45

and what's a stent business

106:46

stent is basically the heart stance like

106:48

as in it would you know I remember Steve

106:50

when I first um wrote an article the

106:53

first sort mainstream media article I

106:55

wrote with in 2011

106:57

was me it was in the Observer newspaper

106:59

I'd met Jamie Oliver having written to

107:01

him saying can you sort hustle food out

107:03

and then I end up writing an article

107:04

called I mend hearts then I see our

107:06

hospitals serve junk food to my patients

107:10

uh basically saying listen we're going

107:11

to tackle this obesity epidemic tackle

107:12

heart disease we need to sort the diet

107:14

out and I remember one of the

107:16

cardiologists um who I knew a senior

107:19

cardiologist I was still a junior doctor

107:20

at this point kind of tongue and cheek

107:23

but many truths spoken in just said it

107:25

seem this is going to affect our our

107:27

business our stent business as in

107:28

operating on people as if it was a bad

107:30

thing that I was saying that we should

107:31

basically prevent heart disease right

107:34

there's a cultural problem here Steve

107:35

honestly I'm not this is this is a this

107:36

is a um a dirty secret if you like

107:39

within medicine unfortunately amongst

107:41

establishment figures is that they have

107:44

become so wedded and so close to these

107:48

tyrannical corporations or their own

107:50

self-interest that you know we're

107:52

missing a big trick here in terms of

107:53

what we can really do for patients.

107:56

In part, your other book here that I

107:59

have, I still can't pronounce this word.

108:01

Pop

108:01

popy diet. Pop.

108:03

Your popy diet, I guess, serves to

108:04

address many of these issues and to make

108:06

sure that our heart hearts stay healthy.

108:09

What I wanted you to do for me is if I'm

108:12

endeavoring to have a a good heart

108:14

health until I die, which I guess is

108:16

kind of impossible, but maybe I'll get

108:18

hit by a truck or something. Um, if I'm

108:20

endeavoring to have good heart health,

108:21

what should my daily rout in an optimal

108:24

situation? What should my daily routine

108:26

look like if I follow

108:28

the diet that you came up with, but also

108:30

if I'm thinking through all of the

108:31

potential things that can cause bad

108:33

health?

108:34

Sleep.

108:35

I wake up. So, I sleep for seven hours.

108:36

So, at least 7 hours sleep. Great. Tick.

108:39

Okay.

108:40

Um, eating real food.

108:42

Okay. So, breakfast is how many how many

108:45

meals a day should I have? Well, two to

108:47

three, whatever suits you. When you feel

108:49

hungry, eat till you're full.

108:50

Not seven, eight, nine, not

108:51

no just eat, you know, and also I

108:53

suppose if you're doing activity, then

108:54

you may, but the most important thing is

108:56

to avoid the ultrarocessed food and not

108:58

to have too much starch.

108:59

What about fasting?

109:01

Um, controversial area, mixed data. Um,

109:06

I think that in terms of, you know, I

109:08

have patients who feel benefit from

109:10

fasting and other people feel more

109:12

stressed. It depends where you are in

109:14

your life. If for example you're very

109:16

active and your sleep isn't good

109:18

actually fasting can make your stress

109:19

levels worse which is not good. It

109:21

causes cortisol to increase and there is

109:23

some data showing that happens. So it

109:25

depends where you are but I'm not averse

109:27

to fasting. It depends you know see how

109:28

people feel.

109:29

Okay. So I've woken up. I've had seven

109:30

hours of sleep. I've eaten breakfast

109:32

which was whole foods. So

109:34

yeah and and Okay. So I would say you

109:36

know whole fruit and vegetables ideally

109:39

um a mixture of of low sugar fruits if

109:41

you can ideally you know berries mixed

109:43

berries for example

109:44

if you can get at least five to seven

109:46

portions in that's great

109:48

um the the foods that are thought so the

109:50

the anti-inflammatory foods that have

109:53

got a reasonable body of evidence behind

109:55

them are things like extravirgin olive

109:57

oil is your base fat okay nuts and seeds

110:00

tree nuts almonds walnuts um hazelnuts

110:04

for example

110:05

oily fish at least a couple of times a

110:07

week,

110:08

right? And then I would say minimizing,

110:12

you know, the starch and the sugar,

110:13

okay?

110:14

And the rest of it doesn't matter. You

110:15

can have, you know, I have heart

110:16

patients ask me whether they can eat a

110:17

steak. I say absolutely it's not going

110:18

to cause, you know, it's very

110:19

nutritious. If you want to have a steak

110:20

two or three times a week, no issue.

110:22

Saturated fat in the diet isn't a

110:23

problem. I busted that myth. So butter,

110:26

cheese, for example, um red meat is not

110:29

going to have any adverse effect on your

110:31

heart.

110:32

Okay. So I've eaten my breakfast. You

110:34

want to enjoy your food as well, right?

110:35

Yeah. So, that's my sleep taking care of

110:37

my meals that day taken care of.

110:39

What else do I need to be thinking about

110:41

to optimize heart health?

110:42

Well, I suppose you got to think about

110:44

what level it's very subjective. You

110:47

know, if you're stressed

110:48

and if you're stressed, then you need to

110:49

think about what can you do about it.

110:51

And there are different things. Some

110:52

people find exercise um is good. Some

110:55

people do yoga, they do pilatees, some

110:58

people do breath work. So, try and find

111:00

I have an app called Calm which I listen

111:01

to. I I do at least 20 to 30 sometimes

111:06

40 minutes of breath work every morning

111:07

when I wake up. It's the first thing I

111:08

do. Actually,

111:09

I'm going to do both. I'm going to do

111:10

breath work and I'm going to go for a

111:12

long workout.

111:14

Could is there such such a thing as too

111:17

much exercise?

111:17

Yeah. Unfortunately, yes. About 11% of

111:21

elite athletes by the time they reach 50

111:23

will have scans that show very severe

111:26

blockages or a heart disease, right? Um,

111:30

and I think Steve, to be honest, it

111:33

might be genetic predisposition, but I

111:34

think a lot of those people, cuz I have

111:35

some of these people coming to me who

111:36

don't know why they've got diagnosed

111:37

with heart disease. And I remember one

111:38

lady I saw a few months ago, and she was

111:40

running something like

111:42

something crazy like 5 to 10 miles a

111:44

day, but on only 4 hours sleep, and she

111:48

was young and otherwise didn't have a

111:49

diabetes or anything else, but had heart

111:50

disease, had developed heart disease.

111:51

And I said, "This is probably because of

111:53

this." So uh I think that when you look

111:56

at heart disease and optimal levels of

111:58

exercise, it is actually the one thing I

112:00

think the guidelines are right about is

112:02

probably that 150 minutes of moderate

112:04

activity a week. So you know I again I I

112:06

follow my own advice and I [snorts] will

112:09

um you know I I used to run but you know

112:12

running can be quite damaging to the

112:13

knees especially as you get older. So I

112:14

I cycle and do exercise bike and

112:16

whatever else and I will get my heart

112:18

rate to about 115 beats a minute for 30

112:20

minutes you know five times a week. One

112:22

study found that marathon runners

112:23

experience a frequency of heart attacks

112:25

and strokes similar to people who

112:27

already have heart disease suggesting

112:28

too much exercise is harmful which is in

112:31

your book statinfree. And um another

112:35

stat here is athletes who do more than

112:37

one hour of intense exercise per day,

112:39

four times um have four times the

112:41

frequency of breathing infections per

112:43

year compared to those who do moderate

112:45

activity.

112:45

Well, that's the other thing about

112:46

overex exercising. Well, it depresses

112:47

the immune system. That's well known. Uh

112:49

and in fact if you look at and a lot of

112:51

people do it for different reasons but

112:52

if you look at the the communities

112:53

around the world known as the blue zones

112:55

Steve you know where people have high

112:56

longevity. These people weren't pounding

112:57

it in the gym. They're just outside you

112:59

know they were gardening they were

113:00

moving. You don't need to be doing all

113:02

of that kind of stuff. People do it for

113:04

different reasons. I mean I I do it

113:05

sometime. Oh also for mental health it's

113:07

good.

113:07

You um didn't you publish an article?

113:09

You co-authored an article for the

113:12

British Journal of Sports and Medicine

113:13

called It's Time to Bust the Myth of

113:15

Physical Activity.

113:17

Yeah. Physical inactivity and obesity.

113:18

You can't outrun a bad diet. That was

113:20

the title of the article. And the point

113:22

was, and we make it very clear at the

113:24

beginning, that exercise has many

113:25

benefits for health, but weight loss is

113:27

not one of them. Because most of what

113:29

determines your weight gain, almost all

113:31

of it is to do with with what you eat.

113:33

And that discussion or that thinking,

113:35

whether it was about burning calories

113:37

actually came from the food industry.

113:38

They they they they manufactured that.

113:40

Um Coca-Cola, McDonald's, they would

113:42

push this. They even the Olympic Games

113:44

2012, they were the main sponsors,

113:46

right? because for them they want to

113:48

distract from their unhealthy products

113:50

and say well the obesity epidemic is

113:51

being driven by people not you know not

113:53

exercising.

113:54

So on that day on this perfect day that

113:56

I'm creating in my mind I'm eating well

113:59

I've moved but not too much. I'm doing

114:01

breath work in the morning. I've slept 7

114:03

hours a day and the last thing that from

114:05

looking at your 21-day immunity plan is

114:08

socializing.

114:10

Yeah. So so important. I think one of

114:12

the most important aspects for mental

114:13

health, physical health is the quality

114:16

of our relationships, friends, family.

114:19

Um people may laugh at this, but I

114:21

actually I actually prescribe hugging to

114:23

a lot of my patients. Uh especially

114:25

couples, you know. Um there is

114:27

definitely some benefits from that, you

114:29

know, uh and uh in terms of reducing

114:31

cortisol levels, etc. I think that's an

114:34

an issue in society in general. You know

114:36

we become we have been conditioned in a

114:38

way to think that we can just be

114:41

dependent on ourselves as individuals

114:43

and we don't need other people but

114:44

actually we do this is that's how we

114:46

evolved partly because it helps us it

114:48

helps us feel safe

114:49

hugging

114:51

I think it just it just yeah there is it

114:54

activates parts of the brain the

114:55

protection soothing mechanisms of the

114:57

brain and I think that is just an extra

114:59

element of it absolutely

115:02

and they it reduces cortisol and all

115:03

those things so it's going to stave

115:05

heart attacks. If we hug, we have less

115:07

chance of having a heart attack.

115:09

It's not not just about that. I think

115:11

it's also good for the immune system. So

115:12

the the people that did the original

115:14

cold studies on on cold viruses, this

115:17

was done, I think, in early 2000s.

115:18

Fascinating study. They inoculated

115:20

people with a cold virus. So they put

115:22

the the traditional cold virus whatever

115:24

into into people's, you know, nasal

115:26

passages and they looked at who

115:28

developed symptoms of a cold and who

115:30

didn't. And they looked and and before

115:32

they did that, they did these

115:33

questionnaires based upon several

115:35

different aspects of people's social

115:37

life. Are they involved in community

115:39

activities? How often do they meet their

115:41

friends? What's it like with their

115:42

partner? Etc., etc. And the people who

115:44

had the highest scores, only one in

115:46

three of those people developed a cold.

115:49

And the people on the other side that

115:51

were more maybe more lonely and weren't

115:52

interacting with other people who didn't

115:54

have good quality relationships, two out

115:56

of three of those people developed

115:58

colds.

115:59

So, it's not just about heart disease,

116:01

it's about the immune system as well.

116:02

Overall health

116:02

makes you think, doesn't it, if

116:03

loneliness really is killing us. Yeah.

116:05

Um,

116:06

we need to try and do more to end the

116:08

epidemic that is loneliness cuz the

116:09

stats seem to suggest it's going one way

116:11

and it's not a good way.

116:13

Um, and part of that issue as well,

116:15

Steve, which is a something we need to

116:17

think about with what's going on society

116:19

[snorts] is I do we've got a worsening

116:21

mental health crisis. We've got less

116:23

trust in government. One of the ways

116:26

societies progress is people being able

116:29

to trust each other by people not being

116:31

afraid to speak the truth. And more than

116:34

ever, and certainly I've seen it in

116:35

medicine, we have got to a situation and

116:38

the COVID vaccine is a microcosm of

116:40

something much bigger where people are

116:42

afraid to speak the truth. And that

116:44

gives us an element of uncertainty,

116:45

distrust, and it makes us more stressed.

116:49

So what this means, what it comes back

116:51

to is ultimately part of the solution to

116:54

the mental, physical, social well-being

116:56

crisis, heart disease is thinking about

117:00

acting from a place of virtue and

117:02

ethics, right? Thinking about, you know,

117:05

your intentions, being honest, not

117:07

manipulating other people for money.

117:08

This is unfortunately where the the the

117:10

capitalist system or the current

117:11

economic system has taken us and the

117:13

corporatization of human beings as a

117:15

result. And that is very detrimental to

117:17

our physical and mental health.

117:19

What was your father's name?

117:21

Kash.

117:21

Kash. Son, never change your loyalties.

117:32

Yeah, loyalty was very important to him.

117:34

You know, those I was brought with those

117:35

core values about um honesty, integrity,

117:39

being loyal to people, how to be a good

117:42

human being, how to be the best version

117:43

of yourself, Steve. Um, that's what it's

117:46

about, you know, for your benefit and

117:49

for the benefit of others.

117:50

He said that to you when you were

117:51

younger and you were considering

117:53

switching football team. Um, he told you

117:56

to never change your loyalties. And you

117:57

des I've got the eulogy you wrote for

118:00

your father when he passed away here.

118:02

And it's interesting some of the the

118:03

words and phrases that you used in the

118:05

eulogy to him because they seem to be

118:06

quite pertinent to our conversation

118:07

today. The ultimate purpose of knowledge

118:09

is to reduce human suffering. And that

118:11

true wisdom

118:14

to achieve that end only comes from

118:16

dialogue.

118:17

Yeah, that was uh that was a Socratic

118:20

Socrates quote. True wisdom comes only

118:22

from dialogue. Um,

118:25

understanding others starts from

118:26

understanding oneself. And to get to a

118:30

greater truth, one has to be able to

118:32

question one's own beliefs. And to get

118:36

there, to achieve that, you have to

118:38

engage uh with other people and have

118:40

discussions. but from a place of

118:41

compassion where you are open to

118:43

listening to other people because we can

118:44

only get to a greater truth if you

118:46

listen to all different sides on a

118:49

particular issue and that comes from

118:52

having that conversation. So I think

118:53

that's a huge component of it all which

118:55

I think we're losing Steven society

118:56

there's so much polarization you know.

118:58

Yeah, and that's yeah, I completely

119:00

agree and you know it's it's difficult

119:01

obviously because um conversation can be

119:05

fatal. It can be um especially medical

119:08

conversation conversation about things

119:10

like vaccines or health information. So

119:13

it's very delicate information but um

119:15

the what can also be fatal is the lack

119:17

of conversation i.e. not seeing ideas

119:20

collide. And it's it's interesting as

119:22

someone who speaks to a lot of health

119:24

professionals on this podcast, I'm not

119:26

an expert in health and people will

119:27

often criticize me for that and say, you

119:29

know, um this they said this thing wrong

119:31

or this thing wrong. But I think the

119:33

place that I've got to now is to present

119:35

every opinion that I can.

119:36

Yeah.

119:37

And hopefully to will people to be able

119:39

to make up their own minds. And I hope I

119:41

tried to uh present some of the other

119:44

side of um the at least the rebuttals so

119:47

people can can be curious and we don't

119:49

we're not pushing in any particular

119:51

direction. I have no bloody dog in the

119:52

fight. I don't you know

119:53

and I'm not I want to have that

119:54

conversation, Steve. I think part of the

119:56

problem with all of this is that that

119:57

conversation isn't even happening. We

119:58

want to have that conversation. We want

120:00

rebuttals. We want a count of view.

120:01

Yeah.

120:02

But the ignoring of even the

120:04

conversation that for me is is is

120:06

unforgivable.

120:07

It's a shame. I think podcasts are doing

120:08

a good job of kind of well I think a lot

120:10

of them are doing a good job of just

120:12

having that conversation.

120:13

Yeah.

120:13

Um because it's long form and there's I

120:16

mean there's a there's a comment section

120:17

so people are going to discuss below and

120:19

there's you know there's lots of podcast

120:21

there's lots of information being thrown

120:23

out into the world and um everything

120:25

we've talked about today will be linked

120:27

below as much of the the links as we

120:29

can. So, if anybody is curious about

120:31

anything we've discussed or any stats,

120:33

you'll send me your stuff. Absolutely.

120:34

And I'll link it below and I'll I'll

120:36

link the NHS and the British Heart

120:38

Foundation and anyone else below so

120:39

people can have a think about that. But

120:41

I am a big fan of conversation and I'm a

120:44

big fan of having both sides of an

120:46

argument and trying to make my own mind

120:48

up on things and find the nuance. I find

120:50

the truth is usually somewhere in the

120:51

middle. Yeah.

120:52

So, I think it's important because, you

120:53

know, one of the things I think we have

120:55

to we have to reflect on is some of

120:57

those amazing people you talked about

120:58

like Martin Luther King and I don't know

121:00

whether it's the suffragettes or Gandhi

121:01

or whoever it might be, their ideas in

121:04

their time were received equally

121:06

horrifically and um now those are things

121:09

that we all consider to be true and very

121:11

important as it relates to maybe science

121:12

or just social issues on equality. So

121:16

with that in mind, we have to also be

121:18

humble to the fact that idea that might

121:20

be important might at first offend us.

121:23

It might trigger us. It might be counter

121:25

to the public narrative or or to the

121:28

current available science. But I don't

121:30

think it should be censored.

121:32

No. And I I would say that people

121:35

listening to this just think about one

121:37

thing. One of the reasons that we seek

121:40

the truth and greater truths is that a

121:43

life lived in darkness has no meaning.

121:48

Amen. We have a closing tradition on

121:50

this podcast where the last guest leaves

121:51

a question for the next guest not

121:52

knowing who they're going to be leaving

121:54

it for. And the question left for you is

121:58

of all the most amazing superstar people

122:01

you have met in your life,

122:04

what was the quality that made them

122:07

superstars? And can it be taught?

122:11

A love for humanity.

122:14

That was a quality that made them

122:16

superstars in whatever they were doing.

122:18

It was to give back to society in some

122:21

way. Whether it was entertainment,

122:23

whether it was music, whether it was

122:24

sport, but based upon a love for

122:27

humanity.

122:34

Stands up to be true. I just went

122:35

through a list of superstars that I know

122:36

of in my brain and I think that's

122:37

certainly the case. And it's funny cuz

122:38

the people that I think of as real

122:40

superstars aren't necessarily famous.

122:41

They're not rich. They're not anything

122:43

like that. They're just like the best

122:44

people, the ones that I really aspire to

122:46

be more like. But the one that made them

122:48

stand out for me, the one quality that

122:50

made them extra special in th those

122:52

particular people um was despite them

122:56

being so excellent is they had a huge uh

123:00

um a wonderful humility about them

123:08

and they can find you everywhere,

123:09

especially on Twitter. Got a big Twitter

123:11

following. Um

123:12

and your books as well. I will link your

123:14

books below in the description for

123:15

anybody that wants to read them. I've

123:17

these two books in particular, the one

123:19

that I can't pronounce, the

123:21

Pop Pop diet,

123:23

a 21-day lifestyle plan. Lose weight,

123:26

feel great, and drastically reduce your

123:27

risk of type two diabetes and heart

123:28

disease, and a statin-free life, a

123:30

revolutionary life plan for tackling

123:32

heart disease without the use of statins

123:34

are two that I'd certainly really,

123:35

really recommend. Thank you, Dr. Thank

123:38

you for the work you do, the way that

123:40

you do your work, and thank you for

123:41

having the courage to be a [snorts] loud

123:45

counteracting voice in society where we

123:47

do need counteracting voices. I don't

123:48

think anybody can ever argue with that.

123:50

And the way in which you do it and your

123:51

intentions of doing it, I think are

123:53

wonderful. Um, and I think they're a

123:56

real credit to the two wonderful people

123:58

that raised you. So, thank you for your

123:59

time today. Thank you for doing the work

124:00

that you do. I'm going to continue

124:01

following it. I follow you on Twitter

124:02

and I've been following you for many,

124:04

many years, I think, I believe. Um, and

124:06

I very much enjoy consuming your

124:09

information because I know that you

124:10

don't you have there's a certain

124:12

fearlessness with you that is going to

124:15

deliver what is true regardless of

124:17

consequence. And that is a useful source

124:20

of information to have in my world where

124:21

I'm I'm trying to advance my thinking

124:22

and I care more about progress and truth

124:25

than I do something feeling comfortable.

124:28

So, I highly recommend everyone go check

124:30

you out on Twitter as well. [music]

124:33

Heat. Heat. N.

124:39

[music]

124:43

Heat. Heat. [music]

124:46

[singing]

124:50

[music]

Interactive Summary

Dr. Aseem Malhotra, a consultant cardiologist, discusses his critical perspective on the COVID-19 vaccine's impact on public health, his concerns regarding the influence of the pharmaceutical industry on medical practices, and his broader advocacy for lifestyle-based healthcare. He explores the potential risks of the mRNA vaccines, specifically regarding cardiovascular inflammation, and reflects on personal losses in his family, including his father's sudden heart attack. Dr. Malhotra also addresses the root causes of heart disease, emphasizing the role of insulin resistance, processed foods, and chronic stress, while advocating for a more holistic approach to medical care.

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