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Insulin Doctor: The Fastest Way To Burn Dangerous Visceral Fat! I'm Finding Mould In My Patients!

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Insulin Doctor: The Fastest Way To Burn Dangerous Visceral Fat! I'm Finding Mould In My Patients!

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3205 segments

0:00

If you have a belly, sticking out, you

0:01

have a problem. Because the fat that's

0:04

in the stomach, that's called visceral

0:05

fat. This is very detrimental fat. And

0:08

that's the epidemic that we have today.

0:10

But the only thing that will make you

0:12

lose that fat very quickly is Dr.

0:14

Pradeep Jamnadas is a world-leading

0:16

cardiologist who has treated more than a

0:18

quarter of a million patients with

0:20

chronic heart disease. Now, he's using

0:22

his voice to help millions more prevent

0:24

ending up on the operating table through

0:25

simple lifestyle techniques.

0:27

>> This is crucial. Glucose actually stops

0:30

sticking inside the bloodstream. And the

0:31

body pours insulin into the bloodstream

0:33

to push glucose out. But frequent

0:35

consumption of carbs, sugar, processed

0:38

foods is causing insulin to stay up,

0:40

which can lead to insulin resistance.

0:42

And you're going to be more prone to

0:43

heart disease, which is the number one

0:45

cause of death all over the world right

0:47

now. But this is where fasting comes in.

0:49

Because after 12 hours, you start

0:51

pulling the fat out. And the first place

0:53

the fat comes out of is going to be

0:55

visceral fat. But with modern living, we

0:57

have lost this physiology of fasting. So

1:00

we'll go into that.

1:01

>> And there must be certain things which

1:03

people aren't aware aren't healthy as it

1:05

relates to my cardiovascular health.

1:06

>> Yes. So I see that people who overly do

1:09

aerobic activity, they end up with more

1:11

coronary artery disease than patients

1:14

who do short sprints and resistance

1:16

exercises. Then there's mold. And almost

1:20

70% of homes these days have some form

1:22

of mold toxicity in them. But also, one

1:25

night of bad sleep, you become insulin

1:27

resistant the next day. And there's

1:29

calcium supplements, excessive fruit,

1:31

white rice.

1:32

>> White rice? You'd be surprised how much

1:34

arsenic there is in rice these days. I

1:36

watch a lot of true crime. People kill

1:37

each other with arsenic.

1:38

>> Yes. And it will slowly kill you. And

1:40

lastly, this is going to surprise you.

1:43

I see messages all the time in the

1:44

comment section that some of you didn't

1:46

realize you didn't subscribe. So if you

1:48

could do me a favor and double-check if

1:49

you're a subscriber to this channel,

1:50

that would be tremendously appreciated.

1:52

It's the simple, it's the free thing

1:54

that anybody that watches this show

1:55

frequently can do to help us here to

1:57

keep everything going in this show in

1:59

the trajectory it's on. So, please do

2:01

double check if you subscribed and thank

2:03

you so much because in a strange way you

2:04

are you're part of our history and

2:07

you're on this journey with us and I

2:08

appreciate you for that. So, yeah, thank

2:10

you.

2:14

Dr. Pradeep Jamnadas,

2:16

what is it you've spent the entirety of

2:18

your career,

2:19

especially the last few decades of your

2:21

career, focusing on?

2:23

Prevention of heart disease,

2:25

which is the number one cause of death

2:27

all over the world right now.

2:30

You see, the heart is made up of many

2:31

parts. So, this is important. It's a

2:33

pump, so it's a muscle.

2:36

So, you have diseases of the muscle and

2:38

it's rampant these days, cardiomyopathy,

2:40

weakness of the muscle. Then you have

2:42

the arteries on top of the heart and

2:44

that's called coronary artery disease,

2:47

diseases of the arteries, they block up.

2:49

Then you have the valves, the valves

2:51

tend to get damaged as well. For

2:53

example, aortic stenosis.

2:55

And then you have the peripheral

2:56

circulation, all the blood vessels that

2:58

go to your carotids, your brain, your

3:00

legs, and to all the organs of your

3:02

body. And then you have microvascular

3:05

disease, which is the tiny capillaries,

3:10

that they become dysfunctional as well.

3:12

>> What's a capillary? Capillary is the

3:13

branch of the branch of the branch.

3:15

>> Okay, so like the tiny little veins.

3:17

>> Tiny ones, yeah. So, when they become

3:19

dysfunctional, that organ becomes

3:22

dysfunctional as well. So, cardiology

3:26

affects all these aspects.

3:29

And how long have you been operating on

3:30

people's hearts? About 35 years now. And

3:33

how many hearts do you think you've

3:35

treated?

3:37

In excess of 30,000.

3:39

And what what's the age range of those

3:41

patients?

3:43

They we used to be mostly older people,

3:45

over 65. Now, I'd say 50% of them are

3:49

under 65 and 50% are over 65. So, the

3:54

demographics is changing. The younger

3:55

people are developing heart disease. So,

3:58

the youngest patient you've ever

3:59

treated?

4:01

28.

4:02

And what what was the predicament in

4:04

that case? He was having a full-blown

4:06

heart attack. So, that means that one of

4:09

his arteries was completely clogged up.

4:11

It had a blood clot in it. There was no

4:13

circulation past that artery, and

4:15

therefore he was having acute chest

4:17

pain, sweating, blood pressure was low.

4:21

He comes straight to the hospital. I

4:22

rushed to the hospital, take him

4:24

straight into the cath lab, and try to

4:26

open up that blockage. And we did open

4:28

up that blockage, and we put a stent in

4:30

it, and got rid of that blockage

4:31

immediately to restore that blood flow

4:34

to the heart muscle.

4:36

And at 28, how does one get a heart

4:37

attack? You said it was blocked up.

4:39

There's a lot of nuance in that.

4:42

Most people don't understand what a

4:44

heart attack is. So, in a nutshell, I'm

4:46

going to tell you what it is.

4:48

When you have a plaque, a plaque is a

4:50

build-up of a little

4:53

fibrous material inside the artery,

4:57

and that doesn't cause a heart attack. A

4:59

heart attack is caused when one of those

5:01

plaques

5:03

cracks. Now, in this diagram, for

5:05

example, it's showing that the plaque is

5:07

closing up the artery, and this will

5:09

cause angina. That means a lack of blood

5:12

supply over here. So, this patient may

5:14

complain of chest discomfort, or he may

5:16

not complain of chest discomfort.

5:18

Because remember, when you have a

5:19

blockage like this, only 20% of the

5:22

patients actually get chest pain.

5:25

That means that you could have a

5:26

blockage like this in a patient, and he

5:29

may not have any chest discomfort.

5:31

And the only way you're going to pick

5:32

that up is by doing a stress test, or a

5:35

coronary calcium score, or some other

5:38

modality to see whether he has blockage

5:41

or not. A heart attack is when a minor

5:44

blockage, which is not causing much flow

5:46

disturbance, suddenly cracks. And what

5:49

happens to the the plaque that that

5:51

cracks off? And when it cracks open, the

5:54

blood that's going past it sees the

5:57

crack and wants to repair it

5:59

and forms a blood clot on it. So, the

6:01

final thing that shuts down that artery

6:03

is a blood clot. It's not the plaque

6:07

that shuts it off. It's a blood clot.

6:09

So, a heart attack is caused by a blood

6:12

clot at the site of a rupture. And what

6:16

causes rupture is inflammation. Okay.

6:19

So, when people say that heart disease

6:22

is the number one

6:24

killer of people generally that are that

6:26

are diseased, is it that process that's

6:28

causing it alone or is there a multitude

6:30

of processes that lead to heart disease?

6:33

There's a multitude of things that lead

6:35

to heart disease.

6:36

Look, they are That was a heart attack.

6:38

That's right. Okay. So,

6:40

one, you're making plaque. You shouldn't

6:43

be making plaque.

6:45

A plaque is an inflammatory

6:48

area inside your arteries. It's reacting

6:51

to something. And I can go into the

6:53

nuance of what's actually What's the

6:55

body reacting to? What's inflammation?

6:57

Inflammation is reacting to something.

6:58

What is that something? And I think it's

7:00

important for your audience and

7:01

everybody to know what that something

7:02

is.

7:04

Now, you've got this pimple.

7:06

Think of it as a pimple on the inside of

7:08

the artery.

7:09

Now, one day when there's enough

7:11

inflammation in your body systemically,

7:13

that pimple is going to crack open, just

7:15

like a pimple on the skin breaks open.

7:19

Now, that artery has this little pimple

7:21

on the inside and it cracks open and a

7:23

blood clot forms on it, and that's a

7:25

heart attack. So, you don't want plaque

7:28

formation. You don't want plaque

7:30

instability, which is rupture of that

7:33

plaque, and you don't want a blood clot

7:34

to form on it. There are factors that

7:37

affect each of these processes, and all

7:40

three processes are going wrong today.

7:42

And that's why we have so much plaque

7:44

formation and instability of the plaque,

7:48

which then leads, of course, to heart

7:49

attacks.

7:50

And our blood, in general, we have more

7:54

coagulation issues today than we ever

7:56

did before, which means our blood,

7:58

because of inflammation, is more ready

8:00

to clot more easily. So, we having the

8:03

perfect triad.

8:05

It's a perfect storm. That's why we're

8:07

seeing an epidemic of this, and that's

8:09

why we're not making a dent in it. Yes,

8:11

you can have a plaque rupture and have a

8:13

heart attack and we'll put a stent in

8:14

you. Okay, that's fine. But what did we

8:17

do

8:18

to look at the underlying reason as to

8:20

why you formed that plaque, and why did

8:22

you rupture it, and why was your blood

8:24

so clotty in the first place that made

8:27

you actually seal off the artery?

8:30

Is this an increasing problem?

8:32

Yes, it's a it's a huge problem, because

8:36

those factors have been identified now.

8:38

And that's what we go for in my

8:40

practice. We look for the factors. Why

8:43

did you make this plaque?

8:45

Why is there so much inflammation in

8:47

your body?

8:48

Inflammation is a reaction to something

8:50

foreign. What is that foreign stuff?

8:53

What is that abnormal physiology in you

8:55

that your body is reacting against?

8:57

Because that's what inflammation is.

8:59

And inflammation, of course, whenever

9:01

there's going to be inflammation,

9:02

there's going to be increased tendency

9:03

to make blood clots. So, we go in, we

9:05

chase all these things in patients to

9:08

find out. And that's the real

9:10

prevention.

9:12

That 28-year-old that came in

9:15

um and you treated him, was it was that

9:18

a a surgery you did? We call it a

9:20

surgery, but it's not. It's a it's a

9:22

angioplasty. So, we go in from the wrist

9:25

Yeah. or we go in from the groin with a

9:28

catheter, and we go into the coronary

9:30

artery. A catheter being like a tube

9:33

>> It's a hollow tube, and we put it into

9:35

the coronary artery. We shoot the dye

9:36

inside, we see where the blockage is,

9:39

and then we thread a tiny wire

9:42

into that artery through the blockage,

9:44

and then bring in a balloon, and squash

9:47

the blood clot out of the way, restore

9:50

the circulation,

9:51

and then bring in a stent. A stent is

9:54

like a spring that opens the artery up.

9:56

We leave the stent inside there, we

9:58

remove everything else, and now you have

10:00

an open artery, and you have blood flow

10:02

going down that artery.

10:04

>> And what had that 28-year-old done to

10:06

end up in your surgery? What were the

10:08

decisions that they had made that

10:10

brought them there?

10:11

So, he was number one, he was

10:14

overweight. But, he wasn't just

10:15

overweight. The overweight was all in

10:17

his belly.

10:19

And he had a fatty liver, and he had

10:20

what is known as visceral fat.

10:23

He was not a diabetic, but he was a

10:25

prediabetic.

10:28

That means he had a lot of insulin in

10:31

his body, and his glucose levels were

10:33

good. So, if you went to his family

10:34

doctor, they'd say, "Oh, you're not a

10:36

diabetic. Your hemoglobin A1C, which is

10:38

a blood test that you do for diabetes,

10:40

is actually okay." But, actually that

10:42

patient had a very high insulin blood

10:45

level.

10:46

Now, insulin

10:48

is a very atherogenic molecule. It

10:51

causes smooth muscle proliferation.

10:53

Smooth muscle is in the walls of the

10:55

arteries. It causes vasoconstriction.

10:58

It makes your blood more clotty,

11:00

and it causes inflammation.

11:03

So, this patient was a prediabetic.

11:05

And this is very important. This is what

11:08

caught my attention.

11:09

Because when I looked at my patients

11:11

that were coming in with heart attacks

11:13

and hardening of the arteries, and when

11:15

I tried to identify why they're doing

11:18

this,

11:19

none of them were diabetics.

11:21

But, then I did a glucose tolerance test

11:24

on them.

11:25

And what happened, it was just by chance

11:28

that I happened to have these tests in

11:29

my office where you can measure the

11:30

blood glucose. So, I had about 120 of

11:33

them. So, I said, "Look, let's just do

11:35

some random testing on these patients."

11:37

And I found that at least half of them

11:40

had no diabetes, but they glucose

11:42

intolerance. That means that their

11:44

sugars went up, but not enough to make

11:47

them a diabetic. But, it did go above

11:49

the normal range. We did not have

11:51

insulin testing at that time.

11:53

About 10 years later, we started testing

11:56

insulin in the office, and I bought a

11:57

machine for this purpose

12:00

in my office to measure insulin levels.

12:02

And life was never the same again. Why?

12:06

Because it's all about insulin when it

12:08

comes to the metabolic derangement. So,

12:10

I found that these patients, their

12:12

sugars were going up 250, 160 after

12:16

giving them a glucose drink. Mhm.

12:18

So, you say, "Okay,

12:19

he's not a diabetic. Look, the sugar

12:21

didn't really go high." But, when you

12:23

measure the insulin level,

12:25

the insulin was off the roof. So, when

12:28

you say this is an insulin problem, can

12:29

you explain this to me like I have no

12:31

idea what glucose and insulin are for

12:33

for any of my listeners out there that

12:34

have a inexperienced understanding of

12:36

these these these terms and what they

12:38

what they do and mean? It's crucial.

12:41

Everybody needs to understand the

12:42

relationship between sugar, which is

12:45

glucose,

12:46

and insulin. When you consume sugar or

12:49

glucose, the body has to get rid of that

12:52

glucose very quickly from the

12:54

bloodstream because glucose actually is

12:56

toxic inside the bloodstream. Even

12:58

though it is what the body uses for

13:00

energy,

13:01

in the bloodstream, it glycates all the

13:04

blood vessels and the walls and the

13:06

components in blood and the hemoglobin

13:08

as well. Glycates it. That means the

13:10

glucose attaches itself to that

13:11

molecule.

13:13

So, now that molecule can't work

13:14

properly. That is why the higher your

13:17

blood glucose,

13:19

all your chemicals don't work well. Your

13:21

enzymes don't work well. Your hormones

13:23

don't work well. Nothing works well, and

13:26

you age prematurely because you're

13:28

getting glycation. Glyca- A glycated

13:31

molecule can't work normally. It doesn't

13:33

function normally.

13:35

So,

13:36

when you consume the glucose, the

13:38

glucose has to come out of the

13:40

bloodstream. And how does the body do

13:41

it?

13:42

It sends a message to the pancreas. The

13:44

pancreas says, "Aha, I'll make some

13:46

insulin." It pours insulin into the

13:48

bloodstream. Insulin comes into the

13:50

bloodstream, pushes the glucose into the

13:52

cells. And where does it push in it?

13:54

Into the liver, into the muscles, into

13:57

every cell in the body. Insulin will

14:00

push glucose out. Now, how much insulin

14:03

is the question.

14:06

If I'm eating every 3 hours,

14:09

and I'm consuming glucose, or I'm

14:11

consuming starchy foods, or I'm

14:13

consuming carbohydrates. Now, watch

14:15

where I'm going with this because you're

14:16

already beginning to know where I'm

14:17

going with this.

14:19

I'm consuming glucose and carbs every 2

14:22

3 hours. I'm stimulating my pancreas.

14:24

I'm stimulating my insulin. My insulin

14:27

goes up, it comes down. But before it

14:29

even gets a chance to come down, it goes

14:31

up again.

14:33

So, the repeated consumption of and

14:36

frequent consumption of glucose is

14:39

causing my insulin to stay up because

14:40

insulin stays a little bit longer in the

14:43

bloodstream than the glucose. The

14:44

glucose will come down in about 2 to 3

14:46

hours, but the insulin stays higher for

14:48

about 4 hours. Now, what happens is that

14:51

you continue this lifestyle for a few

14:53

years.

14:55

Now, the body, because these are all

14:57

hormones,

14:59

will say, "Well, you know, it's I'm

15:01

going to need to make more insulin."

15:03

Now, you become insulin resistant. Any

15:05

hormone that stays in your body for a

15:07

long time, the body becomes immune to

15:09

it.

15:10

So, the next time I eat the sugar, I'm

15:12

going to have to make more insulin to

15:14

produce the same effect.

15:16

That is called insulin resistance. So,

15:19

now you got this patient who's been

15:21

eating carbs,

15:24

sugar,

15:26

processed foods.

15:28

What does that mean, processed food?

15:29

That means foods that are quickly

15:30

absorbed into the bloodstream. Processed

15:33

foods.

15:34

These are products

15:35

without fiber.

15:37

So, the absorption is very quick. So,

15:40

the poor pancreas has to react just like

15:42

that. Produces a whole bunch of insulin.

15:46

And then the frequent eating and the

15:48

frequent consumption makes you insulin

15:50

resistant. So, now you produce a whole

15:52

bunch of insulin in order to bring that

15:54

sugar level down. So, then you say,

15:56

"Okay, well, it doesn't matter because

15:58

the insulin the insulin is bringing the

15:59

sugar level down. So, what's the harm

16:01

done?" Because your A1C is still good.

16:03

You're not diabetic. No, but it's that

16:05

background high insulin that is

16:08

destroying your metabolism.

16:10

It's that high insulin level in the

16:12

background. So, insulin

16:14

pushes glucose

16:16

into the liver and you develop a fatty

16:18

liver.

16:19

It pushes the calories into production

16:22

of new fats around your viscera. The

16:24

viscera means in your belly.

16:27

Around your pancreas.

16:29

You get visceral fat. Now, this fat is

16:31

produced from glucose. It's a different

16:34

kind of fat. Look, if I gave you a high

16:36

calorie diet right now of all sorts of

16:38

foods, you'd put on weight everywhere,

16:40

okay?

16:42

But, if I give you glucose, you put it

16:44

on mostly in your stomach. And your

16:46

stomach will protrude. And that's called

16:48

visceral fat. It's on the inside. You

16:50

can't pinch it. It's on the inside.

16:53

>> Around your organs. Around the organs.

16:55

This

16:56

is very detrimental fat. And that's the

16:59

epidemic that we have today. And that's

17:01

a direct result of eating, you know,

17:03

starchy glucose carbohydrates, which

17:06

creates insulin, which creates this

17:07

downstream effect on

17:09

>> Wrong foods and eating too frequently.

17:12

Too frequently, okay. Because remember,

17:13

it's also the frequency of eating.

17:15

Because before that insulin level gets a

17:16

chance to down, you're already popping

17:18

yourself with more food. And hence you

17:20

develop insulin resistance. You get a

17:22

very high insulin. It takes this much

17:25

insulin now just to bring that sugar

17:27

level down. And then the one day

17:29

when you cannot control that sugar, now

17:33

that sugar will go out and now your

17:35

doctor will say you're a diabetic. But

17:37

by that time, you've already had 10

17:39

years of hyperinsulinemia.

17:41

So, what happens if by the time you make

17:43

a diagnosis of diabetes to say Well, now

17:45

your sugar is really high. It is high

17:48

because your body has not had the

17:49

ability to keep it down. Why? Because

17:52

even that high insulin could not keep

17:54

your sugar level down. You became a

17:56

diabetic. You lost that whole

17:58

opportunity of prevention. It's those 10

18:00

years. Look, by the time you're a

18:01

diabetic and you come and see Dr. J in

18:04

his cardiac clinic, you already have

18:06

coronary artery disease. Like that

18:08

28-year-old. The 28-year-old didn't have

18:11

diabetes. He already has coronary artery

18:12

disease.

18:14

By the time you are diagnosed as having

18:16

diabetes, you already have coronary

18:18

artery disease. We have a great

18:19

opportunity here to actually start

18:22

screening these patients with insulin

18:24

levels very early on. But most doctors

18:27

don't have the ability or knowledge to

18:29

do the insulin level testing.

18:32

But it should be done.

18:33

And someone like me, I'm 33 years old

18:36

now.

18:37

When does the damage begin?

18:41

It starts right now.

18:43

It starts right now. As soon as you

18:45

around 30, you're already starting to

18:47

have trouble. You have to But you know,

18:50

this is what I do. I look at that

18:52

patient walking into my room.

18:54

And if I see that he's got a belly

18:56

sticking out,

18:57

I already know he's probably got insulin

18:59

resistance.

19:01

Because all the fat is in here.

19:03

Because the fat that's in the stomach,

19:06

sideways he looks terrible, from the

19:08

back he looks great. His waist is

19:10

increased. Mhm. Mhm. He doesn't have all

19:14

the fat everywhere else in his body.

19:16

That's the phenotype of somebody who has

19:18

hyperinsulinemia.

19:20

That same person goes on a cruise, he'll

19:22

come back 5 to 10 lb more because he's

19:25

got so much insulin in his body. Insulin

19:27

is a storage molecule. Puts everything

19:29

away.

19:30

And it's very hard for him to lose

19:31

weight. Why is it harder? You The only

19:35

thing that will make you lose that fat

19:36

very quickly is to change your diet, of

19:38

course, but you have to do fasting.

19:40

Because fasting brings your insulin

19:42

level See, this is where fasting comes

19:43

in.

19:44

So, what does fasting do? Do you mean

19:46

fasting or a calorie deficit? Is it Or

19:48

is it the same thing in your view?

19:49

>> No, they're not the same thing.

19:51

They are not the same thing.

19:53

You see, when you don't eat, your

19:55

insulin levels come down because you're

19:57

not stimulating your pancreas anymore.

20:00

So, you want to bring your insulin

20:01

levels down. The best thing you can do

20:03

in the world is to do fasting.

20:05

Because there's no Look, if I just

20:07

simply cut down on my calories,

20:12

then there's a different physiology

20:14

that's going to take place in the body.

20:16

And when you fast, there's a totally

20:19

different physiology. When you cut down

20:21

on calories, the body senses that

20:23

there's caloric deficit. Your metabolic

20:26

rate changes. It actually slows down.

20:28

And the body will start breaking down

20:30

everything, muscles included. So, you

20:34

lose fat and you also lose muscles.

20:36

On the other hand, when you're fasting,

20:38

it's a different physiology.

20:40

Fasting is I've put on fat, now I'm

20:43

going to take it out of the bank. Now,

20:45

the bank is going to be available for me

20:47

to pull out my calories and use it now,

20:49

and you start burning the fat. So, in

20:51

the first 12 hours of a fast, you take

20:54

out all the glucose in the form of

20:56

glycogen from your muscles and your

20:57

liver.

20:58

After 12 hours, you start pulling the

21:00

fat out. And the first place the fat

21:03

comes out of is going going visceral

21:05

fat. That is why fasting benefits you so

21:09

much cuz it gets rid of that worst fat,

21:11

the fat that is very inflammatory. You

21:13

see, if I did a biopsy of your visceral

21:15

fat

21:16

versus a biopsy of, let's say, a fat

21:18

from on your buttock, Mhm. two different

21:20

types of fat. One is full of

21:22

inflammatory molecules,

21:24

the other one is not full of

21:25

inflammatory molecules.

21:27

One is producing interleukin 6 and tumor

21:30

necrosis factor,

21:32

and this other fat is not. These are two

21:34

different fat storages.

21:37

Visceral fat is very toxic. It's very

21:39

inflammatory.

21:41

And that is why patients who have

21:42

visceral fat make all these molecules.

21:45

So, when I do the blood test, I see

21:47

that, "Oh, you've got so much

21:48

inflammation. You've got interleukin 6

21:50

is high, tumor necrosis factor is high,

21:52

your CRP is running high." And one of

21:55

the reasons for this

21:56

is not just leaky gut and other things

21:58

that I look at, but in your case, it is

22:00

also because you have a lot of ectopic

22:03

fat. Ectopic fat. Ectopic fat is now

22:07

we're realizing is not just only around

22:09

the liver and around your pancreas, it's

22:11

also around your heart.

22:13

So, when we look at the coronary

22:14

arteries and we see all those fat around

22:17

the coronary arteries, we had a very

22:19

nice diagram right there, and you can

22:21

see that around each artery,

22:26

there's that yellowness.

22:28

That yellow is all plaque.

22:32

Fat. That's fat around that, and it's

22:34

plaque forming. It's plaque forming. It

22:37

stimulates plaque. It's inflammatory.

22:40

And now we can do CT scans that will

22:43

actually detect how much inflammation is

22:45

in the fat around the arteries as well.

22:49

Ectopic fat is in the

22:52

around the coronary arteries, it's in

22:54

your liver, and in your pancreas, and it

22:56

is very inflammatory. On this point of

22:58

fasting, so if you have someone come to

23:00

you and they're, you know, they have

23:02

that physique where there's a bit more

23:03

of that protruding belly fat, you said

23:05

that fasting is a much better approach

23:07

than just sort of a calorie restriction.

23:09

I was looking at some studies that said

23:11

research shows that calorie deficits of

23:13

any kind can reduce visceral fat, but

23:14

fasting will give it an edge because of

23:17

the insulin sensitivity stuff that you

23:18

talked about as well. What kind of fast

23:21

should one be doing? Because there's so

23:23

many different names for these fasts.

23:24

People do these 40-day water fasts and

23:26

they do intermittent fasting. It's a

23:28

great question. There are many, many

23:30

different types of fasting.

23:32

Depending on your goal on

23:35

what do you want for that particular

23:37

patient? So, if a patient is just simply

23:40

looking to reduce his visceral fat, then

23:43

I start with 12 12, which means 12 hours

23:47

you don't eat anything, you just drink

23:49

liquids with no calories in it, and then

23:52

12 hours is your feeding period. We

23:54

start with that.

23:56

And we do that for about 2 to 3 weeks,

23:58

then we quickly move to 18 6.

24:01

18 6 means 6 hours you get to eat. 18

24:05

hours it's only

24:08

water, black tea, black coffee, green

24:12

tea, no calories. 18 6. 18 6. And then

24:18

that's one type of fasting. Now, if a

24:20

patient is very overweight, a patient

24:23

has diabetes, and your goal is to

24:24

reverse the diabetes, the patient needs

24:27

to lose 60 lb, then those patients have

24:30

a special type of need. For them, I will

24:33

take them to a 48-hour fast once a week.

24:39

Sometimes I'll go to a 3-day water fast

24:42

every 9 days.

24:44

Every 9 days you will do OMAD. OMAD

24:48

means one meal a day only every day for

24:50

9 days.

24:52

And then you're going to give me a 3-day

24:53

water fast.

24:55

Is the same advice applicable to women?

24:58

Because obviously they're contending

24:59

with

25:00

a variety of hormone fluctuations and

25:02

estrogen and I know that the the the

25:05

female body responds differently to

25:06

these kinds of stresses like fasts.

25:09

I've been asked that question so many

25:11

times from patients as well. Most of the

25:13

women can actually handle it.

25:16

The only women that cannot are those who

25:18

are trying to become pregnant or or they

25:20

have um

25:22

or they're already pregnant. I I think

25:24

that women are not that different when

25:26

it comes to the fasting programs. So in

25:28

my experience, I've been able to get

25:30

women to fast. I just finished a fast on

25:32

one lady just now for a 72 days. 72

25:37

days. She was terribly overweight. She

25:40

had diabetes. She had hypertension. She

25:43

had hyperlipidemia.

25:45

She was having hip replacements, knee

25:48

replacements, joint problems. She had

25:49

skin problems. And we fasted her for 72

25:53

days. What did she have in those 72 days

25:55

in terms of drinks, electrolytes,

25:57

coffee? What was she Great question. So

25:59

she would have black tea, black coffee,

26:02

water.

26:04

And in the water once a day, I tell her

26:06

to put some electrolytes in there. So

26:08

there's an electrolyte called Element T

26:10

or sometimes I just tell them to go buy

26:12

some Celtic salt and put half a teaspoon

26:14

in there. And once a day you take that.

26:17

If you get cravings

26:19

and you feel really really hungry,

26:21

take some MCT oil, a teaspoon, and put

26:24

it in your water and you can drink that

26:26

as well. I think it's worth saying that

26:28

probably shouldn't try this at home,

26:30

ladies and gentlemen, because obviously

26:32

medical supervision is critical here.

26:33

But in the case of that lady, what was

26:35

the before and after of that 72-day

26:38

fast?

26:39

So

26:40

diabetes gone.

26:42

Blood pressure

26:43

normalized.

26:45

Weight loss, tremendous weight loss. I

26:47

mean, I think she lost about 55 60 lb.

26:50

Wow. Okay. And not only did she lose all

26:52

that weight, yes, all that weight from

26:54

her belly was gone, but even her face,

26:57

under the arms. So, when you lose weight

27:00

in a fasting program, it's very

27:01

different from losing weight when you

27:05

are restricting calories. You actually

27:07

retract your

27:09

your skin. So, you get real changes in

27:13

your entire body. These people I had one

27:15

patient that fasted for 183 days

27:19

under supervision.

27:21

Okay. So, he went from 400 lb to 210 lb.

27:26

And when he walked into the office, you

27:27

would not recognize that he's just lost

27:29

all this weight because he did not look

27:32

like skin on top of the bones and then

27:35

having to have surgery to remove all

27:37

that excess skin.

27:38

Fasting is a totally different

27:40

physiology.

27:41

In fasting, you you you you you the body

27:45

is doing a whole lot of things that are

27:47

very different and we can go into the

27:49

physiology of fasting because that's

27:51

fascinating. It's a physiology that has

27:53

not been used by us. We've lost it. So,

27:56

one of the things about modern living,

27:58

modern living, we have lost this

28:01

physiology of fasting. First of all, why

28:03

do we still have that physiology in us?

28:05

It's because it's supposed to serve a

28:07

purpose

28:08

because we're supposed to be fasting and

28:09

feasting.

28:11

But now we're only feasting all the time

28:13

and we're not fasting.

28:15

In order to go back to normal,

28:18

you should be fasting. Fasting is

28:20

supposed to be a normal part of your

28:22

existence. That's the way you were

28:24

designed, not this modern industrialized

28:27

living that we've been doing in the last

28:30

fraction of a millisecond in the total

28:33

existence of the human race.

28:36

We've changed our lifestyles so much,

28:39

modern man has, but our genetics and our

28:41

physiology has lagged behind. We are

28:44

supposed to use fasting and feasting as

28:47

part of our normal program, as our

28:49

normal physiology. That's why we still

28:52

have it. That is why after 12 hours, you

28:55

start making some more ketones in your

28:57

body. And the ketones come from fat.

29:00

You're moving that fat out of storage.

29:02

>> What is a ketone?

29:03

Ah, ketones. So, ketones are a energy

29:06

molecule produced by the liver. How does

29:09

it make it? It makes it from fat. So,

29:11

the fat gets liberalized and by the way,

29:13

the fats only start moving when your

29:15

insulin levels are down. So, because of

29:16

fasting, your insulin levels are really

29:18

low now, okay? Because you've been

29:20

fasting, right? So, now the fats start

29:23

dissolving. So, you get free fatty

29:25

acids. The free fatty acids float into

29:27

the bloodstream. Free fatty acids are

29:29

fat products. They float into the

29:31

bloodstream. They go to your liver. Your

29:33

liver converts those into ketones. Now,

29:36

ketones are an energy source of the

29:37

body.

29:39

An alternative source to glucose. So, in

29:42

general terms, you are either going to

29:44

be running on glucose and glucose

29:46

metabolism

29:47

or you're running on ketones.

29:49

Okay? And so, ketones basically show up

29:52

in the when glucose isn't around. That's

29:55

absolutely right. So, ketones and

29:57

ketones are actually a cleaner fuel for

29:59

the body.

30:01

And in terms of producing reactive

30:03

oxygen species in the metabolism, the

30:05

way your mitochondria work, you actually

30:08

produce less reactive oxygen species,

30:10

which is damaging to your physiology,

30:13

when you're in ketones. And ketones are

30:15

signaling molecules that also change

30:17

your physiology in a number of ways.

30:20

Number one, it causes the production of

30:23

brain-derived neurotrophic factor.

30:26

That occurs under ketogenesis.

30:28

Brain-derived neurotrophic factor, which

30:30

means that you become smarter.

30:33

You're growing new cells. Your reflexes

30:35

are better. Your visual acuity is

30:36

better. Why? Because nature wants you to

30:39

become a better specimen so you can go

30:41

out and get your next kill. So, why

30:43

don't we all just stay in a state of

30:44

ketosis then if it's so miraculous?

30:47

Well, that's a great question.

30:49

You don't want to be in ketosis all the

30:51

time cuz that's not what ketosis what

30:53

what our physiology was made for because

30:56

then you'll just be burning fats,

30:57

burning fats, burning fats all the time

30:59

and that's not a good state to be in

31:01

constantly either. So, you want to be

31:04

able to do both. You're supposed to get

31:06

your current account and your deposit

31:09

account. You're supposed to go work with

31:10

both of them. That's the normal

31:12

physiology. So, not only brain derived

31:15

neurotrophic factor increases, but also

31:17

stem cells. Stem cells are amazing.

31:21

And in my patients, of course, I see

31:24

that What What is a stem cell? Ah, stem

31:27

cells. So, stem cells are We all have

31:30

stem cells.

31:31

And we all still make stem cells.

31:34

And they're produced by the bone marrow.

31:36

These are pluripotent cells. Cells made

31:39

that will then go out and become

31:41

whatever they need to become.

31:44

So, they can go out into your

31:45

circulation, become a muscle cell, they

31:47

can become a retinal cell, a skin cell.

31:49

They can transform into anything. So,

31:51

what happens then when you break your

31:53

fast? Mhm.

31:55

You get a surge of stem cells coming out

31:57

of your bone marrow. Some of them become

31:59

immunocytes. That is why we know that

32:01

fasting also boosts your immunity.

32:04

People who fast get less infections, get

32:06

less sore throats and coughs and colds

32:08

and the viruses that are going around.

32:11

The immunity is better. The stem cells

32:13

are not I'm particularly interested in

32:15

the stem cells because of a thing called

32:19

the progenitor cells, endothelial

32:21

progenitor cells. Progenitor cells are

32:24

You see, you're always hurting your

32:25

blood vessels, the lining of the blood

32:27

vessels and the lining of your blood

32:29

vessels have to be constantly repaired

32:31

and they

32:31

they are repaired by the progenitor

32:33

cells. When you do intermittent fasting

32:36

and time restricted feeding,

32:38

you will produce more, and this has been

32:41

shown in numerous studies, more

32:43

progenitor cells. And these progenitor

32:46

cells go and repair your blood vessels.

32:48

So, I would do what Why am I interested

32:50

in this? Because I'm a vascular doctor.

32:52

I want my blood vessels to constantly be

32:54

repairing themselves from the damage

32:56

that we do in day-to-day life. So, stem

33:00

cell mobilization, brain-derived

33:02

neurotrophic factor,

33:04

growth hormone. You make more growth

33:06

hormone in fasting.

33:08

So, when should you exercise when you're

33:10

fasting?

33:12

At the peak of your fast. So, if I'm

33:14

going to break my fast at 6:00 p.m.,

33:17

I tell patients to go out to the gym at

33:19

4:00 in the afternoon.

33:22

But they say, "But doc, how can I do

33:23

that?"

33:24

You will find that you will put on more

33:26

muscle, and you will retain more muscle

33:28

as well, cuz you have higher growth

33:30

hormone. The fastest way and the best

33:32

way to actually increase your growth

33:33

hormone production is to do intermittent

33:36

fasting. When I start with um Dr. Stacy

33:39

Sims, who does a lot a lot of um

33:41

work on women's physiology and women's

33:44

performance, one of the things she said

33:45

to me is that if a woman in particular

33:50

tries to do a workout fasted, the body

33:54

will break down the muscle

33:56

in a woman because she said a woman's

33:58

body is much more the hip- hypothalamus

34:00

is much more sensitive to changes in a

34:03

woman's glucose levels cuz it's trying

34:05

to defend against, you know, pregnancy

34:06

and you know, all these kinds of things.

34:07

So, it shuts down the menstrual cycle if

34:09

there's not enough glucose in the blood.

34:11

And

34:11

and so, she said for women in

34:12

particular, you do you do want to have

34:14

eat something before you work out or it

34:16

will

34:17

your body will

34:18

take from the muscle. And it will

34:19

definitely won't The body is very

34:21

sensitive. It won't want to to help you

34:23

build muscle if there's not enough

34:24

glucose energy in the body. She said for

34:26

men, it's slightly different because

34:27

we're a little bit more robust, and we

34:29

were meant to hunt. So, if we were

34:31

fasted, our body is, you know, designed

34:33

to

34:34

kind of keep up its normal processes to

34:36

help us complete a hunt, per se. So, I

34:39

guess I throw this question out to you

34:40

about about women, exercise, and

34:42

fasting.

34:42

>> Yeah. I I think that's a great

34:44

observation. So, again, it's what kind

34:46

of exercise is the woman doing? You see?

34:49

Now, there's two kinds of basic

34:50

exercises. One is aerobic activity,

34:53

and one is uh resistance exercises. And

34:55

you can also throw into that

34:57

high-intensity interval training. Mhm.

35:00

So,

35:01

when it comes to the training that I'm

35:03

talking about in a fasting state, I like

35:06

resistance training.

35:07

So, in resistance training, that glucose

35:10

issue is not going to become a problem.

35:11

Now, if you're going to put that female

35:12

patient on a treadmill and make her run

35:14

for a long period of time, let's say

35:15

you're going to make her run for 40

35:17

minutes on the treadmill, I think that

35:18

yes, you are absolutely right. You are

35:20

going to deplete her glucose, and you

35:21

may cause some problems, and then the

35:23

muscle will start breaking down. But I

35:25

do believe, from all the experience that

35:27

I've had with my patients, that when I

35:28

tell them to do a hit, which is

35:30

high-intensity

35:32

interval training,

35:34

all the women do just as well as the

35:35

men. Because all you're doing is you're

35:37

intensely working the muscles, and

35:39

you're doing some resistance exercise,

35:40

and then you're totally resting as well.

35:43

And I see fantastic results with that.

35:45

In terms of ketosis,

35:47

do you

35:49

cycle in and out of a ketogenic diet?

35:51

And just for anyone that doesn't know

35:52

what know a ketogenic diet is a diet

35:53

that's very, very low in carbohydrates.

35:55

So,

35:56

less than 50 g of carbohydrates a day,

35:58

typically.

35:59

Do you cycle in and out of that diet?

36:01

Yes.

36:02

Yes, you have to. Because I don't want

36:04

them to stay in a ketogenic state all

36:07

the time. So, I do. I do. I do. I tell

36:10

them, "Look Look, what is your goal

36:12

here? What is your goal? If your goal is

36:15

that, okay, I need to lose 70 lb, then

36:17

you're going to do this till you get to

36:18

your goal. Once you get to your goal,

36:19

then you're going to go back to

36:20

time-restricted feeding, which means

36:22

18:6. So, you'll still make some

36:24

ketones, and then intermittently, you

36:27

can still do a 36-hour fast.

36:30

That means

36:31

normal healthy people, you and me, we

36:34

should still be doing one 36-hour fast

36:37

at least once a month. So, we'll go to

36:39

that. We'll go to that.

36:41

Fasting must still become part of your

36:43

pro- But, you don't need to stay in

36:44

ketogenesis all the time.

36:46

Remember also that when you are in

36:48

ketogenesis, you are

36:50

you are also in autophagy.

36:54

How much autophagy can you do? That

36:56

means you're recycling all your your

36:59

particle all your organelles inside your

37:01

cells. What does that mean in simple

37:03

terms for someone that doesn't know the

37:05

term autophagy?

37:06

>> So, bottom line is the cell senses

37:10

that

37:12

there is no new parts coming in here.

37:15

So, it takes the redundant

37:17

organelles inside the cells

37:20

and breaks them down.

37:23

Packages them up into these little

37:25

packets and exports them out of the

37:27

cells. So, these cells, all your cells

37:30

in the body, now are functioning at a

37:34

much more efficient level.

37:36

So, mitochondria

37:38

also have autophagy. So, you're getting

37:40

new mitochondria, you're repairing your

37:43

mitochondria, and you know it's all

37:44

about mitochondria, right? Your energy

37:46

Mitochondria are those organs

37:48

organelles inside each cell.

37:51

And we just think of them as only an

37:53

energy source. That only are my

37:55

mitochondria, they make ATP.

37:57

That's not the whole story.

38:00

Yes, they produce ATP.

38:02

But,

38:03

they also produce this thing called

38:05

reactive oxygen species.

38:07

And thereby, they influence

38:11

the metabolism of your of your cell as

38:14

well. They send signals. They send

38:16

signals to your to your chromosomes. So,

38:19

you start producing new proteins. You

38:21

start producing

38:22

molecules. So, your mitochondria are

38:26

very important. If you have old

38:27

mitochondria, you're going to have

38:28

fatigue, tiredness. But, when you get

38:31

mitophagy, one reason why you feel so

38:34

good after a fast, because you have new

38:36

mitochondria. They are much more

38:37

efficient in producing ATP and less

38:39

reactive oxygen species. So, you have

38:42

mitophagy,

38:43

recycling of your parts, cellular

38:45

function improves.

38:47

Toxins get out of your cells as well.

38:50

Toxins.

38:52

Do you think there's a

38:54

preferable or a best exercise for heart

38:57

health? Like, if you were going to say,

38:58

"Steven, to make, you know, my my I

39:00

think my family has a history of

39:02

heart-related issues. One of my my uncle

39:04

died from a heart attack. So, I'm very

39:06

aware of

39:08

my own susceptibility to heart-related

39:10

illnesses. I think there's other people

39:11

in my family um that have heart-related

39:13

issues, as well.

39:15

Is there a particular exercise that is

39:17

best for the heart? Cuz I'm guessing

39:19

resistance training, i.e. training,

39:22

might not be the

39:23

the number one recommendation for good

39:25

heart health.

39:27

You mentioned hit. I'm wondering if

39:28

that's

39:30

Yes.

39:31

So, I'm just going to tell you, broadly

39:33

speaking, what I see in my practice.

39:35

I see that people who do a overly do a

39:39

aerobic activity, that means they cycle

39:42

100 miles a day, or they're running on

39:44

the treadmill for 2 hours at a time

39:46

every day, or they're doing a lot a lot

39:48

of marathon training all the time, they

39:50

actually end up with more inflammation

39:52

in the body, and they end up with more

39:54

coronary artery disease than patients

39:56

who do short sprints, and patients who

39:59

do resistance exercises, and patients

40:02

who do hits.

40:04

So,

40:05

in terms of cardiology, you Look, you

40:07

need some aerobic training. Why? Because

40:10

you want to develop some endurance,

40:12

right? You don't want to be running

40:13

behind the bus and and get short of

40:14

breath just, you know, a few hundred

40:16

feet. So, for some endurance you do

40:19

aerobic activity. How much aerobic

40:21

activity, which is running on the

40:22

treadmill, for example, or just

40:24

sprinting?

40:25

Only about 15 to 20 minutes. That's it.

40:29

So, I tell patients, you want to run on

40:31

that treadmill, you want to bicycle, 15

40:33

to 20 minutes. And then you got to go

40:35

into resistance exercises. The

40:37

resistance exercises that I have seen

40:39

work best in my patients is simple

40:41

things. You don't have to go crazy. You

40:43

do floor exercises. You use your own

40:45

body weight

40:47

as resistance. So, you can do the planks

40:49

and leg lifts and all these other

40:51

exercises. And you can do hit. But they

40:54

have to be very specific. So, you

40:56

exercise really hard for about 30

40:59

seconds to 45 seconds and then you

41:00

completely rest for another 30 to 45

41:04

seconds.

41:05

Okay. And that's allowing your body

41:09

to clean up the reactive oxygen species

41:11

you just created through that exercise.

41:13

Because your rate limiting step in

41:16

cleaning up the metabolic mess that you

41:18

get when you get too much exercise. It's

41:20

a rate limiting step. Your glutathione

41:22

production, your superoxide dismutase

41:24

production.

41:25

These are chemicals that clean up the

41:28

metabolic mess that you get when you get

41:30

too much activity. That's a rate

41:31

limiting step.

41:33

So, when you rest, you exercise and you

41:36

rest, you exercise, you rest, you are

41:38

going to get a cleaner physiology.

41:41

With your patients, how many patients do

41:43

you think you've treated or worked with

41:44

in your career?

41:46

Ooh.

41:48

That's a

41:48

>> to guess into the nearest thousand in

41:50

your

41:50

>> have to be at least

41:52

nothing less than

41:54

nothing less than quarter million. How

41:57

do you deal with the heartbreak? That's

42:01

no pun intended, but I'm saying like how

42:03

do you deal with the heartbreak of

42:05

watching people's lives

42:08

either them losing their life or their

42:11

their lives being changed permanently

42:12

forever or dealing with the families of

42:14

of people who lose their husband or wife

42:17

or or son or daughter.

42:21

It it is very difficult. Especially when

42:23

we've been trying everything and still

42:25

things can go wrong.

42:27

Based on that, I realized that sometimes

42:29

you can do everything to try to extend

42:31

life, but you cannot life is still

42:33

finite.

42:34

We will pass, yes, but we want

42:37

better health span and we want a longer

42:40

life span. I want to give the patients a

42:43

better quality of life also. I can't

42:46

make you live forever.

42:48

I will see people die. Sometimes they

42:50

die in front of me while I'm even

42:52

holding their hands. They'll pass away

42:54

in the hospital right there.

42:56

But what I do want to do

42:59

is to give them a quality of life until

43:01

then that's going to change. And in all

43:03

this education,

43:05

it's all so that you can live an optimal

43:08

life

43:10

and then pass away just like that. Have

43:13

you seen many people that made all the

43:14

right decisions? They were healthy, they

43:18

had a good diet, they were doing

43:19

exercise, and they still end up

43:23

passing away on your hospital table

43:25

because of heart-related illnesses? Yes.

43:28

Can you give me give me one such

43:29

example?

43:30

Typical example, a fellow in the 60s.

43:33

His coronary calcium score was over

43:35

1,000. So, you know that's a CT scan

43:37

that we do

43:39

and it looks at calcium on the arteries

43:40

of the of the wall. So, all this plaque

43:42

that you you you you you showed the the

43:45

this yellow stuff here, all that has

43:46

calcium in it. So, the more calcium you

43:49

have, the more plaque you have.

43:52

So, he had a calcium score of over

43:53

1,000.

43:54

So, sit down and talk with him and say,

43:56

"Okay, so tell me, why do you have all

43:58

this calcium? No diabetes, no high blood

44:01

pressure. Doc, I don't even drink. I

44:03

don't do anything. I don't smoke. I

44:07

exercise regularly. I go to the gym. And

44:09

then the question is, why do you have

44:11

all this coronary calcium in your

44:12

coronary arteries now?

44:14

And

44:15

it turned out that ever since he was a

44:18

kid, he had a bad microbiome.

44:21

And in him I found that that was the

44:23

reason why he got it. So he was shocked.

44:25

When he left the office, he was totally

44:28

shocked cuz he thought that being

44:30

constipated and having a bowel movement

44:32

every 3 or 4 days

44:35

is just normal cuz it was always like

44:37

that for him.

44:39

Until I dug into it and found out that

44:42

he's actually got a lot of GI problems.

44:45

And that's what probably caused him to

44:47

have inflammation. And that's what's

44:49

probably caused him to have coronary

44:51

artery disease. That's why he's got all

44:53

this coronary calcification.

44:56

Yeah. So let's talk about the gut

44:57

microbiome then.

44:59

You you mentioned earlier that that's

45:00

really pertinent in

45:02

cardiovascular issues. Can you explain

45:04

to me exactly why and how that happens?

45:08

The biggest difference between you and

45:10

your outer environment is actually your

45:13

gut.

45:15

Not outside your skin.

45:17

It's actually the gut. There's a hundred

45:19

trillion bacteria sitting in there. And

45:21

there's probably 400 trillion viruses

45:23

sitting in your gut.

45:25

And they're not just hitching a ride for

45:26

you. They are there doing something.

45:29

There's a symbiotic relationship between

45:31

the gut

45:33

and you. They produce micronutrients

45:36

which get absorbed into your

45:37

bloodstream. If the right microbiome is

45:39

not present, you are being deprived of

45:41

those micronutrients. For example,

45:43

short-chain fatty acids which are a

45:45

special kind of uh thing. But also, they

45:48

maintain the integrity of the lining of

45:50

the intestines.

45:52

If the lining of the intestines is

45:54

breached,

45:55

then bacterial wall products get into

45:58

the bloodstream. And the first place

46:00

they'll get to is the liver.

46:02

Hence, you get a fatty liver.

46:05

There are basically three reasons for

46:07

fatty liver. Either you're drinking too

46:09

much alcohol,

46:10

or you get a leaky gut,

46:12

or you have too much insulin, cuz

46:14

insulin causes the conversion of glucose

46:17

into fat, right? So, you get a fatty

46:19

liver. When you say leaky gut, So,

46:21

that's a leaky gut.

46:22

>> Ah, okay. So, leaky gut is when

46:24

the microbiome is dysfunctional. The

46:26

microbiome that is dysfunctional causes

46:29

the integrity of your intestinal lining

46:32

to be compromised.

46:33

>> Ah, okay.

46:34

>> Yeah. So, now molecules that are

46:37

supposed to stay in the gut,

46:39

dead bacterial wall products, they're

46:41

called lipopolysaccharides,

46:44

they're supposed to stay in the gut, but

46:46

now they get into the bloodstream, they

46:48

go up the portal vein,

46:50

and up the portal vein, they'll end up

46:52

going straight to the liver.

46:54

And when they end up in the liver, they

46:55

cause that fatty liver. Now, you get

46:58

this big fatty liver, which causes a lot

47:00

of inflammation. A fatty liver

47:03

is an epidemic of this going on today.

47:06

At least 25% of the population now have

47:08

a fatty liver. And what is upstream from

47:11

that? Is it Is it alcohol and all these

47:14

processed foods and things like that?

47:16

It's basically processed foods,

47:18

sugar,

47:20

and leaky gut. And of course, toxins,

47:22

alcohol being the biggest toxin. Okay.

47:26

This is the seat of metabolism.

47:28

So, as a cardiologist, I became

47:29

interested in this because I found that

47:32

if you have a fatty liver, you're going

47:34

to be more prone to inflammation,

47:37

and you're going to be more prone to

47:38

coronary artery disease. When we fix the

47:41

fatty liver, we change the diet through

47:43

fatty by by changing your diet, and now

47:46

you start eating real food and not

47:48

processed foods and products, when we

47:50

increase the fiber intake so that the

47:52

good bacteria start coming back into

47:54

your gut, mhm? And when I give you

47:57

certain supplements to restore all your

48:00

nutrition in your body, then all of a

48:02

sudden the progression of coronary

48:04

calcium flatlines out. You see, let's

48:07

say you came to me for test and I did a

48:09

coronary calcium score and it's 500.

48:12

And 2 years from now it's 800.

48:15

That means, wait a second, something's

48:17

not right here, right? Your coronary

48:19

calcium should not be going up.

48:22

Now, I can't get rid of the coronary

48:23

calcium because that's plaque in the

48:25

walls of your arteries, but it shouldn't

48:27

keep going up because one day it's going

48:29

to be 2,000. So, how do I follow how

48:32

well my treatment for you is is I follow

48:35

the coronary calcium score. And again,

48:37

the coronary calcium score is a marker

48:39

of the amount of plaques Correct. around

48:43

the heart and in the vessels of the

48:45

blood. Yeah, in the wall.

48:47

>> Okay, so you want low

48:49

coronary calcium. Like my calcium score

48:50

is zero. You shouldn't have any coronary

48:53

calcium in the walls of the arteries.

48:55

So, we have the yardsticks now We should

48:58

probably We should probably

48:59

differentiate between calcium

49:01

supplements and the calcium that you're

49:03

talking about. So pertinent. Calcium

49:06

supplements actually increase the risk

49:08

of cardiovascular events. Calcium

49:11

supplements should not be taken. The

49:12

problem is not calcium supplements. The

49:14

problem is lack of D3 and lack of K2.

49:18

So, we shouldn't be taking calcium

49:19

supplements.

49:20

>> Absolutely not. Absolutely. I stop all

49:22

calcium supplements on all my cardiac

49:24

patients.

49:25

And I tell them, you should take vitamin

49:27

D3 so you'll absorb calcium better into

49:30

your gut and you take vitamin K2 because

49:34

vitamin K2 is going to make sure that

49:37

you don't get the calcium build up in

49:39

the wrong places, in your vasculature

49:40

for example. Just to give some context

49:42

on the research. Some Some large

49:44

observational studies suggest that

49:45

high-dose calcium supplements,

49:47

which is especially, um, above 1,000 mg

49:50

a day from pills, may increase the risk

49:52

of heart attack and stroke, while others

49:54

find no clear link.

49:55

Yeah. Okay.

49:57

Yeah, no. I Look, also they advise high

50:00

calcium supplements in women to prevent

50:03

osteoporosis, for example, right? And

50:05

osteopenia. Now, I live in Florida, and

50:07

all these young women, they come to me

50:09

in their 30s, they all have osteopenia.

50:11

And the question is that they've got

50:13

calcium supplements put inside the milk,

50:15

in the orange juice has calcium in it,

50:18

and they're taking calcium supplements,

50:20

and why would they And they have

50:21

sunshine, and why would they have

50:23

osteopenia? And the reason is D3.

50:26

You need D3 and K2. That's what you

50:28

need. It's a hormonal thing. It's not

50:30

the elemental thing.

50:33

So, when people are on blood thinners,

50:35

do they typically have low vitamin K1?

50:40

If you're taking Coumadin, Coumadin is a

50:42

type of blood thinner Yeah. that lowers

50:44

your your vitamin K1 levels. And it will

50:47

also lower K2. So, Coumadin will lower

50:50

K2 as well.

50:52

So, you will get increased coronary

50:54

calcification, and that's been

50:56

documented. So, patients who have

50:58

who are taking Warfarin, for example, or

51:00

Coumadin, have been shown to have

51:02

increased coronary calcification because

51:05

of the fact that the K2 levels come down

51:08

as well as the K1 levels. Both come

51:10

down. Both So, you need vitamin K2. And

51:13

do you just do supplement that, or would

51:15

you get it from dietary? I supplement it

51:17

on all my patients because diet comes

51:19

from fermented foods. And we don't eat

51:21

enough fermented foods to contain enough

51:24

vitamin K2.

51:26

One of the reasons why I do like my

51:28

supplements is because today's food is

51:30

not as rich

51:32

as it was in the past.

51:35

So, we're not eating the variety of

51:36

foods, but also we're not getting enough

51:38

vitamin K2 in our regular diet. So, if

51:41

you want to give me advice then on

51:42

having a great gut so that I don't end

51:43

up with leaky gut, which will cause all

51:45

of these downstream effects, what is

51:47

What is the advice you'd give me to have

51:49

a perfect gut?

51:50

Number one,

51:52

lots of fiber.

51:54

Because the fiber is going to be eaten

51:56

by your bacteria, and you're going to

51:58

get a wide variety of good bacteria

52:01

eating fiber. If you eat a lot of sugar,

52:04

you will get bacteria, too, but they are

52:05

the bad ones, the ones that you don't

52:07

want. So, you're going to get a

52:09

condition called SIBO, for example.

52:11

But you want to eat lots of fiber, so

52:14

because our diets today don't give us

52:17

enough fiber,

52:19

I do advise all my patients to take

52:20

inulin with FOS. It's called inulin with

52:24

FOS. It's a powder, it's a soluble

52:26

fiber, and I just tell them to take one

52:28

scoop in your water every day, or put it

52:31

in your whatever you're going to have,

52:33

whatever liquid you're going to have.

52:35

Number two,

52:36

fermented foods.

52:38

The fermented foods will give you not

52:41

only the bacteria themselves, because

52:43

they are For example, called kefir has

52:46

so many different strains of good

52:47

bacteria, but they also have the

52:49

postbiotics.

52:51

What is a postbiotic? A postbiotic, for

52:53

example, vitamin K2. It's a product that

52:56

the bacteria make.

52:59

Short-chain fatty acids. That's a

53:01

product that the bacteria make. So, you

53:03

want bacteria, you want good bacteria in

53:06

your gut, and they will stabilize the

53:09

ratio of all the other bacteria in your

53:11

gut. So, the keystone species have to

53:14

come back into your gut. I was reading

53:16

some

53:17

research around fiber,

53:19

and it says that in the United States,

53:22

women are 40% fiber deficient, and men

53:25

are roughly 50% fiber deficient. And in

53:29

the UK,

53:30

we're about 40% fiber deficient.

53:33

I don't think people know that. Where do

53:35

I find fiber? What What What do I need

53:37

to be eating to increase my fiber?

53:40

Fiber is found in anything that's

53:41

non-processed

53:43

because the very act of a processed food

53:46

is to take out the fiber so that you can

53:48

increase the shelf life of it. So, any

53:50

processed food, anything that's

53:52

manufactured in a factory, for example,

53:54

anything that already comes ready-made,

53:56

they've taken the fiber right out of it.

53:58

So, you got to eat real food. So, you're

54:01

going to eat real vegetables. And that

54:03

too, a variety of vegetables. You see,

54:05

you're supposed to eat 30 to 40

54:06

different types of vegetables a week and

54:09

we don't tend to do that. So, you want

54:10

fiber from a variety of sources. So, we

54:13

got to eat a little bit of all these

54:15

different different type of vegetables,

54:16

including spices. They all count as one

54:19

vegetable. So, that's something I I

54:21

stressed to everybody that you got to

54:22

have variety. You got to have all these

54:24

different fibers because they will

54:25

foster the growth of different types of

54:27

bacterial species in your gut. What

54:29

about lifestyle recommendations for

54:32

improving my gut? Things like, I don't

54:34

know, stress, uh sleep, exercise.

54:36

Anything

54:36

>> Sleep is absolutely important. So, the

54:39

the the next thing is actually actually

54:41

sleep and stress because lack of sleep

54:44

causes a change in your gut microbiome.

54:46

You got to sleep 7 hours a night. And

54:51

cutting down on sleep is going to affect

54:53

your entire physiology in your body and

54:56

your repair processes and your vagus

54:57

nerve and your delta sleep and but also

55:01

your gut microbiome. One night you'll

55:03

get insulin resistance. One night of bad

55:05

sleep, you become insulin resistant the

55:07

next day.

55:09

If you're changing

55:12

If you're changing time zones very

55:14

frequently,

55:15

the

55:16

liver dysfunctions

55:19

and the gut microbiome also

55:21

dysfunctions.

55:23

So, we got to be more cognizant of that.

55:26

That lifestyle is very important.

55:29

And we have to have plenty omega-3 in

55:33

the diet.

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56:37

You talked you talked earlier on about

56:38

toxins. Yeah. What do you mean by

56:41

toxins, and how what role is toxins

56:42

playing in my physiology, in my heart

56:44

health, in my gut health?

56:46

Yeah. So, and what is a toxin?

56:49

A toxin is a molecule

56:52

that should not be in your body. You

56:54

should not have been exposed to it.

56:57

So, let's take a pesticide or a

57:00

herbicide. These are chemicals

57:03

which man has made

57:05

and not perhaps adequately tested it in

57:08

in um

57:10

in rigorous studies, and yet these

57:12

chemicals get into the body. And we know

57:14

that many of them are estrogen receptor

57:16

disruptors, and that causes inflammation

57:19

as well.

57:20

So, if you look at pesticides and

57:23

herbicides,

57:25

we look at plastics,

57:27

forever chemicals, for example, PFAS and

57:31

other chemical BPA.

57:34

These chemicals all disrupt our

57:36

metabolism in our body.

57:38

And then, of course, mold is a toxin.

57:42

And lastly, heavy metals are toxins.

57:46

When the gut is not working properly,

57:49

you're going to be more prone

57:51

to toxicity because the gut bacteria

57:53

normally grab a lot of those things and

57:55

take them out in your stools. So, when

57:57

we look at toxicity, we look at gut

58:00

health and the liver health. The liver

58:02

is supposed to get rid of a lot of

58:04

toxins, but

58:06

in today's day and age, because of the

58:08

foods that we're eating, the environment

58:10

that we breathe, the environment that we

58:12

walk out into, we're getting too many

58:15

toxins and this poor organ here is

58:17

getting overwhelmed and cannot detoxify

58:20

adequately. So, when I do a blood test,

58:24

urine sample, and a stool sample, I get

58:26

a very good bird's-eye view of what's

58:29

going on with their ability to detoxify

58:32

and which levels are high. And I'm

58:34

finding

58:36

that many of my patients who have gut

58:38

issues and liver issues are very, very

58:40

high with not only heavy metals, but

58:43

they also have a lot of

58:46

pesticides, herbicides, and mold in the

58:50

body.

58:51

And mold, I'm finding a lot of mold. And

58:54

mold and all these extraneous toxicities

58:58

cause inflammation. Inflammation is a

58:59

reaction to something that your body

59:01

shouldn't have. Where is the mold coming

59:03

from?

59:04

Mold is very interesting. I've only

59:06

started researching it in the last 1

59:08

year.

59:09

Basically,

59:11

almost 70% of homes these days have some

59:14

form of mold toxicity in them, either

59:17

from previous water damage or exposure.

59:21

And mold is ubiquitous. It's It's just

59:24

about everywhere. And it can reinoculate

59:27

you every time you're in that

59:28

environment and it causes your innate

59:32

immune system

59:33

to start reacting to it and causes

59:36

low-grade inflammation

59:39

in your body.

59:40

And when I get rid of the mold, all of a

59:42

sudden I'm noticing that the patients

59:43

are not only feeling better but the rate

59:46

of progression of coronary artery

59:47

disease flattens off. I have this uh

59:50

steam room in one of my houses and uh I

59:52

went in there the other day and I

59:53

smelled I could smell it smelled a bit

59:55

funny.

59:56

And I recognize that smell from when I

59:58

was younger as being mold. So I like I

60:01

Googled it. I said what what is that

60:02

like earthy strong pungent smell in my

60:05

steam room? I think what's happened is

60:08

part of the like seat has come away from

60:09

the wall so it's not sealed anymore and

60:12

I think the moisture has got in there

60:14

with the water and I think it's moldy

60:16

and ChatGPT said to me when I was asking

60:18

ChatGPT, said it smell near the like

60:20

cracks. Yes. And if you smell it if it

60:22

smells really strong near the cracks

60:24

then that's mold and I smelled near the

60:25

crack and it was really really strong. I

60:27

remember thinking oh like so I had I not

60:29

known that would have been sitting in

60:32

there 30 minutes a day inhaling all of

60:35

that strong mold smell.

60:39

And from doing this show I've I've

60:40

learned that the downstream impacts that

60:42

can have on your body and your

60:44

cardiovascular health and everything in

60:45

between is really really bad. So I

60:47

I had a team come over and just uh fix

60:49

everything, repair it and do a deep

60:50

clean but I don't think people are very

60:52

aware of

60:53

how mold which is the kind of thing you

60:55

think of as like oh the the the bread

60:57

has gone green whatever can have a

60:59

chronic downstream impact.

61:01

>> And also this mold can come in and then

61:04

become part of your microbiome. So it

61:06

can actually come into your So let's say

61:08

you get out of this place

61:09

but if the mold is still in your gut or

61:11

if it's in your sinuses for example and

61:14

you get chronic sinusitis, you're

61:16

re-inoculating yourself over and over

61:18

again with this mold. And this is an

61:20

area of great interest and a lot of

61:23

research needs to be done. It is real

61:26

and we've known about it for a long

61:27

time, but the medical profession as a

61:29

general has not really taken this and

61:31

run with it, but it is very important.

61:33

And we certainly

61:36

looking at mold toxicity in our practice

61:38

because we find that it is actually more

61:40

prevalent than we ever ever imagined

61:41

before and we know the chemistry. We

61:44

know that it causes a a systemic

61:46

inflammatory reaction in the body and

61:48

it's reflected in the blood work. We see

61:50

that the CRP is up. We see that the

61:52

complement levels are high. We see that

61:55

the certain ratios of certain

61:56

inflammatory molecules are off. So, we

61:58

will do want to treat mold.

62:00

We want to treat sources of mold

62:02

toxicity both in the gut as well as in

62:04

the sinuses. And yes, people are

62:07

surprised when I tell them that that

62:09

affects the cardiovascular health. When

62:11

we talk about the clogging of arteries,

62:14

why is it that healthy people still end

62:17

up sometimes with these clogged

62:18

arteries? I think it's because they have

62:21

sources of inflammation that we have not

62:23

identified. For example, we say, "Oh,

62:26

you have no cholesterol, you have no

62:28

smoking, you have you you're exercising,

62:30

you're not overweight, you don't have

62:31

diabetes. Well, then I don't know why

62:33

you got all these odds." Well, there's

62:35

always if you dig deeper inside, there's

62:37

always something. And we'll find the

62:39

source of the inflammation. There's

62:41

always a reason why you get hardening of

62:43

the arteries. What's in that list? In

62:45

that list is mold. Mold, yeah.

62:48

Leaky gut. Yeah. Huge. Food

62:51

sensitivities. Oh, let me tell you about

62:53

food sensitivities. So,

62:55

another reason for the leaky gut

62:58

is that certain foods punch holes into

63:02

your intestines in a in a in a sense and

63:04

causes a leaky gut. And I've had at

63:06

least one patient in the last 6 weeks

63:09

that I can remember

63:10

who

63:11

actually has celiac disease.

63:14

And he didn't even realize he had celiac

63:15

disease because he had no constipation

63:17

diarrhea and he had premature coronary

63:20

artery disease and we identified that he

63:23

had celiac disease. We cut out all wheat

63:25

from his diet and that's going to stop

63:28

this inflammatory process from

63:30

occurring. See, it's all about

63:31

inflammation. We need to get rid of

63:32

inflammation in order to get rid of

63:34

coronary artery disease. Any source of

63:36

inflammation.

63:37

Get rid of the source. So, if the

63:41

pesticide levels are high, herbicide

63:43

levels are really high, get off

63:45

that source. Maybe you may maybe you're

63:48

living on a golf course and you smell

63:49

those chemicals every day. Maybe you

63:51

using it yourself in your yard. Maybe

63:53

you have toxicity coming from mold as

63:55

you mentioned, but we got to find the

63:57

source of it. So, a thorough evaluation

64:01

of the gut microbiome, a thorough

64:03

evaluation

64:05

of toxins in your body and a nutritional

64:08

status.

64:10

Your body is able to work and get rid of

64:14

this type of pathology if you have good

64:17

nutrition.

64:18

We don't have good nutrition today.

64:20

Let's talk about nutrition then. So, if

64:21

I wanted to end up on your surgical

64:24

table, if I if that was my goal,

64:28

what would you recommend

64:30

that I ate

64:32

and yeah, consumed? You should eat

64:35

nutrient

64:37

deficient

64:39

processed

64:40

foods

64:41

every day.

64:43

Give me some examples of of things.

64:45

Pringles and

64:47

Correct. So, it'll be things made out of

64:50

wheat, refined wheat, biscuits, cookies,

64:56

cakes,

64:57

lots of bread,

64:59

basically

65:01

fast foods,

65:03

foods that come in a box, ready-made

65:06

food with labels,

65:09

anything that comes in a packet that

65:11

you're open up.

65:13

Including some of these

65:16

that are

65:18

high protein bars or some of these

65:20

because they have all sorts of other

65:21

chemicals in them.

65:23

And I would have

65:25

lots and lots of um

65:27

artificial colorings in my food in all

65:30

those processed foods.

65:32

Um artificial sweeteners

65:35

lots and lots of diet drinks and sugary

65:37

drinks. Orange juice? Orange juice is is

65:41

on my list because that produces such a

65:43

profound increase in my glucose level

65:45

and then my insulin spike that I'm going

65:47

to get afterwards will cause me to

65:49

become very hungry 3 to 4 hours later.

65:52

So, yeah. I would not eat real food.

65:57

And you'll end up on my table.

65:59

Also, alcohol, smoking

66:01

>> Alcohol

66:03

No fiber because none of those foods

66:04

will have fiber in them. None of those

66:06

processed foods have fiber in them. Your

66:08

patients must

66:11

come to you at times and tell you what

66:12

they're doing

66:13

uh in terms of what they're eating and

66:15

lifestyle choices. And there must be

66:17

certain things which

66:18

very people aren't aware aren't healthy.

66:22

You actually mentioned bread.

66:24

Yeah.

66:25

Is all bread bad? No, not all bread is

66:28

bad. Most bread is bad. I don't mind

66:31

some sourdough because it's fermented.

66:33

So, it's going to get rid of some of

66:35

those lectins that are already in the

66:36

flour. So, those lectins are gone out

66:38

because they've been fermented. So, it's

66:40

probably a little bit okay. But again,

66:43

not large amounts of it, you know, a

66:44

slice once or twice a week is fine.

66:48

But really, it's a survival food. Bread

66:50

is a survival food, just empty calories.

66:52

That's all it's going to do.

66:53

>> Is there like a worse bread? Is there

66:55

like one bread which is

66:56

Yeah, pure white bread, simple white

66:58

bread. The simple light white bread is

67:00

pure calories, 70 calories right there.

67:03

You're talking about just eat 10

67:05

teaspoons of sugar instead in one slice.

67:08

I mean, it just doesn't make any sense

67:09

whatsoever. White rice?

67:11

White rice. I

67:13

White rice is a staple food of many

67:15

people and they cannot live without it.

67:17

So, I run into this problem all the

67:18

time. So, I tell them that this is what

67:20

you're going to do. You're going to

67:21

first and foremost soak your rice in

67:23

water and then discard the water after

67:26

an overnight soak because it contains

67:28

arsenic in it and other heavy metals.

67:31

You'd be surprised how much arsenic

67:32

there is in rice these days.

67:34

You see? See the problem we have? Our

67:36

sourcing of our foods.

67:38

I mean, here is a cardiologist has to

67:40

talk to his patients about how to cook

67:41

rice.

67:43

What is arsenic for people that don't

67:44

know?

67:45

It's a heavy metal that is found in the

67:48

soil and the water. It's in contaminated

67:51

soil and water and in this paddy fields

67:54

are now full of arsenic. It's a poison,

67:56

isn't it? People I watch a lot of true

67:58

crime. People kill each other with

67:59

arsenic.

68:00

It's a very, very strong poison and

68:03

it'll slowly kill you.

68:05

Is there anything else on that list of

68:07

thing misconceptions? Cuz

68:09

>> So, so, so the rice has to be soaked,

68:11

then you'd get rid of the water, then

68:12

you cook it

68:14

with lots of water, throw away the rice

68:15

again. I mean, the I throw away the

68:17

water, not the rice. And then, you cool

68:20

the rice in the refrigerator.

68:23

And then, you eat the rice the next day

68:25

by reheating it. Now, what you've done

68:26

is you've created resistant starch.

68:30

Now, what's going to happen is that that

68:31

rice is not going to have the same

68:34

insulin effect in your body and weight

68:36

gain effect and changes in your

68:38

metabolism because it's now got

68:39

resistant starch. Resistant starch means

68:42

that those

68:43

molecules have all bound to each other.

68:45

They don't They resist absorption and

68:47

digestion until they get to your colon.

68:49

And then, who's going to eat it? Your

68:50

bacteria. So, you're basically feeding

68:53

the bacteria with your rice. So, if

68:55

you're going to have rice, this is the

68:57

way you're going to do it. So. Is there

68:59

anything else on that list that

69:02

people should be thinking about that

69:04

they probably consider to be healthy

69:05

right now. Yeah, yeah, yeah, yeah, yeah,

69:07

yeah. So, it's also what you do to your

69:09

food. Look, you take a nice piece of

69:12

fish, for example, and if you burn it

69:16

and you blacken it really bad,

69:18

you're creating what is known as

69:20

advanced glycation end products.

69:22

You fry something, you're creating

69:24

advanced glycation end products. So, let

69:26

me define that for you.

69:28

If nature does not have the ability on

69:31

its own

69:33

to take

69:35

glucose,

69:36

protein, and fat and combine them at

69:38

very high temperature.

69:41

But you can do it in your air fryer

69:44

and you can do it outside in in in you

69:46

when you're blackening your food and

69:48

over blackening your food, it's called

69:49

advanced glycation end products. So,

69:51

when you over burn your food, when you

69:53

overcook your food, you're creating

69:55

these molecules. Now, you're consuming

69:57

these molecules and they've been shown

69:59

to cause a radical increase in the

70:01

inflammation in your body cuz your body

70:03

reacts to those products. There's

70:05

receptors to get rid of those, they're

70:06

called RAGEs, and they get rid of these

70:09

products, but you overwhelm them and now

70:11

you get inflammation.

70:13

And is there any other watch out foods

70:15

that

70:16

you know, people think are healthy but

70:18

maybe if if I was trying to have an

70:19

optimally healthy heart, I should avoid?

70:22

This is going to surprise you.

70:25

Excess of fruit.

70:27

Cuz I'm not a very big

70:29

follower of fruit. Fruit should be eaten

70:31

only in season cuz it has too much

70:32

fructose in it. Fructose being the

70:35

sugars. Fructose is a sugar and fructose

70:37

causes fatty liver.

70:39

So, our over consumption of fruit is

70:41

another factor

70:44

that is contributing to coronary artery

70:46

disease and diabetes and fatty liver.

70:48

And we think of fruit as being a healthy

70:50

thing.

70:52

You see? But fruit should only be

70:53

consumed really in the fall and in

70:56

season and in small amounts. Cuz that

70:58

high fructose level really causes major

71:02

changes in your metabolism.

71:04

So, I'm not too fond of fruit. You can

71:05

eat fruit in season, small amounts of

71:07

it. But, you know, people go crazy about

71:10

fruit.

71:11

I mean, I had a patient who was having

71:13

mental problems, as well as

71:15

cardiovascular disease, and he That's

71:17

all he lived on, is fruit. Morning,

71:19

lunch, and dinner. And the moment we

71:21

stopped that, his health changed

71:22

completely. He got so much better.

71:25

So, excess of fruit is also no good.

71:28

Excessively cooked foods, processed

71:30

foods, refined products. And another

71:32

one, vegetable seed oils.

71:35

So, any food that you prepare or you buy

71:38

that contains vegetable seed oils is a

71:41

no-no.

71:42

Cuz vegetable seed oils are a product.

71:45

And if you look at all of them, they're

71:48

exceedingly high in omega-6. And the

71:51

ratio of omega-6 to omega-3

71:54

is what the problem we have today.

71:57

We have far too much omega-6, very

72:00

little omega-3. So, anything that

72:02

contains vegetable seed oils So, I

72:05

always tell my patients, get rid of that

72:07

vegetable seed oil bottle from your

72:08

home.

72:10

So, what oil should I use instead? The

72:12

oil at home, if you're going to use any

72:14

oil, is going to be olive oil,

72:16

extra-virgin olive oil for your

72:19

for your salad.

72:20

And if you're going to do high

72:21

temperature, put a small little pat of

72:23

butter. Or use a little bit of ghee.

72:26

Or you can use a little bit of coconut

72:28

oil.

72:29

This is a bit more of a different

72:30

question, but you said earlier on that

72:32

when a patient walks into your office,

72:33

you can kind of look at them

72:35

and generally see if their

72:36

cardiovascular health is intact. And

72:39

yeah, we're generalizing here. But, if

72:41

I'm sat at home right now,

72:43

and I'm trying to figure out if I'm

72:45

healthy

72:46

as it relates to my cardiovascular

72:48

health, what are the symptoms of

72:51

cardiovascular poor health. Like what

72:54

throughout the whole body? What what

72:55

should I can I sort of obviously don't

72:57

want to anyone to self diagnose at home,

72:59

but what kind of symptoms should I be

73:00

looking at when I just you even when you

73:02

look at me. Let's do this on me to make

73:04

it easier.

73:04

>> Yeah. So number one, look at your

73:06

weight. Okay. If you are overweight,

73:09

that's a that's a

73:11

that's one strike against you already.

73:12

Number two, if you overweight, where is

73:14

it? It's mostly on the belly? I'm

73:15

overweight according to the BMI.

73:19

But again, you got to look at

73:21

BMI, you know, is a little crude. It's

73:23

not a very accurate way of actually

73:25

looking at it because some people are

73:26

very muscular and the BMI of 28 is

73:28

actually perfectly okay for them. It's

73:30

the constitution I look at and it's

73:32

visceral fat. Now I can't measure your

73:34

visceral fat by looking at you, but I

73:36

can tell if your belly is protruded. Now

73:38

in my office, I do visceral fat

73:40

analysis. So what else I I I don't have

73:43

a protruding belly per se.

73:45

Um

73:46

what else should I be looking for to

73:48

understand if my cardiovascular health

73:50

is intact. Is there I know

73:52

Just by looking at you physically? Yeah.

73:54

No, I think that's it basically is your

73:55

BMI and your overall weight and must not

73:58

have a belly. Just don't have a belly.

74:00

If you have a belly, you have a problem.

74:03

And what about my joints and stuff like

74:05

that? Is that an indicator of Yes. If

74:08

you have joint now symptoms, if you

74:09

having joint pains, look, if you having

74:12

joint pains, people think oh it's just

74:14

degenerative joint disease.

74:16

In my experience, what I have seen, it's

74:18

all inflammation. Because when I change

74:20

the diet, when I fix the gut issues,

74:23

when I fix

74:24

the inflammation in the body, they all

74:26

come back and say the joints got better.

74:28

All of them uniformly. Especially

74:30

patients who have autoimmune disease.

74:32

For example, many patients have

74:34

rheumatoid.

74:35

And the moment we change the diet, the

74:37

lifestyle, the frequencies of eating,

74:40

introduce a little bit of fasting. So we

74:41

do everything, right? Right? We've

74:43

changed the gut microbiome. We've we've

74:45

made the liver better by by by giving

74:48

them some supplements. We do

74:49

intermittent fasting. We do some

74:51

exercises. We give them some stress

74:53

management. We improve their sleep. All

74:55

of a sudden,

74:57

the joint pains get better. Rheumatoid

74:58

gets better. Skin diseases get better.

75:00

Psoriasis gets better. Cuz it's the

75:02

fundamental underlying part is all

75:05

inflammation. This is a random one, but

75:08

I'm just connecting dots from different

75:09

podcast guests I've had on the show.

75:10

What about bad breath?

75:13

Bad breath?

75:14

Bad breath is real.

75:16

But what is it is my oral microbiome

75:18

linked to my heart health? Yes. There's

75:21

a There's a microbiome that goes all the

75:23

way from your nose, mouth, all the way

75:25

down to the anus. And each one is

75:27

specific and can predict whether you're

75:30

going to get cardiovascular disease or

75:31

not. There's

75:32

unequivocal data to show that if you

75:34

have bad teeth, bad dental hygiene, bad

75:36

bacteria in your mouth, you're going to

75:39

get valvular disease such as aortic

75:41

stenosis, premature calcification of

75:42

your aortic valve, and you're going to

75:44

get coronary calcification. That's been

75:46

proven unequivocally. Patients who have

75:48

chronic sinusitis in your So, you think

75:50

that that's benign? If you have chronic

75:52

sinusitis, What's chronic sinusitis?

75:55

Chronic sinusitis, infections inside

75:57

your maxillary sinuses, frontal sinuses,

75:59

constantly, and you get these headaches,

76:02

and you get the cold, and your blocked

76:04

nose, and you you constantly clogged up

76:06

inside here.

76:07

That's inflammation. And oftentimes,

76:10

that's also linked to premature coronary

76:13

arteries and inflammation in your body,

76:14

especially when it's fungal. There's a

76:16

condition called fungal sinusitis.

76:19

Again, it comes down to mold. And this

76:22

mold, it causes that low-grade

76:23

inflammation in your body. And this has

76:26

been linked to coronary artery disease.

76:27

You see, what's happening is we're

76:28

transforming the whole definition of the

76:31

causes of coronary artery disease.

76:34

We have been so myopic

76:37

in our definition of what causes

76:38

coronary artery disease.

76:40

It's everything. entire lifestyle.

76:45

Mental, physical, eating, everything

76:48

will affect your coronary arteries.

76:50

Everything. Everything con- Do you think

76:52

much about

76:54

overstimulation? So, I mean really mean

76:56

like too much caffeine.

76:58

Does does too much caffeine play a role?

77:00

And I say this in part because I think

77:01

there's been a few times where I've had

77:03

like a pre-workout or

77:05

too many glasses of coffee and I've got

77:07

like heart palpitations and you almost

77:08

like feel like you're dying. Yes. Yes,

77:11

yes. Caffeine Caffeine is very similar

77:13

to the adrenaline molecule.

77:15

Now, why would you want to have too much

77:17

adrenaline in your body? Because then

77:19

you're living in a state of existential

77:21

threat

77:22

constantly.

77:24

And then what happens? You don't get the

77:26

offset either, so you don't get the

77:28

parasympathetic. You're only in

77:29

sympathetic all the time. That's fight

77:31

or flight. All the time. So, caffeine

77:33

generates that and the physiology it'll

77:36

do Look, caffeine is not as benign as we

77:39

think it is. Now, there's nothing wrong

77:40

with having one or two coffees a day.

77:42

That's fine, okay? But I know people who

77:44

drink about six glasses or six cups of

77:46

coffee a day and and I think that that's

77:48

just That's toxicity. You see? That's

77:51

toxicity. Too much caffeine. And it's a

77:53

diuretic. And a diuretic will cause

77:56

intravascular volume depletion and that

77:58

also turns on your neurohormonal

78:00

activation. So, you become more

78:01

sympathetic. You see? So, it's a vicious

78:04

cycle. Makes you dehydrated. I was

78:07

reading about this earlier because I did

78:08

have heart palpitations one day because

78:09

I think I drank too much coffee um

78:11

and

78:13

what I what I see here in the research

78:14

says that moderate caffeine

78:16

is safe and even heart protective for

78:17

most people, but excessive caffeine when

78:19

you get, you know, above 600 mg a day

78:23

um can

78:24

raise blood pressure, trigger

78:27

arrhythmias

78:28

>> Arrhythmias, yeah.

78:29

and stress the heart.

78:30

>> Yes. Now, I want to tell you why coffee

78:32

is good. It's not good because of the

78:33

caffeine. And so, it's not good at all,

78:35

"Yeah, I'm getting that caffeine, so

78:37

it's good for me. No. Cuz believe it or

78:39

not, coffee has a lot of soluble fiber

78:41

in it. So, there is some soluble fiber

78:43

in it.

78:44

And it has polyphenols.

78:46

So, coffee has polyphenols, just like

78:49

how cocoa has. You know, cocoa, cacao,

78:51

85% cacao chocolate. It's fantastic.

78:53

It's got soluble fiber. Cuz who's going

78:55

to consume the soluble fiber? It's

78:57

actually the bacteria. So, a coffee a

78:59

day is actually good for you for a

79:01

different reason. It's good because it's

79:02

good for your bacteria.

79:04

Cuz it's getting soluble fiber and it's

79:06

getting polyphenols. And all those

79:07

polyphenols are consumed by your

79:09

bacteria. They They call cacao, don't

79:11

they, the something of the heart? My My

79:14

girlfriend's quite spiritual and

79:16

in her practice, they call it like the

79:17

heart medicine, cacao. Cacao, it is.

79:20

Because it has a lot of antioxidants and

79:23

it has a lot of soluble fiber. So, wait

79:25

a second. Is it soluble fiber? Yes,

79:28

because that's fostering the growth of

79:29

the good microbiome. The good microbiome

79:31

is then going to produce postbiotics.

79:34

And the postbiotics

79:36

are going to give you all the benefits

79:37

that you're going to need. So, it's

79:38

going to cause less leaky gut, less

79:40

inflammation,

79:42

and your enteric nervous system, your

79:43

vagus nerve Your vagus nerve.

79:47

is going to be protected cuz we're

79:48

destroying our vagus nerve right now

79:50

with the leaky gut. When people get

79:52

heart palpitations, I imagine a lot of

79:54

them message you because it's quite

79:56

scary, isn't it, when you feel your

79:57

heart beating?

79:59

What is typically going on and when is

80:01

it cause for concern if you've got heart

80:03

palpitations? Yeah. If you have

80:05

underlying structural heart disease

80:07

Let's say you have blocked arteries.

80:09

Let's say you have a cardiomyopathy, a

80:11

valvular disease, and you're having an

80:12

arrhythmia, which is palpitations, that

80:14

is definitely life-threatening. They

80:15

need to come straight to the office and

80:17

we need to monitor you and find out

80:18

what's going on. But let's say you're

80:19

otherwise perfectly healthy, young

80:21

person, perfectly healthy, no heart

80:24

disease whatsoever, and you're having

80:25

palpitations, then it's usually an

80:27

imbalance of your sympathetic and

80:29

parasympathetic nervous system. You're

80:31

too stressed.

80:32

It's basically either too much

80:34

sympathetic or

80:36

not too much sympathetic, but you have

80:37

too little parasympathetic. And that is

80:40

one thing that I want people to realize

80:41

that

80:42

if you've knocked your vagus nerve off

80:45

because you have a leaky gut or because

80:48

of your lifestyle and your vagus nerve

80:50

is not working very well,

80:51

you will have this imbalance cuz the

80:53

imbalance is not with absolute levels.

80:55

It's the relative levels of sympathetic

80:56

versus parasympathetic. So, for example,

80:58

I see a lot of young women in their 30s

81:00

and 20s who come to me with tachycardia.

81:03

They have a rapid heartbeat all the time

81:05

and especially when they stand up, they

81:06

get a rapid heartbeat. This is called

81:08

postural orthostatic tachycardia. And

81:11

when they come to me,

81:13

I find out that it's actually the gut. I

81:15

fix their gut and the tachycardia gets

81:17

better because by fixing the gut, I'm

81:20

fixing the vagus nerve. By fixing the

81:22

vagus nerve, the heart rates come down.

81:26

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81:42

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83:42

Let's talk about the vagus nerve then.

83:44

Um, before we do that, I had a little a

83:47

little incident actually caused by

83:49

Jemima, who's out there.

83:51

Jemima,

83:52

we are in this little fitness

83:53

competition, and Jemima decided that she

83:55

wanted to beat me. And so, she cycled

83:57

100 km one day. She cycled for 4 hours,

84:00

which meant that she had more minutes in

84:02

this fitness competition than I did. So,

84:04

my rebuttal

84:06

was to do the same, but more. So, the

84:08

next day I cycled just over 100 km on my

84:11

Peloton at home. Funnily enough, it took

84:13

me like 4 hours to do it. And then I

84:15

came off the Peloton, I was celebrating,

84:16

whatever, you know, won this

84:19

gold medal emoji as my reward that

84:21

month.

84:22

But also I won heart palpitations that

84:25

stayed with me for a little while. And I

84:26

was wondering like why that happened. So

84:28

I did this big vigorous exercise which

84:30

was kind of outside of my usual exercise

84:31

regime. And then for a a couple of weeks

84:35

I could like feel these heart

84:36

palpitations.

84:38

And is that because of what you were

84:39

saying about my parasympathetic vagus

84:42

nerve What do you think happened there?

84:43

Yes. Yes. Yes. Yes. Over exercise.

84:48

This has been shown. When you do what

84:50

you just did,

84:52

you're diverting blood from your gut to

84:54

your muscles. Because your muscles at

84:57

that point really need all that blood

84:58

supply. And this has been well

85:00

documented. So what happens when you

85:02

overly exercise like that, you're

85:04

causing relative ischemia to your gut.

85:07

Ischemia Ischemia, what does ischemia

85:08

mean? Lack of circulation to your gut.

85:11

So your poor gut at that point had lack

85:13

of circulation. Relative. Not to the

85:15

point where it's going to go in

85:16

gangrenous. No. Don't Don't Don't get me

85:18

wrong. But it affects your gut. And in

85:21

the gut is the enteric nervous system.

85:23

The vagus nerve ends in your gut lining.

85:27

And the vagus nerve got affected. So

85:29

your vagus nerve didn't work very well.

85:30

When you get vagus nerve not working

85:33

well, you get too much sympathetic

85:34

you're going to get tachycardia. You're

85:35

going to get arrhythmias. Palpitations.

85:38

So tell me what the vagus nerve is and

85:41

what I can do to get my vagus nerve in

85:43

check. By the way, Jemima, it was

85:45

totally worth it. But just tell me what

85:47

I what I can do to get my vagus nerve in

85:48

check and like what role my vagus nerve

85:50

plays. I've got this photo here of the

85:51

vagus nerve.

85:53

Um Yeah. You know, the vagus nerve is

85:55

fascinating.

85:57

It is the largest nerve in the body.

86:00

And it it can it basically is sending

86:04

messages from the brain

86:06

to the whole body and receiving messages

86:09

back to say what is the state of

86:11

affairs. And the largest distribution,

86:13

believe it or not, is all in in gut.

86:16

Now that tells you something right there

86:18

that

86:20

what is the brain most concerned about?

86:23

It's most concerned about the most

86:26

treacherous border in your body, which

86:28

is your gut.

86:30

Your gut health is so important that the

86:34

body has dedicated a huge nerve called

86:37

the vagus nerve just to take care of

86:40

your gut.

86:41

Because your gut is that important. That

86:43

is why By the way, it also has endings

86:47

on the heart, in your lungs, in your

86:49

face. So, there's lots of endings of the

86:52

vagus nerve. And what is it doing? Is it

86:53

telling Is it basically the

86:55

communication channel between all your

86:57

gut, your heart, your brain?

86:58

>> affairs. What is going on in the gut?

87:01

What should I be doing? Back and forth

87:03

traffic information going back and

87:04

forth. So, when you when you

87:08

the body is supposed to be in a state of

87:10

sympathetic followed by parasympathetic.

87:13

What does that mean? You run away from

87:15

the tiger.

87:16

You hide behind the rock. The tiger is

87:18

gone now. And now you're supposed to be

87:20

parasympathetic.

87:22

>> So, sympathetic is the fight.

87:24

Parasympathetic is the the relaxation.

87:26

>> Rest, relaxation, repair.

87:29

Guess what? We're not getting enough

87:30

repair these days because we're

87:32

constantly in the fight fight fight

87:33

fight fight. We never give our chance to

87:35

go into repair. That's what the problem

87:38

with the vagus nerve that we have today

87:40

is. So, we don't get a chance. So, that

87:42

is why why don't we do that? Well, one

87:45

is our lifestyle. Two, our gut is not

87:49

right because it has just made our vagus

87:52

nerve dysfunctional. When I fix the gut,

87:54

one of the things I notice is heart rate

87:57

variability gets better. What does that

87:59

mean? Heart rate variability. That means

88:01

your pulse

88:03

with each breath in

88:05

and each breath out, there's a little

88:06

variation in your heartbeat. Mhm? That's

88:09

called heart rate variability. Now,

88:12

when you lost your heart rate

88:13

variability, it means that your vagus

88:14

nerve is not working very well. When all

88:16

patients when I fix their gut, their

88:18

heart rate variability gets better as

88:20

well.

88:21

When you fix the gut, your vagus nerve

88:23

will be able to work more efficiently

88:25

and you'll be able to repair yourself

88:27

after the sympathetic drive. So, you get

88:30

the yin and then you get the yang as

88:32

well. And the nice thing about the vagus

88:33

nerve that I find is that you can repair

88:35

it through the gut, but you can also

88:37

hack it and you can give it some

88:39

nutritional supplements.

88:41

The nutritional supplements are omega-3,

88:44

which right now 50% of the population is

88:46

very low in omega-3, and you need DHA

88:49

and omega-3 to to to make your vagus

88:52

nerve work optimally. You need to fix

88:53

the gut. And thirdly, you can hack the

88:57

vagus nerve. So, how do you do that? You

88:59

do the breathing exercises. So, when you

89:01

do the deep expiration, you stimulate

89:05

all those parasympathetic nerve endings

89:08

and your heart rate goes down, blood

89:11

pressure goes down. What kind of

89:12

breathing exercises?

89:14

So, breathe in to the count of four,

89:16

breathe out to the count of eight. Very

89:19

simple. I tell it to all my patients.

89:20

Breathe in to the count of four, breathe

89:22

out to the count of eight. It's a

89:24

deliberately long expiration. You do

89:26

that for about 10 minutes a day.

89:28

And when you do that, your vagus nerve

89:30

is getting stimulated and any nerve that

89:33

is stimulated frequently enough will

89:35

work more efficiently. What about this

89:37

eyeball thing that I've heard you talk

89:39

about where you

89:40

Eyeball does affect the vagus nerve

89:42

function, too. So, when you look to the

89:44

right, look to the left at the top, look

89:45

down, look left, it stimulates the vagus

89:48

nerve. There is a nerve on the ear.

89:50

There's also the vagus nerve that can be

89:52

stimulated here in the neck, that can be

89:55

stimulated the facial nerve also

89:58

has branches that go to the vagus nerve.

90:01

So, there are a few hacks that people

90:03

can use. I even have a small contraption

90:05

that I give patients that wear on the

90:07

neck and it it it it releases little

90:10

signals onto the vagus nerve over here

90:12

and I've placed it on patients and they

90:15

in front of me and they say, "Oh my god,

90:18

I already feel so relaxed and nice." And

90:20

it's

90:22

I was shocked when they did that.

90:23

>> I heard you talk about massaging the

90:25

eyeball.

90:26

Yes. And cold water on the eyeballs. So,

90:29

I don't advise massaging too often, but

90:31

cold water and gentle massage on your

90:34

eyeballs to release the vagus nerve and

90:36

you can do it yourself. You'll notice

90:37

that if you're on a monitor and you do

90:38

that, your pulse rate goes down. It's

90:40

the deep dive reflex that we have in all

90:44

of us dive into water. What about

90:46

putting an ice pack on the back of your

90:48

neck? I think that's fantastic. Back of

90:50

the neck, but I would prefer the front

90:52

of the neck. Okay. Cuz the front of the

90:54

neck is close to the vagus nerve. It's

90:55

very close to the karate chief over here

90:58

and when you put ice packs on it, the

91:00

vagus nerve is stimulated. Humming for

91:02

10 minutes? 10 minutes, both sides. What

91:05

do you mean by humming? Can you give me

91:06

an example?

91:06

>> Oh, humming. Oh, humming. Oh, yeah. That

91:09

sound is transmitted

91:12

to the vagus nerve. So, it's like

91:14

shaking that vagus nerve and it creates

91:15

electrical impulses

91:17

in the vagus nerve. That's been shown.

91:19

Singing singing

91:21

singing, humming

91:23

>> Laughing? You know that there are

91:24

laughing clubs?

91:27

Maybe you should start a laughing club.

91:28

But laughing clubs Yeah, laugh for no

91:31

reason. Just sit there and laugh. So,

91:32

that diaphragmatic movement that

91:35

constant diaphragmatic movement in

91:37

laughing stimulates the vagus nerve.

91:39

I've heard you talk about this Valsalva

91:41

maneuver. Yeah. What's that?

91:43

>> The Valsalva is when you breathe in

91:46

and then you breathe out, but don't let

91:48

the air out and you're straining.

91:51

And that straining compresses the

91:53

thoracic cavity, but also the abdominal

91:54

cavity and stimulates the vagus nerve.

91:57

And that is also found to be very, very

92:00

very

92:00

And remind me again, if I have a healthy

92:02

vagus nerve, if I have a

92:04

calm, healthy vagus nerve, what is the

92:06

downstream impact going to be across my

92:08

life?

92:09

Downstream impact is going to be faster

92:11

healing,

92:12

less coronary disease, less high blood

92:14

pressure.

92:15

Your blood will not be so sticky

92:18

and make blood clots, less inflammatory

92:21

markers.

92:23

The blood test that we do, the

92:24

interleukin-6, the tumor necrosis

92:25

factor, the CRP levels, small dense LDL.

92:30

We didn't even talk about LDL, but LDL,

92:32

the bad cholesterol,

92:34

all those get better. And what is the

92:36

the the role of LDL as it relates to

92:38

heart disease? Cuz I've heard this word

92:40

cholesterol, I really don't really

92:42

understand what it is or I know there's

92:44

good cholesterol, there's bad

92:45

cholesterol. I know I guess the bad

92:46

cholesterol comes from fast food and

92:48

processed foods and stuff. But simply,

92:51

what do I need to understand about the

92:53

role cholesterol plays in heart disease

92:54

and how to not have bad cholesterol?

92:58

The LDL carries more so of the

93:00

cholesterol in the body. It's not

93:02

complicated. LDL is what? Is

93:05

LDL is a lipoprotein. Yeah. Right? And

93:08

you have HDL, which is good cholesterol,

93:11

LDL, bad cholesterol, total cholesterol,

93:13

and triglycerides. Okay, these are the

93:14

four tests.

93:15

So, the LDL is a reflection of your

93:17

cholesterol. Now,

93:20

cholesterol is a normal molecule in your

93:22

body.

93:23

And you should have cholesterol because

93:26

if you don't have cholesterol, I don't

93:27

think you'll live too long.

93:29

And you need to move this cholesterol

93:31

around your body. Every cell in the body

93:33

makes cholesterol. Your liver makes most

93:34

of the cholesterol, actually.

93:36

Now, what happens is that when that LDL

93:38

molecule becomes damaged, it becomes a

93:41

small dense LDL particle.

93:44

A damaged LDL causes inflammation

93:48

because now you have a molecule that's

93:50

floating around your body. It's been

93:51

damaged.

93:52

It's small, it's dense.

93:55

Now, your white cells, your macrophages,

93:57

see that and want to gobble it up.

94:00

That's number one. Number two, these

94:02

molecules get oxidized in the lungs and

94:05

then they the first place they come out

94:07

from after the lungs is the coronary

94:08

arteries. So, it's the small dense LDL

94:11

particles, which are now oxidized. They

94:16

destroy the glycocalyx, which is the

94:18

lining of the coronary arteries, and

94:19

they activate the endothelium.

94:22

When they activate the endothelium,

94:24

which is the lining of your coronary

94:26

arteries, that's when you're going to

94:28

start getting atherosclerosis. So,

94:30

atherosclerosis and LDL are related, but

94:33

it's not the the total cholesterol. It's

94:36

when you have small dense damaged LDL.

94:40

So, let me explain the impact in my

94:43

practice.

94:44

Patients come to me because

94:46

they either cannot tolerate cholesterol

94:48

medications, or they don't want to take

94:50

cholesterol medications. What I do then

94:53

is I look and say, "Okay, your LDL

94:55

is it high or low? Is it Is that what

94:58

I'm really concerned about, or is it

94:59

that it's small dense LDL? So, I do a

95:02

subfractionation." And this is important

95:04

for you to understand because they don't

95:07

do it in every country in the world, and

95:08

it's very difficult to get some of these

95:09

blood tests abroad. But in the United

95:11

States, it's easy to do it. It's called

95:13

subfractionation of your LDL. So, it

95:15

tells you whether you have small dense

95:17

LDL or not. So, let's say you came to me

95:19

and your LDL is 150.

95:22

But if it's all made up of large,

95:25

fluffy, good, normal LDL,

95:29

it's not going to be implicated in your

95:31

inflammation or in your coronary artery

95:33

disease. I leave that alone.

95:36

But if you have small dense LDL, that

95:39

also is a problem because it's pointing

95:42

me towards inflammation, cuz what causes

95:44

small dense damaged LDL are glucose,

95:48

omega-6,

95:50

advanced glycation end products,

95:52

toxins,

95:54

and lipopolysaccharides that come from

95:56

the gut, leaky gut.

95:58

So, now I just gave you the primer

96:02

for treating coronary artery disease.

96:05

These five things

96:07

promote coronary artery disease, and

96:10

it's small dense LDL. Those macrophages,

96:14

they engulf small dense LDL, and those

96:17

macrophages become foam cells. The

96:19

endothelium has been

96:21

already activated, so they uh

96:24

uh bind to the endothelial lining.

96:27

>> what is the endothelium

96:28

>> The endothelium is the lining of the

96:29

artery in your coronary artery. So,

96:31

these white cells that are full of fat,

96:34

the bad fat, attach themselves to it,

96:37

and then become endothelialized. That

96:38

means they become part of the wall. So,

96:40

they become a plaque.

96:41

>> That's Now you got it. That's making the

96:43

plaque. You just hit it right on the

96:45

head.

96:46

So, how would I naturally lower my LDL

96:49

without taking statins or any of these

96:51

other medications? Is there a way to to

96:53

do it naturally?

96:55

You You stay away from from meat, yes.

96:57

You've gone a vegetarian diet, yes.

96:59

It'll come down. But there's no need to.

97:01

All meat? All meat, because they all

97:03

have cholesterol. Even chicken? Even

97:04

chicken. Yeah, if you want to bring it

97:06

down. But the question is, do you really

97:08

want to bring it down? Do you really

97:09

want to bring it down? Later, the most

97:11

recent data is very scary. It shows that

97:14

patients who have the highest LDLs

97:16

actually make it into the 90s

97:18

and live a healthier life.

97:20

It's not the LDL, it's the damaged LDL

97:23

that's the problem. So, this

97:26

the medical profession as a whole and

97:28

all of us are having a problem grappling

97:30

with this. It's Is it the total LDL that

97:32

we should be concerned about?

97:34

Point to your question. What is a normal

97:36

cholesterol?

97:38

And should we be lowering cholesterol?

97:40

And the answer really is not that

97:41

simple. What we should be doing is not

97:45

to have small dense LDL. So, the

97:47

question is that's the issue. And small

97:49

dense LDL are five things. Sugar causes

97:52

it, omega-6,

97:54

advanced glycation end products, toxins,

97:56

and leaky gut. They make your

97:58

cholesterol become small dense

98:00

particles. A lot of people are concerned

98:01

about eating eggs because they think it

98:03

might raise their cholesterol levels.

98:05

Absolutely no concerns whatsoever. And

98:07

what about statins? So, a lot of people

98:09

are prescribed statins when they have

98:10

heart-related issues. Are they

98:12

safe in the long run? Are they

98:14

effective? Okay. First part, are they

98:17

safe? For the most part, they are. But

98:20

at least 20 to 30% of patients will

98:22

suffer from sarcopenia, which is loss of

98:24

muscles. So, they'll get weakness,

98:27

aches, pains.

98:29

They'll get diabetes because it causes

98:32

mitochondrial dysfunction. So, they'll

98:34

they'll do a sugar problems. And many of

98:37

them also do develop mental diseases.

98:40

So, I've seen that they become forgetful

98:42

and they just can't think right and I

98:43

stop the statins and all of a sudden

98:45

they start feeling better. So, no. Not

98:48

all statins are safe.

98:50

You need to monitor statins. Also, they

98:52

can cause liver dysfunction. So, you

98:53

need to watch that. Number two is that

98:57

the statins

98:58

are they effective? Effective in what?

99:01

If the end point is to reduce your LDL,

99:05

yeah. Statins will bring your LDL down.

99:08

But does that translate to a decrease in

99:10

cardiovascular event rates? That's a

99:12

totally different question. You see? So,

99:15

is your goal just to reduce the LDL or

99:18

is your goal to reduce the damaged

99:20

oxidized small dense LDL levels?

99:24

And when people say they have high blood

99:26

pressure,

99:27

that's because there's potentially a

99:30

narrowing of their blood vessels. So,

99:32

the

99:33

the the blood has less space to go

99:35

through. So, it's kind of like squeezing

99:36

a hose pipe that it gets it gets shoots

99:38

out faster.

99:39

Is that typically what it means by high

99:40

blood pressure? Sort of. Let me explain.

99:43

When your insulin levels are running

99:45

high, all your blood vessels and your

99:48

capillaries all don't vasodilate

99:50

properly. Oh, okay. And by vasodilation

99:52

is the expansion and shrinking.

99:54

>> Yeah. So, they don't vasodilate

99:55

properly. So, you will get high blood

99:57

pressure. So, one of the biggest things

99:59

I've noticed with diabetes patients is

100:00

when I bring their insulin levels down

100:02

through my fasting program and dietary

100:04

changes, the blood pressures go away.

100:06

See, that's another thing. There's no

100:08

such thing as essential hypertension.

100:11

Essential hypertension meaning that

100:14

you just have high blood pressure.

100:15

There's always a cause. Either you have

100:17

sleep apnea or you have

100:18

hyperinsulinemia.

100:19

Cuz sleep apnea also causes

100:21

hypertension. So, hypertension does not

100:23

always necessarily mean that you're

100:25

going to be committed to blood pressure

100:26

medications for the rest of your life.

100:29

I've heard you talk quite a lot about

100:30

breakfast. People ask you about what

100:31

they should have for breakfast cuz

100:33

everybody wants to know

100:35

um how they should be starting their

100:36

day.

100:37

What is your advice on what we should be

100:38

eating for breakfast to have a healthy

100:40

heart and cardiovascular system?

100:44

Breakfast is a tough one.

100:46

I tell most of my patients that if you

100:47

absolutely have to have a breakfast,

100:49

have a couple of eggs. And if you are a

100:52

vegetarian, it makes it a little bit

100:54

more tough.

100:55

Um but you can have some kefir with two

100:59

to three blueberries, two to three

101:00

blackberries, and about four blueberries

101:03

in it. And then you put your inulin

101:06

powder in it, and that's your breakfast.

101:08

That's all you should have. But a heavy

101:10

breakfast and also breakfast

101:13

in my opinion, breakfast should be

101:16

should pass breakfast for most adults

101:18

and just go for lunch and dinner only.

101:21

Cuz then you can do your 18:6. If you

101:23

absolutely going to have breakfast, then

101:25

you have breakfast and lunch and then no

101:27

dinner.

101:28

But you got to plan 18 hours. 18 hours

101:31

is optimal and we can do it. A lot of

101:33

people say they can't do it, but most of

101:35

us can do it.

101:36

You will notice how much more energy you

101:38

have. You know, when I do my OMAD, which

101:41

I do at least three to four times a

101:43

week, which I only have one meal a day,

101:46

at 6:00 in the evening, I actually have

101:48

more energy than I do in the morning.

101:50

What does your diet look like, doctor?

101:52

My diet? Yeah.

101:54

I do eat meat, and I do eat fish,

101:57

chicken, and turkey. And but I'll have

101:59

Yeah, source it right. Always make sure

102:02

that it's grass-finished. Always make

102:03

sure it's organic. And I have small

102:05

amounts of it. Grass-finished,

102:06

grass-fed?

102:08

Grass-finished. What does that mean?

102:10

Grass-fed just means they took it out

102:12

one day and put it out in the field.

102:15

And they fed it some grass.

102:17

So, this advertising that industry does

102:19

is a little deceiving.

102:21

And I had to investigate this to find

102:23

out.

102:24

It's a lie. It has to be grass-finished

102:26

from beginning to end. So, then it's

102:28

going to have more omega-3 in it. It'll

102:30

have more K2 in it. It'll have more

102:32

nutrition and it'll have less toxins

102:33

also in it.

102:35

So,

102:36

less omega-6. Because if your cows are

102:38

eating corn,

102:41

then you're going to get all that

102:41

omega-6. And the cow is not supposed to

102:44

be eating corn.

102:46

Cow is supposed to be eating grass.

102:48

So, I do eat some meats and I eat some

102:50

vegetables with it.

102:52

Coming from a background, my

102:54

great-grandfather's from India, we do

102:56

eat uh Indian food as well. So, we have

102:58

some lentils

103:00

and some curries, of course. So, we can

103:02

cook the meats in a in a curry form as

103:04

well. I don't eat much chapatis, because

103:07

those are very full of carbs.

103:10

I love the taste of it. I love naan once

103:12

in a while. And what do you supplement

103:14

with? What's your supplements diet look

103:15

like?

103:16

>> I take D3,

103:18

K2,

103:19

omega-3, fish oil. So, I take two two

103:21

capsules every day. Vitamin C, 1 g,

103:25

inulin,

103:27

kefir, I drink that every day. And I

103:29

count it. Magnesium,

103:32

and I take nattokinase.

103:35

Nattokinase. Nattokinase, I take about

103:39

8,000 units a day. What it is is that

103:43

it does thin the blood a little bit, so

103:46

that you're less likely to make blood

103:48

clots. Part of metabolic syndrome is a

103:50

tendency to make blood clots and have a

103:52

high fibrinogen level. My fibrinogen

103:54

level was at the upper limits of normal,

103:56

so I just take nattokinase. It doesn't

103:58

cause any bleeding or any problems like

104:00

that. And then I also take

104:03

a probiotic.

104:05

And I it's it's basically is called Mega

104:08

Spores

104:10

probiotic.

104:12

So, this is spores.

104:14

There's no real bacteria in there. It's

104:16

all spores. What's a spore? A spore, a

104:19

bacteria

104:21

can survive

104:23

by sporulating. So, it becomes a spore,

104:25

so it's no longer bacteria, it's a

104:26

spore, but it'll germinate under the

104:29

right circumstances. So, these little

104:31

things, they have they are the spores of

104:35

the bacteria that are in soil.

104:38

So, soil has certain species of

104:40

bacteria, the bacillus species.

104:43

The spores

104:45

I consume, they go in my gut because

104:48

remember, if I take a probiotic,

104:51

the acidity in my stomach is going to

104:53

kill most of the bacteria.

104:56

So, what does the spore do, sorry? Do

104:57

you So, the spore passes through the

104:59

stomach, goes all the way down to your

105:01

intestines, and then it germinates

105:04

there. It grows there. So, now the spore

105:06

becomes a bacteria. Okay. So, it gets

105:09

past that acidity of your stomach. So, I

105:12

do take that as a supplement as well.

105:14

Dr. J, what is the most important thing

105:15

we didn't talk about that we should have

105:17

talked about?

105:19

For the people at home that have clicked

105:20

on this episode. And you know, I guess

105:22

one way that you might tackle this

105:24

question is just by thinking about the

105:26

most popular questions you get asked

105:28

from your audience on a daily basis

105:30

that we might not have covered today.

105:33

How can I screen myself right now? What

105:36

test should I do right now to see

105:41

at what stage of inflammation and

105:43

coronary artery disease am I in? Because

105:45

coronary artery disease is the number

105:46

one cause of death, and inflammation is

105:48

the underlying cause. So, what test

105:50

should I be doing right now?

105:52

That is an important question. If you

105:54

over the age of 30

105:56

and you have concerns,

105:58

one, get a coronary calcium score.

106:02

If your coronary calcium score is zero,

106:05

you have no calcium,

106:07

then you are in a good place. If you

106:09

have coronary calcium,

106:12

you need to go see a good cardiologist

106:14

that's going to do a prevention program

106:15

because it means you already have

106:16

atherosclerosis.

106:18

Atherosclerosis meaning plaque Hardening

106:20

of the arteries.

106:21

Number two, you need a good blood test

106:25

to look for inflammation. The blood test

106:27

that I order is called a Cleveland Heart

106:30

Labs.

106:32

In it, you get your HDL, LDL,

106:35

cholesterol, but you look at particle

106:37

sizes.

106:39

Is my LDL small dense? If it is small

106:42

dense particles, is it oxidized? Yes, I

106:45

have a problem.

106:46

My CRP level,

106:49

other inflammatory interleukin-6,

106:51

tumor necrosis factor,

106:53

hemoglobin A1C,

106:56

sugar level.

106:58

All these are part of that panel of

107:00

tests. So, you should get a good

107:02

inflammatory panel, which is the

107:05

Cleveland Heart Labs. So, those are the

107:07

two things that everyone must do. Get a

107:09

coronary calcium score, all adults,

107:11

and do an inflammatory panel.

107:14

Inflammation. You got to know whether

107:16

you Look, you know

107:17

you may not feel inflammation. It's not

107:19

like you get a big pimple and it's going

107:21

to hurt you. But if you have

107:23

inflammation in the body, yeah, you

107:24

could know about it. How? You have

107:26

mental fog.

107:28

I'm not optimal, doc. My memory is going

107:31

down. I'm depressed. Depression is

107:34

inflammation. Depression is not

107:36

something psychological, okay?

107:38

Now, this I have to stress to you and I

107:39

tell all my patients, you come in and

107:41

you say I'm depressed.

107:42

You're depressed because

107:45

that very symptom of depression is a

107:47

symptom of your inflammation.

107:50

You can be depressed about something

107:52

that oh my car's not working very well.

107:55

That's a difference from depression.

107:58

Depression is

107:59

inflammation in the brain.

108:01

So, you'd be surprised how many patients

108:03

go on my anti-inflammatory diet, which

108:05

is I change their diet, fix them all up,

108:07

and the depression goes away.

108:09

So, ask yourself not just questions

108:12

about physically how you're feeling, but

108:14

mentally also.

108:17

And if the answer is that doc, I'm not

108:19

optimal, I'm forgetful, I'm depressed,

108:21

I'm having sleep disturbance, I'm

108:23

relation things, man. I've changed. My

108:26

body's also changed.

108:28

You have inflammation.

108:30

We

108:31

have a closing tradition on this podcast

108:33

where the last guest leaves a question

108:34

for the next guest not knowing who

108:35

they're leaving it for. And the question

108:37

that's been left for you is what was the

108:39

most difficult day of your life and how

108:41

did you overcome it?

108:43

The most difficult day of my life

108:46

was a very personal one for me.

108:49

You know, when when when

108:50

my when my dad passed away and you know,

108:54

and I'm Dr. J.

108:57

And uh

108:59

and when he he was sick and he didn't

109:02

want to go to the hospital

109:05

and then he passed away in his sleep.

109:08

And then you always ask yourself, could

109:09

I have done something differently, you

109:11

know?

109:12

You second-guess yourself. And that

109:14

second-guessing happened with him, of

109:16

course. It was very very painful.

109:19

But it happens with patients as well.

109:21

But

109:22

how did I overcome it? It taught me a

109:25

lot about my own limitations as well, as

109:28

a physician, as a son,

109:30

as a scientist, as a reader.

109:33

And that's why I'm motivated to do what

109:36

we are doing here also is to

109:38

do the best you can to educate people.

109:41

What was he sick with? What was his

109:42

predicament?

109:44

He was a cardiac patient already. And

109:46

he's he's got a very interesting story,

109:48

actually. He was told that he only had a

109:50

few years to live after he had bypass

109:52

surgery. But then I put him through all

109:55

the things we talked about today.

109:57

He lost a lot of weight. He went from

109:58

185 lbs to 135 lbs. He changed his diet,

110:01

and he lived another 30 years. And he

110:03

did another six world tours, and

110:05

traveled, and he had a wonderful life.

110:08

Okay?

110:09

The impact of simple things that we can

110:11

do to change his life. So, he stopped

110:13

drinking all alcohol as well, and he he

110:15

really had a very clean diet and

110:17

everything else. But that night, he came

110:19

down with some sort of infection. He had

110:22

massive diarrhea and massive massive

110:25

explosion. And I think that what really

110:27

happened is he got overwhelmed. It's

110:29

it's the gut.

110:30

He got overwhelmed with a

110:32

toxin from his gut.

110:34

And then he passed away.

110:37

But But here's a man who who lived to

110:39

89, was supposed to have passed away in

110:42

his 60s.

110:43

And the impact But But thing is that,

110:46

you know, we

110:47

we we shouldn't second-guess ourselves.

110:49

We don't want to live in the past.

110:52

I want to live in the present moment,

110:54

not even in the future.

110:56

So,

110:57

all these things have taught me to live

110:59

in the moment.

111:01

And one of the skills I've developed is

111:03

just that.

111:04

That when I'm with you, I'm only with

111:06

you.

111:08

If I'm doing your surgery, I'm only

111:09

doing your surgery. If I'm having a

111:11

conversation with you, you and I are the

111:14

only people that matter right now.

111:16

And this, being in the moment,

111:19

that ability to be that is something I

111:22

learned over time. Because before, you

111:24

know, something bad happens, your next

111:26

case is still reminiscing about that and

111:28

you're regretting that. You can't do

111:31

that. Life is only expressed in this

111:33

moment.

111:34

Right now. So, learning how to live in

111:37

the moment applies not only to me, but

111:39

to everybody.

111:40

We all living in the past otherwise.

111:43

And or we worried about the future.

111:46

So, how do you learn present moment

111:49

awareness and live in this moment so

111:51

that we will be happier, we can do what

111:55

we want to do, make the changes we need

111:57

to make, get the courage and make the

111:59

and have the willpower to go out and

112:01

make a difference to yourself and to

112:03

everyone around you, starts with you

112:06

being here. Are you here?

112:09

Or are you in your past?

112:11

Or have you gone off into the future?

112:13

That's a skill

112:15

that medicine has taught me and I've

112:17

become humbled by it.

112:19

And this is something

112:21

that I have learned.

112:23

And I want to pass on to all my patients

112:25

to as well. That they okay, you've got

112:26

all these things going on, but

112:28

learn to live in the moment.

112:31

Dr. J, thank you so much. Thank you so

112:32

much for the wisdom that you've shared

112:34

with so many people. If people aren't

112:35

familiar with you, you're extremely

112:37

prolific and well-loved across the

112:39

internet the

112:41

the public education work you've done

112:42

and the the heightening of awareness of

112:45

people's heart and their cardiovascular

112:46

health more broadly and until really

112:48

before you started making videos online,

112:50

a lot of this stuff was

112:52

opaque. It was a mystery to a lot of

112:54

people and your YouTube channel has

112:56

almost a million subscribers now and you

112:58

you go through some of these subjects

113:00

that we've talked about today in even

113:01

greater detail. So, I'd highly recommend

113:03

people go and check you out there, but

113:04

also just thank you for saving so many

113:06

people's lives cuz

113:07

you know, that's what you do day in and

113:08

day out and that's an incredibly I mean,

113:10

there's probably not a higher calling

113:12

one can answer. So thank you for doing

113:14

what you do and I can see now why people

113:16

love you so much on online and on

113:18

YouTube because you're so incredibly

113:20

engaging, I guess is the word. Engaging,

113:23

wise and you make the information

113:25

accessible in a way that

113:27

I don't think it's been done before. So

113:28

thank you so much. It's a pleasure to

113:29

meet you and to have this conversation

113:30

with you and thank you for enlightening

113:32

my audience. The pleasure was mine.

113:34

Thank you.

113:35

This has always blown my mind a little

113:36

bit. 53% of you that listen to this show

113:39

regularly haven't yet subscribed to this

113:41

show. So could I ask you for a favor? If

113:43

you like this show and you like what we

113:44

do here and you want to support us, the

113:46

free simple way that you can do just

113:47

that is by hitting the subscribe button.

113:49

And my commitment to you is if you do

113:51

that, then I'll do everything in my

113:52

power, me and my team, to make sure that

113:54

this show is better for you every single

113:56

week. We'll listen to your feedback,

113:57

we'll find the guests that you want me

113:59

to speak to and we'll continue to do

114:01

what we do. Thank you so much.

Interactive Summary

Dr. Pradeep Jamnadas, a cardiologist with over 35 years of experience, emphasizes the importance of addressing the underlying causes of heart disease—primarily inflammation, insulin resistance, and visceral fat—rather than just treating symptoms. He advocates for lifestyle interventions such as intermittent fasting, resistance exercise, and proper nutrition, including fiber-rich foods and fermented products, to optimize cardiovascular health, repair the gut microbiome, and manage toxins like mold.

Suggested questions

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