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The Man Who Can Predict How Long You Have Left To Live (To The Nearest Month): Gary Brecka | E225

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The Man Who Can Predict How Long You Have Left To Live (To The Nearest Month): Gary Brecka | E225

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2892 segments

0:00

If you want to strip fat off your body,

0:02

there is nothing, no type of

0:03

cardiovascular or weight training that

0:05

comes anywhere close to

0:10

He's spent 20 years working in life

0:11

insurance, predicting when people were

0:12

going to die to the nearest month. And

0:14

now he's on a mission to extend your

0:16

life.

0:17

Most people, they're walking around

0:19

right now at about 55% of their true

0:21

state of normal. There is an element

0:23

missing from their body that would make

0:25

the difference between them being an

0:27

average person and being a superhuman.

0:29

How? Everything that we put into our

0:31

bodies gets converted into a usable

0:33

form. If you cannot make this

0:35

conversion, you have a deficiency. And

0:36

it is this deficiency that leads to

0:39

these conditions.

0:41

They have accepted something as either a

0:43

consequence of aging, stress, or their

0:44

environment. This not a consequence of

0:46

any of those things. I'm going to tell

0:48

you exactly how to find out what it is

0:49

that's missing, so you could thrive in a

0:51

way that you probably never thought

0:52

possible. Let's use Dana White as an

0:54

example. So, LabCorp calls us, so says,

0:56

"Hey, we have a life-threatening alert

0:57

on a patient." He had all of these

1:00

conditions.

1:01

I've been talking to doctors. None of

1:02

them could fix any of my problems. I

1:04

said, "I'm surprised you could even

1:05

sleep through the night without choking,

1:07

gagging." He's like, slammed his hand

1:08

down, "How did you know that?" And I

1:10

said, "If you don't do what we're going

1:12

to ask you to do, if you have a life

1:13

expectancy of 10.4 years." And in 10

1:16

weeks, he has such a material change,

1:19

and he was like, "I had no idea I could

1:21

feel this good."

1:21

And his life expectancy? Almost tripled.

1:24

Someone who's just heard that at home,

1:25

where do they start? So,

1:30

I just want to start this episode with a

1:32

message of thanks. A thank you to

1:34

everybody that tunes in to listen to

1:35

this podcast. By doing so, you've

1:37

enabled me to live out my dream, but

1:39

also for many members of our team to

1:42

live out their dreams, too. It's one of

1:43

the greatest privileges I could never

1:45

have dreamed of or imagined in my life

1:46

to get to do this, to get to learn from

1:48

these people, to get to have these

1:49

conversations, to get to interrogate

1:51

them from a very selfish perspective,

1:53

trying to solve problems I have in my

1:54

life. So, I feel like I owe you a huge

1:56

thank you for being here and for

1:58

listening to these episodes and for

1:59

making this platform what it is. Can I

2:01

ask you a favor?

2:02

I can't tell you how much um you can

2:04

change the course of this podcast, the

2:06

the the course of the guests we're able

2:07

to invite to the show, and to the course

2:10

of everything that we do here just by

2:11

doing one simple thing. And that simple

2:13

thing is hitting that subscribe button.

2:15

Helps this channel more than I could

2:16

ever explain. The guests on this

2:18

platform are incredible because so many

2:20

of you have hit that button and I know

2:23

when we think about what we want to do

2:24

together over the next year on this

2:26

show, a lot of it is going to be fueled

2:28

by the amount of you that are subscribed

2:29

and that tune into this show every week.

2:31

So, thank you. Let's keep doing this and

2:34

I can't wait to see what this year

2:34

brings for this show, for us as a

2:36

community, and for this platform.

2:45

Gary. Yes.

2:47

I This is the first time I've started

2:49

this podcast in this particular way, but

2:51

I was

2:52

but this is where I wanted to start it.

2:54

It's with a slightly curious question,

2:56

which is

2:58

there's people that have just clicked

2:59

onto this podcast to listen now, right?

3:01

YouTube, Spotify, Apple, wherever

3:03

they're listening.

3:04

Um

3:05

why should they

3:07

stay and listen to the message that you

3:10

have committed your life to spreading?

3:13

Because

3:14

everybody wants to be better, right? I

3:17

mean, everybody wants to improve their

3:19

health. And most people, especially

3:21

young people,

3:23

probably don't realize that they're

3:25

walking around right now at about 55%,

3:28

maybe 60% of their true state of normal.

3:31

There is

3:33

a nutrient, an amino acid, a substrate,

3:35

a compound, um

3:37

an element missing from their body

3:40

that if they knew what it was

3:43

would make the difference between them

3:44

being an average person and being a

3:45

superhuman. They have accepted something

3:48

as either a consequence of aging or a

3:50

consequence of stress or a consequence

3:52

of their environment, something like

3:54

brain fog, or repeated poor sleep, or

3:57

weight gain, or water retention, or not

3:59

the healthiest response to exercise, or

4:02

brain fog, or poor short-term recall, or

4:04

any number of things that they've

4:06

accepted as consequence of life, of

4:08

aging, of stress, or what have you,

4:11

it's not a consequence of any of those

4:12

things.

4:13

It's a consequence of missing raw

4:14

material.

4:15

And I'm going to describe that in detail

4:17

and tell you exactly how to put that raw

4:19

material back into your body,

4:21

and how to find out what it is that's

4:22

missing,

4:23

so you could thrive in a way that you

4:25

probably never thought possible. Who are

4:27

you? Um I'm a human biologist,

4:31

um and a researcher, biohacker. My

4:34

background is in

4:36

uh of all things mortality science. You

4:38

know, my undergraduate degrees were in

4:40

biology, my postgraduate degrees are in

4:41

human biology. I'm fascinated by the

4:44

human body, um its form, its function,

4:47

how we can improve its performance, and

4:51

I'm a researcher. You know, I spent 22

4:52

years um

4:54

as a mortality expert in the insurance

4:55

industry,

4:57

which meant that if we got 5 years of

4:58

demographic data on you, and 5 years of

5:01

medical records, we could tell the

5:03

insurance company how long you had to

5:04

live to the month. To the month? To the

5:07

month. And I get a lot of flak about

5:09

that. People say, "Oh, if you could

5:10

predict death to the month, you'd win a

5:12

Nobel Prize, or you know, only God can

5:13

tell you when you're going to die." I

5:14

mean, and that's very true. I mean,

5:16

obviously, if we do a mortality

5:17

prediction on you, it doesn't mean

5:19

December 12th, you know, 2065, you're

5:22

going to drop dead on that day.

5:24

But it is very accurate science. You

5:26

know, if you think of the most

5:27

successful financial institutions on the

5:29

planet as a category, are insurance

5:32

companies.

5:33

If you look at the failures during the

5:34

2008-2009 financial crisis, which was a

5:37

global crisis, you know, in the US, we

5:39

had 364 banks fail. You didn't have a

5:41

single life insurance company fail. No

5:43

other financial institution, no other

5:46

investment enterprise, hedge fund, angel

5:48

investor, um venture capital firm would

5:51

ever put tens of millions of dollars at

5:53

risk

5:54

on any kind of investment on one single

5:57

variable.

5:59

Yet every time a life insurance company

6:00

bets on your life or or issues an

6:03

annuity or a reverse mortgage or any

6:04

number of other financial services

6:06

instruments that are based on mortality,

6:08

they're only betting on one factor. They

6:09

don't care where you are on a mortality

6:11

curve. They want to know how many more

6:13

months does this person have left on

6:14

Earth? And they needed some of the most

6:15

accurate science on the planet.

6:17

And

6:19

they have perfected this and the good

6:22

news is

6:23

that this science is very accurate and

6:25

I'm going to share a lot of the details

6:27

of that today, but

6:28

the downside of this industry and the

6:30

reason why I left it was that

6:32

during my tenure in this career

6:34

I was not allowed to have any contact

6:36

with the patient or any contact with the

6:37

treating physician. Now, I'm not

6:39

licensed to practice medicine. I'm a

6:40

human biologist.

6:42

But if I was reading a medical record

6:44

and saw a life-threatening drug

6:45

interaction, I could not contact the

6:47

patient.

6:49

And this database where this information

6:51

is drawn from, if this database could

6:54

see the light of day

6:56

I believe it would permanently change

6:58

the face of humanity. It would upend

7:00

modern medicine in a way that would be

7:02

completely catastrophic because you see

7:04

they have information that no other

7:05

database has, no other research study,

7:07

no other you know, financial

7:09

institution, no other

7:11

university has and that is that they

7:13

know the day, the date, the time, the

7:14

location, and the cause of death for

7:17

hundreds of millions of people that they

7:19

have all of these records on.

7:22

You know, if I if I'm a cardiologist and

7:23

you come in to see me and I put a heart

7:25

stent in your heart. Well, when you

7:27

leave my office, I don't know what

7:28

happens to you two months later, two

7:29

years later, 12 years later. I don't

7:31

know if you died as a consequence of

7:32

something that went wrong with the

7:34

procedure I did or if you died of

7:35

something completely different. I don't

7:37

know exactly what kind of impact on your

7:40

life that procedure had or didn't have

7:42

Um, unless it's contained in a very

7:43

short-term clinical study.

7:45

And

7:46

uh, not that I want to go into the whole

7:48

science of mortality, but

7:50

if you had access to this database, you

7:53

would realize that the most common

7:54

ailments that we suffer from

7:57

are not diseases and pathology that are

7:59

happening to us. They are things that

8:00

are happening within us.

8:03

You know, I always say that if I was to

8:04

boil my entire career down to a single

8:07

sentence, it would be

8:09

that the presence of oxygen is the

8:11

absence of disease.

8:14

And nothing is more impactful than that

8:16

statement.

8:17

The presence of oxygen is the absence of

8:19

disease. I have yet to find a single

8:21

disease etiological pathway, something

8:23

that's happening in in the human body

8:25

causing pathology, causing disease,

8:26

dysfunction, that doesn't have its roots

8:28

in a lack of blood oxygen or its roots

8:32

is not aggravated by something called

8:33

hypoxia, lack of oxygen. In fact, all

8:35

human beings die of the same thing. We

8:37

all leave this earth the same way.

8:39

Right? We all die of hypoxia, lack of

8:42

oxygen to the brain. That's the

8:43

definition of death. Only we tend to

8:45

think of it as an event. Right? A

8:47

gunshot wound, a bus, car, a stroke,

8:49

heart attack. But the truth is, we are

8:51

on a hypoxic curve.

8:53

Meaning we are accelerating quickly or

8:55

accelerating slowly towards the grave.

8:57

And the second thing that we discovered

8:58

in this

8:59

um, voluminous data-driven industry

9:03

was that

9:04

when you deplete certain raw materials

9:07

in the human body,

9:09

you get the expression of that

9:11

deficiency. So, what I mean is, you

9:14

know, we've all heard that um,

9:16

uh, disease, you know, travels in

9:18

families or you might have genetically

9:20

inherited hypertension or genetically

9:22

inherited hypothyroid, genetically

9:24

inherited drug and alcohol addiction,

9:26

diabetes, um, depression, anxiety. These

9:28

things tend to run in families. So,

9:31

we've accepted the myth in most cases

9:33

that these are genetically inherited

9:35

diseases.

9:36

But think about this, the next time one

9:38

of your listeners gets told that, "Oh,

9:41

you have genetically inherited

9:42

hypertension." What we call uh

9:45

idiopathic hypertension, or genetically

9:47

inherited hypothyroid, or if you have

9:49

this genetically inherited disease, or

9:50

that genetically inherited disease. Stop

9:52

your physician and say, "Well,

9:54

wait a minute. We We've mapped the

9:56

entire human genome.

9:58

Um so, we know every gene in the human

9:59

body. Can you tell me what disease I

10:01

inherited from my ancestor that's

10:02

actually causing this condition?"

10:05

And watch their face go blank.

10:07

Because in the majority of cases, we

10:09

have accepted that disease travels in

10:11

families when it's actually not the

10:13

disease being passed from generation to

10:15

generation. It is the inability for

10:17

their body to refine a certain raw

10:18

material,

10:20

which causes a deficiency,

10:22

which leads to that disease.

10:24

So, in other words, there's not a single

10:26

compound known to mankind, not one,

10:29

no mineral, vitamin, amino acid,

10:31

protein, carbohydrate, no nutrient of

10:33

any kind, no element known to man, that

10:35

enters the human body

10:37

and is used in the format that we put it

10:39

in. Everything that we put into our

10:41

bodies gets taken in by the body and

10:43

converted into the usable form.

10:47

If you cannot make this conversion,

10:49

you have a deficiency.

10:51

And it is this deficiency

10:53

that leads to these conditions.

10:56

And so, if you go hunting for that

10:58

deficiency and you supplement, not for

11:00

the sake of supplementing, but you

11:01

supplement for the sake of deficiency,

11:04

magic things happen in human beings.

11:06

There is a superhuman inside of every

11:08

person listening to this podcast, and if

11:10

they actually were able to test

11:12

themselves to a genetic test and look at

11:14

what's called their methylation, how

11:16

their body refines raw materials, and

11:18

they were able to replace the deficiency

11:21

that is holding their body back from

11:22

creating adequate levels of

11:24

neurotransmitter and achieving normal

11:25

new mood, moving um

11:28

contents through the gut and achieving

11:30

normal gut function,

11:31

um elevating emotional state, deepening

11:34

their sleep, quieting their mind. All of

11:35

these conditions that society has

11:37

labeled different pathologies and

11:38

disease.

11:40

Uh

11:40

then they would begin to thrive in a way

11:43

that they just never thought possible.

11:45

It's so blatantly clear how passionate

11:47

you are about the subject matter, which

11:48

begs the question. You know, I remember

11:50

when I was reading through

11:51

your story

11:53

I I uncovered that you'd read

11:55

hundreds of peer-reviewed papers. I

11:57

think the quote was something along the

11:58

lines of Um if there's a peer-reviewed

12:02

paper on the subject matter of

12:03

biohacking and um disease and longevity

12:08

and those kinds of things, you've

12:09

basically read it. So this begs the

12:11

question to me, like why is one

12:12

individual so unbelievably passionate

12:14

about this subject matter? And

12:17

I would like you to take me as early as

12:20

you possibly can to answer that question

12:22

in your own life. Where did your

12:23

obsession, passion, where was it

12:25

fostered fostered and nurtured so that

12:28

you spent and committed your life to

12:29

this subject matter? Where did that

12:31

begin? So it actually began I I grew up

12:33

on a

12:34

very large tobacco farm. My my father

12:37

was a Navy captain, my mom was a flight

12:39

attendant, she worked for the airlines.

12:41

But we had a

12:43

a farm that we leased out to other

12:45

farmers. And surrounding this 300-acre

12:47

tobacco farm were all kinds of animal

12:50

farms. There were cattle farms, there

12:51

were chicken farms, there were

12:53

horse farms.

12:54

And I was an only child. My nearest

12:57

neighbor was miles away. In fact, from

12:59

my home you couldn't even see another

13:00

house from my parents' house until I was

13:02

in my early 20s.

13:04

And so for me to to play with another

13:06

child, I had to get on a bike and spend

13:09

half an afternoon biking to their house.

13:12

So I got very familiar with the farm and

13:14

I was always just fascinated

13:16

by the veterinarians that would come

13:18

onto these farms and fix horses and fix

13:21

cattle and fix sheep and fix chickens.

13:24

And I always thought it was so cool that

13:26

these animals could be laying on the

13:27

ground seemingly about to die and the

13:30

veterinarian would come come on board

13:32

and do a bunch of stuff and the next

13:33

thing you know this horse was up and a

13:34

few days later it was galloping back in

13:36

the field or

13:37

um you had

13:39

all kinds of strange things that would

13:41

happen on this farm and there was always

13:42

someone arriving to just fix it and I

13:44

started to get fascinated with medicine

13:47

I guess in that way. I always found it

13:49

as a way to take something that was sick

13:51

and helpless and sort of help it get

13:53

back to normal function.

13:55

And I found out in the eighth grade uh

13:58

sometime during my eighth grade year

14:00

that uh I was clinically photographic.

14:03

So I have a clinical level of

14:04

photographic recall which is different

14:05

than just having a

14:07

uh visual memory. I'm I'm clinically

14:09

photographic so I have a voluminous

14:10

capacity to recall things that I read

14:13

even if I don't understand them which is

14:15

why I never read for pleasure. Um I'm

14:17

very cautious about what I read and very

14:19

cautious about what I look at

14:21

um because I record everything. So I

14:23

can't be flying on American Airlines and

14:25

take the seat back magazine out and read

14:27

the seat back magazine because

14:30

3 months later I'll tell you where the

14:31

sales center is for a

14:32

condo project in Buenos Aires. You know

14:34

it's so I can I can fill my brain with

14:36

senseless things like that and record it

14:38

and regurgitate it um or I can fill it

14:41

full of things that that fascinate me

14:43

and so I naturally gravitated towards

14:45

science. Is that what they call a

14:46

photograph photographic memory?

14:48

Photographic memory is usually someone

14:50

that can remember seven numbers in

14:52

sequence. They have extremely good

14:55

recall for things that they visualize.

14:58

Clinically photographic is being able to

15:01

regurgitate voluminous amounts of

15:02

information with incredible accuracy. So

15:05

I still remember

15:07

you know section 15.2 sub paragraph B of

15:09

my first employment contract that I

15:10

signed 28 years ago.

15:12

Really?

15:13

Really? Yeah. I remember I remember

15:16

that. It was actually with a trading

15:17

firm

15:18

um that I signed it with so

15:20

you know when I read peer reviewed

15:21

papers um in scientific journals, uh it

15:24

doesn't make me more intelligent than

15:26

someone because very often I can recall

15:28

information that I don't understand. So,

15:30

I can regurgitate voluminous amounts of

15:32

information. So, subjects that rely on

15:35

rote memorization like chemistry,

15:36

biology, neurobiology, microbiology, a

15:40

lot of these sciences don't actually

15:42

make sense.

15:43

Right? That you just have to memorize

15:45

how they operate. Chemistry in a lot of

15:47

ways doesn't make sense. Right? Um you

15:50

just have to remember what happens when

15:52

you put these two elements together,

15:53

they create this. So, you know, when you

15:55

pull a carbon bond or you double a

15:57

carbon bond here or hydrogen bond there

15:59

that you you that the molecule shifts in

16:01

this way.

16:02

And so, I gravitated towards science. I

16:04

got my undergraduate degree in biology.

16:07

How did you do? Um I did very well. I

16:09

actually found it quite the breeze. Um

16:12

my you know, when I was an undergrad, my

16:14

you know, biology, chemistry were

16:16

considered the toughest majors. And I

16:19

went to branch campus University of

16:20

Maryland. A lot of my roommates were

16:22

political science, not investing on

16:24

political science majors at all, or

16:25

philosophy, or psychology, and they got

16:27

to party all the time, and you know, I

16:29

had

16:30

really intense classes, you know,

16:32

morphology of thallophytes and chemistry

16:34

and and you know, biomechanics and and

16:38

science and

16:40

and a lot of plant biology courses and

16:41

human biology courses.

16:43

But I was able to have the same kind of

16:45

social life that they were because I

16:47

would, you know, record a lecture and

16:49

then I would regurgitate the lecture

16:50

when I needed it during during a test.

16:52

Is it a gift or a curse? Because with

16:54

all things in life that appear to be

16:55

gifts, there's often some kind of hidden

16:57

curse. Well, the curse is that I can't I

16:59

I rarely read for pleasure. Um it's very

17:02

difficult to read for pleasure because I

17:03

record all of that information. So, when

17:06

I'm going in to read a book, you know, a

17:08

lot of people

17:09

read a book that they didn't really

17:11

like,

17:12

it doesn't stay with them. Um or they

17:14

read a fiction novel, it doesn't really

17:15

give them any benefit. It's not doesn't

17:17

doesn't feed them, but they do it to

17:19

relax or they do it for entertainment.

17:22

For me, it would take up storage.

17:25

I would record that information. So,

17:27

there's no need for me to have that

17:29

information in my possession. I find

17:31

other ways to

17:32

to relax and

17:34

and and meditate and unwind, but reading

17:37

for pleasure is just just not one of

17:39

them. So, I guess that could be a curse.

17:42

But, you know, then I went to grad

17:43

school for human biology. I went to

17:45

another 4 years of school for human

17:47

biology. So, I had 8 years of science.

17:49

And then I was um

17:51

either going to go into chiropractic or

17:53

into orthopedic medicine. I really like

17:56

the idea of orthopedics because again,

17:57

an orthopedic surgeon was somebody that

17:59

fixed people. And

18:01

you know, an internist was somebody that

18:03

just managed disease. You know, they

18:05

took a

18:06

obese hypertensive diabetic uh patient

18:09

and they just kind of managed their case

18:11

throughout their lifetime, but I felt

18:12

like an orthopedic was somebody that you

18:14

know, you came in and you had a broken

18:15

leg and

18:17

they

18:18

you know, fixed your broken leg and and

18:20

you were back to normal. I like the idea

18:21

of fixing things and and kind of

18:23

returning function back to people. And I

18:26

took some time off, uh you know, after I

18:27

got my human biology degree and I ended

18:29

up going into

18:31

a rare area of science, um this

18:33

mortality science,

18:35

um

18:36

for life insurance and secondary life

18:38

insurance

18:39

where you would take medical records and

18:42

demographic data and you would use this

18:44

in what's called a probabilistic model

18:46

to predict life expectancy.

18:48

And I was fascinated by it cuz I loved

18:50

the big data nature of it. And for years

18:52

I actually subscribed to the fact that

18:55

this was just data.

18:57

And

18:58

that

19:00

I had no influence on this person's

19:02

outcome. I didn't put them in this

19:04

position nor am I responsible for

19:05

getting them out of this position.

19:08

But, eventually I think as both my faith

19:10

grew and my awareness of the fact that

19:13

there were human beings on the other

19:14

side of these spreadsheets,

19:16

I finally made a conscious decision to

19:18

say, "What am I doing? I'm going to

19:19

spend the rest of my life just

19:21

predicting death um for the monetary

19:24

gain

19:25

of an investor

19:26

versus taking this information and

19:29

helping people live healthier, happier,

19:31

longer, more fulfilling lives. Because

19:33

by studying medical records and you

19:35

know, I read you know,

19:37

8 to 10 hours a day, 6 days a week,

19:40

thousands and thousands and thousands of

19:42

medical records and extracted from those

19:45

how we would impact somebody's outcome

19:48

of their life, you know, how how it

19:50

would impact the length of or the the

19:53

shortness of the duration of their life,

19:54

their health span or their lifespan.

19:56

And once you realize there's human

19:57

beings on the other side of the

19:58

spreadsheet, you start to feel like

20:00

you're sitting behind a thick glass wall

20:02

just watching blind people walk into

20:04

traffic.

20:05

And

20:06

towards the tail end I used to

20:08

artificially kind of manipulate the

20:10

record. I would say, "Well, what if I

20:11

what if I artificially fixed this

20:13

person's D3 deficiency? What if I cured

20:15

the anemia? What if I actually

20:18

um noticed that they had a gene mutation

20:20

called MTHFR instead of trying to fix

20:21

them with folic acid, I fixed them with

20:23

methylfolate?" And you would just see

20:26

the life expectancy jump. In the model?

20:28

In the model. Yeah. Right. And this

20:30

model was based on very large data. So,

20:33

it was very likely to have a real life

20:34

impact. And when I say we predicted

20:36

death to the month, it was based on that

20:38

patient's current position. And doesn't

20:41

mean that they couldn't walk out the the

20:43

next day and and and get hit by a bus or

20:45

or die in a commercial airline, you

20:47

know, disaster or something like that.

20:49

But there's there were standard

20:50

deviations that would account for those

20:52

kinds of risks. What the insurance

20:55

company wanted to quantify or the

20:56

annuity company or the reverse mortgage

20:57

company wanted to quantify was based on

21:00

this person's everything that we know

21:01

about this person right now, the

21:03

function of their liver, the slightly

21:05

hypoxic anemic profile that they have,

21:07

the deficiencies that they have in

21:08

vitamin D3, the suppressed immune

21:10

system,

21:11

um you know, the elevated visceral fat,

21:13

the body mass index, the bone mineral

21:15

density. Based on all of these things,

21:17

when do these things all meet and

21:20

actually cause a catastrophic event

21:21

catastrophic event? And there were

21:23

things called mortality debits and

21:25

comorbidity debits and you know, we put

21:27

all these together and I really like the

21:28

data of it. But realizing that there

21:31

were human beings on the other side of

21:32

the spreadsheet just woke me up and I

21:33

said, you know, I have this

21:38

chasm of knowledge. Even though I'm not

21:40

a physician,

21:41

I have a fundamental understanding of

21:43

human physiology. I'm fascinated by

21:46

human physiology and ways that we can

21:48

improve um lifespan and health span.

21:52

And so why wouldn't I take this

21:54

gift and get into an industry

21:57

like wellness or functional medicine

21:59

where I could actually make a

22:00

difference. Where instead of predicting

22:02

death, we could extend life. And in

22:04

doing so, help people live healthier,

22:06

happier, longer lives, more fulfilling

22:07

lives, get rid of a lot of the ailments

22:10

that people suffer from. I mean, when

22:12

you when you start to peel back the way

22:14

that modern medicine defines a lot of

22:16

conditions that we have, you know, take

22:18

um depression or anxiety or ADD or ADHD

22:21

because a lot of your listeners are

22:22

probably entrepreneurs. And a lot of

22:25

entrepreneurs have very active minds and

22:27

they've been told they have attention

22:28

deficit disorder, right? Or attention

22:30

deficit hyperactivity disorder. Well,

22:32

attention deficit disorder is not an

22:34

attention deficit at all.

22:36

Um we misdefine these things. It's an

22:38

attention overload disorder because you

22:40

see in the human brain, we don't just

22:41

create thought.

22:43

We also dismantle thought. It is just as

22:45

important for you to be able to create a

22:47

new thought or feeling as it is to

22:49

dismantle it. Right? And if you don't

22:52

degrade thought called catecholamines,

22:54

if you don't degrade them, then there's

22:56

a gene that governs this,

22:57

catechol-O-methyltransferase.

22:59

If you create thought at a faster rate

23:01

than you degrade thought, then the mind

23:02

gets very clouded. And so, attention

23:05

deficit disorder is attention overload

23:07

disorder. It's too many windows open at

23:09

the same time.

23:11

So, modern medicine says, "Well, if the

23:12

mind's racing, let's put an amphetamine

23:15

into the body, race the central nervous

23:17

system to match the pace of the mind."

23:20

And this is a very poor choice, right?

23:22

Because eventually this will burn you

23:23

out. Can actually change the

23:24

neuroplasticity of the brain. Rather

23:26

than put the right amino acids back into

23:28

the body, the right complex blend, um

23:31

the right methylated folates, so that

23:34

the mind can actually begin to quiet.

23:36

What would you say to someone that says

23:37

that ADHD is also in some ways a result

23:40

of some early trauma?

23:42

Well, you know, trauma's always

23:44

fascinating to me. Trauma can trigger

23:46

methylation. Trauma can interrupt the

23:48

methylation cycle.

23:50

Right? But the the idea in modern

23:52

medicine that you have some kind of

23:54

trauma, you have a disrupted

23:55

relationship with your

23:57

mother, for example, and that somehow

23:59

we're going to go and put

24:00

neuroplasticity altering chemicals into

24:02

the brain, and it's going to fix this

24:03

30-year broken relationship you have

24:05

with your mother. To me,

24:07

it doesn't Yeah, yeah, yeah. make any

24:09

sense, right? And so, it's not at all to

24:13

poopoo trauma, to to to put trauma down.

24:15

Depression really exists, anxiety really

24:17

exists, but but if you actually look at

24:19

how we define these conditions, take

24:20

depression, for example. We define

24:22

depression, at least in America, we

24:23

define depression as an inadequate

24:25

supply of serotonin.

24:27

Right? So, if you're a low in serotonin,

24:29

you're by definition depressed. So, then

24:31

you would think that the solution would

24:33

be to raise serotonin.

24:35

Right? If we define depression as low

24:36

serotonin, you'd think that the solution

24:37

would be to raise serotonin, but that's

24:38

not what we do. We take people that are

24:40

depressed and we put them on SSRIs,

24:42

selective serotonin reuptake inhibitors.

24:45

And what these do is they ration what

24:46

little serotonin these people have.

24:48

So, by definition, it never raises

24:51

serotonin. So, by definition, it never

24:53

ends depression. I mean, I have I have

24:55

clients come in to see me all the time,

24:57

and and and and our clinical team and

24:59

then I'll say, "Well, how long have you

25:00

been on antidepressants?" They say, "15

25:03

years, 18 years." And my first question

25:05

is, "Well, when did you think it was

25:06

going to kick in?"

25:08

Right? So, if we understand that

25:10

serotonin is actually methylated in the

25:11

gut, this process that I'm talking about

25:13

is called methylation. We actually make

25:14

serotonin from an amino acid called

25:16

tryptophan, the one that's famous for

25:18

making you sleepy after Thanksgiving

25:19

dinner. I know you guys don't have

25:20

Thanksgiving in the UK, but I'm from

25:23

America, but um so, you guys we eat a

25:25

lot of turkey on Thanksgiving and turkey

25:26

has a lot of tryptophan. So, when you

25:28

take um tryptophan methylate it into the

25:31

neurotransmitter serotonin, of which 90%

25:34

resides in your gut.

25:35

Methylate is basically processing.

25:36

Yeah, the processing. It's like the

25:38

refining process. Crude oil gets refined

25:40

into gasoline.

25:41

Amino acids get methylated into

25:43

neurotransmitters. Okay. And so, this

25:46

process of methylation

25:48

when it's broken and and it can be

25:50

relatively easy to fix. When it's broken

25:53

it means that we have an impaired

25:54

ability to create. We have an impaired

25:56

ability to um refine a raw material and

25:59

it leads to this deficit. Well,

26:01

serotonin for example, 90% of it resides

26:04

in your gut. So, if you don't have it

26:05

here, you can't have it here. So,

26:07

depression rarely begins in the outside

26:09

environment. It usually begins in the

26:11

gut. Now, it may be trauma that led to

26:13

the deficiency.

26:14

But, the fix is not in a chemical or

26:18

synthetic or pharmaceutical blocking the

26:20

brain's capacity to uptake these

26:22

neurotransmitters. The fix is in

26:24

restoring adequate levels to the body so

26:26

it can naturopathically make its way

26:28

back up the vagus nerve and and arrive

26:30

to the brain. Sim- similar things are

26:32

true with anxiety. I mean, if you

26:34

actually have ever suffered from or know

26:36

somebody who's suffered from anxiety, if

26:38

you ask them three questions, you can

26:40

find out very quickly that their anxiety

26:43

is not coming from a cluster of

26:44

symptoms. It's not coming from their

26:46

outside environment. It is coming from

26:47

within them. It's coming from their

26:49

physiology.

26:50

Right? I mean, if you know someone who's

26:52

suffering from anxiety and you say well

26:54

have you had anxiety on and off

26:56

throughout your lifetime?

26:58

The most of the time they'll say yes.

27:00

And then if you say can you point to the

27:01

specific trigger that causes it?

27:03

Very often they'll say no.

27:05

I mean yes I know some of my triggers

27:07

but I could be sitting in a podcast just

27:09

like this in a very calm environment

27:11

there's no threats around and all of a

27:12

sudden I get overwhelmed by anxiety. I

27:14

can be driving home from work on an

27:15

otherwise innocuous day and I can be

27:17

overwhelmed by anxiety. Well that is not

27:19

coming from your outside environment.

27:21

Right? This is coming from a process

27:22

called methylation and it is caused from

27:25

excess catecholamines entering the brain

27:27

and an inability to down regulate these.

27:30

So the body's entering this mild fight

27:31

or flight response without the presence

27:34

of a fear.

27:36

See remember that as sophisticated as we

27:38

like to think our brains are it's really

27:40

not. Our brain is very primal.

27:43

You know what the brain cares about? The

27:43

brain cares about survival.

27:45

And so doesn't care how fat or skinny

27:47

you are how pretty or

27:49

ugly you are it doesn't care about your

27:50

skin your hair it cares about survival.

27:52

And so when we understand that the brain

27:54

does not know the difference between

27:56

perception and reality

27:58

we start to understand how it can play

28:00

tricks on us. So I always use the

28:02

example that let's say you drove home

28:04

tonight

28:05

and you got out of your car when you got

28:06

home you got out of your car and

28:08

somebody was standing in front of you

28:09

with a knife.

28:10

It's a very real threat. Right? You'd

28:11

have a fight or flight response. Your

28:12

pupils would dilate your heart rate

28:14

would increase your extremities would

28:15

flood with blood your hearing would get

28:17

very acute your brain would flood with

28:18

catecholamines you're getting ready to

28:19

fight or flight.

28:21

But you could also be laying on the 30th

28:24

floor of a condo building in bed and

28:27

start thinking about getting eaten by a

28:28

shark.

28:30

There is zero chance of a shark getting

28:31

out of the ocean going up a 30th floor

28:33

elevator. Right? Coming into your condo

28:35

and biting you in that bed but you can

28:37

have the exact same response.

28:38

If you're watching a movie or something.

28:40

Exactly. So one is entirely real one is

28:42

entirely perceived. The physiological

28:45

response is identical.

28:47

So now once we understand this, now we

28:50

begin to understand how I can feel the

28:52

presence of a fear, which is what

28:54

anxiety is. It's a fear of something

28:55

happening in the future. Usually it's

28:57

not going to happen. Usually hasn't

28:58

happened in the past and is not likely

29:00

to happen. But it's it's this fear

29:03

starts to build up. You start to get

29:05

very anxious. It can actually change

29:07

your heart rate um to the point where

29:09

you can panic attacks can land you in

29:10

the hospital.

29:12

Um or it can be mild enough that it just

29:13

causes you anxiousness. Mild anxiety.

29:16

But there's no presence of a fear. And

29:18

so you start trying to correlate it to

29:19

your outside environment. Starts to

29:20

drive you crazy because you go, "Well, I

29:22

don't get it. I'm on vacation with my

29:24

wife and or my spouse and my kids and

29:26

I'm in the resort of a lifetime. I've

29:28

been here a thousand times. I love this

29:29

place. There's no reason I should feel

29:31

like this." But all of a sudden you have

29:33

this feeling of anxiousness, anxiety. So

29:36

these are these are lack of raw material

29:39

in the human body. My mission is to try

29:40

to help people by taking a genetic test

29:44

um once in their lifetime, find out

29:47

where is methylation broken, and then

29:50

stop supplementing just for the sake of

29:52

supplementing, and start supplementing

29:54

for this deficiency so your body can

29:55

thrive. In the case of people that are

29:57

listening to this now and they can

29:59

pinpoint the moments where they've

30:01

gotten anxiety. So say they've I

30:03

remember I had one guest on the podcast

30:05

maybe two years ago

30:06

and after he became famous, he developed

30:09

social anxiety.

30:10

Mhm. So he whenever he would be with

30:11

around a lot of people, he'd feel that

30:13

sense of anxiety. And then from that

30:15

sort of capitalistic moment, then when

30:18

he's at home, he'd get the same rush of

30:19

anxiety, but he would point to that

30:20

capitalistic moment of becoming famous,

30:22

and then some things had happened in his

30:24

life, and then he'd get anxiety at home

30:26

when nothing was going on. Right.

30:28

In that situation, what's the the

30:30

nutrient

30:30

you go. So so now you've you've you've

30:32

interrupted methylation because there is

30:34

one where there is the presence of fear

30:36

and there's one where there isn't. Yeah,

30:37

there's the absence of a fear. So to be

30:40

very specific, anxiety, true anxiety

30:42

does exist, but you can point to the

30:44

specific trigger that causes it. So, for

30:46

example, if you if you have a fear of

30:47

heights and you walk to the edge of a

30:49

30th floor balcony and look over it,

30:50

you're going to feel anxiety. If you're

30:52

claustrophobic and you step on a really

30:53

crowded elevator, you're going to feel

30:55

anxiety. But, if you're claustrophobic

30:58

and you're sitting at home

31:00

and you start to become overwhelmed with

31:02

anxiety, this is actually not coming

31:04

from that trigger. This is coming from

31:06

your physiology. And the way that we

31:09

deal with stress, right? And like

31:11

cortisol, when you measure cortisol

31:13

levels, cortisol's not really a measure

31:14

of how much stress is in your life, it's

31:16

a measure of your body's reaction to

31:17

stress. So, why are some people more

31:19

resilient to stress and don't have

31:21

anxiety attacks and why are other people

31:23

not as resistant? Again, this is not to

31:26

say that if you didn't have

31:28

a violent attack in your life or or a

31:31

terrible car accident that sometimes

31:33

when you've had a

31:34

you know, a a vehicle accident and you

31:35

approach an intersection,

31:38

the the the

31:39

the memory of having been T-boned, you

31:42

know, recently and badly injured is

31:44

going to give you anxiety. But, the

31:46

majority of people are not suffering

31:47

from that type of hyper-specific

31:49

situational anxiety. They're suffering

31:51

from something called generalized

31:52

anxiety or idiopathic anxiety, which

31:55

means of unknown origin. So, for

31:57

somebody who's very famous and gets into

32:00

a a crowd and doesn't know who's coming

32:03

at them, that's a very I wouldn't even

32:05

define that as anxiety. That's a very

32:07

primal instinctual reaction to a real

32:09

fear. Mhm. Right? Just like walking to

32:12

the edge of a 30th floor balcony. What's

32:14

not a primal reaction to a real fear is

32:17

when there is no presence of a fear,

32:19

especially if that incident has never

32:21

happened and you aren't even sure what

32:23

you are afraid of or why you are anxious

32:25

or why you have anxiety, then this is

32:28

coming from your physiology. So, how

32:30

would you treat that? You look at the

32:32

different There are five major

32:34

actionable genes that I like to look at

32:36

and they're what's it called their sub

32:38

alleles. And when you find out what

32:39

they're deficient in, you start to

32:42

supplement with things like SAM-e,

32:44

S-adenosylmethionine,

32:46

methylated forms of vitamins,

32:48

L-methionine, the the proper balance of

32:51

B complex,

32:53

methylated forms of folic acid or folate

32:56

called methylfolate. And what happens is

32:58

now the body has the capacity to degrade

33:01

these neurotransmitters that are causing

33:04

this fight or flight. This group of

33:06

neurotransmitters called catecholamines

33:08

and the anxiousness that follows. And

33:11

you'll find that the majority of people

33:13

that suffer from idiopathic anxiety or

33:15

generalized anxiety because of low

33:17

serotonin, they also have gut issues.

33:19

You show me a person that's truly

33:21

depressed and I'll show you somebody

33:22

that's also suffering from severe gut

33:25

issues. Either gas or bloating or

33:26

diarrhea, constipation, irritability,

33:28

cramping because the same

33:30

neurotransmitters that affect these

33:31

emotional states also are responsible

33:34

for the motility of the gut, the speed

33:36

of the gut. This is the most overlooked

33:38

thing in all of bariatric medicine

33:40

because

33:41

people that believe that they have all

33:44

of these allergies. Well, I'm allergic

33:45

to wheat, soy, corn, dairy, blueberries,

33:47

bananas,

33:48

uh you know, gluten.

33:50

Yes, sometimes those individual

33:52

allergies do exist, but the majority of

33:53

time even if you talk to somebody who

33:56

says, "Yeah, I I I get bloated or I deal

33:59

with gas or cramping or diarrhea or

34:01

constipation or irritability. I deal

34:02

with all of these gut issues, irritable

34:04

bowel syndrome, uh Crohn's Crohn's

34:07

disease, ulcerative colitis." All these

34:08

names that we give to conditions of the

34:10

gut. When you ask them, "Well, what are

34:12

you allergic to?"

34:13

And they give you this laundry list of

34:15

things. And then you ask them another

34:16

question and say, "Well, if you're

34:18

really allergic to corn,

34:20

is there ever a time that you can eat

34:21

corn and not have a reaction?" Majority

34:23

of time people will say yes.

34:25

Okay, well, right there you know you

34:26

don't have an allergy. Allergies are not

34:28

transient, allergies are consistent.

34:30

Right? You don't wake up Monday morning

34:31

and being allergic to milk and then

34:33

you're unallergic on Wednesday afternoon

34:35

and then reallergic on Saturday morning.

34:37

But what happens when people have gut

34:38

issues that they can't explain is they

34:40

always correlate it to what they last

34:41

ate. And it's hard to make this

34:43

connection. They're like, "Well, wait a

34:44

second. I ate the same thing Monday and

34:47

I was fine and I ate the identical food

34:49

on Wednesday and I blew up like a tick."

34:51

So, this is not an allergy. This has to

34:53

do with the motility of the gut. So, if

34:55

you don't know what gene mutation you

34:58

have that is causing a deficiency, then

35:01

you don't know what to supplement with

35:02

to restore gut motility. But once you

35:04

do,

35:06

the gut goes back to its normal pace.

35:07

What's gut motility? It's the pace of

35:10

the gut. So, if you remember Henry Ford

35:13

was actually not made famous for the

35:14

automobile. He's made famous for

35:15

something called the assembly line. So,

35:17

the assembly line was just a glorified

35:19

conveyor belt, right? And when you

35:20

walked into his factory, they put a part

35:22

on it at one end and about every 6 ft

35:25

somebody stood and tinkered with that

35:27

part. So, it went to me, I tinkered with

35:29

it, it moved to the guy to my right, he

35:31

did something to it, moved to the guy to

35:32

his right, and by the time it reached

35:34

the end of that conveyor belt, it's

35:35

fully assembled. This is very analogous

35:37

to how the human intestinal tract works.

35:40

It's 30 ft long. It's a giant conveyor

35:42

belt. You put parts on it at one end as

35:44

they exit the stomach in a very acidic

35:47

environment and it moves slowly towards

35:50

the rectum and before it exits the

35:51

rectum, it's in a relatively alkaline

35:53

environment. So, instead of having

35:55

people standing along a conveyor belt,

35:57

you have bacteria that are graded by pH.

36:01

This sequence is very important.

36:05

So, imagine what would happen if Henry

36:06

Ford walked into his factory one day and

36:08

doubled the speed of the conveyor belt.

36:10

The entire assembly line would break

36:12

down. Not because there's anything wrong

36:14

with the parts, the contents, not

36:16

because there's anything wrong with the

36:17

people that are working there, the

36:18

bacteria, but because you changed the

36:20

speed. What if he went in there one day

36:22

and reversed the the conveyor belt? What

36:24

if he just ran it in the opposite

36:25

direction?

36:27

Screw up. Just It would screw the whole

36:29

thing up, right? So, by changing the

36:32

pace of the gut, the speed of the gut,

36:35

the conveyor belt, I've I've ruined this

36:38

sequence of events. And I spend a

36:41

lifetime trying to figure out what's

36:42

wrong with the parts, what's wrong with

36:44

the workers, what's wrong with the

36:46

conveyor belt itself. Nothing.

36:48

It's how quickly or slowly it's running

36:50

because the motility, this peristaltic

36:53

activity, is affected. And once you

36:55

supplement for this deficiency and you

36:57

return that activity to normal, you find

37:00

that

37:01

all of a sudden these strange allergies

37:03

eviscerate and all of this gas and

37:05

bloating and diarrhea and constipation

37:06

and irritability and all of this

37:08

inability to equate things that I'm

37:10

eating back to what is going on in my

37:12

gut seem to go away.

37:14

It's It's true with all kinds of

37:16

conditions. You know, we we have

37:17

subscribed in this world to the fact

37:20

that we are so affected by disease and

37:22

pathology. And once I get you to

37:25

to subscribe to the fact that you have a

37:27

disease and you can get you to subscribe

37:29

to a lifetime of medication.

37:31

You know, this is true in hypertension.

37:33

You know, it's another huge, you know,

37:35

we think genetically inherited disease.

37:36

And a lot of families have, you know,

37:38

grandfather had high blood pressure, um

37:40

dad had high blood pressure. Now the son

37:42

has high blood pressure. So, they go,

37:43

"Oh, it's genetic. It's genetically

37:45

inherited high blood pressure." Okay,

37:47

well,

37:48

what gene is being passed down from

37:49

these generations to cause this? Well,

37:51

they don't know.

37:52

Okay, well, then we know all of the

37:53

genes. So, if we don't know what gene it

37:55

is, then let's look at the methylation

37:57

genes. Let's look at the genes in the

37:59

human body that refine raw materials.

38:02

Because if you have a certain

38:03

deficiency, for example, in

38:05

hypertension, very often, not always,

38:07

but very often it's it's caused by

38:10

elevated levels of an amino acid called

38:13

homocysteine.

38:14

You have in your bloodstream right now,

38:15

I have in my bloodstream, every listener

38:17

to this podcast has homocysteine in

38:18

their blood. Because we we in in the

38:21

process of normally refining this amino

38:24

acid into something called methionine,

38:26

we

38:27

um we keep this level at a reasonable

38:30

level. But when you can't break

38:33

homocysteine down, it rises. When

38:35

homocysteine rises, it's very irritating

38:38

to what's called the endothelial lining

38:40

of the blood vessel. It in it irritates

38:42

the lining of the blood vessel. Well,

38:44

when you irritate a blood vessel, it

38:46

clamps down.

38:47

Well, if you make the pipes smaller in a

38:50

fixed system,

38:52

pressure goes up.

38:53

You have 63,000 mi of blood vessel in

38:55

your body. It doesn't take much

38:56

narrowing to drive pressure up. And so

38:59

these people end up with hypertension,

39:00

high blood pressure, and they go to the

39:01

doctor, and the doctor does an EKG, it's

39:03

normal. An EEG, it's normal. Stress EKG,

39:06

it's normal. Cardiac cath, normal. Heart

39:08

and lung sounds, normal. Dye contrast

39:10

study, normal. The entire cardiac

39:11

workup's normal.

39:13

And then they still start pounding on

39:14

the heart with medication because they

39:16

can't figure out why

39:18

the blood pressure's up, but they never

39:20

actually looked at what raw material was

39:22

missing in their body not allowing them

39:24

to bring homocysteine down and allowing

39:26

the vascular system to relax.

39:28

As the vascular system relaxes, pressure

39:30

returns to normal.

39:32

You spent 22 years

39:34

at that insurance company looking at the

39:36

database that you described. Um

39:39

and as you're as you're saying there,

39:41

you know,

39:42

much of the sort of medical profession

39:44

and I think most of society

39:45

Yeah, and I don't want to attack the

39:46

medical profession. I really you know,

39:47

I'm in no I am a huge lover and believer

39:50

in modern medicine. It's saved people

39:52

very close to me. I mean and trust me,

39:54

if I had a windshield at 20 miles an

39:55

hour, I want a surgeon, I want

39:57

painkillers, I'm going to the ER. But

39:59

there's been this kind of this it's not

40:01

a preventive approach to disease that

40:02

we've taken over the last couple of

40:03

decades. It really is, you know, to to

40:05

put a band-aid on something that's

40:07

that's emerged. And as you said at the

40:08

start of this conversation, these

40:10

predicaments, these um diseases emerge

40:12

decades before we even see the symptoms

40:14

often times. Um at the end of your 22

40:17

years at the insurance company, you

40:18

started to

40:20

look at these things which you call

40:21

modifiable risk factors.

40:23

Yes. What is a modifiable risk factor?

40:26

And what are the most common modifiable

40:29

risk factors in your view? Well, I mean,

40:31

one of the most common ones that I saw

40:32

was um modifiable risk factor is

40:35

it's a risk factor that you have

40:38

and if you changed it, it would have

40:41

modified it, it would have a

40:42

demonstrative impact on the trajectory

40:45

of your life. So, for example, anemia,

40:48

right? Low low hemoglobin, low red blood

40:50

cell count, low oxygen transport in the

40:53

blood will exacerbate just about any

40:55

condition that you have, right? So, if

40:57

you are hypertensive and anemic, if you

41:00

are diabetic and anemic, if you are

41:02

morbidly obese and anemic, um meaning

41:05

you have low blood oxygen because you

41:06

have low red blood cells, low

41:07

hemoglobin,

41:09

what if you could modify the anemia?

41:12

Well, if you could modify the anemia,

41:13

carry more oxygen in the blood, you'd be

41:15

much more resilient to all of these

41:17

conditions.

41:18

Right? So, the same thing is true with

41:21

dementia and Alzheimer's and and uh

41:24

cognitive function. As you impair

41:25

cognitive function, you increase the

41:27

incidence of all-cause mortality. So, a

41:30

modifiable risk factor would be

41:32

something like um looking at the levels

41:35

of vitamin D3 in the blood blood in the

41:38

body. So, it's estimated that roughly

41:40

50% of the world's population is

41:42

clinically deficient in vitamin D3. Why

41:44

is that important? Um well, vitamin D3

41:46

is the only vitamin that a human being

41:48

can make on our own. There's hundreds of

41:50

vitamins in your bloodstream right now.

41:52

You're only capable of making one.

41:54

And it's vitamin D3, cholecalciferol. We

41:57

make it from sunlight and cholesterol.

41:59

You don't even need to eat to make this

42:01

vitamin. There is not a single cell in

42:03

the entire human body that does not have

42:05

a receptor site for this vitamin. It

42:07

also acts like a hormone. It's calcium

42:09

transport molecule. It's enormously

42:11

impactful in your um immune system.

42:15

Clinical deficiency in vitamin D3 was at

42:17

one time the second leading cause of

42:19

morbidity in COVID. You know, when they

42:21

said COVID disproportionately affected

42:22

minorities. If you ever heard that, it's

42:24

true. So, how did COVID

42:26

disproportionately affect minorities? It

42:27

wasn't like the virus didn't start going

42:30

after certain minority populations.

42:32

Well, it actually did, but it wasn't

42:34

because they were not minorities. It had

42:35

to do with the pigment of their skin.

42:37

The darker the pigmentation of your

42:38

skin, the lower the vitamin D3. The

42:40

lower the vitamin D3, the more

42:42

compromised the immune system. The more

42:44

compromised the immune system, the more

42:45

susceptible you are to attack. That

42:47

suggests that if you have darker skin

42:49

like I do, that you should spend more

42:51

time in the sunlight. Yes, it does.

42:53

It absolutely does. I mean, you and I,

42:55

just based on our skin tone, if you and

42:57

I in a pair of board shorts went out and

42:58

spent 30 minutes in sunlight, I would

43:00

manufacture about 25,000 IUs,

43:03

international units of vitamin D3, you

43:05

would manufacture about 3,500. [ __ ]

43:07

hell, that's not fair.

43:09

It's not fair.

43:10

We can run faster.

43:11

But, you also

43:12

you also look better older and you don't

43:14

age, you don't get wrinkles as much, you

43:16

have better oily skin. There's some

43:18

trade-offs. So, so when you look at, you

43:20

know, African-American populations,

43:22

Latino populations, Middle Eastern

43:23

populations, they're the percentage of

43:25

vitamin D3 deficiency approaches 85%,

43:28

right? The truth is most of us are not

43:29

getting enough sun. It's not where that

43:31

we're getting too much. So, let's talk

43:32

about the depletion of this one

43:34

nutrient. Can I just ask them before we

43:36

move on to the depletion of that

43:37

nutrient?

43:38

Cuz I want to understand why that is.

43:40

I'm my guess was that because people

43:43

with darker skins have grown up in

43:44

sunlight, we spend more time in in the

43:47

sunlight, I'm guessing, which meant that

43:49

we adjusted our production of vitamin D3

43:54

to suit the environment we lived in.

43:55

Yeah, you actually have a better

43:57

barrier, right? You are um less

44:00

susceptible to skin damage from sunlight

44:03

than someone with lighter skin, right?

44:05

And so, if you actually look because I

44:07

used to be a population mortality

44:08

expert, um you know, the hot the longest

44:11

life expectancies um were centered right

44:13

around the equator of the earth. If you

44:15

look at most of the blue zones, they're

44:16

going to be

44:17

sort of closer to the equator of the

44:19

earth. They're going to be closer to

44:21

places where the sun shines longer

44:23

periods of time throughout the year.

44:24

The blue zones? The blue zones are

44:26

Sorry, the blue zones are areas of the

44:28

world where people live um

44:30

extraordinarily long life expectancies,

44:33

right? Mediterranean diets. So,

44:36

the longest life expectancies are

44:38

centered around the the equator. So, for

44:40

every 20° um uh

44:44

latitude, um cuz this is longitude,

44:46

every 20° latitude, there's a

44:48

precipitous drop in life expectancy

44:50

until you get to the poles,

44:53

where when I started in this industry, a

44:56

um the lowest the shortest life

44:58

expectancy on earth existed. So, when I

45:00

was born in 1970, a true Eskimo had a

45:02

56-year life expectancy. A 56-year life

45:06

expectancy had 56.

45:06

True Eskimo. So, these are these are at

45:09

the poles. Now, why? Because they would

45:10

go months without seeing the sun. And

45:13

when they did see the sun, they were

45:14

layered up. So, they had brittle bone

45:16

disease, they had autoimmune disease,

45:17

they had immunocompromised

45:19

conditions, all kinds of conditions that

45:21

go with severe depletion in vitamin D3.

45:24

And so, I would see this in the medical

45:26

record all the time, right? I would see

45:27

medical record after medical record, you

45:29

know, a patient has been had a decade or

45:32

longer of clinical deficiency in vitamin

45:34

D3. Well, when you deplete this nutrient

45:36

and you don't supplement for it. By the

45:38

way, vitamin D3 is so easy to supplement

45:40

for. Everybody should be taking vitamin

45:41

D3 unless you're getting adequate

45:42

sunlight.

45:43

5,000 IUs of D3 with 80 micrograms of

45:46

K2.

45:47

And you know, you you deplete this

45:49

nutrient, eventually you will develop

45:51

rheumatoid arthritis-like symptoms. Now,

45:54

you don't have rheumatoid arthritis, but

45:57

you have you have the same identical

45:59

symptoms when you're depleted in vitamin

46:01

D3. You start waking up sore and achy

46:02

like you had a workout the night before

46:04

when you haven't. The soles of your feet

46:06

and your ankles are tender when you get

46:07

out of bed in the morning to walk to the

46:08

bathroom, take your first pee. Um, your

46:11

your knees and hips and ankles start to

46:13

stiffen up. Then it goes to cross the

46:15

shoulders and eventually it's hard to

46:17

make a really tight fist. Well, if you

46:19

go to the wrong physician,

46:20

family medicine practitioner that's

46:22

seeing a lot of high volume of patients,

46:24

you start to give them that description,

46:25

they go, "You know what? You got you got

46:28

rheumatoid arthritis. I'm going to hit

46:29

you with something called

46:30

corticosteroids. I'm going to put you on

46:32

some prednisone first and then we're

46:33

going to transition you to a

46:34

corticosteroid like a methotrexate or

46:36

one of these

46:37

and and you're going to be fine. You

46:39

know, you you know, it's it's going to

46:40

push this off into the future." Well, we

46:42

knew that if you started corticosteroids

46:44

that at first they had an

46:45

anti-inflammatory effect, but you had

46:48

roughly 6 years and 1 day until you're

46:51

having a joint replacement. Because

46:53

first they're anti-inflammatory, but

46:54

then they eat the joint like a termite.

46:56

And so it was so accurate that if you

46:58

were a 60-year-old female and you were

47:00

misdiagnosed with rheumatoid arthritis

47:02

because you had a vitamin D3 deficiency,

47:04

I would artificially advance your age 6

47:06

years. I would schedule the joint

47:08

replacement that wasn't required, by the

47:10

way. I'll schedule the joint replacement

47:12

and then I would begin to reduce what's

47:13

called your ambulatory profile, how well

47:15

you ambulate, how well you move.

47:17

And we know now that sitting is the new

47:18

smoking, right? Sedentary lifestyle is

47:20

the leading cause of all cause

47:21

mortality. And so as I start to reduce

47:24

your ambulatory profile, I'm bringing

47:26

all of the diseases that exacerbate with

47:28

reduced motility.

47:30

So, if I back this up,

47:32

you were diagnosed with a condition that

47:33

you didn't have

47:35

because you had a deficiency in a very

47:37

simple nutrient. So, then you were put

47:39

on a medication that wasn't required.

47:41

This caused you to have surgery that you

47:43

didn't need because to replace a joint

47:45

that wasn't initially going to be

47:46

defective. This reduced your mobility

47:49

and it brought diseases forward that you

47:50

never would have caught and you died

47:52

early from a condition you never would

47:53

have had because you had a deficiency in

47:55

a nutrient that was misdiagnosed,

47:58

mistreated, and led to your early

48:00

demise. And I could give you hundreds of

48:02

examples like this, and this is why, if

48:04

you look at the 2016 Harvard study, the

48:06

2019 Johns Hopkins study,

48:09

medical error is the third leading cause

48:11

of death. Medical error?

48:12

Medical error. Is that misdiagnosis?

48:14

Misdiagnosis and medical error.

48:17

The average American

48:20

over 50, 52,

48:23

is on five prescription medications.

48:26

These are synthetic chemical

48:27

pharmaceuticals,

48:29

right? And in a lot of cases, we don't

48:32

know the long-term side effects of

48:33

these. In a lot of these cases, we

48:35

actually don't even know the mechanism

48:37

by which they work. If you actually open

48:38

the PDR that comes with the the the

48:41

disclaimer that comes with a lot of

48:42

these medications, you'll see that they

48:44

say mechanism of action unknown.

48:47

Well, it's a sleep medication. How does

48:48

it make me sleep? I don't know.

48:50

Just makes you sleep, right? In fact,

48:52

most people that are suffering from from

48:53

sleep issues are suffering from sleep

48:55

issues not because they can't sleep, not

48:57

because they're not tired, because their

48:59

mind is keeping them awake.

49:01

Right? If you have a gene mutation

49:03

called COMT,

49:04

catechol-O-methyltransferase,

49:06

then what happens to you is you lay down

49:07

to go to sleep,

49:09

and there's nothing more frustrating

49:10

than one spouse having this gene

49:12

mutation and one not, right?

49:13

god, I think you're talking about me and

49:14

my partner.

49:16

I'm the one that that I need some like

49:18

sound or something to go to sleep

49:20

because she's just out like a light.

49:21

There you go. See, and nothing's more

49:22

frustrating because so annoying. I

49:24

sometimes sleep in another room Okay, so

49:25

the reason why you're not falling asleep

49:26

is because when your environment quiets,

49:28

your mind wakes up.

49:29

And if you actually hone in on the kind

49:31

of things you're thinking about, you're

49:32

thinking about the most innocuous little

49:34

nonsense,

49:35

right? I mean, it's like did I get

49:37

everything on my grocery list? Did my

49:39

belt match my shoes today? Did I return

49:42

that email? It's nothing that couldn't

49:44

wait till the next day. 100%. Yeah, and

49:46

so and sometimes you can even catch

49:48

yourself going, "What am I doing?"

49:50

Thinking about the color of dishes I'm

49:52

going to have if I threw a party, which

49:54

I'm probably not going to throw. I just

49:56

like, how did I get down this rabbit

49:57

hole, right? And um this is because, you

50:01

know, the you're not quieting the mind

50:04

at the pace that you are

50:06

exciting the mind. So, in other words,

50:07

when you lay down to go to sleep and

50:08

your environment quiets, your mind

50:10

starts to wake up. So, you need to be

50:12

distracted so that your mind doesn't

50:14

continue to run. But, if you just put

50:16

the right amino acid balance, in fact, I

50:18

would bet my career you have this gene

50:20

mutation.

50:22

I will pay for your test if you don't.

50:23

It's $599. It's a cheek swab you do once

50:26

in your lifetime and it will tell you if

50:28

you have this gene mutation, COMT. And

50:30

if you do, I'll tell you exactly what

50:32

supplement to take and it will become a

50:33

permanent thing of your past.

50:36

What supplement should I take?

50:38

Don't end your sentence there, Gary.

50:40

So, it's called L-methionine.

50:42

Oh, I've heard And you take

50:43

L-methionine, you take methylfolate,

50:46

and depending on where the gene break is

50:48

and how severe it is, you add you may

50:50

add something called SAM-e,

50:52

S-adenosylmethionine.

50:54

And these sound like fancy names,

50:55

they're just fancy names for vitamins

50:57

and amino acids. They you all of these

50:59

are in your bloodstream right now. So,

51:01

when you look at the sequence of breaks

51:03

that somebody has, then you can tell

51:05

them exactly what supplement to take.

51:07

When they take the supplement, their

51:08

body's no longer deficient.

51:11

Right? Deficiencies over time express

51:14

themselves as all kinds of things. You

51:16

know, there's a lot of people that can't

51:18

reach elevated emotional states for any

51:19

prolonged period of time. This is

51:21

because mood and emotional states are

51:23

recipes, just like a

51:25

you know, a chef

51:26

bakes a recipe. But, if you went to a

51:28

bakery chef and you said,

51:30

"You can bake whatever you want.

51:32

You just can't use butter."

51:34

Okay, doesn't sound like a big deal.

51:35

It's just one ingredient. But, think of

51:38

the number of cookies, pastries, pies,

51:39

brownies, you know, that it would

51:40

affect. It would affect so many

51:42

different recipes just by removing one

51:44

ingredient. This is the same thing that

51:46

happens in human beings. If you couldn't

51:47

use serotonin to assemble mood, okay,

51:50

now any emotional state that requires

51:52

that neurotransmitter as a part of its

51:54

structure, you can't assemble.

51:57

And as it relates to that mutation, you

51:59

when when you people hear the term

52:00

mutation, they think of it as being

52:02

something that's happened in me. And I'm

52:04

maybe, you know,

52:06

one of a few that have that mutation.

52:08

Mutation sounds like a uh Yeah, it does

52:10

sound it sounds it does sound like an

52:11

alien's about to start like growing out

52:13

of your stomach.

52:14

it sounds like it you know, my sister

52:16

won't have it and it's just me.

52:17

Okay, so a gene mutation means that, you

52:20

know, we we get a copy of our genes from

52:22

each parent. Yeah. Right? And then these

52:26

the the copy of these genes is passed

52:27

down to us. And we know you know, most

52:29

of us are familiar with basic genetics.

52:32

Eye color, skin color, detached ear

52:34

lobes. Um but there's nothing you can do

52:37

with that information.

52:38

So beyond your ancestry, you know,

52:40

whether you got Irish heritage or Native

52:43

American Indian or or what have you. I

52:45

mean, those are if you did a 23andMe,

52:47

you'd be able to find your ancestry,

52:48

which is also your genetic history.

52:51

That's not actionable information.

52:53

When you look at the genes of

52:55

methylation,

52:57

right? And there are several of them. I

52:58

look at five majors and a few minors.

53:01

When you look at the genes of

53:02

methylation, these are the genes that

53:04

code for how materials are taken into

53:07

the body, are refined into the usable

53:10

form.

53:11

And these are not mutations that you're

53:15

going to suffer from. What they do is

53:17

they they they

53:19

cause deficiencies to arise in the body.

53:22

And it's this deficiency that leads to

53:26

the expression of these conditions.

53:28

And so when you can't adequately

53:32

methylate neurotransmitters, you have a

53:35

deficiency in a certain

53:36

neurotransmitter. Any mood, any

53:38

emotional state that requires that

53:39

neurotransmitter, you can't assemble. If

53:42

you have a deficiency in the ability to

53:43

quiet the mind, then you have excess um

53:46

neurotransmitters in the brain, which

53:48

cause it to stay in awaken state. So,

53:51

that gene that is responsible for my

53:53

loud mind when I go to sleep.

53:55

Mhm.

53:56

Do you have any idea the amount of

53:57

people that have that particular gene?

53:59

Well, 44% of the world's population has

54:02

a gene mutation called MTHFR,

54:04

methylene tetrahydrofolate reductase.

54:07

This is a

54:08

genetic mutation that impairs the

54:10

ability to convert folate into

54:12

methylfolate or folic acid into

54:14

methylfolate.

54:16

And that might not sound like a big

54:17

deal, but folic acid is one of the most

54:20

prevalent nutrients in the human diet.

54:22

And the thing about folic acid is that

54:24

folic acid is an entirely man-made

54:25

chemical.

54:27

Folic acid is not found anywhere on the

54:29

surface of the earth. You can't find

54:30

folic acid anywhere naturally in nature.

54:33

And yet, we give folic acid

54:36

to pregnant women when they get pregnant

54:38

because we tell pregnant women that

54:40

folic acid prevents neural tube defects.

54:42

Well, that's patently false. Folic acid

54:43

doesn't prevent anything. Folic acid has

54:45

to be converted

54:47

first into tetrahydrofolate then

54:48

dihydrofolate, but eventually it becomes

54:50

something called methylfolate. And that

54:52

prevents a neural tube defect. Well,

54:54

what if like 44% of the population and

54:57

44% of women, this woman has this gene

55:00

mutation and you give her folic acid,

55:02

the man-made version of folate, and she

55:05

can't convert it into methylfolate. This

55:07

is where postpartum depression develops.

55:10

And you know, I have not seen a single

55:12

peer-reviewed published clinical study

55:14

linking elevated levels of of

55:16

uh

55:17

hormones in female pregnancy to

55:19

postpartum depression. But if you give a

55:22

woman that can't methylate folic acid

55:24

1,400% of the daily allowance of folic

55:27

acid and she can't break it down, then

55:30

she goes nuts. And then eventually the

55:31

pregnancy ends, the depression goes

55:33

away, and so So blames it on the

55:35

pregnancy, not on the vitamin.

55:38

And you know, we do this over and over

55:40

and over again. You know, we have in in

55:42

in the modern industrial world, we try

55:44

to synthesize what occurs naturally in

55:46

nature in a laboratory. You know,

55:48

there's three types of B12, right?

55:50

Adenosylcobalamin, Hydroxocobalamin,

55:52

Methylcobalamin. These forms of B12

55:54

occur naturally in nature. There's one

55:56

that we make synthetically in a

55:57

laboratory. It's called Cyanocobalamin.

56:00

We make it from hydrogen cyanide.

56:03

It is useless in the human body. The

56:04

human body actually has to take it and

56:06

convert it into the active form of B12

56:09

called Hydroxocobalamin. And so, a lot

56:12

of times the supplements that we're

56:13

taking us are causing more harm than

56:15

good and we're taking them because the

56:17

doctor said, "Well, I'm pregnant. My

56:18

OBGYN told me to take this synthetic

56:20

chemical called folic acid which it's

56:22

always beyond me why anyone would think

56:25

that something that we make in a

56:26

laboratory that's entirely synthetic

56:27

that isn't natural, that isn't found

56:29

anywhere on the surface of the earth

56:30

could ever be required for optimal

56:32

health."

56:33

I mean, just in its on its surface that

56:34

sounds

56:36

strange to me. But,

56:38

you know, yet we do and we don't

56:40

understand this process of methylation.

56:42

So if you Once you understand where your

56:45

genetic mutations are, where the genetic

56:48

inability to refine raw materials is,

56:50

you can go about supplementing targeted

56:53

supplementing for that deficiency. So

56:55

you would recommend people take a

56:57

genetic test to find out these answers,

56:59

right?

57:00

everyone take that test once in their

57:02

lifetime. You know, I

57:04

I'm I'm in the camp that we're as humans

57:06

we're not broken. Like we're not broken.

57:08

You know what I mean? And so whenever

57:09

there's something going on in my body or

57:12

you know, a way that I am or a habit I

57:14

have that I'm like, "Why do I do that?"

57:16

I'm always trying to figure out the sort

57:17

of I guess the ancestral reason why that

57:19

might have helped me to survive or

57:21

what's going on. So when I hear

57:22

mutation, it's it sounds a little bit

57:24

like broken to Yeah, yeah. So I

57:25

shouldn't use the I mean, we we use the

57:27

term mutation, but basically, you know,

57:30

in our ancestry, right? We pass on our

57:33

entire genetic code. Which a lot of that

57:36

is hair color, eye color, skin color,

57:38

you know, our our our heritage. But we

57:40

also pass on how our body refines

57:43

certain raw materials. And it's not to

57:45

say that it's good or or or bad or or

57:48

broken or fixed, but there are certain

57:52

families and human beings that have an

57:54

inability to refine certain raw

57:55

materials. And if we could be aware of

57:58

this, it is astounding what happens to

58:00

human beings when you just give their

58:02

body the raw material to do its job. You

58:05

know, lots of people that have attention

58:07

deficit disorder or have trouble

58:09

sleeping will

58:10

also have a trouble trouble with

58:12

prioritization. They just don't know it.

58:14

They'll say things like, "I work really

58:16

well under pressure." Right, most

58:18

entrepreneurs say, "I work really well

58:19

under pressure." Do you say that? Oh my

58:21

god. Okay. So let me I only work when

58:23

there is pressure.

58:24

So I was like getting to school that

58:25

we'd only do the test when you know,

58:27

there was an hour left to go or you

58:28

know, the homework or whatever. Yeah, so

58:30

many entrepreneurs are that way. And and

58:32

when you say you work really well under

58:33

pressure, physiologically what you're

58:35

saying is, "I lack the ability to set

58:38

priorities internally. So I use external

58:40

pressure to set my priorities." And why

58:43

is that? It's because when you don't

58:46

have the right methylated nutrients in

58:48

the body, you lend equal weight to every

58:50

thought that comes into your mind.

58:52

Right? So you could be working on

58:55

you know, the deal, the joint venture,

58:57

business closing of a lifetime. Right?

58:59

And you got the contract and you have 45

59:01

minutes to get it back to your lawyer

59:03

you know, the deal's permanently going

59:04

to go away. This is a life-changing

59:06

deal. You've

59:07

waited for this your entire life. It's a

59:08

very meaningful event. You're working

59:10

your way through this legal document and

59:13

you look over at your phone and there's

59:14

an Instagram message. And you're like,

59:16

"Oh, that's my neighbor's cousin's kid

59:20

fishing in the lake.

59:21

You know, I wonder if he catches a

59:22

fish."

59:23

So you start looking at it and you're

59:25

like to he's still casting. Oh, he

59:26

didn't catch it that time. Now,

59:27

meanwhile,

59:29

this has gone from 45 minutes to 35

59:31

minutes.

59:32

And now it's down to 25 minutes. And all

59:34

of a sudden you go,

59:35

"Oh, no crap." You turn the phone off,

59:37

which didn't mean anything anyway, and

59:39

you focus all of your attention and

59:41

because you're very intelligent and you

59:43

and you execute and you can hyperfocus,

59:45

you hyperfocus on this and you worm your

59:47

way through it. And 1 minute before the

59:48

deadline, you hit the send button.

59:50

Lawyer gets it. The deal goes through

59:51

and you go,

59:53

"I work really well under pressure."

59:56

What what's that called? That process

59:58

you described there is many people

60:00

describe that as procrastination. Mhm.

60:02

It's procrastination, but it's

60:03

procrastination because you give equal

60:04

weight to all of these different

60:07

variants that come into your and that

60:09

come into your your field. So, you

60:10

actually give equal priority

60:13

to very disequal events. But also, you

60:17

know, sometimes if I'm looking at that

60:18

contract, the contract clause is a

60:20

little bit difficult. And my attempt to

60:23

maybe dis escape discomfort would be

60:25

I'll just go do the dishes. Mhm. Right?

60:27

And that And so, I had Nir Eyal on the

60:29

podcast. He wrote a book called

60:30

Indistractable and he says we're

60:31

creatures that we're not we're not

60:33

pleasure-seeking creatures. We're

60:34

creatures that are avoiding discomfort

60:36

essentially. I think that aging

60:39

is the aggressive pursuit of comfort.

60:42

Could not agree with you more.

60:44

Let's use Dana White as an example.

60:45

Okay. Because, you know, the reason why

60:48

I came across your work is because of a

60:50

clip that

60:52

you know, involved Dana White and I'm

60:53

sure you I know you get this a lot

60:54

because I've seen you um be asked about

60:57

this in interviews, but for context for

60:58

anybody listening, I don't know where I

61:00

was or Ah, now I know what happened. My

61:03

friend sent into a WhatsApp group a clip

61:05

of Dana White talking about you. Mhm.

61:08

And that's the clip that made me

61:11

go down the rabbit hole. Oh. I watched

61:13

that clip. I then did some research. I

61:16

then watched a series of videos of you

61:17

online talking about health and I

61:19

watched you asking audience members to

61:22

stand up and name the you know the

61:24

health issue they were suffering and you

61:26

on the spot told them what was missing

61:28

from that their life, their diet,

61:29

whatever it might have been. You kind of

61:31

diagnosed them in a way of Um and then I

61:34

reached out to you on Instagram and

61:35

that's why you're here. But if we go

61:36

back to the start of that, it was that

61:38

story that Dana White told that had me

61:40

so compelled to to reach out to you. For

61:42

anybody that doesn't know and there will

61:44

be some people that don't know, Dana

61:45

White is the president of the UFC which

61:47

is the the

61:48

the the big fighting tournament where

61:50

everybody kicks each other's heads in.

61:51

So

61:52

what's your take on the the Dana White

61:54

story? So, Dana White is a an example, I

61:59

mean he's just a celebrity example, but

62:01

he is a shining example of the vast

62:06

number of people, you know, men and

62:08

women in his age category

62:11

that have given up on the capacity to

62:14

thrive. They've accepted that they have

62:16

hypothyroidism, hypertension, they wake

62:19

up sore and aching in the mornings, that

62:21

they don't have a response to exercise,

62:23

they have a little bit of spare tire,

62:24

they're brain foggy, they're on three or

62:26

four medications. In his case he was on

62:28

seven

62:29

medications at the time, three of which

62:31

were for blood pressure.

62:32

Um he was on, you know, I think a

62:35

thyroid medication, was also on He's

62:36

been very public about this by the way.

62:38

And and again I have to say I'm not

62:39

licensed to practice medicine, so it was

62:41

my clinical team that that came up with

62:42

the diagnosis and I communicated it to

62:44

Dana. Um I do train physicians to read

62:47

blood work and genetic testing, but I

62:48

can't practice medicine.

62:50

But the the point is that

62:52

when I met Dana

62:54

all he wanted to do was for me to

62:57

predict his life expectancy. And I

62:59

hadn't done that in almost seven years.

63:01

I left that industry for a reason, I

63:03

don't do it anymore. The test that I do

63:04

does not predict life expectancy. The

63:06

genetic test and the blood work that I

63:08

do will not tell you how long you're

63:09

going to live. I have no interest in

63:10

predicting death anymore. I only have an

63:13

interest in extending life.

63:15

And

63:16

you know, when when Dana was only

63:18

interested in me predicting his death.

63:20

So, I said, "Okay, for Dana White, I'll

63:23

come out. I'll meet with you.

63:25

I'll do a blood test on you, a gene test

63:26

on you. I'll pull all your medical

63:28

records, and I'll give you your life

63:29

expectancy."

63:30

But, what I did was went out and got his

63:32

blood work and his gene test, and

63:34

um I was actually in bed

63:38

at 1:30 in the morning when the lab was

63:40

running his blood work. And I've had

63:42

seven life-threatening alert calls um in

63:44

the middle of the night because when you

63:45

drop blood work off at the lab, the lab

63:47

runs it through the night. If they find

63:49

a life-threatening alert, they call the

63:51

account holder, right? So, um I own the

63:55

company. I was on the account. So,

63:57

LabCorp calls us at 1:00, 1:30 in the

63:59

morning, says, "Hey, we have a

64:00

life-threatening alert on a patient." I

64:01

was like, "Whoa.

64:03

Um

64:04

what's this patient's name?" They said,

64:05

"Last name's White." I said,

64:08

"Dana White?" They said, "Yeah." And I

64:09

go, "Whoa. What's the uh

64:11

life-threatening alert?" They said,

64:12

"Triglycerides are almost 800."

64:15

Now, triglycerides are a measure of

64:16

blood fat. Okay? It shouldn't be above

64:18

149.

64:20

At 200 or 300, this is a

64:22

cataclysmic level in the blood,

64:24

especially in a fasted state. Okay, we

64:27

pulled his blood in a fasted state. They

64:29

weren't 400. They weren't 500. They

64:31

weren't 600. They weren't 700. They were

64:33

like 768.

64:36

So, they were I mean, this is an

64:38

enormous number. And so, I said,

64:41

"Okay, uh I need to get the blood work

64:43

over to the, you know, to the doctor."

64:45

And when they sent the um the blood work

64:47

into the portal,

64:48

I then saw that he was insulin

64:50

resistant. He was hyperinsulinemic. He

64:52

was prediabetic. He had skyrocketing

64:55

levels of cholesterol. He had He was

64:56

hypertriglyceridemic. He was hyper um

65:00

homocystinemic. This homocysteine that I

65:02

told you elevates and causes the blood

65:03

vessels to constrict. Um I mean, he had

65:07

all of these conditions. I literally at

65:10

that moment booked a flight

65:13

for 7:30 or 8:30 in the morning

65:16

to head out and see him.

65:18

And

65:20

because I said I need to go see him in

65:21

person. And I remember I think his

65:24

assistant called me and I was at the

65:26

airport and she said, "Hey, Dana wants

65:27

to know if his life expectancy is in." I

65:30

go, "Well,

65:31

I'm on my way to see him, you know. Um

65:34

And she goes, "Oh god, is it like that?"

65:35

I said, "Yeah, it's like that."

65:37

And uh so I flew out to see him and I

65:38

sat down with Dana.

65:40

And

65:41

when we talked about the blood work, I

65:43

didn't even explain the levels. I

65:45

explained the symptom.

65:47

Um I did not know that he was on a CPAP

65:49

machine, but I said, "I am surprised

65:52

that you can actually sleep through the

65:53

night because he was so hypoxic.

65:55

Um red blood cell count, hemoglobin

65:57

levels.

65:58

Uh I'm surprised you could even sleep

65:59

through the night without like just

66:01

waking up choking, gagging." He's like,

66:03

"Dude, I'm on a CPAP machine. I wake up

66:04

every night. I throw up in the middle of

66:06

the night. I throw up so much I'm losing

66:07

my voice."

66:09

Um And I said, "This level of

66:10

coagulation triglycerides in the

66:12

bloodstream will surprise you can't even

66:13

bend down and tie your shoes. That's not

66:15

painful to tie your shoes. Not that it's

66:16

not restrictive to tie your shoes. It's

66:18

not painful. Like it doesn't feel like

66:20

the skin's going to peel off your legs."

66:21

And he went, "What the

66:23

I mean, he

66:24

slammed his hand down. He was like, "How

66:26

did you You know, how did you know

66:27

that?" And I said, "Dana, your level of

66:29

brain fog and fatigue right now has got

66:31

to be at a crushing level of fatigue. I

66:34

don't know how The only thing getting

66:35

you through the day is your own stubborn

66:37

willpower. And I'm surprised you can

66:39

remember anything from one minute to the

66:40

next." And his whole staff was like,

66:41

"Dude, he's so forgetful. He passes out

66:43

in meetings. He's sleeping on the

66:44

planes. He's gagging, snoring."

66:47

Um these were not things I necessarily

66:49

knew about him. So, I began to describe

66:50

all the

66:51

outcomes of these kinds of conditions.

66:55

And I said, "Look,

66:56

um

66:58

if if you don't do what we're going to

67:00

ask you to do for the next 10 weeks,

67:03

you know, based on this blood work and

67:05

the medical records that we pulled for

67:07

the previous 10 years and the

67:08

demographic data we pulled for 10 years,

67:10

you have a life expectancy of 10.4

67:12

years.

67:13

Um, you know, for a 52-year-old man to

67:16

realize he's not going to make it out of

67:17

his 60s,

67:19

a big realization. And he flipped a

67:21

switch, a level of discipline that, you

67:23

know, I haven't seen in a patient a long

67:25

time. He goes, "Dude, I'll do whatever

67:27

you tell me to do."

67:28

So, we wrote a prescription ketogenic

67:29

diet. I I I'm a fan of the keto diet. I

67:32

don't think everybody needs to be on the

67:33

keto diet, but

67:35

um, my prescription ketogenic diet, we

67:37

wrote a keto diet right down to the

67:39

grocery list, keto reset diet. And I

67:42

said, "If it's literally if it's not on

67:43

here, you can't eat it, Dana. This is

67:45

your grocery list. You go to the store,

67:47

you buy this. You send your chef to the

67:48

store to buy this. You make this. If

67:50

it's not this recipe, if it's not on

67:51

here, you literally can't eat it. Your

67:53

only leeway is water

67:54

and the supplements."

67:56

And um,

67:57

and we started uh a process of of

68:00

balancing hormones, controlling his

68:01

glycemic index, of using amino acids to

68:04

bring down his level of homocysteine, to

68:06

actually try to fix the insulin

68:07

resistance, to reduce his triglycerides.

68:10

And in 10 weeks,

68:12

he had a such a material change in his

68:15

blood work. I forget how much weight he

68:17

lost. I think he had lost almost 28 or

68:20

30 lbs at that time. He's over 40 lbs

68:22

now.

68:23

Um, by the end of the fifth month, he

68:26

was completely off of every prescription

68:28

medication he was on. He's down 44 lbs.

68:30

He lost the you know, he's no longer

68:32

using the CPAP machine. He no longer is

68:34

pre-diabetic. He no longer has insulin

68:36

resistance. He no longer has

68:38

life-threatening levels of triglyceride.

68:39

In fact, they're normal. His kidney

68:41

function improved. His liver function

68:42

improved. His immune system

68:43

strengthened. He feels like a

68:45

35-year-old man again. His skin tone all

68:48

improved. Um, his blood pressure

68:50

returned to normal. He's not on any

68:51

blood pressure medication.

68:53

Uh, so his blood pressure returned to

68:54

normal and he was like,

68:57

"Dude, I had no idea I could feel this

68:59

good. I feel freaking amazing." And his

69:01

life expectancy?

69:03

Almost tripled.

69:05

Almost tripled?

69:05

Almost tripled. Just under 30 years.

69:09

When I heard the story about Dana White

69:11

and I saw he'd gone from respectfully

69:13

being a man that had a little bit of

69:15

weight to to having this these six-pack

69:18

abs on Instagram. Of course, the

69:19

six-pack isn't the the outcome. It's as

69:21

you've said, it's the stuff going on

69:22

inside him. That's really the

69:24

transformation.

69:26

It it left me with the question like,

69:27

okay, I I heard the keto bit. But what

69:30

can someone who's just heard that at

69:31

home, where do they start with getting

69:33

extending their life by triple and

69:35

getting the

69:36

So, you know, he also started something

69:38

called the superhuman protocol.

69:40

And superhuman protocol is using

69:42

magnetism, oxygen, and light, right? So,

69:45

the only things that we really get from

69:47

Mother Nature, the the the big benefit

69:49

we get from Mother Nature is we get

69:51

magnetism from the Earth, we get oxygen

69:53

from the air, we get light from the sun.

69:55

The truth is, most of us are not

69:56

contacting the surface of the Earth that

69:59

much anymore. So, he bought $150,000

70:02

worth of equipment, a PMF mat, an oxygen

70:06

what's called a hypermax oxygen to do

70:07

exercise with oxygen therapy, and a red

70:10

light therapy bed. And I had him use

70:11

that equipment every single day, 7 days

70:13

a week.

70:15

But if your listeners want to do it for

70:16

free,

70:18

you can take off your shoes and contact

70:21

the surface of the Earth. And I'm

70:22

talking about bare feet on soil, dirt,

70:24

grass, sand. Because earthing and

70:27

grounding is a very real thing. We

70:28

actually discharge into the Earth. We

70:30

actually human beings built up a charge.

70:32

Do you know that pH, the acid alkaline

70:34

scale, pH stands for potential hydrogen.

70:37

It's a charge. It's a complete fallacy

70:39

that you can get alkaline by drinking

70:41

alkaline water. That's the biggest

70:42

marketing myth ever sold to the public.

70:45

Um

70:45

but you can get alkaline by contacting

70:47

the surface of the Earth. So, if you

70:49

don't have 150 grand, which I don't

70:50

expect anybody listening to this podcast

70:52

to spend 150 grand, but he did. I said,

70:54

you need a magnet you need a PMF mat so

70:56

that you can be alkaline. You need to

70:59

spend 10 minutes a day breathing um 95%

71:03

O2 under mild exercise and you need to

71:05

lay in a red light therapy bed. So,

71:08

in the absence of the superhuman

71:09

protocol, you can become superhuman by

71:11

contacting the Earth and by learning to

71:13

do breath work.

71:15

Let's talk about breath work. I spend 8

71:17

minutes every day doing a very specific

71:19

series of breath work and I'll teach it

71:21

to you now. You're You said your wife is

71:22

certified in? Yeah, my partner she's a

71:24

she's a breath What practitioner? Um

71:27

I've done breath work with her. I've

71:27

done breath work with a few people but

71:30

no one's ever had the profound impact on

71:32

me through breath work that she has.

71:33

I've never shouted her out before so I

71:34

probably should. Her Instagram is

71:36

@meloai

71:39

for anybody that's interested in breath

71:40

work. People do not realize the power of

71:43

something that is so accessible, so

71:45

free, and so easy to do, right? They

71:48

They want things to be more complicated

71:50

but it's not. And when I said the

71:52

presence of oxygen is the absence of

71:54

disease, it's absolutely true. Remember

71:56

that every elevated emotional state that

71:58

a human being can experience actually

72:00

has in its molecular structure

72:03

oxygen is a component of that emotion.

72:06

So, if you look at the difference

72:07

between passion, elation, joy, arousal,

72:09

libido

72:10

and anger for example, it's usually only

72:13

one neurotransmitter and the presence of

72:15

oxygen. The reason why no human being

72:17

has ever woken up laughing is because

72:19

you don't have the oxidative state to

72:21

experience laughter right out of deep

72:22

sleep. But can you wake up angry?

72:25

Yes. Because anger doesn't require

72:27

oxygen. So, every morning contact the

72:29

surface of the Earth and then spend 8

72:31

minutes doing I do a Wim Hof style

72:33

breath work. I give credit where

72:34

credit's due. He's the father of breath

72:36

work as far as I'm concerned. So, I do

72:38

three rounds of 30 deep breaths.

72:41

Like obnoxiously deep breaths.

72:44

And I start by trying to take my belly

72:46

button and pull my belly button out

72:48

towards the wall. Imagine there's a

72:49

string pulling your belly button towards

72:51

the wall

72:53

and then you

72:54

you fill from the lobes of the lung to

72:56

the apex of the lung and then you exhale

72:58

and just relax.

73:06

God knows what they think we're doing

73:08

out there.

73:09

Right outside this podcast. They're

73:11

like, "A bunch of freaks. I knew it was

73:12

a cult." Um

73:14

I knew I knew he was a cult leader. But

73:17

um so you do three rounds of 30 breaths.

73:19

On the 30th breath you exhale and you

73:20

hold. Allow the carbohydrate receptor to

73:23

reset. When you don't feel you can hold

73:25

anymore, you take a deep breath in.

73:28

You hold again and then you let it out

73:30

slow.

73:32

And you start again. I would suggest

73:34

that you start with three rounds of five

73:35

breaths. Then work to 10, 15, 20, 25,

73:38

and 30. If you get light-headed, this is

73:41

a good sign that the oxygen tension is

73:43

changing in your brain. If your fingers

73:45

and toes get tingly, this is a good sign

73:47

that you're changing the oxygen tension.

73:49

If you feel some kind of heat

73:51

temperature change in your neck, these

73:52

are all great signs. You will get to the

73:55

point where you can actually hold your

73:56

breath for two or three minutes,

73:58

sometimes four minutes between rounds of

74:00

breath work. Um and then the last thing

74:02

is to expose yourself to natural

74:04

sunlight. First thing in the morning,

74:06

the first 45 minutes of the day God

74:08

gives us a very, very special type of

74:10

light. It's called first light. There's

74:11

no UVA, there's no UVB rays in this

74:13

light. Um so that it's not the damaging

74:15

rays from the sun. It still generates

74:17

vitamin D3. It has a positive effect on

74:20

cortisol, on vitamin D3. First light is

74:23

the best way to reset your circadian

74:25

rhythm. So by contacting the surface of

74:27

the earth, doing breath work, and

74:28

getting first light, you can get to the

74:30

same place that Dana White did with 150

74:32

grand in equipment. What about oxygen

74:35

masks? Cuz I I'll be honest, when I read

74:37

when I read um about the Dana story, I

74:39

went on Amazon soon after and I was

74:40

like, "I'm just going to buy an oxygen

74:42

canister." Good idea, bad idea? Um so

74:45

what you want to do is um you know, you

74:46

get an oxygen concentrator, which takes

74:48

21% oxygen from, which is what the

74:50

concentration at sea level. It turns it

74:52

into 95% O2 and it fills this bag. And

74:55

it can refill this bag over and over and

74:57

over again. Okay, I use one called a

74:59

HyperMax. You can see it on my

75:01

Instagram.

75:02

And, um, you turn you plug it in, you

75:04

turn it on, it fills this bag, and then

75:06

you go and you put an oxygen mask on,

75:07

and you exercise for 10 minutes. Only 10

75:10

minutes. Cycle for 3 minutes, s- uh,

75:13

sprint for 30 seconds, cycle for 3

75:14

minutes, sprint for 30 seconds, cycle 3

75:17

minutes, sprint 30 seconds, and you're

75:18

done. And what this does is it raises

75:20

something called the partial pressure of

75:21

the the storage of oxygen in your blood.

75:24

The only two-time two-time Nobel

75:26

laureate prize winner in in medicine,

75:27

Dr. Otto Warburg, won both of his Nobel

75:30

prizes for his work in exercise with

75:31

oxygen therapy. You want to be a

75:33

superhuman? Do mild exercise every day

75:36

while breathing 95% O2. And it's

75:38

important that you're exercising.

75:40

And then, after that, you move into a

75:42

red light therapy bed,

75:43

photobiomodulation.

75:44

Um, so, you know, if you don't have

75:46

access to a HyperMax oxygen machine,

75:48

just do the breath work. Get the breath

75:49

in, you know, exchange the oxygen

75:51

tension in the tissues, and expose

75:53

yourself to first light.

75:54

What about cold cold water plunging? So,

75:57

I'm a huge fan of cold water plunging,

75:58

but probably not for the reasons why you

76:00

think. You know, um, I also sit on the

76:03

board of the NFL, um,

76:05

uh, alumni association athletica as a

76:06

health services director. You know,

76:07

there there was a time when we used to

76:09

think that putting athletes in cold

76:10

water after exercise was good because of

76:12

its anti-inflammatory effects. We know

76:14

now that that's only about 15% of the

76:16

benefit. The majority of the benefit

76:18

comes from something called a cold shock

76:20

protein. If you really want to be

76:21

fascinated, Google cold shock proteins.

76:24

These are reserved proteins that are in

76:25

your liver. They're dumped into the

76:27

bloodstream in effort to save your life

76:29

when you put yourself in cold water.

76:30

They scour the body of free radical

76:32

oxidation. They increase the rate of

76:34

protein synthesis, muscle repair. They

76:36

are free. You get them when you put

76:38

yourself in cold water. Um, I don't know

76:40

what the Celsius conversion is, but I

76:42

use 50° for 3 minutes.

76:45

Minimum 6 minutes maximum. Cold? Yes.

76:47

It's It's actually not that cold. I

76:49

mean, you know, I see people getting in

76:51

37, 38° water. There's no evidence that

76:54

I've read that shows that colder is

76:55

better. You get a peripheral

76:56

vasoconstriction, so it forces all the

76:58

oxygen into the core and up to the

77:00

brain. Um, and you ask you get an

77:02

activation of something called brown

77:03

fat, right? Um, thermogenesis comes from

77:06

brown fat. And for the women that are

77:08

listening, for some reason I seem to

77:09

insnare the women when I say this.

77:11

Remember that the definition of a

77:12

calorie is a measure of heat. Right? I

77:15

mean, the definition of a calorie is the

77:17

amount of energy it takes to raise 1

77:18

cubic centimeter of water 1° centigrade.

77:21

So, if if if a calorie is a measure of

77:23

heat, then this means that when heat's

77:25

leaving your body, calories are leaving

77:27

your body. So, if there is nothing

77:30

nothing, no amount of exercise, hits

77:33

cardio, no type of cardiovascular or

77:35

weight training that comes anywhere

77:37

close to immersing yourself in cold

77:39

water in terms of what will strip fat

77:41

off your body fast. If you want to strip

77:44

fat off your body,

77:46

get in cold water 3 to 6 minutes a day.

77:49

That's fascinating. Because because the

77:50

oxygen rushes to my head, that's why it

77:52

has a really profound impact on mood.

77:54

That's why it has a very profound impact

77:55

on mood. Because if you think about it,

77:56

what's the reason why we need deep

77:58

sleep? What happens in deep sleep that's

78:00

so special? There's a secondary oxygen

78:02

transfer. We transfer oxygen from the

78:04

periphery, from the extremities, to the

78:05

brain. Remember, the brain's a

78:07

non-metabolic organ.

78:08

So, in other words, it's unlike a

78:10

muscle. If I pick up a weight and start

78:11

to work out my muscle, my arm, my body

78:14

will send more blood, more amino acids,

78:15

more oxygen to that muscle because it's

78:17

working. Well, if I'm sitting at my

78:19

computer and I'm watching a reruns of

78:21

The Simpsons,

78:22

or I'm sitting at my computer and I'm

78:23

solving the most complex joint venture

78:26

agreement, partnership agreement with

78:28

all kinds of mathematical equations, my

78:31

brain gets the same amount of nutrients.

78:33

Same amount of blood flow, same amount

78:35

of oxygen. So, it eats the same meal

78:36

whether or not it's in a dead sprint or

78:38

whether or not it's just chilling on the

78:39

couch. Except in deep sleep and when

78:44

you're in cold water because it's

78:45

forcing the oxygen up to the brain.

78:48

You said earlier about

78:50

about comfort. Yes. I I was speaking to

78:52

someone yesterday about this thing

78:54

called we he referred to it as the

78:55

comfort crisis and how

78:57

you know, as we've become more

78:59

civ- I'd say civilized but I don't know

79:01

if that's the right terminology. As

79:02

we've become more advanced

79:03

technologically as humans, we can make

79:04

our lives increasingly more comfortable.

79:06

Correct. Sounds like a good thing.

79:09

Terrible. It accelerates aging in every

79:11

form. I mean, aging is the aggressive

79:13

pursuit of comfort. We have got to stop

79:15

telling grandma not to go outside it's

79:16

too hot, not to go outside it's too

79:18

cold, just to lay down, just to relax,

79:21

to eat at the very first pang of hunger.

79:23

This is collapsing all of our own

79:25

natural defense mechanisms. You know, if

79:27

we don't load our bones, they don't

79:28

strengthen. If you don't tear a muscle,

79:30

it doesn't grow. If you don't challenge

79:32

the immune system, it weakens.

79:35

And so, stress is very often very good

79:38

for the body.

79:39

Thermal stress, um you know,

79:42

weight-bearing exercise, breathwork,

79:44

these things put stressors into the body

79:46

that are very they have a very positive

79:48

effect at strengthening you. We want to

79:50

regulate everything now. We regulate our

79:52

temperature. We go from a you know,

79:54

temperature-controlled office to a

79:56

temperature-controlled car to a

79:57

temperature-controlled home.

79:59

You know, we don't we don't

80:00

thermoregulate anymore. I mean, you

80:02

know, usually when you when I ask people

80:04

to start taking cold showers, they take

80:06

their first cold shower, they never do

80:07

it again. Why? Cuz they don't want to be

80:08

uncomfortable.

80:10

And so, when you learn to deal

80:13

and become comfortable with being

80:14

uncomfortable,

80:16

this is like a metaphor for life. It's

80:18

almost like yoga. If you've ever done

80:19

really intense yoga and you're holding a

80:21

yoga pose and you're you're trying to

80:23

remain calm and focus on your breath

80:25

while your body's in intense pain. Now,

80:27

you're not in any risk, but your ass

80:29

feels like it's going to peel off your

80:31

legs and your hamstrings are firing and

80:33

you're sweating and you're shaking and

80:34

you're doing this thing that's called

80:36

the candlestick, but it's really

80:37

painful. And if you can mean maintain

80:40

calm and and and breathe through a

80:42

situation like that, what happens 4

80:44

hours later when you get a nasty

80:45

Instagram message? Nothing.

80:47

Doesn't shift your mood. And if we don't

80:49

learn to control our emotional state, we

80:52

will never control our future.

80:55

You know, MIT did an incredible clinical

80:57

study that showed that the amygdala of

80:59

the brain, which is where we experience

81:01

emotion,

81:02

is the sole gateway to an area of the

81:04

brain called the hippocampus, which is

81:06

where it we hold our memories.

81:09

So, just imagine that the emotional

81:12

center of the brain is the sole gateway

81:14

to the memory of the brain.

81:17

This is why if you've ever had an

81:18

argument with your spouse, you can

81:19

always recall with incredible accuracy

81:21

every other time they've made you feel

81:23

this way. You know, you did this on

81:24

September 21st. You did this when we

81:26

were on the boat with my boys. You did

81:27

this at

81:28

um you know, our Christmas holiday party

81:30

four Christmases ago cuz because that

81:32

emotion is linked to that memory. So,

81:34

you can recall that memory very

81:36

accurately. Well, our memory, our

81:37

hippocampus, is what projects into the

81:39

prefrontal cortex. It determines our

81:41

future. It's our conscience.

81:44

So, this means if emotion is the only

81:46

gateway to memory and memory projects to

81:48

our conscience, which is our which is

81:49

our future,

81:51

this means that your current emotional

81:53

state

81:54

determines your future.

81:57

That's a biophysiologic fact.

81:59

So, like for example, if you had an

82:00

argument with your spouse on the way to

82:02

work and you get out of the car and you

82:03

slam the door and you walk into the

82:04

office, when you break the plane of the

82:05

door of that office, the only memories

82:07

you can recall about the office at that

82:09

moment are negative.

82:10

You're going to walk through the door of

82:11

the office, you'll be like, they don't

82:12

respect me around here. I'm going to

82:13

have a stern talking to management

82:15

today. You know, my office better not

82:17

you know, you know, nobody better be at

82:18

my desk and you know what, Mary better

82:20

not run into me today cuz she doesn't

82:21

respect me. You can just start going

82:23

through all the negative things about

82:24

the office. The The didn't do anything

82:25

to you. How do I prevent that?

82:27

your

82:28

You learn to control your emotion. How?

82:32

Well, first you start by putting the

82:33

right nutrients into the body that

82:35

allows you to achieve elevated emotional

82:37

states.

82:38

And you learn to do things like when you

82:41

feel like you are beginning to lose

82:43

control of your emotional state, you you

82:45

you actually break that cycle. I usually

82:47

do it with breathwork. Mhm. Um and so

82:51

um you know, first it begins by having

82:53

the right raw materials, but this is

82:55

just taking you back to the cold plunge.

82:56

If you can start your day in an elevated

82:58

emotional state, if anybody listening to

83:00

this has ever really done a cold plunge,

83:02

tell me if you were ever in a bad mood

83:03

getting out of a cold plunge. Just try

83:05

to be in a bad mood getting out of a

83:06

cold plunge. They say if you want to

83:07

cure depression, push somebody in cold

83:08

water. You know, um

83:10

and it's so true. You're in such an

83:12

elevated emotional state. You're like,

83:13

"Wow." Now you go cruising into the day

83:16

and get a little negative, you know,

83:17

Instagram message and your you know,

83:19

spouse calls you and tells you she

83:20

forgot what you wanted to get at the

83:21

grocery store and you get to work and

83:23

you got a little problem at the office.

83:24

These things roll off your back instead

83:25

of shifting your state, which now shifts

83:28

your memory, which now changes the

83:30

trajectory of your prefrontal cortex,

83:31

which affects your future.

83:33

I do a lot of traveling, Gary.

83:35

I travel all over the world all the

83:36

time. Oh, it's one of my favorites.

83:38

Yeah, you travel a lot? Tons. You've got

83:40

a bit of a system for traveling because

83:42

when I travel, I feel like [ __ ] Oh, I

83:44

feel amazing when I travel.

83:46

What do

83:46

And I post all about it on my Instagram.

83:48

You know, all I do is teach on

83:49

Instagram, but you know, I went Miami,

83:51

Atlanta, New York, London, Stadt,

83:54

Switzerland, Dubai, Dubai, Miami, Miami,

83:56

Vegas. I would I got I I I got up at

83:58

4:00 this morning to come here. I mean,

83:59

I I landed here at I think 7:15 this

84:03

morning. And you're flying out of here?

84:04

I'm flying out of here now. In 5

84:05

minutes? I'm flying out of here in 5

84:07

minutes and I'm going on a red-eye back

84:08

to Miami.

84:09

And you feel good when you travel and I

84:11

feel like [ __ ] What do you think we're

84:12

doing differently if you were to guess?

84:14

Okay, so there's three things that I

84:16

that you can do when you travel. First

84:19

and foremost, and I don't know why

84:20

anybody talks about this, is that you

84:22

know everybody talks about waking with

84:24

the sun which I'm a big believer in

84:26

or forcing yourself to stay up to try to

84:28

get onto a new time zone. When you

84:29

change time zones, the single most

84:31

important thing that you can do

84:34

is preserve your sleeping window. Do not

84:38

eat during your normal sleeping window.

84:40

Let me tell you what I mean. Let's say

84:42

that you're on the East Coast, you live

84:44

in New York

84:45

and you go to bed at 10:00 and you get

84:47

up at 6:00 a.m.

84:48

Okay, you go to bed at 10:00 p.m. Get up

84:50

at 6:00 a.m. Most people go to bed at

84:51

midnight get up at 6:00 a.m. So let's

84:53

say you go to bed 10:00 you get up 6:00

84:54

a.m. And now you go to London.

84:56

Okay, so now London is depending on the

84:59

time of the year 6 hours ahead.

85:01

If you eat

85:03

during 10:00 p.m. to 6:00 a.m. New York

85:06

time

85:07

there is zero chance you will adjust to

85:09

that time zone. We are more tied to our

85:10

digestion in terms of our circadian

85:12

rhythm than we are to the sleep-wake

85:14

cycle of the sun. So in other words, if

85:16

I fly to London and I start eating

85:20

when it's 3:30 in the morning my time

85:23

my body goes what the heck are you

85:24

doing? We're having steak and eggs and a

85:26

champagne? It's 3:30 in the morning.

85:28

Right? Your circadian rhythm is screwed

85:30

up. So shift your sleeping window and

85:33

preserve that sleeping window in your

85:36

new time zone and do not eat during

85:37

those times. So in other words, in

85:40

London that would be

85:42

6:00 a.m. to noon. So between 6:00 a.m.

85:45

and noon I'm not going to eat. I'll have

85:46

coffee or I'll have water

85:49

or fluids, but I will not start to eat

85:51

until noon. And how long do I preserve

85:53

that window?

85:54

Well,

85:55

depending on how long you're going to be

85:56

there. Usually I'm only if you're there

85:58

a week or less, preserve your sleeping

86:00

window the entire week that you're

86:02

there. When you're there for more than 7

86:04

to 10 days, then you need to really

86:05

adjust to that time zone.

86:07

What happens physiologically if I don't?

86:10

If you don't, you will you will

86:12

irrevocably mess up your circadian

86:14

rhythm. I I just sleep just just just

86:16

imagine it. So let's say you lived in

86:18

New York and you didn't travel. You go

86:19

to bed at 10:00 and you wake up at 6:00

86:20

a.m. Try for three nights setting an

86:22

alarm for 3:30 in the morning. Get up,

86:24

eat a big breakfast at 3:30, and try to

86:26

go back to bed.

86:27

And watch what happens to your sleep

86:29

cycle, right? You'll destroy it. So, we

86:31

don't we preserve our sleeping window.

86:33

The other thing is I fast on domestic

86:34

flights.

86:36

I don't care what time or where I'm

86:37

going. If I fly anywhere in the

86:38

continental United States, I fast on

86:40

airplanes. So, I I allow myself um you

86:44

know, water. I I hydrate and I have

86:45

black coffee. On international flights,

86:47

I just came back from Dubai. It was 16

86:48

hours. I ate on that plane.

86:50

Um and then for flights that are more

86:52

than an hour, every hour on the hour, I

86:54

get up out of my seat. I don't care how

86:56

weird it looks. I go to the back of the

86:57

plane. I do 25 air squats. I go either

86:59

into the bathroom or in the back of the

87:01

plane. I do 25 deep breaths.

87:03

On a 10-hour flight, I'll do 250 deep

87:05

breaths and 250 air squats over the

87:07

course of a 10-hour um flight. Feel

87:09

amazing.

87:11

And then I eat fats and proteins on

87:14

flights.

87:15

Carbohydrates at altitude are terrible

87:17

for you, and that's usually where all

87:18

the salt hides. Remember that you know,

87:20

there's there are essential fatty acids,

87:23

meaning they're essential for life.

87:24

There's two of them. Um if you don't get

87:26

these fatty acids, you'll die. There's

87:28

nine essential amino acids. They're

87:29

proteins. They're essential for life. If

87:31

you don't get these nine

87:33

essential amino acids, you'll die. There

87:34

is no such thing as an essential

87:36

carbohydrate.

87:38

Why Why are carbs so bad at in the air?

87:40

Um carbs are bad in the air because as

87:41

soon as you divert blood from your brain

87:45

to digestion. Let's not forget, it's a

87:47

30-ft long tube. The higher the the the

87:50

consumption of carbohydrate, the more

87:51

blood floods to your gut.

87:53

going to feel like crap.

87:54

So, now you're seated and your gut is

87:57

flooded with blood. It's all come from

87:59

your brain. So, now you're tired, you're

88:01

not focused, you're exhausted, and

88:02

you're seated. So, you're it this isn't

88:05

a good place to be tired cuz you're not

88:06

going to get good sleep.

88:08

Right? So, you So, I put I set a huge

88:10

priority on energy. Energy is a huge

88:12

priority to me. So, when I look at food,

88:14

I look at it two ways. It's going to

88:15

serve me or it's going to steal from me.

88:17

When I'm I'm flying first class back to

88:19

Miami tonight. It's a red eye. I'm going

88:21

to actually use that time to sleep. I'm

88:22

not going to waste it on eating because

88:24

I know that I'm first of all, I'm past

88:25

my feeding window now cuz I preserve my

88:28

East Coast time feeding window. And this

88:30

is what keeps your the train running on

88:31

time. What is energy? You said energy

88:33

there. What is it?

88:34

Energy Energy is oxygen in your blood.

88:36

Everything that you perceive about

88:37

energy is nothing more than oxygen in

88:39

your blood. If you told me, "Gary, I had

88:41

a lot of energy today." Physiologically,

88:43

what you're saying is I had a lot of

88:45

oxygen in my blood today.

88:47

Oxygen equals energy.

88:50

At the start of this conversation, I

88:51

asked you a question. I said, "Why

88:52

should people listen to your message?" I

88:54

think we're now at the end of the

88:55

conversation. What have I missed that is

88:58

pertinent, important to your message

88:59

that we haven't discussed? You know, I

89:01

feel like I could talk about this

89:02

forever. Yeah, yeah, yeah. I can

89:04

imagine. Like to me, I feel like the

89:05

podcast is just getting going. Um

89:07

you know, I I I

89:09

I believe in human beings and and and

89:12

the ability of the body to heal itself.

89:13

I believe in the power of the mind and

89:15

frequency and the power of this to heal

89:17

this. Um I guess my

89:19

my message would be and I don't think

89:21

that you've missed anything. My message

89:22

would be that optimal health is found in

89:24

the basics.

89:25

Not in the complicated

89:28

fancy nootropics or some rare root

89:30

that's buried deep in the Amazon jungle.

89:31

It's found in the basics. The further we

89:33

get away from the basics, magnetism,

89:35

oxygen, light, whole foods,

89:37

the more unhealthy we become.

89:40

And you know, my message what I try to

89:42

teach on Instagram and what I the

89:43

message that I get to the world is, you

89:45

know, not that you need a lot of fancy

89:46

equipment. Sure, if you can afford it,

89:48

you can have it. But if you get back to

89:49

the basics with Mother Nature and back

89:51

to the basics with, you know, our foods,

89:53

a good

89:54

rule of thumb is if your

89:55

great-grandmother wouldn't recognize it,

89:56

don't eat it.

89:57

Um

89:59

then you'll find a state of optimal

90:01

health that's beyond

90:02

anything that you thought imaginable.

90:04

There's a superhuman inside of everybody

90:06

listening to this podcast.

90:09

Gary, we have a closing tradition on

90:10

this podcast where the last guest asks a

90:11

question for the next guest not knowing

90:12

who they're asking it for. The question

90:14

that's been left for you is, what is the

90:17

unobvious thing

90:19

that you struggle with?

90:22

Mhm.

90:24

What is the unobvious thing that I

90:26

struggle with?

90:27

You know, I

90:29

I believe that

90:31

I'm so committed to my craft

90:34

and I'm so committed to being authentic

90:37

that I struggle sometimes like everyone

90:40

else when I

90:42

I have an

90:44

extra burden of guilt whenever I want to

90:48

just have any kind of marginal

90:50

enjoyment, all right? Like if I just

90:52

want to have a cocktail or I want to eat

90:54

some birthday cake, um which I know is

90:57

not going to hurt me and it's not, um

91:00

you know, it's not going to throw me

91:01

off, but I

91:02

I really struggle with that. I feel like

91:04

I'm letting the whole world down when I

91:06

do that. Um and I know that it's not and

91:08

it's ridiculous and you shouldn't be

91:09

that, you know, being that disciplined

91:11

actually

91:12

is a you know, it's not sustainable over

91:15

over a long period of time.

91:17

Um

91:17

I think that like a lot of people

91:19

listening to this podcast, I

91:21

I'm I'm very hard on on myself. I'm my

91:24

own worst enemy sometimes.

91:26

And I have an insatiable appetite to do

91:30

what I'm doing and I think that if I

91:32

just

91:33

wasn't so hard on myself, I

91:36

probably would find it a lot easier.

91:39

Mhm.

91:41

Thank you so much. You're welcome.

91:42

Everybody listening to this should go

91:43

check out 10X Health. Um much of the

91:45

information you've discussed here lives

91:46

within that ecosystem and your Instagram

91:49

and your website and the those channels

91:50

which I've explored, you know, in in

91:51

depth are incredible resources to

91:53

understand how to start your journey to

91:55

living a more healthy life and that's

91:57

that's everything that you you espouse.

91:59

You're incredible, Gary. Thank you so

92:00

much. And you're you know, it's very

92:02

very rare that I find someone online. I

92:04

then DM them and I nagged your DM for a

92:07

couple of months to get you here. But

92:09

but I think everybody listening to this

92:11

can understand why.

92:12

You're doing incredible work and

92:13

incredibly important work in an

92:14

incredibly important time to shift the

92:16

narrative. And as I said to you I think

92:19

before we start recording or in an

92:20

interval, I I realized that this is

92:22

literally just the start for you in the

92:24

journey and the mission that you're on.

92:26

That's so clear to me. And thank you. I

92:28

feel the same way.

92:33

I've now been a Huel drinker for about 4

92:36

years roughly. So much so that I ended

92:38

up investing in the company and I play a

92:40

role on the board of the company. But

92:42

they also very kindly sponsor this

92:44

podcast. And to be honest, I've never

92:45

said this before, but Huel believed in

92:46

this podcast before anybody else. The

92:48

CEO Julian um

92:50

told me before we even launched the

92:51

podcast how successful it would be and

92:53

that Huel would back it. And I

92:54

absolutely have a huge amount of

92:55

gratitude for them for that support. But

92:58

an even greater sense of gratitude for

92:59

the fact that they've helped me stay

93:01

nutritionally complete throughout the

93:02

chaos and hecticness of my tremendously

93:05

busy business schedule. So if you

93:07

haven't tried out Huel, which I hope

93:08

most of you have at least given it a go

93:09

by now, try it out. It's an unbelievable

93:11

way to try and stay nutritionally on

93:13

course if you have a hectic busy

93:15

schedule. And let me know what you

93:17

think. Send me a tweet and a DM. Tag me.

93:19

Let me know what you think.

Interactive Summary

The video features an interview with a human biologist and mortality expert who discusses his transition from analyzing life expectancy data in the insurance industry to helping people thrive through biohacking. He argues that many chronic conditions, often accepted as consequences of aging or genetics, are actually the result of nutritional deficiencies caused by an impaired ability to process raw materials, a process known as methylation. He explains the importance of oxygen and basic natural elements, shares his work with high-profile clients like Dana White, and provides practical advice on breathwork, sunlight exposure, and cold water therapy to optimize health and extend longevity.

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