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Vitamin D Expert: The Fastest Way To Dementia & The Big Lie About Sunlight!

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Vitamin D Expert: The Fastest Way To Dementia & The Big Lie About Sunlight!

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3693 segments

0:00

He was literally two days away from

0:02

dying. So, this is a story where a

0:04

15-year-old boy was diagnosed with blood

0:06

cancer, but he developed a flesh-eating

0:08

infection in his lung. He wasn't going

0:10

to make it. So, he has one request. He

0:13

wants to go outside, and that's exactly

0:14

what they do. And this was actually

0:16

mind-blowing to me. After the second

0:18

day, the infection is probably 60-70%

0:20

gone, and it became clear to me that

0:22

sunlight has so many important benefits.

0:25

For instance, if you're the bed closer

0:27

to the window, you get discharged from

0:28

the hospital faster. So, I want you to

0:30

give me any any information you have as

0:32

it relates to light health. For example,

0:34

do you recommend these kinds of things?

0:38

Dr. Roger Seholt is a board-certified

0:40

critical care physician who breaks down

0:42

complex science into clear, life-saving

0:44

advice. I see people at the very end of

0:46

their lives, so I know what prevents

0:48

them from getting this ill and how to

0:50

extend life. So, let's distill it down

0:52

into eight pillars. The first thing that

0:54

exercise, it reduces stroke, it reduces

0:57

depression. Next, sunlight. Did you know

0:59

that infrared lights from the sun is

1:01

able to penetrate up to about 8 mm and

1:04

stimulate and upregulate melatonin,

1:06

which prevent a lot of diseases like

1:07

dementia, cardiovascular disease,

1:09

diabetes.

1:10

>> What if you live in a cloudy country?

1:11

>> There's some very actionable things that

1:13

you can do, and we'll talk about that.

1:15

Next one, water. For instance, people

1:16

who use sauna are more likely to have

1:19

less death from cardiovascular disease.

1:21

Next, air. There are studies that show

1:23

that just going out one day a week can

1:25

elevate our immune system and make us

1:27

more relaxed. And then there's

1:30

But finally, trust. This is something

1:31

that can't be ignored because studies

1:34

have shown that people who have a good

1:36

faith and trust in a God are good.

1:41

I see messages all the time in the

1:43

comment section that some of you didn't

1:44

realize you didn't subscribe. So, if you

1:46

could do me a favor and double-check if

1:48

you're a subscriber to this channel,

1:49

that would be tremendously appreciated.

1:50

It's the simple, it's the free thing

1:52

that anybody that watches this show

1:54

frequently can do to help us here to

1:55

keep everything going in this show in

1:57

the trajectory it's on. So, please do

1:59

double check if you subscribed and uh

2:01

thank you so much because in a strange

2:02

way you are you're part of our history

2:05

and you're on this journey with us and I

2:06

appreciate you for that. So, yeah, thank

2:08

you.

2:12

Dr. Roger Schwelt.

2:14

With the work that you do, what is it

2:16

that you're aiming to accomplish?

2:19

Outside of my clinical duties, uh and

2:21

maybe even part of that, I would like to

2:24

clearly

2:25

explain

2:28

very easily graspable tools that can be

2:31

implemented to make people live their

2:34

best life and it's specifically in terms

2:36

of their health and their well-being.

2:38

And as we dig down into health and

2:39

well-being cuz that's quite a broad

2:41

Yeah.

2:41

>> basket, what is it within health and

2:43

well-being that you've spent your

2:44

career, your life focusing on? So, I'm a

2:47

board-certified internal medicine

2:49

specialist. Then I did an extra 3 years

2:51

of training uh here in the United States

2:53

on pulmonary and critical care. So, I

2:55

deal with all of the issues that are

2:56

related to the lungs and the critical

2:58

care aspect. So, if you are uh

3:00

if you were admitted to a hospital and

3:02

you're critically ill, you'd go to the

3:04

intensive care unit. I'm the doctor that

3:05

you see. So, I'm putting in, you know,

3:08

the lines, putting them on vasopressors,

3:10

intubating them. Uh I worked in the

3:12

clinic this morning.

3:13

And what are some of the unappreciated

3:15

things that most people don't think of

3:16

cuz we think of, you know, don't eat

3:18

processed food and exercise. But, is

3:20

there things outside of that that you

3:22

don't think the average person

3:24

appreciates enough? Yes, absolutely.

3:26

What are those things? So, if we look at

3:28

those things that extend life

3:32

and are beneficial, we could put them

3:35

into what I call eight pillars of

3:37

health. If you can imagine your life is

3:41

uh a chain with a bunch of links, okay?

3:43

And I'm talking medically.

3:45

Each one of those links is an organ

3:47

system.

3:48

So, your heart is a link. Your lungs are

3:51

a link. Your liver is a link. Your

3:52

kidneys, etc., etc.

3:54

As you go through life, imagine those

3:56

links starting to erode.

3:59

So that at some point in your life,

4:01

you're going to get some sort of a

4:02

disease or a diagnosis that focuses on

4:05

one organ system of your life. For many

4:07

here in the United States and in the UK,

4:09

it's the heart.

4:10

And as that link gets more eroded,

4:14

we can see very clearly that this is

4:15

going to be the link that's going to

4:16

break first. And therefore attention is

4:18

paid to that link. For many, that

4:21

diagnosis comes with medications. So

4:24

here here is where the first truism that

4:26

I would say going forward, all

4:28

medications have side effects.

4:31

And what the aim is to do in modern

4:33

medicine is to utilize the knowledge of

4:36

those medicines and their effect on the

4:39

human body so that we leverage the other

4:42

links to protect that weakest link. So I

4:45

could go through a whole bunch of

4:46

medications that I give all the time to

4:48

patients in the intensive care unit,

4:50

where I'm focused on saving their life

4:52

because I can clearly see which link is

4:54

the weakest,

4:55

but I'm doing it knowing that there's

4:57

side effects because I'm trying to save

4:59

that link to save that life. And I'm

5:02

leveraging those other things. For

5:03

instance,

5:04

somebody comes in with a stroke.

5:06

And

5:08

they have they've they've lost weakness

5:11

on the on the right side of their body

5:13

or on the left side, whichever side.

5:14

I can give them a medication

5:17

immediately that will break up all of

5:20

the clots in their body. It's called TPA

5:22

or TNK. And it will restore blood flow

5:25

to the brain and it will reverse many of

5:27

their symptoms. If they come in soon

5:28

enough, we can do this.

5:31

What's What's amazing is that that

5:33

medication has an effect that can do

5:35

that, but it also has a pretty

5:37

significant side effect in that it could

5:38

break up clots somewhere else and cause

5:40

bleeding.

5:41

So we have to be careful about what it

5:43

is that we're doing.

5:45

Clearly there, what we're doing is we're

5:47

saving one link at the expense of other

5:49

links, but that's what you have to do in

5:50

an emergency situation. But if after

5:53

that, I simply send that patient home

5:56

without telling them why they had that

5:58

stroke and what they need to do to

6:00

prevent themselves from getting that

6:02

stroke again, and what are the lifestyle

6:04

factors that caused that to happen,

6:07

I haven't done my job. So, what is what

6:09

are the interventions that we can do

6:11

hopefully early on in life

6:13

so that we don't have those links

6:15

eroding, so that all of the links are

6:16

strong, and as we get older, we can

6:19

continue to strengthen all of the links.

6:21

Here are the eight. So, nutrition.

6:22

That's That's nutrition. Basically, you

6:25

know, we know there are studies that

6:27

have done that showed that that

6:28

depending on what we put into our bodies

6:30

as food can have a dramatic impact in

6:33

terms of our well-being. Okay? So,

6:34

that's nutrition. Exercise.

6:37

As I was saying, exercise not only I

6:39

mean, drugs and things have side

6:41

effects. Exercise have side benefits.

6:43

So, exercise obviously is going to make

6:45

you more fit. It's going to make you

6:46

have better endurance.

6:48

But did you know that it reduces stroke?

6:50

Did you know that it improves

6:53

uh well-being? It reduces depression.

6:55

There's so many benefits. So, all of

6:57

these links are actually improving.

6:59

There's no leveraging here.

7:01

Exercise is incredible.

7:03

Water.

7:04

So, this may sound obvious, right? Like

7:06

you drink because you're thirsty. Where

7:08

I would like to go today is to talk a

7:10

little bit about what the effect of

7:11

water externally on your body can do.

7:13

And you're talking there about like hot

7:15

and cold

7:15

>> Exactly. usage. Exactly.

7:18

>> or cold plunges, saunas, that kind of

7:19

thing?

7:20

>> Exactly. Okay. And we'll get into the

7:21

actual evidence for this. We'll get into

7:23

it later, but what is the potential

7:25

>> system.

7:26

Which changes your probability of

7:27

diseases and stuff like that.

7:29

>> Correct. Absolutely. Especially in the

7:30

acute setting. Especially. So, not only

7:32

in the acute setting, but we also have

7:34

pretty good data from Finland, where

7:36

they have more saunas than almost than

7:39

people,

7:40

uh where they've actually done the

7:41

research and and shown with dose

7:43

response curves that this is actually

7:44

very beneficial. We won't get into too

7:46

much of that, but what I want to focus a

7:47

little bit more on because of my job in

7:50

the intensive care unit

7:52

and what we're seeing right now with

7:53

viruses and mutations and the innate

7:55

immune system, why something like this

7:57

may actually be very important, uh, as

8:00

we look forward. Interestingly, as we

8:02

look forward to future famous pandemics,

8:06

we can look backward and see what we had

8:08

done in the past. And we've got some

8:09

really actually really good information

8:10

on that. Let's go to

8:13

sunlight. This is something that, um,

8:16

I've really become more and more

8:17

involved with because of some of the

8:20

benefits that that this can do.

8:22

A very big misconception that people

8:24

have is that sunlight equals vitamin D.

8:27

And therefore, if you take a vitamin D

8:29

supplement, you don't need to go in the

8:31

sun.

8:32

This is really something that's uh, now,

8:35

um,

8:35

being debunked. Sunlight has far more

8:38

benefits than just vitamin D. Not saying

8:41

that vitamin D is is not something that

8:43

you want to supplement with. I

8:45

supplement with vitamin D. I think it's

8:47

there's a benefit to supplementing with

8:48

vitamin D, no question. But sunlight has

8:51

so many important things and I and I

8:53

really would like to spend the bulk or

8:55

the lion's share of the time talking

8:56

about this because this is really

8:57

important. Okay.

9:00

Um,

9:01

this is a this is this is amazing. Uh,

9:04

and it really made me think about

9:06

sunlight.

9:07

So, this is a story

9:09

by a lady by the name of Amy Hanmeyer.

9:11

Her 15-year-old boy

9:13

was diagnosed with lymphoblastic

9:15

leukemia, went into uh, the hospital,

9:18

actually started chemotherapy for it.

9:20

Which is blood cancer.

9:21

>> It's a blood cancer, yeah. And so, this

9:24

the treatment for blood cancer is

9:26

chemotherapy, which he started. The side

9:28

effect of chemotherapy is suppressing

9:31

the immune system.

9:32

And unfortunately, he didn't realize it,

9:35

but he developed uh, he didn't realize

9:37

it immediately, but he developed a a

9:40

fungus flesh-eating infection in his

9:42

lung.

9:43

And went into the hospital in June of

9:45

2024.

9:47

Uh this is in Minnesota, tertiary care

9:49

hospital.

9:51

And he

9:53

got worse and got worse and got worse to

9:56

the point where the only way that they

9:58

could control this infection was to

9:59

actually remove his left lung, which

10:02

they did.

10:03

So, this is a 15-year-old boy.

10:05

He is without his left lung. He only has

10:08

a right lung.

10:09

And he starts to decline even after

10:11

that. They do a CT scan and it shows

10:14

that now the infection has moved to the

10:15

his remaining right lung.

10:18

They have a family conference.

10:20

And as Amy is telling me this story, I

10:22

could hear her choking up. She's telling

10:24

me this on the phone.

10:25

She's saying that he's 15, he's

10:27

completely awake, he's completely alert.

10:29

He knows everything that's going on

10:31

around him. He's on a ventilator like a

10:33

a machine that they use for for sleep

10:35

apnea. It's like a BiPAP machine that's

10:37

breathing for him. It's not intubation,

10:38

but it's on his on his mouth.

10:41

And the doctors, you know, have done

10:43

everything they possibly can and they

10:45

say, "Look, he's getting worse. We can't

10:47

take Obviously, we can't take the right

10:49

lung out. We can't put him on a

10:51

heart-lung machine because there's no

10:53

sort of destination to where he's

10:54

going."

10:56

Uh

10:56

we recommend not intubating him and

10:58

making him what they call DNR, do not

11:00

resuscitate.

11:03

So, they're like, "Wow." They were not

11:05

expecting this coming. So, they have a

11:06

big conference. Somebody they they call

11:08

in help to like, "How do you explain to

11:10

a 15-year-old boy that you're dying?

11:12

And how how's that going to feel?"

11:15

So, they asked the doctors, "How how

11:17

much time does he have?" They say, "2

11:18

days."

11:20

So,

11:21

in this in this situation,

11:24

they ask this boy,

11:26

"Okay, you're going to die.

11:28

What do you want to do with your life in

11:30

the in the next 2 days? What do you want

11:31

to do?

11:32

And surprisingly, he says, "I want to go

11:36

outside."

11:38

"I just want to go outside."

11:40

This this guy grew up probably on a farm

11:42

or something and he spent his time

11:43

outside. So, he wants to go outside.

11:46

So, you know how

11:47

I mean, I don't know if you know this,

11:48

but like if you're a nurse or you're a

11:50

doctor

11:52

and you've done everything you can and

11:54

this and you're just completely

11:56

horrified at the fact that this

11:57

15-year-old is going to die and he has

11:59

one request, you're going to move heaven

12:01

and earth

12:03

to fulfill that one request. And that's

12:05

exactly what they do. They

12:07

get this boy the hospital bed outside.

12:10

He's on a BiPAP machine the respiratory

12:12

therapists have put together. So, this

12:14

guy is outside.

12:16

And they're not putting him outside to

12:18

get better.

12:19

He's just this is his dying wish. They

12:21

also use this thing called a Firefly.

12:23

It's like a a light device that they

12:25

were using. I to I'll be honest, I don't

12:27

know which did it. The Firefly was the

12:30

It's a It's a light device that gives

12:31

off light at different wavelengths and

12:33

they would use that for about three

12:35

times a day for 5 minutes.

12:39

This guy

12:40

does not die.

12:42

After the first day

12:44

his white count starts to come down.

12:46

That's like a a measure of the infection

12:48

that's going on in the lung.

12:49

And and and by the way, they do a CT

12:51

scan of his lung before this all starts

12:52

and the and it's just the the the

12:55

remaining lung on the right is just

12:57

filled with infection. It's horrible.

13:00

By the By the second day, the white

13:02

count comes down even more. And and by

13:04

the way, they haven't changed any of the

13:05

other treatment. He's been in By this

13:06

point, he's been in for 6 weeks.

13:08

He has not seen the light of day.

13:10

And and he's still getting the powerful

13:13

antifungal medication amphotericin B and

13:16

posaconazole. All of these things are

13:18

are really high-powered drugs that that

13:20

just completely fight fungus, but it's

13:22

not working. He's getting worse.

13:24

But now, he's out for the second day.

13:26

The white count's coming down, which is

13:28

good. That's a good sign.

13:30

His oxygen requirement is coming down.

13:32

That's a good sign. He's requiring less

13:33

and less oxygen. By the time he gets to

13:35

day five,

13:37

okay, we're already past two days.

13:39

He's off the BiPAP. He's on regular

13:42

just nasal cannula oxygen that you see

13:44

people wearing on their nose.

13:47

The doctors are scratching their head.

13:49

They're like, we we should get a CT scan

13:50

to see what's going on.

13:52

So So, Amy tells me that they they get a

13:54

CT scan of this guy, and they're in the

13:56

room. And some some even like swear

13:58

under their breath. Like they they're

13:59

completely

14:01

amazed.

14:03

Because on the CT scan, obviously the

14:05

left lung is still gone.

14:06

But the right lung,

14:08

the disease is probably 60 70% gone. And

14:14

he he's still alive.

14:16

He goes home. There's no sign of the

14:18

disease after after treatment. And he he

14:22

uh I I just she just uh communicated

14:25

with me to tell me that he just got his

14:26

Make-A-Wish

14:28

uh thing for for his cancer. He's

14:30

continuing treatment.

14:32

And to And she just can't believe that

14:35

he was literally two day two days away

14:37

from dying.

14:39

They changed nothing. They changed none

14:41

of his treatments. The only thing that

14:42

they did was they took him outside and

14:45

they they were using this Firefly before

14:48

inside, but they were using it more

14:49

consistently when he got outside.

14:52

Maybe hospitals should be outside.

14:54

>> This is exactly what

14:56

Okay, so if you wanted to know what my

14:57

drive was, what what it is my purpose

14:59

that I'm doing right now, I'm working at

15:01

three different hospitals. And

15:04

I'm trying to work in each of those

15:05

three different hospitals to try to get

15:07

patients outside. The biggest barrier

15:09

that we have is staff taking those

15:11

patients outside. That's the thing

15:12

that's the hardest. But this is what we

15:14

used to do.

15:16

Steven, this is what we used

15:17

When we built hospitals at the turn of

15:19

the century, we had hospital rooms where

15:21

beds could be taken out onto the veranda

15:24

and people could get sunlight. I would

15:26

love to see a time where we could go

15:27

back to that type of effect. There are

15:29

studies that have been done. People in a

15:31

two-bed room,

15:32

if you're the bed closer to the window,

15:35

you get discharged from the hospital

15:36

faster, on average.

15:39

Really? Yes.

15:42

I need to swap sides of the bed with my

15:44

girlfriend.

15:45

She's on the sunny side.

15:46

>> There There's so much evidence for this.

15:48

People who are in hospitals that are

15:50

have bigger windows, they give better

15:52

surveys.

15:53

And hospitals

15:54

reimbursement is tied

15:56

to the surveys that they get from

15:58

patients. So, it's literally a

16:00

win-win-win. If hospitals started to, I

16:03

believe, get patients outside. And And

16:05

they're already doing this. There

16:06

I don't want to say that this isn't

16:08

happening. There are hospitals that are

16:09

have programs to get patients outside.

16:12

Um I think we just ought to be doing it

16:14

a lot more.

16:16

Um

16:18

temperance. Temperance? What does that

16:20

mean?

16:20

>> It's an old term, isn't it? It really

16:23

means

16:24

moderation. And And I I would say in

16:26

this sense, temperance really means to

16:30

avoid toxins in the body.

16:32

Um As somebody who is a pulmonologist,

16:34

who is What's a pulmonologist?

16:36

>> is someone who takes care of the lungs.

16:39

And so, as a result of that, I see a lot

16:41

of issues with lung cancer

16:45

related to smoking.

16:47

Um I see it in the intensive care unit,

16:48

people with liver failure as result of

16:51

alcohol abuse. I also see people

16:54

on amphetamines. Here in Southern

16:55

California, where I reside and where I

16:58

work, we we have quite a bit of that.

17:00

And uh temperance, if if you want to

17:02

live a long and wholesome life, there

17:05

are some toxins that you want to avoid.

17:07

And uh And And And understanding that is

17:10

really important. So, this is something

17:11

that, if you stop some of those things

17:14

that we're talking about, all of those

17:16

links are going to be improved.

17:19

Air.

17:20

Seems kind of obvious. Early on I used

17:23

to think that that what this meant was

17:25

getting pure air with absolutely nothing

17:28

in it except for just nitrogen and

17:30

oxygen.

17:32

That's not true anymore.

17:33

We now understand that for you to have

17:36

the best type of air,

17:38

it actually has to come with some things

17:40

in it. Just like our our gut has a

17:43

microflora that you may have heard

17:44

about, so too does the air that we

17:47

breathe also must have that. And the

17:49

best type of air that you can have is

17:52

actually outside.

17:54

Rest.

17:56

This is really interesting because we

17:57

just mentioned that exercise was a

17:59

pillar, but rest also is. How can rest

18:02

and exercise at the same time be pillars

18:05

of health? And it really comes down to

18:08

knowing when to do what. Sleep, which is

18:11

also part of rest. So we're not just

18:13

talking about a daily rest when you go

18:16

to sleep. And and as a sleep physician I

18:17

can tell you quite a bit. We have lots

18:20

of information about how long we should

18:21

sleep, the quality of sleep, some of the

18:23

diseases that prevent us from sleeping.

18:26

Sleep is so important. I can't I I

18:29

couldn't over express it enough. Not

18:31

just a a daily rest, but I would also

18:34

say and and venture out we can talk more

18:36

about this, a weekly rest. A weekly

18:39

rest?

18:40

>> A weekly rest.

18:41

What do you mean weekend? Yeah. Yeah,

18:44

absolutely. How many times do we even on

18:47

the weekend do we put down our our

18:48

phone? Never. Or we stop reading emails.

18:53

And we take the time out to do things

18:54

that we would never be able to do.

18:56

Finally, trust.

18:58

So this is something that really just

19:00

can't be ignored. And and I'll say this

19:03

up front that um

19:07

in the world of research and science

19:09

there is a there is a silo of science

19:12

and there is a silo of faith. But,

19:15

what's what we can't ignore is the

19:18

growing body of evidence

19:20

from the scientific world that's peeking

19:22

over and looking at faith, that people

19:25

who have faith and people who have faith

19:27

in God,

19:28

uh whether that is uh their God in in

19:31

that particular denomination, are better

19:33

apt and able to deal with stress and

19:36

depression and anxiety.

19:38

So, this is something scientifically

19:40

that has been shown. Now,

19:42

if you you may have noticed that I I did

19:44

these in a particular order.

19:47

Um and if you go through them, you've

19:49

got nutrition,

19:51

you've got exercise, you've got water,

19:54

you've got sunlight, you've got

19:56

temperance, you have air, you have rest,

19:59

and finally you have trust. You put that

20:01

together and it spells out NEWSTART.

20:04

So, interestingly, these these

20:06

particular topics are not copyrighted,

20:09

but there is a um there is a university

20:11

in Northern California called Weimar

20:12

University that has actually put these

20:14

together in that very pattern as called

20:17

it NEWSTART. They actually have a

20:17

NEWSTART program.

20:19

Um and so, this is something that uh

20:21

that is actually being used uh

20:24

internationally.

20:25

So, of these subjects that you just went

20:28

through there for this NEWSTART um

20:30

framework,

20:32

where do you want to start?

20:34

I think actually sunlight is is one of

20:37

those things where I'm excited about all

20:39

of those, but I think sunlight is

20:42

is really where we have It's the Let's

20:44

put it this way, it's the lowest-hanging

20:45

fruit. Okay. Explain to me why sunlight

20:48

is the the place where your focus is at

20:51

the moment.

20:52

It's a long trip that has gotten me to

20:55

that. Um and I and I think part of it

20:57

has to has goes through right through

20:58

COVID.

20:59

So, as a critical care intensivist, when

21:02

I heard that there was this virus that

21:04

was coming, they all told us that it was

21:07

going to be people with respiratory

21:09

illnesses, which I was certainly

21:11

comfortable with.

21:12

But, that's not what it turned out to

21:14

be. We certainly saw people with

21:15

respiratory illnesses, but what we saw

21:17

in the intensive care unit, the people

21:18

that were dying around me

21:21

were people with obesity, people with

21:23

heart disease, people with kidney

21:25

disease, people with dementia, people

21:27

with chronic diseases.

21:29

And it made me think, why was that the

21:30

case?

21:32

All of those things

21:34

have one thing in common.

21:36

And many other things, too. But,

21:38

specifically, they're rooted in

21:41

something called mitochondrial

21:42

dysfunction. So, let me unpack that for

21:44

you.

21:45

And and this has to do with longevity,

21:47

this has to do with aging. This is a

21:48

huge topic that is now just emerging.

21:51

And we're we're now finding more about

21:53

this. So, what when we were when I was

21:55

in high school biology, when I was in

21:58

college, uh we all learned about this

22:00

little organelle in all of our cells,

22:02

except for red blood cells, called the

22:03

mitochondria. And and and I have to say

22:05

it, what is the mitochondria? It's the

22:07

powerhouse of the cell, right? So, it's

22:09

the thing that makes energy.

22:11

What we didn't know at the time is that

22:13

as we get older, the output from these

22:15

batteries in our cells drops by about

22:17

70%. Damn. Can you imagine running your

22:21

house on 70% less energy? Well, how

22:23

fundamentally that would change what

22:25

change what happens in your house? Like,

22:27

you could not run the laundry the same

22:28

way. You could not run the microwave and

22:30

the laundry at the same time.

22:32

>> And what does that look like in terms of

22:33

symptoms? Excellent question, because

22:35

what it looks like is depending on the

22:37

cell type that we're talking about,

22:38

that's going to have the issue. So, if

22:40

we're talking about the liver, the

22:41

liver's going to get more fatty. If

22:43

we're talking about the heart, the

22:44

heart's going to is going to become more

22:45

congested. If we're talking about the

22:46

brain, it's going to have more dementia.

22:49

And so, what's what's happening here is

22:51

that as we get older, the batteries in

22:54

our cells are not working the same way

22:56

as they used to. Metabolism is slowing

22:58

down.

22:59

And and so, these are these are huge

23:01

issues.

23:02

And all of these diseases that I just

23:03

talked about, all the ones that we saw

23:05

in COVID, if you look in a lot of these

23:08

diseases, they are rooted in

23:09

mitochondrial dysfunction. So, the

23:11

question is is why is that the case and

23:13

what can we do about it?

23:15

So, there was a paper that came out in

23:17

2019

23:19

that fundamentally changed the way I saw

23:21

this. It was written by Russell Reiter

23:23

who is the

23:25

executive editor of melatonin research.

23:28

It's a he's out of University of Texas.

23:31

And Scott Zimmerman who's a light

23:32

engineer. And what they set forth was to

23:34

show that basically

23:37

sunlight

23:39

is

23:40

made up of so many different types of

23:42

wavelengths. You've got ultraviolet on

23:44

one end which of course makes vitamin D.

23:47

And it's very beneficial. It it it's the

23:49

type of light from the from the sun that

23:51

is very short wave

23:53

and but cannot penetrate very deeply.

23:55

Let let me let me back up a little bit

23:56

and explain.

23:57

You pull up to a stop sign and somebody

24:00

pulls up next to you and they're playing

24:01

the latest hip-hop music. How does that

24:04

sound to you in your car?

24:06

It's very boom boom, right?

24:08

>> Yeah, muffled. It muffled. And the

24:10

reason why is because low wave frequency

24:14

has the ability to travel very far.

24:18

Go to the Grand Canyon and there's a

24:19

thunderstorm at the other end of it.

24:20

What do you hear? It's like a rumbling.

24:22

And then as it gets closer, you hear the

24:24

higher pitched sounds. This is a

24:25

fundamental physics

24:27

principle. And so when the sun is is

24:30

shining, there's very short wavelengths,

24:33

ultraviolet B involved in vitamin D. But

24:36

at the other end, there's this infrared

24:37

light which we'll talk about or red

24:39

light. It's very long wavelength and it

24:42

can penetrate very very deeply.

24:45

That's very important because what we're

24:46

talking about is the human body. And if

24:48

the sun is going to have an effect on

24:49

the human body, it's got to be more than

24:51

just the skin.

24:53

So, that's exactly what what this paper

24:55

showed is that basically

24:57

infrared lights

25:00

from the sun

25:01

is able to penetrate probably up to

25:03

about 8 cm according to Scott Zimmerman

25:06

in this article.

25:07

And it fundamentally interacts with

25:10

specifically the mitochondria.

25:12

And what does it do to the mitochondria?

25:13

So let's let's back up and talk about

25:15

the mitochondria cuz this is central.

25:17

The mitochondria to the cell is like the

25:20

engine in your car.

25:21

The engine produces locomotion that

25:25

causes this the wheels to spin. But in

25:27

the process of doing it

25:29

it causes heat to surround the engine.

25:32

And if you don't deal with that heat

25:34

it will shut down the engine. It will

25:36

make it more inefficient and eventually

25:38

it will shut it down. So what do all

25:40

internal combustion engines have? They

25:42

have a cooling system. They have a

25:43

radiator. They have a an oil pan. They

25:45

have a water pump. And that's exactly

25:47

what the cell has to have for the

25:49

mitochondria. It's not heat in the

25:51

mitochondria. It's called oxidative

25:53

stress. And it's specifically oxidative

25:55

stress that causes destruction and

25:59

and yeah, destruction of the

26:01

mitochondria and leads to these types of

26:03

diseases. So oxidative stress causes the

26:05

mitochondria not to work well. This

26:07

leads to diabetes. Oxidative stress

26:09

makes the mitochondria not work so well.

26:11

This leads to dementia. So there's a

26:13

This has already been laid out. This is

26:14

not that controversial. The

26:16

controversial part is what do we do

26:17

about it? So what these guys in this

26:20

paper showed

26:21

was that and and not just them but look

26:24

reviewing the literature is that the

26:26

mitochondria makes its own cooling

26:28

system.

26:29

And that cooling system is melatonin.

26:33

Now you might think be thinking, wait a

26:34

minute, melatonin, isn't that the isn't

26:36

that the stuff that we take that our

26:38

brain makes right before we go to sleep?

26:40

Yeah, that you're it's absolutely

26:41

correct. That's what happens.

26:44

The problem is is that this is not

26:46

melatonin that's made in the brain. This

26:47

is not melatonin that goes through the

26:49

blood supply and goes to It through our

26:51

blood and tells us it's time to go to

26:52

sleep. This is melatonin that's made in

26:55

the cell in the mitochondria.

26:57

And it's a powerful antioxidant that

26:59

basically prevents the oxidative stress

27:02

from occurring.

27:03

What Scott Zimmerman and Russell Reiter

27:05

showed and and proposed in this was that

27:08

basically the infrared radiation that's

27:10

coming in

27:11

to the body is able to stimulate and

27:14

upregulate melatonin and a number of

27:16

other factors that keep the mitochondria

27:19

cool and can actually improve the energy

27:22

output of the mitochondria.

27:24

So this is This was actually

27:26

mind-blowing to me and I'll tell you why

27:28

I resonated with this as a critical care

27:30

physician because there was two things

27:31

that bothered me the most. Number one.

27:36

SARS-CoV-2 virus. When it comes into the

27:39

body, it interacts with something called

27:40

the ACE2 receptor. You may have heard

27:42

about the ACE2 receptor. Okay. This is

27:44

where the the virus actually latches on

27:47

to the cell

27:48

and gets internalized. So what is this

27:51

ACE2 receptor? Is this Was this there

27:53

for all of humanity just to be a

27:55

receptor or does it actually have a

27:56

role?

27:57

It turns out it actually has a role and

27:59

mind-blowingly

28:01

the ACE2 receptor is involved in

28:03

mitigating oxidative stress.

28:06

So in other words, it's another part of

28:08

the cell's cooling system for the

28:09

mitochondria.

28:11

What

28:12

what's happening is that the

28:15

the virus when it attaches to the cell

28:18

is basically eliminating that action.

28:21

And so

28:23

imagine you have a bunch of people with

28:25

various different engines running at

28:27

different temperatures. In other words,

28:29

you've got some people with chronic

28:30

disease and we know their engines are

28:31

running hot. We have some other people

28:33

who are completely healthy and they're

28:35

doing quite well. Their engines are nice

28:37

and cool. They have no problems at all.

28:39

Now imagine COVID comes and SARS-CoV-2

28:42

is infecting everybody.

28:45

What that tendency is to do is because

28:47

it's knocking out everybody's ACE2

28:49

receptor, which has the ability to cool

28:51

down the engine if in other words, it's

28:53

causing everybody's engine to run hot.

28:56

Right? So, but in somebody So, in other

28:57

words, picture it this way. You're

28:59

You're You're driving along in your car

29:01

and your thermometer is there and all of

29:02

a sudden there's this big hill that you

29:03

have to climb, called COVID-19. Who's

29:06

going to make it over that hill and

29:08

who's not going to make it over that

29:09

hill?

29:10

The people that make it over the hill

29:12

are those with great cooling engines

29:14

that whose temperatures are running

29:15

great. The ones that don't make it over

29:17

that hill are the ones that have the

29:19

thermometer on their engine running hot.

29:22

Those are the ones that poop out at the

29:23

top and can't make it and they're

29:24

they're the ones pulled over to the side

29:26

of the road with the hood up and the

29:27

steam coming out of the out of the

29:29

engine. Do you understand what I'm

29:30

saying?

29:30

>> yeah. So, this makes perfect sense to me

29:32

why I wasn't seeing what they were

29:34

predicting, which is respiratory

29:35

patients coming into the ICU. Who was I

29:37

seeing in the ICU? I was seeing people

29:39

with dementias, as we talked about,

29:40

diabetes, kidney disease. These are the

29:43

ones that were that that were sick. The

29:45

other thing that that really hit me

29:47

and resonated with this

29:50

was and this was this was not even

29:52

controversial. We knew early on in the

29:54

pandemic that people who came into the

29:56

hospital and had higher levels of

29:59

vitamin D

30:00

did really well.

30:02

They didn't die.

30:05

They They didn't have the same chances

30:07

of dying. People who had low vitamin D

30:09

levels, they had much higher levels

30:12

chances of dying.

30:14

So, we would check these vitamin D

30:15

levels. And so,

30:17

think about this. You're You're there at

30:18

ground zero and you're taking care of

30:20

these patients and you see this data

30:22

over and over and over again that

30:24

vitamin D is very predictive of who's

30:27

going to die. Obviously, what are you

30:29

going to do? Even though this is an

30:30

associative study, that association

30:32

doesn't mean causation, you're going to

30:33

be giving people vitamin D

30:35

and try to get those levels up.

30:38

The problem is is that we gave vitamin D

30:40

and it really didn't have much of an

30:42

effect.

30:44

So, you gave it in supplement tablets.

30:46

>> Yeah, when people would come into the

30:48

hospital exactly.

30:50

Exactly, just like that. In fact, I was

30:51

supplementing myself. I mean, what have

30:53

you got to lose, right?

30:58

I already took my vitamin D tablet this

31:00

morning. I don't There's not There's

31:01

very hard to overdose, but it's

31:02

possible. So, you noticed that it was

31:05

hard to treat people with vitamin D, but

31:07

giving them a a tablet didn't really do

31:08

much. That's correct. Why?

31:11

Well, this is what I believe is the is

31:12

the is the fact is we saw that people

31:15

with high levels of vitamin D or normal

31:17

levels of vitamin D did better than

31:19

those that had low levels. I believe

31:21

that that was a marker of something

31:23

else.

31:25

In other words, people who had higher

31:28

levels of vitamin D meant

31:30

that they were out in the sun more. They

31:32

were outside more.

31:35

Than those the people that had very low

31:36

vitamin D levels. The people with low

31:38

vitamin D levels were telling me these

31:40

are people that were not getting outside

31:42

into the sun. And so, what's the real

31:44

factor here? What's doing the heavy

31:46

lifting? And I would propose and Scott

31:48

Zimmerman and Russell Reiter would

31:49

propose, and I can tell you a number of

31:51

other scientists that would agree with

31:52

me on this,

31:54

is that infrared radiation

31:56

from the sun

31:58

is causing an effect at the

32:00

mitochondrial level in terms of

32:02

oxidative stress.

32:04

And that vitamin D was just the marker

32:07

of who was getting the infrared light

32:08

and who was not. Who was going outside

32:11

and who was not going outside. So, when

32:13

the sun is shining for the most part,

32:16

you're getting infrared light. You're

32:18

getting the entire the entire biological

32:20

spectrum from the sun. We can go to the

32:22

longest wavelength, which is far

32:24

infrared,

32:25

all the way to the shortest wavelength,

32:27

which is ultraviolet B, okay, which

32:30

makes vitamin D.

32:31

So, in other words, when you are outside

32:33

in a natural environment, you're getting

32:35

a very broad spectrum of light.

32:38

And so, because of that, if you're

32:40

getting infrared light, you're also

32:42

going to be making vitamin D.

32:43

Yeah, you're getting both.

32:45

Um

32:47

Now, that can change because in the

32:49

wintertime

32:51

when the sun is lower in the sky,

32:53

especially, you know, in England, this

32:55

is this is a special issue at that

32:57

latitude. When the sun is low in the

32:59

sky, it's got the light has to penetrate

33:01

through obliquely through the

33:03

atmosphere. And because of that,

33:05

shortwave

33:07

radiation from the sun, like ultraviolet

33:09

B, does not make it very well. So,

33:11

there's times of the year where you're

33:13

not getting enough ultraviolet B or

33:15

maybe even no ultraviolet B from the

33:17

sun. Which makes the vitamin D. Which

33:19

makes the vitamin D. And that you're

33:21

you're going to be deficient, you need

33:22

to supplement. However,

33:24

during that same period of time

33:26

when you're not getting enough vitamin D

33:28

because there's no ultraviolet B

33:29

radiation, that sun is low, but that's

33:32

it's still enough to allow that long

33:35

wavelength penetrating infrared light to

33:38

still come through. So, is the longwave

33:41

infrared light the type of thing that we

33:44

see

33:45

these gadgets doing? Absolutely. And and

33:48

I would say just to be specific is

33:50

because you can see that as red light

33:53

there, that's not infrared light because

33:55

you can see it.

33:56

So, infrared light technically is

33:58

completely invisible. But this these do

34:01

give out infrared light, but you just

34:02

can't see it.

34:03

>> So, it's toward that red end of the

34:05

spectrum. And people like Glen Jeffery

34:07

out of UCL

34:09

is actually doing research at 670

34:11

nanometers of red light and has shown

34:14

in randomized controlled trials that

34:16

that type of light right there at 670,

34:18

the type that you can even see,

34:19

actually does improve mitochondrial

34:22

efficiency. He's shown this in a number

34:23

of randomized controlled trials. It

34:25

improves eyesight. And and you have to

34:27

realize that the retina at the back of

34:28

your eye is very rich in mitochondria.

34:32

He's shown this in terms of managing

34:34

glucose and and output for mitochondria.

34:38

And and the reason why these things work

34:40

so well is because what's going on here

34:42

is as you get older, your skin starts to

34:45

become more saggy because the the

34:48

fibroblast or the cells in your skin,

34:50

they're designed to make collagen and

34:52

collagen is the skeleton that makes your

34:55

skin soft and subtle. Yeah. Don't mind

34:58

me. Please carry

34:59

>> No, no. So this is this is exactly My

35:01

wife uses the same thing. This is a fun

35:03

charge one day.

35:04

>> So what what's going on right now is

35:05

that red light

35:07

which can penetrate very deeply down is

35:09

going into the skin and it is activating

35:12

the mitochondria in your fibroblast to

35:14

produce more energy which those cells

35:16

need to deposit collagen. And so when

35:19

you deposit collagen, that's going to

35:20

give the skin a more tight uh feel

35:24

because as you get older, that collagen

35:25

deposition is going to get less and less

35:27

and less.

35:28

>> So this is going to help keep me looking

35:29

young. That's the whole point of it. And

35:31

you're saying that the the light in

35:33

these penetrates what, 6 or 7 cm or is

35:36

it

35:37

>> does about 8 cm. Uh this red light would

35:39

be a little bit less because this

35:41

light obviously you can see it, so it is

35:43

a little bit shorter wavelength, but

35:44

yes, this light, the red light can

35:46

penetrate deeper than for instance

35:48

yellow light or blue light. And it's

35:50

this light particularly that interacts

35:52

with the mitochondria to increase that.

35:54

So should we be putting this all over

35:56

our body because okay, it's good for my

35:57

skin, but if it's penetrating deeper,

35:59

presumably those other parts of my body

36:01

that would benefit from that, another

36:02

mitochondria. It's interesting you say

36:04

that. Um the the the study that I'm

36:07

referring to with Glenn Jeffery out of

36:09

University College London, he took uh

36:12

young people in this study. He gave them

36:14

a a bunch of glucose. And everybody who

36:17

gets a bunch of glucose should have a

36:18

spike in their blood sugar.

36:21

And he randomized them on their backs

36:24

to see what would happen when he shined

36:26

red lights uh on their backs. And the

36:28

people that got the red light had lower

36:30

spikes. In other words, it seemed as

36:32

though the mitochondria

36:34

were metabolizing faster, which caused

36:36

less of a spike of the glucose in their

36:38

blood. The way he confirmed that is

36:41

looking for the byproducts of the

36:43

mitochondrial metabolism, which is

36:45

carbon dioxide. So, when we breathe,

36:47

when we metabolize, we're breathing out

36:49

carbon dioxide, which is the result of

36:51

of a mitochondrial metabolism. And in

36:54

fact, in those people that had the light

36:57

on,

36:58

it showed a higher level of carbon

37:00

dioxide in the exhaled breath. The whole

37:02

point of that is to get back to your

37:04

question is whether we should putting

37:05

this all over your body. He was able to

37:07

get that effect systemically with just

37:10

putting the light on the back.

37:13

Um that was a that was a systemic

37:15

ability. What We don't understand

37:17

everything about the mitochondria, but

37:18

what we do seem to understand is that

37:20

they can communicate with each other.

37:22

And that you don't need to have this all

37:25

over the body to have systemic effects.

37:28

In this particular case though, if you

37:29

want to have the skin here to be more

37:33

you know, younger-looking, then it makes

37:36

sense that this is where you need to

37:37

have it. If you want to have a

37:38

particular other part of your body to

37:39

look younger, then then perhaps that

37:41

that's where the light needs to go. So

37:43

interesting. So how long did it take in

37:45

those studies to see the effect of red

37:47

light therapy like this? Well, that's a

37:49

very good question. If you talk to Glenn

37:51

Jeffery, which I have, he noticed a an

37:53

improvement in 15 minutes.

37:55

15 15 minutes? What did he notice in 15

37:58

minutes? He said he has studied the

38:00

mitochondria in fruit flies and in

38:03

mosquitoes and bees and in human beings

38:05

and it's the same every time. He says

38:07

after about 15 to 20 minutes of this

38:10

type of light in that type of setting,

38:12

there is a switch

38:14

that turns on and it and you and you

38:16

don't need further stimulation. Further

38:18

stimulation doesn't do anything more.

38:21

It's a it's a very bizarre thing. You

38:22

would think that the more light that you

38:24

gave, the more the effect would be. It's

38:26

not. After about 15 minutes, it it

38:29

there's something that changes in the

38:30

mitochondria.

38:32

There are certain theories about where

38:33

this might be. This might be in the

38:34

electron transport chain

38:36

complex four. Uh these are very

38:38

technical things. There's a lot of

38:39

studies that are actually There's a

38:40

number of groups that are actually

38:41

looking at this.

38:42

Uh there's a whole area of science

38:44

called photobiomodulation, which is

38:46

looking at this. But, 15 minutes is

38:50

really what it takes. So, we're not

38:51

we're not talking about a long period of

38:53

time.

38:55

This is really really interesting. So,

38:57

getting back to my

39:00

experience in the intensive care unit.

39:03

The vitamin D wasn't working.

39:05

These patients were dying, and it became

39:07

clear to me that COVID was a metabolic

39:10

issue

39:11

for these patients.

39:13

By the time I had realized this,

39:16

the sir I mean, the pandemic arguably is

39:18

still going on because people are still

39:20

becoming infected, but the rush to come

39:22

into the hospital and the and the number

39:24

of bodies that we were seeing

39:25

circulating through the intensive care

39:27

unit had dropped dramatically.

39:29

And at that point, I was able to

39:33

see that potentially

39:35

infrared lights may be very beneficial

39:39

in these patients with COVID-19. Now,

39:42

there was a study in Brazil. They took

39:45

COVID patients

39:46

that were that were sick enough to be

39:49

admitted to the hospital, but not too

39:50

sick to be intubated in intensive care

39:52

unit.

39:53

And they did something tremendous. They

39:55

actually manufactured a jacket that they

39:58

could put on patients. And on the inside

40:00

of this jacket were these LED bulbs that

40:03

gave off

40:04

infrared radiation at exactly 940

40:07

nanometers. They put the jackets on,

40:10

and they randomly randomized assign

40:13

which jacket was turned on and which

40:15

jacket was turned off. It was blinded

40:16

because the light coming from this

40:18

jacket could not be seen by the human

40:20

eye. It wasn't even enough to produce

40:21

enough heat.

40:22

And so they they did this on 30 subjects

40:25

and they randomized them. 15 did it 50

40:27

50 all 15 had or all 30 had the jackets

40:30

on. 15 had it turned on, 15 did not have

40:33

it turned on. And they watched them.

40:34

What happens to these patients?

40:37

Every single endpoint that they looked

40:38

at was statistically significant.

40:41

And What does that mean?

40:42

>> It means that the differences between

40:44

these two groups could not have been

40:46

from chance.

40:47

There was a real difference.

40:50

The group that had the jacket turned on

40:52

had improvement in their oxygen

40:54

saturation.

40:56

Had could take breaths in more deeply

40:59

and stronger. Had improvements in their

41:02

blood white blood cells.

41:04

Um and not only that, had improvements

41:06

in their heart rate, their respiratory

41:08

rate, all of these statistically

41:09

significant. But the most important and

41:11

mind-blowing statistic was the length of

41:14

stay in the hospital.

41:15

So,

41:16

they had these jackets on

41:18

for 15 minutes

41:21

once a day

41:22

for 7 days.

41:25

In the group that did not have the

41:27

jacket turned on, their average length

41:29

of stay was 12 days in the hospital.

41:31

For those that had the jacket turned on,

41:33

it was eight days. That was four day

41:37

difference. That's tremendous when you

41:39

realize that it costs thousands of

41:41

dollars

41:43

to hospitalize patients.

41:45

It's it's a huge amount when you think

41:47

about the fact that there are certain

41:49

drugs that get FDA approved for

41:52

influenza for instance by just cutting

41:54

short the symptoms for 24 hours. This is

41:57

not just 24 hours for people were

41:59

discharged

42:00

from the hospital four days faster. When

42:03

I saw that study, that was enough for me

42:05

to convince me. I mean obviously it was

42:06

30 subjects, right? We should do a

42:08

bigger study. We should do a hundred a

42:09

couple hundred, right? That would be

42:11

that would be ideal to do. But the fact

42:12

that with just 30 patients they could

42:15

show statistical significance. That was

42:16

enough for me to say every patient from

42:19

now on that I see

42:20

that comes in with COVID-19 that's

42:22

hospitalized, that they're asking me to

42:24

go intubate, to bring to my ICU, these

42:27

patients are going to get outside.

42:29

I don't have that jacket that they made

42:30

in Brazil.

42:31

I I I don't even know how I would make

42:33

that jacket. They made it for the study.

42:35

And it's not commercially available.

42:36

There's no 940 nanometer lights, which

42:39

is what they did in the study. But I do

42:41

know this. I do know that sunlight has

42:43

940 nanometers in it.

42:46

And if I could just take these patients

42:47

outside

42:49

maybe they could improve. So I had I got

42:51

my wish.

42:53

I had a patient on the floor.

42:56

He was on 35 liters a minute.

43:00

100% oxygen through high flow. Through

43:03

his nose. Through his nose. Barely

43:05

barely saturating because he had

43:06

COVID-19.

43:08

And uh I was asked to go see him because

43:10

he was potentially needing to be

43:11

intubated or brought to the intensive

43:13

care unit. I could not believe it cuz I

43:15

had not seen one of these in months. So

43:17

I went down, walked into the room,

43:18

opened the door. It was in isolation. I

43:20

had a mask on, the whole nine yards. The

43:22

room was completely dark. The blind was

43:24

closed. His daughter was there. And the

43:26

first words out of his mouth to me was

43:29

"Doc, how much time have I got?"

43:32

I mean, it was a catastrophe. Like

43:34

there's no light, no circadian rhythm.

43:36

This guy was depressed.

43:38

I immediately called my respiratory

43:39

therapist, immediately called the charge

43:41

nurse. We got everybody together. And I

43:44

said, "We need to get this guy outside."

43:45

It was a bright and sunny day.

43:47

How are we going to get this guy

43:48

outside? 35 liters, 100%. We My

43:50

respiratory therapist, Kim, managed to

43:53

put a couple of oxygen tanks together.

43:55

And we were able to get this guy into a

43:57

wheelchair.

43:58

And we wheeled him outside.

44:00

And uh he told me this uh weeks later.

44:04

But he says, "You know, that first day

44:06

that you got me outside in the sun cuz

44:08

we did this for like 7 days in a row.

44:10

He said, "That felt so good."

44:13

Um he after just 1 day dropped down from

44:16

35 L to 15 L.

44:18

15 L of of oxygen. And then down to 12,

44:21

and then down to 8 the next day, and

44:23

then down to 5. 5 days The amount of

44:25

oxygen he was inhaling to keep him

44:27

alive. Correct. So, in other words, we

44:29

were titrating down the amount of oxygen

44:30

that we had to give him to maintain a

44:32

saturation in the 90s. In 5 days he was

44:35

discharged home without oxygen.

44:38

Now, obviously that's an anecdote,

44:40

right? That's not a study. But, I'm

44:42

looking at the risks of getting people

44:44

out in the sun for 15-20 minutes.

44:46

There's not a lot of risk to that. And

44:48

if there's a benefit, I thought it was

44:49

worthwhile doing. We we need to have

44:51

larger randomized controlled trials, but

44:54

it got me down the road to looking to

44:55

see what what was it

44:58

about sunlight

45:00

that was affecting this change. And you

45:01

know what? There was There's There's

45:03

ample data.

45:04

There There was a study actually that

45:06

was done

45:07

in in Europe where they looked They

45:09

said, "Okay, here's COVID. COVID's going

45:12

up. When does COVID go up? Is it because

45:15

of temperature that changes? Is it

45:17

because of humidity?" And the answers to

45:18

both of those were no. Do you know what

45:20

predicted when countries were to have

45:22

their first surge in the autumn of 2020?

45:24

There was a study that was actually done

45:25

on this.

45:26

It was latitude.

45:28

It started in Finland

45:30

and then real it went down the entire

45:32

continent. The last country in the in

45:34

the autumn of 2020 to have a COVID surge

45:37

was Greece.

45:38

So, as the sun is literally pulling down

45:41

into the southern hemisphere, as the

45:43

shadow starts to go over Europe, that's

45:45

when we start to see

45:47

COVID surges one by one by one. There

45:49

was Yeah. Is that Is that because COVID

45:53

and the sun aren't friends? So, if I it

45:55

makes it harder to spread because, you

45:57

know, if I put COVID on this table and

45:58

then I put sunlight on the table, the

46:00

COVID is going to die. Yeah, it's

46:01

possible. Although we now know that

46:04

COVID probably doesn't spread too much

46:06

through contact. It's it's more of an

46:08

airborne thing. So there there was a

46:10

study that was done at the University of

46:12

Edinburgh.

46:13

And they looked at this very question

46:16

that we had talked about earlier about

46:17

vitamin D. They looked at the United

46:19

States in the in the wintertime.

46:21

So and they eliminated the southern part

46:23

of the United States because in the

46:24

southern part of the United States you

46:25

can actually get some vitamin D in the

46:27

wintertime. So they just looked at the

46:29

sort of the northern portion of the

46:30

United States and they were able to show

46:32

that the more sunlight there was in

46:36

particular areas, the lower the

46:38

mortality from COVID-19.

46:41

So they said, "Well, this is

46:42

interesting.

46:43

What about in England?" So they they did

46:45

the exact same study in England and and

46:47

sure enough of course they didn't have

46:49

to eliminate any part of England because

46:50

the whole country doesn't get in any

46:52

vitamin D in the wintertime. What they

46:54

showed was that again, certain parts of

46:56

the country in England as you know, get

46:58

more sunlight than other parts. Well,

46:59

those areas that got more sunlight had

47:01

lower

47:03

mortality from COVID-19. Then they took

47:05

the same

47:07

They predicated the same study and they

47:08

looked in Italy.

47:10

Exactly the same finding and they

47:12

published this. And they said in their

47:14

study and this is what really amazed me.

47:16

They said,

47:18

"The fact if this is causal,

47:20

they say." They said that

47:23

this might actually show a possible

47:27

public health intervention. The fact

47:29

that it is completely independent of

47:31

vitamin D means that there's something

47:33

else going on.

47:35

There was a study in 2011 in Sweden.

47:38

Yes.

47:39

Is that linked to this?

47:40

>> No, this is a completely different

47:42

study. But that that's also a very

47:43

important study. So the Swedish study is

47:45

is is groundbreaking. Um

47:48

This was a study where they asked 20,000

47:51

20 to 30,000 Swedish women

47:54

about their habits in sunlight.

47:56

And they divided these women into three

47:59

categories. Those women that did not get

48:01

a lot of sun, those that got a moderate

48:03

amount of sun, and those that got a lot

48:05

of sun. And they followed them for 20

48:07

years. And they they kept a track of

48:10

each one that died and what they died

48:12

of.

48:13

And when they were done with that, they

48:14

were astonished because what they found

48:16

was that

48:17

the women who had spent most spent the

48:20

large amount of their time outside or

48:21

that spent the most amount of time

48:22

outside

48:24

had

48:25

the least amount of mortality from

48:27

cancer,

48:29

from cardiovascular disease, and

48:30

non-cardiovascular disease. And those

48:32

that spent the least amount of time

48:34

outside had the highest levels of that.

48:36

The magnitude difference between those

48:38

two was so much

48:41

that they were able to show that women

48:43

who in Sweden

48:46

who spent the most amount of time

48:47

outside and smoked

48:51

had the same mortality as those women

48:53

that did not spend as much time outside

48:56

and did not smoke.

48:59

They were equal. They were equal. In

49:01

other words,

49:02

being in that category of not spending

49:04

much time outside in the sun was the

49:07

same risk factor for death as smoking.

49:10

How do they know it wasn't linked to

49:12

exercise? How are they able to establish

49:14

causation cuz that's a You're absolutely

49:16

That's an excellent question. So, the

49:18

the difference here as you go up is this

49:20

is a this is an association study, okay?

49:23

So, the question is is how can you get

49:25

causation from association? You can't.

49:28

But, if you look at the Bradford Hill

49:30

criteria, there is a way that you can

49:33

potentially make a a strong argument for

49:36

causation if there's something called a

49:37

dose-response curve. In other words, if

49:41

you can show you're not just comparing

49:43

two things, but you're comparing three

49:45

or more, if you can show that as you

49:48

increase

49:49

the variable, that there is a change in

49:51

the

49:53

the outputs that is strongly suggestive

49:55

of potentially causation. By the way,

49:56

this is exactly what we did to show that

49:58

smoking causes lung cancer. Obviously,

50:00

we can't do a randomized control trial

50:02

here. You get to smoke, you don't get to

50:04

smoke. We'll follow up in 20 years to

50:05

see who has lung cancer. This is exactly

50:07

what we did. We showed that that there

50:09

was such a strong association with

50:11

cancer risk with smoking that we were

50:13

able to say through association that

50:15

smoking causes lung cancer. By the way,

50:18

Richard Weller

50:20

was a dermatologist in England uh did

50:23

just last year a very similar study as

50:25

to the Swedish study except it was 10

50:26

times bigger

50:28

and he did it with both men and women.

50:31

He found the same results.

50:34

It was a UK Biobank study.

50:36

What did he discover? He discovered that

50:39

either from

50:40

from solariums or from um or being

50:44

outside using solar radiation data

50:47

uh he was able to show both on their

50:50

their questionnaire and also where they

50:51

lived

50:53

that the more light that they had the

50:55

lower their risk as of of um mortality

50:58

uh and um and cancer mortality. So, the

51:01

question was

51:02

does it increase melanoma?

51:05

What's melanoma? Melanoma is is a skin

51:07

cancer. So, that's the big risk. That's

51:09

the big risk that everybody's concerned

51:10

about. You go out into the sun sun and

51:12

you're going to get skin cancer.

51:13

And he was able to show in that study

51:15

this was like three three 400,000 people

51:19

in this study. UK Biobank study, Richard

51:21

Weller.

51:22

He was able to show that

51:24

there was no increased there's no

51:26

statistical increased risk of melanoma

51:29

incidence but there was

51:32

a reduction in non-skin cancer

51:35

mortality.

51:37

Okay, so so here's the trade-off. If you

51:39

want to go out into the sun in England

51:42

okay?

51:43

The benefits are you're going to have a

51:45

reduction

51:47

in non-skin cancer mortality. So,

51:49

everything other than skin cancer?

51:51

>> Correct.

51:52

On the other hand, there's no increase

51:54

in melanoma incidence. So, that caused

51:57

him to write an a an op-ed and publish

52:00

it. And actually, you can look up this

52:02

op-ed. It's a great op-ed. Uh it

52:04

published in a um

52:05

in the Journal of Investigative

52:07

Dermatology called Sunlight: Time for a

52:10

Rethink. Where he goes through the

52:12

arguments. And he's actually shown And

52:14

there's been a number of changes that

52:16

people have making around the globe. So,

52:18

public health uh

52:20

organizations that are saying now, you

52:23

know, before we have said

52:25

that, you know, the the sun is a deadly

52:27

laser.

52:29

And you should avoid it at all costs.

52:31

We may need to rethink that.

52:33

So, you telling me that essentially 15

52:35

minutes in the sun every day turns on a

52:37

switch in my body that improves my

52:40

mitochondrial function, which is going

52:42

to impact a variety of different parts

52:44

of my health. Is that essentially what

52:46

you're saying? Essentially, yes. Uh and

52:48

and we're we're looking And this is in

52:50

an environment where where we are

52:52

spending less and less and less time.

52:55

To give To give you To put in

52:56

perspective, if we were on a British

52:58

ship 300 years ago, and I came to you

53:01

and I said, "Do you see this little

53:02

yellow fruit? Just by eating a little

53:05

bit of this yellow fruit, all of this

53:06

disease that you're seeing around you

53:07

with your fellow shipmates is going to

53:09

go away." That that would seem almost

53:10

incredulous, right? But, that's exactly

53:12

the case.

53:14

We We are

53:16

The scurvy of the 21st century is the

53:18

lack of sunlight.

53:20

Everything is inside.

53:23

Uh we we avoid the outside. We avoid

53:25

discomfort. We avoid high temperatures.

53:27

We avoid low temperatures. We uh are We

53:30

used to go out and play sports. We now

53:32

are are playing virtual sports on on

53:35

pads.

53:36

We have windows

53:38

that are specifically designed,

53:39

especially here here Southern

53:40

California, to eliminate infrared

53:43

lights. Because why? Infrared lights

53:45

comes in and it heats up. One of the

53:47

interesting things we didn't mention

53:49

this about infrared light is the way

53:50

that we interact with infrared light.

53:52

You can You can tell this on your own.

53:53

You go outside

53:55

and

53:56

close your eyes. You can tell which side

53:59

of your body the sun is on. And the

54:00

reason is is because that infrared light

54:03

not only can penetrate through your

54:04

body, it's also penetrating through

54:06

clothes

54:07

very easily.

54:08

And you can feel that. That That heat

54:10

that you're feeling is the infrared

54:12

light going through this going through

54:14

the clothes, going through the skin, and

54:15

interacting with your heat receptors

54:17

that are well below the surface.

54:19

Um so all of this

54:22

All right, guys. Pretty good. I got

54:23

Steve. The guest is here.

54:25

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How long does the average American spend

56:15

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56:16

indoors? Good question. They're almost

56:19

identical. I think the Brits spend a

56:21

little bit more time outside than

56:22

Americans. The The last number for

56:25

Americans was 93%

56:27

and uh Brits is 92%.

56:29

>> Were we born to be outside? I think so.

56:32

You think our ancestors probably spent a

56:33

huge amount of time outside? Yeah. And

56:35

And And if you think about when I say

56:37

outside, that also brings into play a

56:40

number of other of the new start letters

56:42

that we haven't talked about. Uh

56:44

exercise. You're much more likely to do

56:46

good exercise outside. You're much more

56:48

likely to get air that's that's the

56:51

right type of air outside.

56:53

The other aspect about infrared light,

56:55

briefly,

56:56

is that trees

56:58

are highly reflective of infrared light.

57:01

In other words, if you're in an

57:02

environment where there are trees are

57:04

present, you're going to get much more

57:06

of this beneficial infrared light than

57:08

if you're in a concrete jungle.

57:10

So So So plants like this. In fact, the

57:14

way that we measure the deforestation of

57:16

the Amazon is through satellite imaging

57:19

that looks at infrared light because it

57:21

reflects infrared light back. So,

57:24

the best thing best situation to be in

57:27

is to be outside

57:29

on a green

57:30

on a day where where there's lots of

57:32

green trees. We've known for We've known

57:34

for decades that people who live in

57:36

green spaces

57:38

do much better in terms of diabetes, do

57:40

much better in terms of hypertension,

57:42

mortality, all of these things.

57:44

>> Depression. Depression, all of these

57:45

things, yeah.

57:46

And And when you think about this, um

57:50

you bring up a point in terms of of

57:52

correlation. How do we know it's not

57:53

depression? We used to say, "Well,

57:55

people who live in green spaces have

57:57

more money."

57:58

that people have They have more access

58:00

to things. Maybe that's what we're

58:01

saying.

58:02

I just have to tell you this study.

58:03

There was something called the

58:04

GreenHeart study in South Louisville,

58:06

Kentucky. They did an amazing thing.

58:08

They took this 4-square-mile area

58:11

in South Louisville, Kentucky, urbanized

58:14

area.

58:16

And they measured everybody's hs-CRP.

58:19

What is hs-CRP? Highly sensitive

58:22

C-reactive protein. It's a marker of

58:25

inflammation, and it's been correlated

58:27

to bad things like stroke and heart

58:29

attack. So, if you have high levels of

58:31

CRP, that's not good.

58:34

So, they measured every They measured

58:35

about 700 people.

58:37

And then they did something

58:39

extraordinary. They purchased 8,000

58:43

mature trees,

58:45

dug holes, and planted 8,000 trees into

58:48

4-square-mile area. And these are trees

58:50

with leaves on them.

58:52

2 years later, they come back and they

58:54

measure all 700 people in their study.

58:57

Repeat the hs-CRP.

58:59

Dropped by 13 to 20%, which correlated

59:02

to about a 10 to 15% reduction in

59:04

strokes.

59:05

These people didn't change their

59:07

socioeconomic status. They didn't

59:09

institute an exercise program. And so,

59:12

really, it kind of shoots in the heart

59:14

the idea that the advantage that we see

59:17

with green spaces has to do with

59:19

something else that we're not measuring.

59:20

I actually believe that we'll talk about

59:23

fresh air, too, that things like

59:26

these plants,

59:28

but much bigger. This is kind of like a

59:29

bonsai plant. But But trees outside,

59:32

they actually have a benefit. And what

59:34

they represent, again, is

59:37

these things that

59:40

don't leverage the other parts of your

59:42

body in the in terms of the chains uh

59:45

that make all of the chains bigger cuz

59:46

they're they're they're they're having a

59:48

benefit. But you can't get the benefit

59:49

of this if you're inside a house. So,

59:52

what should we do about this in terms of

59:54

how what changes should I make in my

59:56

life to capitalize on this? Um this is a

59:59

This brand here is called Bon Charge.

60:01

They do these Yeah. these red light

60:03

devices. They do like red light saunas,

60:05

blankets, um masks. They're actually a

60:07

sponsor of mine because I I started

60:09

wearing this and I think they they found

60:10

out. And I started wearing it because of

60:13

my girlfriend. Yeah. She was wearing it

60:15

every day and I got curious. And so, I

60:16

as I always do, I'm always super

60:17

skeptical. Sure. So, I went on online

60:20

and started looking at some of the

60:21

research. And I was shocked. Yeah.

60:23

>> It made no intuitive sense to me that a

60:25

red light mask or any like red light

60:27

device could have a profound like what I

60:29

see is a profound impact on my health.

60:30

Like I didn't believe it Yes.

60:32

>> to start with. Yes.

60:33

>> like woo-woo stuff to me. I I Yeah.

60:34

>> But I couldn't disprove it. Right. All

60:36

the studies, many of which you've

60:37

referenced, um supported that it was

60:40

having a profound impact. And as I said

60:41

on this podcast before, my girlfriend's

60:42

always right.

60:44

She's like always ahead of the curve and

60:45

always right. So, I started wearing her

60:47

mask and now I have my own from Bon

60:48

Charge.

60:49

>> Yeah. Ish. Do you recommend these kinds

60:50

of things? I I think it's reasonable to

60:53

do. I will say this. Um if you are

60:56

getting enough infrared light from the

60:58

sun,

60:59

what we find in studies, not

61:01

particularly with the mask, but we find

61:02

in other things, is that these these

61:04

other areas don't have as much efficacy.

61:07

It's almost to say, if you're on a ship

61:09

with a bunch of people with scurvy and

61:10

you already have a diet that's rich in

61:12

vegetables and fruits, eating an extra

61:14

lemon is not going to be that

61:15

beneficial. So, what do you do? Like a

61:17

lot of doctors do, we have shifts that

61:19

go from 7:00 a.m. to the p.m. So, you're

61:21

in the hospital, you're not going to get

61:22

outside. So, at lunch, I try to get

61:24

outside as much as I possibly can into

61:26

the sun for my 15 minutes.

61:28

>> What if you live in a cloudy country?

61:30

So, that's a good point. Uh clouds,

61:33

because they are water molecules, will

61:35

absorb a lot of the infrared lights. And

61:38

the problem is is that that's the

61:40

exactly the type of light that you want

61:42

to get. However, even on a cloudy day,

61:44

being outside, you're going to get more

61:46

infrared lights than if you were inside.

61:49

Okay, so I still get the light I need

61:53

when it's cloudy, but I just don't get a

61:55

lot.

61:56

Yes, exactly.

61:57

>> Is there anything I can do to get if

61:59

it's super cloudy and I know I'm going

62:01

to be indoors,

62:03

what do I do then? Yeah. So, the light

62:05

the type of lights that we have inside,

62:07

like these ones? Like these. Uh and and

62:10

and actually, I think the UK and the

62:12

United States are very similar in this

62:13

regard, is that we really can't get the

62:16

old incandescent bulbs. We're on LED or

62:19

or fluorescent. And if you think about

62:21

what they've done in terms of of these

62:23

bulbs,

62:26

the old incandescent bulbs used to give

62:29

you a very broad uh

62:31

spectrum. So, all the way from, you

62:33

know, just near blue all the way down

62:36

into the into the infrared. The way that

62:38

they've made the bulbs more efficient,

62:40

they said, "Hey, let's stop using energy

62:43

to give off this light that we can't see

62:46

and give a very narrow spectrum of light

62:48

that we can see."

62:50

So, think about what they've done. They

62:51

have for the first time in the history

62:53

of humanity,

62:55

they are now we are now being exposed to

62:57

light

62:59

in a very narrow spectrum without

63:01

anything else. Whatever in the history

63:03

of of humankind, when we light a candle,

63:06

when we would go outside into the

63:08

sunlight, when we would have a kerosene

63:10

lamp,

63:11

we were getting full spectrum.

63:13

In other words, we were never getting

63:15

blue lights without red lights. Now,

63:18

we're starting to get blue light without

63:19

red light.

63:20

So So do I change my bulbs?

63:23

It's difficult to do that because you

63:25

can't pick up in these incandescent

63:27

bulbs. Which brings me to I mean at

63:29

least at least in the United States, we

63:31

have laws now that outlaw the regular

63:33

selling of incandescent bulbs because of

63:36

energy efficiency. Oh yeah. I'm on

63:38

Google now and I've typed in

63:39

incandescent bulb. So there's something

63:41

called a general service lamp, which is

63:43

what the type of bulbs that you could

63:44

plug in. But if you decide that you want

63:47

to get a

63:48

bulb that you put into your microwave or

63:50

a bulb that you would do into a type of

63:52

chandelier that's a special type of

63:54

chandelier, those are still available.

63:56

You can still get incandescent bulbs for

63:57

those. What about these kinds of bulbs?

64:00

Is that an incandescent bulb? That's a

64:03

That is a That is an incandescent bulb.

64:04

Again, for these special type of lights,

64:06

but I'm I'm talking about the light like

64:09

the like the the good old fashioned A90,

64:11

I think it's called, light bulb that you

64:12

just screw in.

64:13

Those are

64:15

the 120 watts, those are getting more

64:16

difficult. You It's harder to find. You

64:18

can't go down to your Home Depot and and

64:20

find them there. So Glenn Jeffrey um and

64:24

this is this is a preprint that he is

64:26

he's done. He actually took

64:29

people

64:30

with that were working in this

64:31

environment with LED bulbs. He's

64:33

actually It's not peer-reviewed, it's

64:35

not published yet, but it's a preprint,

64:37

it's available on the internet, so I'm

64:38

not speaking out of class.

64:40

And what he did with 22 people is he

64:43

switched out

64:45

these LED bulbs and put in incandescent

64:48

bulbs.

64:49

And there was a there was a 25%

64:51

improvement in in color differentiation

64:53

in his study.

64:54

What does that mean? They were able to

64:57

distinguish colors 25% better than they

65:00

were when they were exposed to LED LED

65:03

When I say LED bulbs, these are the

65:04

bulbs that are high on the blue end.

65:08

Um so why would that be?

65:10

The retina, which is the back of your

65:12

eye, where in the light is coming in,

65:13

there's these cones that are

65:16

tremendously metabolically active.

65:19

They're constantly updating, sending

65:21

neural signals to the brain.

65:23

And there's It's the It's the one tissue

65:25

in your body with the most amount of

65:26

mitochondria. And it's because they have

65:28

to supply a lot of energy. As somebody

65:30

gets older,

65:33

that mitochondria is not producing the

65:35

same amount of energy. And so, the the

65:37

ability of the energy that those cones

65:40

have to draw on to do their work is

65:42

less. And so, they're not going to do

65:43

the job as well.

65:45

If you can perhaps increase the

65:47

the

65:48

the amount of output of energy from

65:50

those mitochondria, you could improve

65:53

the the the ability to visually

65:56

perceive. And and and

65:58

Glenn Jeffery has done this study

65:59

already, where he

66:01

for just 3 minutes,

66:03

670 nanometer light, very similar to

66:05

that mask, in the eye, only in the

66:07

morning,

66:09

improved

66:11

those people's ability to to to to

66:14

visualize and actually see.

66:16

And what does that mean for the broader

66:18

picture of our health?

66:19

>> would be able to distinguish colors

66:21

better and actually improve their

66:22

vision.

66:24

That's basically what it means.

66:26

Um and so, the question goes back to the

66:27

first question that you had at the very

66:29

beginning of the podcast, which is what

66:31

is the effect of low

66:33

energy output from the mitochondria.

66:34

Well, it depends on what tissue the

66:35

mitochondria is in. And so, if it's in

66:37

the eye,

66:40

then it's going to be a bit better

66:42

visual perception. If it's in the brain,

66:43

it's dementia. If it's You know, you see

66:45

You see what I'm saying? So, what we

66:47

start to see

66:48

is we start to see that a myriad of

66:51

different diseases are affected by the

66:53

sun.

66:55

I I challenge anyone to do this. If you

66:57

look at a publication in the United

67:00

States, I've seen it, where they map out

67:02

the amount of deaths

67:04

per in a calendar day.

67:07

Cardiac disease,

67:08

respiratory disease, kidney disease,

67:11

pneumonia,

67:13

all sorts of diseases, infectious

67:15

diseases, non-infectious diseases. You

67:17

will see a very clear pattern.

67:20

The maximum amount of deaths every year

67:23

occurs

67:24

within a month after the shortest day of

67:26

the year.

67:28

So, we're talking December, January.

67:32

We see the most amount of influenza

67:33

deaths at that time. We see the most

67:36

amount of cardiac deaths at that time.

67:39

We see the most amount of kidney deaths

67:41

at that time.

67:42

So, you might ask, well, that that's

67:44

because

67:45

that's because people get together at

67:47

Christmas time and they spread the germs

67:49

around more and we have Thanksgiving in

67:51

late November here in the United States

67:53

and and and and that's what's going on.

67:55

The problem is is if you look at

67:57

Australia,

67:58

which is on the other end. So, when is

68:00

their longest day of the year?

68:02

Their longest day of the year is in

68:05

December.

68:06

And that's when they have the least

68:07

amount of deaths, despite the fact that

68:09

they're all getting together for

68:10

Christmas in December. So, that doesn't

68:12

fly.

68:14

It's exactly the opposite. The most

68:16

amount of deaths occur in Australia, in

68:19

the Southern Hemisphere, in June to

68:21

July. That's their winter.

68:23

And so, what you see is deaths are

68:27

correlated to the length of the day.

68:29

This is the reason why whenever they

68:30

have to whenever they show you deaths in

68:33

the year, they always have to seasonally

68:35

adjust it. And the length of the day is

68:37

a proxy for the amount of sunlight.

68:39

>> Absolutely. You're much more you're much

68:41

more likely to get sunlight

68:44

on the longest day of the year than the

68:45

shortest day of the year, especially

68:46

when and and this is well known. There

68:49

there are some months, especially in

68:51

people who are doing shift work, like

68:53

7:00 a.m. to 7:00 p.m., there's

68:55

literally like December and January, you

68:56

will not see the sun because you are

68:58

going off to work before the the gets up

69:00

and you're you're coming home after the

69:02

sun is long set.

69:04

So, you're not you're not able to see

69:05

the sun, and so you could go literally

69:07

weeks

69:08

without seeing the sun at all.

69:11

Is there an optimal time of day to get

69:14

sunlight? Yes.

69:16

So, optimal time of day to get sunlight

69:18

would be for those that are concerned

69:20

about getting damage from ultraviolet

69:23

radiation. As we talked about, when the

69:26

sun is low in the sky, that's going to

69:29

be beneficial because the ultraviolet

69:31

cannot penetrate obliquely through the

69:33

atmosphere as well as long wavelength

69:35

radiation. So, when the sun is coming

69:37

up,

69:38

So, in the mornings? In the mornings,

69:39

and when the sun is going down in the

69:41

evenings. That's going to be the time

69:43

where you're going to get proportionally

69:44

more infrared light and

69:47

the least amount of ultraviolet light.

69:48

Now, when the sun is directly overhead

69:50

at noon, you're going to be getting the

69:52

most amount of infrared light at that

69:53

time, but you're also going to be

69:55

getting a lot of ultraviolet radiation.

69:56

And so, if you're not someone that's

69:58

gone out into the sun a lot, you may

70:00

want to avoid this period of time. Or,

70:03

as we talked about,

70:05

put on a broad-brimmed hat, put on

70:07

clothes. I mean, more clothes because

70:10

as we said, ultraviolet light does not

70:12

penetrate through clothes very well.

70:15

But, infrared light can. Does it matter

70:16

where the sun is hitting on my body?

70:18

Shouldn't. So, if I go outside and I'm

70:20

wearing a big hat, it's obviously going

70:22

to cover my eyes, my face, Yes. but

70:24

it'll be hitting my legs. For the

70:25

purposes of we're talking about with the

70:27

mitochondria, it will not matter.

70:28

However, if we're talking about

70:30

circadian rhythm, if we're talking about

70:33

getting circadian rhythm, that pathway

70:35

is through the eyes. So, you want to

70:37

maximize light through the eyes. Yeah,

70:39

so so this type of a light is called the

70:41

SAD light. So, your question had to do

70:43

with what part of the body does it need

70:44

to touch?

70:45

So, or need to be

70:47

touching. So, for the effect of the

70:49

mitochondria and and the metabolic

70:51

effects, it it should not matter, okay?

70:54

For this type of a light though, what

70:56

we're looking at is circadian rhythm.

70:58

And that's a that's a completely

71:01

different system that we're talking

71:02

about. That's not mitochondrial. That

71:04

has to do with the internal clock that's

71:06

in your brain that is regulating when

71:08

all of these things in your body

71:09

happens. And this light is about 10,000

71:13

lux. Lux is a way of measuring the

71:14

brightness of light. And what studies

71:17

have shown is that when you shine this

71:19

type of a light into your eyes, it had

71:22

it it's the way of of adjusting your

71:25

circadian rhythm. You don't have to If

71:26

you have a clock and it's not set to the

71:27

right time, there's a little thing at

71:28

the back that you can pull out and you

71:29

can change the time. Yeah. That pulling

71:32

out and and changing the time about when

71:34

things happen in your body is affected

71:36

most

71:37

by light. And light can actually shift

71:40

it one way or the other depending on

71:42

when you're shining that light. If

71:43

you're shining the light in the morning

71:45

time,

71:46

and this is what a lot of people do is

71:47

they'll use these these what they call

71:49

SAD lights. SAD stands for seasonal

71:51

affective disorder.

71:53

These lights, especially in the morning,

71:55

have a way of of not only setting your

71:58

circadian rhythm and making sure it's on

72:00

track, but also reducing depression.

72:04

There's a there's a portion of your

72:05

brain that receives light information.

72:07

It's called the perihabenular nucleus.

72:10

It's a long name, but it's it's it's

72:11

back there. And if it doesn't get

72:13

stimulated, it can cause depression.

72:16

And so so for people who live at high

72:18

latitudes,

72:19

uh further away, closer to the poles,

72:22

where the sun is getting up very late in

72:24

the morning, they're already off work

72:25

inside, this can actually be very

72:27

beneficial. So, what I would recommend

72:29

doing, you can pick these up pretty

72:30

cheaply on Amazon for about 20 bucks.

72:33

But they should generally be about 11 to

72:35

16 in from your face.

72:37

And uh what people should be getting is

72:39

about 3,000 lux hours.

72:42

Uh and what I mean by lux hours is you

72:44

multiply the lux times the amount of

72:47

hours that you're wearing it. So, 3,000

72:49

is where you ought to be. Because this

72:51

is 10,000 lux, you only have to look at

72:54

it for about a third of an hour or 20

72:56

minutes, and that should be enough. So,

72:58

is this a replacement for going outside?

73:00

It's a replacement for going outside

73:02

because of the fact that you're living

73:04

at a very high latitude and the sun is

73:05

not up, and because of the job that you

73:07

have,

73:08

it's going to uh it's going to have that

73:10

effect, but realize that this will not

73:12

replace the effect that the sun has on

73:14

your mitochondria. This is only to

73:16

affect the effect that lack of sunlight

73:19

has on depression. Okay. What if I'm

73:21

looking at the sun out of a window?

73:24

It depends on the window. So, you're not

73:26

still the window is going to be reducing

73:28

the amount of lux, so I would not

73:29

recommend if you can, I would not

73:31

recommend I would not say that staying

73:33

inside looking out the window is the

73:34

same as going outside. That's number

73:36

one. The other thing that you have to

73:38

understand is a lot of these windows,

73:39

especially if they're modern windows,

73:42

will be specifically designed to reduce

73:44

infrared light. I want you to give some

73:46

me any information you have yeah as it

73:48

relates to light health Yeah. that will

73:51

improve my life, things that I can act

73:52

actionably do tomorrow. Obviously, one

73:54

of them is that I'm going to go outside

73:55

and make sure I get some sunlight

73:56

ideally in the morning. Yes. We talked

73:58

about this sad lamp

74:00

for people especially that live in

74:02

certain countries which it which have

74:04

less sunlight

74:05

>> Correct. to set their circadian rhythm

74:06

and to help with things like mental

74:08

health. Is there anything else I should

74:09

be thinking about or can do or change?

74:11

Yes. So, just like we had in our

74:15

mnemonic of new start, rest and exercise

74:17

both at the same time and then yet the

74:19

short of opposite of each other, it's

74:21

important to have darkness.

74:24

Okay. It's important to have darkness,

74:25

and this is a real issue. This is one of

74:27

the biggest issues is the fact uh there

74:29

was a study that was published recently

74:31

and the title was dark days and bright

74:33

nights,

74:35

and that correlated with

74:37

uh increased mortality.

74:39

I mean, that's how most of us live.

74:40

That's the problem. We have dark days

74:42

and we have bright nights, and what we

74:43

really should be having is bright days

74:45

and dark nights. So, just as important

74:49

as it is to have bright sunlight and

74:51

getting outside in the middle of the

74:52

day, we also need to start working on

74:54

getting darker nights as well. And how

74:56

do we do that? Turning things off.

74:59

Uh getting the screens away from our

75:01

eyes. Um these are really important

75:03

because the screens have a lot of light

75:06

and the light what's going on here? This

75:08

is the reason why it's important.

75:09

There's two reasons actually. Is the

75:11

light that's going into our eyes is is

75:14

doing two things at night. Number one,

75:16

it is shutting down melatonin production

75:18

from the pineal gland. And as we just

75:20

talked about, melatonin's a very

75:21

powerful antioxidant that's very

75:23

beneficial.

75:25

The second thing that it's doing is it's

75:27

confusing your circadian rhythm. You

75:29

see, your circadian rhythm is designed

75:31

to see light as day.

75:33

If your If your eyes are seeing light,

75:36

your brain thinks it's the day. So, if

75:38

it's 10:00 at night and your eyes are

75:41

seeing light, your circadian rhythm is

75:43

saying, "I must have made a mistake. I

75:45

thought it was 10:00. It must not be

75:47

10:00 because look, there is light."

75:49

And so, what it's going to do is it's

75:51

going to adjust itself

75:53

and delay everything because it's

75:55

saying, "Well, it can't be 10:00 at

75:56

night. It must be 6:00."

75:59

And so, therefore, when you would

76:00

normally feel tired and sleepy at 10:00

76:04

at night,

76:06

after a number of days of doing this,

76:08

you're not going to feel sleepy until

76:09

1:00 in the morning.

76:11

These devices we have, they spit out a

76:13

lot of blue light, right?

76:14

>> Yes. Is there a way to like turn that

76:15

off? Yeah, actually a lot of these come

76:18

with uh with with tied to the clock

76:21

where after a certain time of night, it

76:23

will shift its its uh spectrum to a more

76:27

red spectrum. So, it's giving you less

76:30

blue light. The problem is is that while

76:33

the sensor in your eyes are tuned more

76:37

to blue light, it's not just blue light.

76:39

So, really the solution The best

76:41

solution is to turn off the lights.

76:43

The next best solution is to have more

76:45

of a red shift

76:47

or you know, put these glasses on at

76:49

night.

76:50

So, these are blue blockers. They're

76:52

trying to eliminate blue, but I'm I'm

76:55

still getting light in and that's enough

76:56

light to shut down melatonin production.

76:59

Even with those on?

77:00

>> Yeah. Absolutely. But you're telling me

77:02

these help? They're better than not

77:04

turning off the light.

77:05

And you're saying I still get light in

77:07

because there's light coming over the

77:08

top.

77:08

>> Even even that light there is still

77:10

going to bleed and it's going to bleed

77:13

into that part of the spectrum and cause

77:14

melatonin to be shut down. Yeah.

77:17

So, it's just the eyes are the sort of

77:19

the barometer for Correct.

77:20

>> what time of day it is.

77:21

>> Correct. And and the problem is is that

77:23

even when you close your lids,

77:25

light can still get through the lids.

77:27

What do you think of these sleep masks?

77:29

>> I think they're I think they're great

77:31

in terms of the fact that we now know

77:33

that closing your eyelids still can

77:37

allow some light to go in. So, if you're

77:39

in if you're sleeping in an environment

77:41

where, you know, light is out of your

77:43

control. If you're living in the city

77:45

and you can close your blinds but

77:46

there's still light that's coming in,

77:48

these things could actually be very

77:49

beneficial. I don't recommend

77:51

nightlights in bedrooms. You don't

77:54

recommend

77:54

>> not. I Even even clock radios or air

77:57

conditioners with LED displays on them.

78:00

That's that's just like total light

78:01

pollution to your bedroom. Your bedroom

78:03

should be as dark as possible. What if I

78:05

have those lamps that don't have blue

78:07

light in them?

78:08

Cuz I think my girlfriend puts some of

78:10

those by the bed. Yeah. Do they do

78:13

they still not great?

78:14

>> Again, the best thing is no light. The

78:16

second best thing is light with no blue

78:18

light in it and then the worst is, you

78:21

know, blue light.

78:23

What about candle light?

78:25

That's interesting.

78:26

Um there was a study that was done

78:29

where they compared someone reading at

78:31

night with a book with a light bulb

78:34

shining on it versus the LED, uh you

78:37

know, like a Kindle or whatever, okay?

78:39

What they found was that there was a lot

78:41

more light coming out of the Kindle than

78:43

there was just reading the book with the

78:44

with the lamp. And it and it delayed

78:48

sleep onset.

78:49

So Which delayed sleep onset? The

78:51

Kindle. The Kindle. Yeah. It delayed

78:53

sleep on enough to actually shut down

78:55

actually delay the circadian rhythm and

78:57

shut down melatonin production. So the

78:58

answer to your question is is candle

79:00

light's great. The only thing I'd be

79:01

concerned about is just the fire risk.

79:03

Yeah, cuz you fall asleep with that

79:05

thing.

79:05

>> Exactly. So the adjacent topic there was

79:07

vitamin D which we we touched on a

79:09

little bit. Do vitamin D supplements

79:11

work? Oh yeah, certainly. They do work.

79:13

I've tested them. Yeah, there's a number

79:15

of studies that have come out. Martineau

79:17

actually published in the British

79:19

Medical Journal. This is back

79:21

before 2020.

79:23

It was a meta-analysis of randomized

79:25

controlled trials showed that people who

79:26

supplement every day with vitamin D had

79:28

lower risks of acute chest syndrome.

79:31

The other one there was a recent study

79:33

that came out that showed that people

79:34

who supplemented with 2,000

79:36

international units a daily of vitamin D

79:39

had a lower risk of all cause autoimmune

79:42

conditions. We're talking rheumatoid

79:43

arthritis,

79:45

Crohn's disease, ulcerative colitis, you

79:48

name it.

79:49

That was that was a

79:51

study that came out that we actually

79:52

reviewed that on our on our Med Cram

79:54

channel. Cuz I've heard before in the

79:55

past that a lot of vitamin supplements

79:57

we take don't even get into our

79:59

bloodstream and into our bodies.

80:01

Yeah, so so vitamin D is very

80:03

interesting.

80:05

It is a supplement and it is a vitamin

80:07

but it's also a hormone.

80:09

Okay. It's it's it actually manipulates

80:12

um

80:14

DNA production. So it's it is quite

80:16

interesting. But these these are are

80:18

well described randomized controlled

80:19

trials. So if you're looking at the

80:21

autoimmune condition, this was actually

80:22

a study that was designed looking at

80:24

cardiac disease. They actually had two

80:27

arms, one with

80:28

omega fatty acids and and vitamin D and

80:31

they showed that in the vitamin D group,

80:34

there was a statistically reduction

80:37

statistically significant reduction in

80:40

autoimmune conditions.

80:42

I supplement with vitamin D. I just

80:44

Here's the

80:44

Here's the concern I have is if you are

80:47

going to supplement with vitamin D,

80:50

make sure that you get your levels

80:51

checked.

80:52

Why?

80:53

The reason is just because it is a

80:55

fat-soluble vitamin and it is possible

80:57

to take too much.

80:59

What happens if you take too much? It

81:01

can affect calcium metabolism and you

81:04

can have issues with calcium, too high

81:06

of levels of calcium.

81:08

It's very rare,

81:10

um but it can happen.

81:12

And I don't mean to say that in a sense

81:14

that I would I dissuade people from

81:15

supplementing because I think

81:17

supplementation can be good, but it at

81:19

some point you want to get a level

81:20

checked to see where you are. The other

81:22

reason is is because

81:23

based on your body habitus, based on

81:26

your skin color, because people with

81:28

darker skin, it's harder for them to

81:29

make their own vitamin D. They need to

81:31

be more time outside, especially if

81:33

they're at high latitudes. So, like me

81:35

living in the UK.

81:36

>> Exactly. I need to be outside more. It's

81:38

going to be harder for you to make as

81:40

much vitamin D as as somebody who for

81:43

instance if you were living at a lower

81:45

latitude or if you had lighter skin,

81:46

yeah. What is vitamin D doing in my

81:49

body? Ah, good question. Lots of things.

81:52

So, vitamin D if you were to look at the

81:54

the structure of vitamin D. Actually, I

81:56

actually did research on this

81:58

interestingly in college. I used to make

81:59

starting material for the graduate

82:01

students. It's It's a lipid-soluble

82:03

molecule and because it's lipid-soluble,

82:06

it's able to go right through into the

82:08

nucleus and actually go onto the DNA and

82:12

combine with proteins that actually

82:14

affects the transcription of your DNA.

82:17

So, in other words, depending on which

82:19

cell type we're talking about, it can

82:20

cause a lot of interesting changes. So,

82:23

it affects calcium metabolism. There's

82:25

vitamin D receptors on your immune

82:27

system.

82:29

So, uh it affects your immune system,

82:31

affects calcium metabolism, a whole host

82:33

of things. My team did some research and

82:35

found that approximately 1 billion

82:37

people globally have a vitamin D

82:39

deficiency. Not surprising.

82:41

>> 50% of the global population has

82:43

insufficient levels of vitamin D.

82:46

Absolutely.

82:48

Yeah.

82:49

Yeah, so the the issue is is that as the

82:51

world becomes more industrialized, as

82:54

the world becomes more well-off,

82:57

they're able to create dwellings and

82:59

they're able to air condition those

83:00

dwellings. And we, as human beings, tend

83:03

to avoid extremes. We don't like things

83:06

too hot, we don't like things too cold.

83:08

Well, I mean, let's face it, in our cars

83:09

we have something called climate

83:10

control. We can set the We can set the

83:13

temperature and that's what the

83:15

temperature is going to be. There is

83:16

this other implications which we can

83:17

talk about in terms of hydrotherapy,

83:19

perhaps, if we get to that. But, um the

83:22

issue is is we don't like those

83:24

extremes. We don't like going out into

83:25

the sun. And when we don't do that, we

83:28

we suffer the consequences. Is there a

83:30

way for me to get vitamin D without

83:32

supplementation and without going into

83:33

the sun? Yes. Yeah, it's in uh certain

83:35

foods as well. Mushrooms, for instance,

83:37

uh certain types of fish. They're um

83:40

they're they they have vitamin D in them

83:42

as well.

83:44

This is a strange question. But, do you

83:46

think our body knows

83:48

which foods we're deficient in?

83:51

And really what I'm saying there is

83:53

if I'm vitamin D deficient, do you think

83:55

there's a part of my body that knows

83:56

that I I need to eat mushrooms?

83:58

>> a good question.

83:58

>> makes me hungry for mushrooms. I don't

84:00

know about that particularly. I can say

84:02

this, though. In people who don't get

84:04

enough sleep,

84:06

we tend to have a predilection to eating

84:09

more carbohydrate-rich foods.

84:12

That one we do know. Okay. And and we

84:14

can and this is the reason why people

84:15

who

84:16

This is the reason why many scientists

84:18

believe that people who don't get enough

84:19

sleep tend to have food choices that

84:22

tend to put weight on.

84:24

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85:20

Of these cards that we have left in

85:21

front of us from the New Start

85:23

framework,

85:24

which one are you compelled to talk

85:25

about next? Water.

85:27

Water. Yeah. Okay, so tell me what you

85:30

mean by water, cuz people will think,

85:31

"Yeah, I drink enough water." Well,

85:33

first of all, we I don't think we do

85:34

drink enough water, but

85:36

I what what everyone talks about, you

85:38

know, the internal use of water, and it

85:40

makes sense.

85:41

But, as I was talking about before, the

85:43

the external use of water can actually

85:45

be very impressive. And it has to do

85:48

with uh body temperature, and it has to

85:50

do with the immune system.

85:51

So, we'll talk about water, but let's

85:53

sort of set the framework for that

85:54

conversation.

85:56

Your immune system is broken up into two

85:58

into two types. There's the innate

86:00

immune system and the adaptive.

86:02

We've become very familiar with the

86:04

adaptive immune system during COVID,

86:06

because all the talk was about

86:07

antibodies and antigens, and the fact

86:10

that SARS-CoV-2 was mutating, and would

86:13

the vaccines that made antibodies

86:15

against them be uh still functional. All

86:18

of that, where we have like where we

86:20

literally have a key with a keyhole that

86:23

it fits into and turns the lock and

86:24

these antibodies and they fit, that's

86:26

all the adaptive immune system. It's

86:28

very important, but it completely

86:31

eliminates or or removes from discussion

86:33

the innate immune system. The innate

86:35

immune system is really the body's first

86:38

defense.

86:39

And what's happening there is there's

86:41

these cells that are circulating, cells

86:43

like monocytes and natural killer cells

86:47

and a number of other other cells which

86:49

scour the body always looking for

86:51

something that looks

86:53

foreign to it. And it can tell based on

86:56

the molecular patterns of these of these

87:00

invaders that they're not supposed to be

87:02

there and they should be eaten up.

87:05

The major effector of this innate immune

87:08

system is something called interferon.

87:12

Interferon is a very

87:14

important molecule in the body and it is

87:18

effective it is so effective

87:21

at preventing viral infections that just

87:24

about every single viral infection that

87:27

plagues the human body today has a

87:30

defense mechanism against interferon. It

87:32

is it is a prerequisite. There's no

87:35

self-respecting

87:37

virus that can think it it can infect a

87:40

human body without dealing with the

87:42

issue of interferon.

87:45

Period. Think about interferon

87:48

as the security guard at the bank. And

87:50

if you are want to rob a bank, you've

87:52

got to have a plan for how you're going

87:54

to deal with the security guard.

87:55

Otherwise, you're not getting the money.

87:57

Does that make sense? Yeah.

87:58

So, there was actually an article that

88:00

was published a couple of years ago

88:02

where they talked about this this battle

88:04

between interferon and emerging viruses

88:07

and what viruses are doing to try to get

88:09

around interferon.

88:10

You may recall that back in 2002, we had

88:14

an outbreak of something called SARS,

88:16

which especially was pretty bad in

88:18

China, but also in Canada. The reason

88:20

why we were able to secure that outbreak

88:24

was because that everybody who came down

88:26

with SARS

88:28

developed a fever.

88:29

And so it was easy to tell who those

88:31

people were,

88:32

and and we were able to hospitalize and

88:35

isolate them.

88:36

The issue with SARS-CoV-2, and indeed

88:38

many infections like the common cold, is

88:41

that you don't necessarily get a fever.

88:44

And

88:45

fever is really important. And you're

88:47

like, what does this have to do with

88:48

water? We're going to talk about this.

88:50

Interferon

88:52

production goes up with temperature.

88:55

And in fact, the body's

88:57

fever mechanism is one of the ways that

89:02

it tells the body that it needs to

89:04

increase interferon to deal with the

89:06

viral infection. Is that why you feel

89:08

hot? You feel hot You may actually feel

89:10

cold. And the reason why you may feel

89:12

cold and even have chills is because the

89:15

way you feel is a product of what your

89:18

temperature is and what your thermostat

89:20

in your body is set to. So, if your body

89:22

and your if your body's thermostat is

89:25

saying, "Okay, here we are at 98.6

89:28

or I guess in in terms of Celsius, 37°

89:32

and you develop an infection, the body's

89:34

going to say, "Whoa, we have an

89:36

infection and we need to increase the

89:38

body temperature. We're going from 37°

89:40

or 98.6 up to 38° or or 100.4."

89:45

Because your actual body temperature is

89:47

below where your body wants it to be,

89:49

you're going to feel cold. You're going

89:51

to shiver to try to increase that

89:52

temperature, so you go up with that.

89:55

Now, once the fever is done and the

89:56

infection is done

89:58

and it comes down, you're going to have

89:59

you're going to break a sweat. So,

90:01

that's that's why when someone Oh, he's

90:03

sweating. That means the fever is

90:04

breaking. That means your temperature is

90:05

coming down. So, you're typically you'll

90:07

feel cold, you'll feel like you're

90:09

shivering, you'll want to get into bed

90:11

and and and put the covers on, and

90:13

that's when your temperature goes up.

90:14

That and and that's for a reason.

90:16

Because what happens when the

90:17

temperature goes up in your body is that

90:20

creates an environment where the virus

90:22

that can't replicate very well. All

90:24

viruses really cannot replicate very

90:27

well at high temperatures, including

90:28

SARS-CoV-2. It's also a signal

90:32

to your body to produce more interferon.

90:35

So, there was a study that was published

90:37

last year where they looked in mice,

90:40

which by the way have the same body

90:41

temperature as we do, and they found

90:43

that there was like five different

90:45

regulatory proteins, all of which led to

90:48

one endpoint, and that was to produce

90:50

this thing called interferon.

90:52

All of them jumped in production when

90:54

your body went from

90:56

30

90:58

7° to 38°. That's basically right below

91:03

a fever,

91:04

right? So,

91:06

the point the take-home point that I got

91:07

from all of that was that we should not

91:10

really be treating fevers unless they're

91:12

so high that there's other complications

91:14

that could occur like, you know, uh

91:17

racing heart rates or or um or having

91:20

seizures. But, we do this all the time.

91:22

We treat fevers because it feel makes us

91:25

feel bad, and we think that by treating

91:27

the fever we'll feel better, but what

91:28

we're actually doing is we're we're

91:30

cutting the legs out from our immune

91:31

system because part of the immune system

91:34

response is to generate a fever, and the

91:36

and the fever generates interferon. Now,

91:38

I I don't want to overstate this, but

91:41

let's compare the innate immune system

91:43

to the adaptive immune system. The

91:45

adaptive immune system is pretty

91:47

specific for a particular variant of a

91:49

virus. And for a virus that mutates very

91:52

rapidly like SARS-CoV-2,

91:54

the immunization may be very good in

91:56

terms of binding,

91:58

but if that

91:59

that virus mutates,

92:01

that binding is going to be affected in

92:04

some way. It may not affect

92:05

hospitalization, but maybe in terms of

92:07

of preventing infection. Do you know you

92:08

understand what I'm saying? So, the

92:10

different variants, we had the alpha

92:11

variant, then we had the the delta

92:13

variant, then we had omicron, etc.

92:17

Those

92:18

are material changes for the adaptive

92:20

immune system.

92:22

For the innate immune system, for

92:23

interferon,

92:25

it doesn't matter.

92:26

Interferon is just as effective against

92:28

alpha as it was for delta as it would be

92:30

for omicron. So, so let's set this up

92:32

again.

92:34

Water.

92:36

We said that water has a very high

92:39

specific heat, which means that if I

92:41

apply hot water

92:44

onto the human body, it's able to

92:46

transfer heat. This is why people get

92:48

burned with boiling water.

92:50

We don't obviously want to burn anybody,

92:51

but if we're able to put them into a

92:53

sauna, if we're able to put them into a

92:54

spa, if we're able to use hot towels and

92:57

apply to the human body to heat up their

92:59

body to cause a sweat,

93:01

in other words, if we're able to induce

93:03

artificial fevers in patients who have

93:05

these infections,

93:07

there seems to be evidence that the

93:10

interferon response will be will be

93:12

better. Uh there was a study that was

93:14

done looking at lymphocytes and taking

93:16

them out of the human body and at

93:17

different temperatures, once it hit

93:19

about 38, 39°, there was off-the-charts

93:23

uh 10 10-fold increase in interferon,

93:26

which is exactly what you would want to

93:28

have. Now, how do I know that interferon

93:31

levels are so important in things like

93:32

COVID-19? Well, there was a study that

93:34

was done that showed that high levels of

93:36

interferon

93:38

correlated with more mild

93:42

uh

93:42

uh SARS-CoV-2 infections and that people

93:44

that had low interferon levels had very

93:47

severe COVID-19 infections. So, you're

93:50

suggesting that we should

93:52

be in the sauna more often? Yes, and

93:54

it's based on data

93:56

that is

93:58

has been well documented in in the

94:00

Finnish sauna

94:03

realm.

94:04

So, people who use sauna

94:07

four, five, six, seven times a week are

94:10

more likely to have less

94:12

uh death from cardiovascular disease

94:14

than people who use sauna once a week.

94:17

And and in Finland, once a week is kind

94:19

of the standard. And why do they say to

94:21

do hot and cold therapy together?

94:24

So, they would

94:26

I would argue that the reason why it has

94:29

been argued to do this, and this goes

94:30

back to uh a number of papers that have

94:32

been written back over 100 years ago, is

94:35

what you're doing when you're doing hot

94:36

for a long period of time, let's say 20

94:38

minutes in the sauna. And what you're

94:40

doing is you're heating up the body,

94:42

and uh and and the whole purpose of that

94:44

is to increase the body temperature.

94:46

What the What the cold at the end does

94:49

is it does two things, they believe. The

94:51

first thing that it does is it causes

94:53

vasoconstriction. So, you put a brief

94:55

amount of cold onto the body, it's going

94:58

to cause vasoconstriction superficially,

95:00

so that when you're done,

95:03

you're not going to lose as much heat

95:05

through those blood vessels. And so,

95:07

you're going to keep the core body

95:08

temperature higher for longer, which is

95:10

exactly what you want to do. The other

95:12

thing that cold water does, again, is

95:14

the vasoconstriction. When you It's well

95:16

known that when you take a cold shower,

95:18

your blood vessels constrict, and when

95:20

when you look at a blood vessel on end

95:23

in in a person who's living and

95:25

circulating, there are a number of white

95:27

blood cells that are latched on to the

95:29

inside surface of that blood vessel.

95:32

When that blood vessel contracts, a lot

95:34

of those white blood cells that were

95:36

stuck get kicked off into circulation,

95:39

and they go off, and they do whatever it

95:40

is that they're going to do. It's called

95:41

demargination. So, two things for cold

95:44

right at the end. Doesn't have to be

95:45

very long, maybe just a minute.

95:47

It causes uh actually to keep your body

95:49

temperature higher for longer,

95:51

ironically, and number two,

95:53

demargination.

95:55

So, that's water, which is the W. Um

95:59

of these,

96:01

which one do you want to pick next?

96:02

Which one do you find most compelling?

96:05

Let's talk about air.

96:06

Uh real briefly. So, we said that air is

96:11

not just the lack of toxins, but

96:13

actually benefits. So, first of all, we

96:15

want to have good oxygen. We want to get

96:17

rid of carbon dioxide, especially in

96:19

buildings when there's no ventilation.

96:21

That's not good. But, there's been

96:23

actually a number of studies looking at

96:24

plants and trees and the fact that they

96:27

can give off things like phytoncides.

96:29

What's that? These are are aromatic

96:31

compounds that the tree actually gives

96:34

off. And when we look to see the effect

96:38

of these compounds on the human body,

96:40

they're actually very beneficial. They

96:41

interact with our immune system and

96:43

elevate our immune system and actually

96:44

can make us more relaxed. There's a

96:46

There's a lot of data in the Japanese

96:48

literature on on this in the what they

96:50

call the hinoki cypress forests, where

96:53

they looked at um these CEOs.

96:57

There's a podcast about CEOs. There's

96:59

these CEOs in Japan, and they took them

97:01

from their their jobs and basically took

97:04

them up into the mountains of the hinoki

97:06

cypress and had them walk around, took

97:08

blood tests, and they found that the

97:10

natural killer cells, which are so

97:12

important in terms of immunity, were not

97:14

only increased in number, but they were

97:17

also the the um

97:19

the the enzymes within them that break

97:21

down diseases or viruses were also

97:24

increased.

97:26

So, when they brought them back down to

97:29

uh

97:29

the city in Japan, they put them up in

97:32

hotels, and they infused

97:34

some of these uh these chemicals, these

97:37

um naturally produced uh phytoncides,

97:40

they're called, and they had almost

97:42

exactly the same effects in in these uh

97:45

in these uh subjects.

97:47

So, you think plants and being out in

97:48

nature could actually be giving us much

97:50

more than just clean air. It's giving us

97:52

chemicals which help us fight disease.

97:54

>> Absolutely. So, so again, here's this

97:56

dichotomy, inside versus outside. What

97:58

do you get when you're outside? We've

98:00

already talked about exercise. We've

98:02

already talked about uh sunlight. And

98:05

now we're adding to it fresh air. Not

98:08

just the fact that you have uh

98:10

low pollutants, which is certainly very

98:12

important, but the fact that when you're

98:14

around green plants, when you're around

98:15

green trees, there could actually be a

98:17

benefit. By the way, the benefit that

98:19

they found lasted for about 7 days. So,

98:23

just going out one day a week um can

98:26

actually have that benefit.

98:28

I think a lot about carbon dioxide at

98:30

you cuz obviously cuz I I spend a lot of

98:31

time sat in the studio recording and um

98:34

this is our big LA studio, but uh

98:36

in the UK, it started in a really small

98:39

room and there wasn't air conditioning.

98:41

And obviously I sit here sometimes for

98:42

several hours with a guest and we're

98:44

recycling CO2 at that point. And then I

98:47

read a couple of studies that showed the

98:48

impact that would have on my cognitive

98:50

performance and

98:51

That's all true. We actually had on our

98:53

on our channel uh with uh Meg Cran, we

98:55

had Dr. Joseph Allen out of the Harvard

98:58

uh public school of health.

99:00

Um and he he showed us. I mean, he

99:02

literally uh had the the CO2 meter and

99:05

just by cracking the window uh just a

99:07

little bit allowed carbon dioxide to

99:10

escape and brought down those carbon

99:11

dioxide levels. So, very important,

99:13

absolutely.

99:14

And for people that work in offices or

99:16

you know, travel in hotel rooms or are

99:18

inside a lot, what should they be

99:19

thinking about and how can they go about

99:21

making sure that the air quality is

99:22

optimal?

99:24

Well, uh

99:25

the surrogate for that is carbon dioxide

99:27

as we mentioned. So, cracking open a

99:29

window if they're able to, if there's a

99:30

door that they can uh open up safely

99:32

without, you know, compromising

99:33

security. These are all things that uh

99:36

would be very very beneficial. Even

99:38

rolling down the window uh in in uh

99:41

in in car and and maybe taking making

99:43

sure that we're taking the uh that

99:45

recirculation button off when we're when

99:47

we're driving. I got friends that won't

99:49

won't stay in certain hotel rooms unless

99:50

the window opens cuz you know in a lot

99:52

of hotel rooms, especially ones that are

99:53

high up, you can't open the windows. And

99:55

there's also a bunch of devices that we

99:57

um

99:58

in our UK studio, which is smaller,

100:00

we found on Amazon for a you know, not

100:02

super expensive, that we just sometimes

100:04

put on the floor in the studio just to

100:05

see Yeah. how we're doing. I'll link

100:07

some of that stuff on screen if anyone's

100:08

interested in getting seeing what the uh

100:10

CO2 are in whatever room you're working

100:12

in. Yeah. What is uh what's next on your

100:14

list then here?

100:15

>> Well, we've talked about I mean,

100:16

exercise, nutrition, temperance, these

100:18

are things that a lot of people talk

100:19

about. Not many people talk about trust.

100:22

When you say trust, you really mean

100:23

religious faith and

100:25

>> Religious faith uh

100:26

something that would give you a a way of

100:30

of dealing with uh stress and and

100:32

anxiety. That's really where this comes

100:34

down. And there's been actually a number

100:35

of studies that have looked at that. So,

100:38

yeah, basically the Bible. Um or it

100:40

doesn't have to be the Bible as well.

100:42

There's there's other faith um

100:44

denominations that that look into this

100:47

as well. Number of studies that have

100:48

looked at trust in God and how that

100:52

relates

100:53

to anxiety. So, a number of studies have

100:55

shown that people who have a good

100:58

faith and trust in a God that is uh or

101:01

or in a religion that is supportive and

101:03

not non-supportive, they have less

101:06

anxiety, less depression, have a faith

101:08

community that they can uh engage with

101:11

and and be supportive.

101:12

Um and I think that that's that the

101:14

literature is uh where where as you have

101:17

it this the science behind that is not

101:19

as strict as it would be for like a

101:21

randomized placebo control trial.

101:23

There's a lot of associations that you

101:24

have to say here. Um that it certainly

101:26

is one of those pillars that I believe

101:28

helps with all of the those links. What

101:30

do you think's going on there? So, you

101:31

were telling me that from what the

101:33

literature's saying, people

101:35

>> who have a faith in a God Yes.

101:39

are insulated from depression and

101:41

anxiety in some interesting way. Yeah,

101:44

that that's a question that a lot of

101:45

people have tried to answer and and they

101:47

believe that it comes down to if you

101:49

have a trust in in a God that is looking

101:52

out for you

101:53

and is there on your side uh then that

101:57

type of relationship does lead is

101:59

associate let's say I shouldn't say does

102:02

lead to because that implies causation.

102:03

Let's say it's associated with

102:06

a reduction in depression, a reduction

102:08

in anxiety particularly.

102:10

Um there's there's an there are some uh

102:12

studies that have been done particularly

102:14

in uh in Christianity where there was a

102:17

study that was published. This is Krauss

102:19

and uh out of I believe University of

102:22

Texas where he did a survey and he asked

102:25

people

102:26

how they forgive.

102:28

And he he divi- he basically divided

102:31

them to two different groups. There were

102:33

people that would forgive give

102:35

conditionally and people that would

102:36

forgive unconditionally. Let me put it

102:38

into practical terms. Someone does

102:39

something to you.

102:40

And you say oh that's okay, I forgive

102:42

you.

102:43

The question is would you forgive that

102:45

There's some people that would only

102:46

forgive if that person came back and and

102:49

you know, did some sort of act of

102:50

contrition. Like okay, I'll forgive that

102:52

person they came back and apologized or

102:55

I'll forgive that person they came back

102:56

and they did you know, whatever it is.

102:58

That would be considered conditional

102:59

forgiveness.

103:01

The other type is unconditional

103:03

forgiveness. So in other words, someone

103:04

does something to you, you don't see

103:06

them again. Or or they've never

103:08

expressed any kind of you know, being

103:10

apologetic for what they did. They still

103:13

get forgiven. So that's unconditional

103:15

forgiveness.

103:16

What they found in the study when they

103:17

divided that is that the people

103:20

that forgave unconditionally

103:23

had less

103:26

depression.

103:27

They had less feelings of inadequacy.

103:29

They had less anxiety regarding uh end

103:32

of life. They had all They had all of

103:34

these They had more The people that

103:36

forgave conditionally

103:38

had more somatization of depression.

103:41

So, these were real medical

103:44

you know, things that they could

103:44

actually diagnose with surveys and and

103:47

and tests that are well validated. And

103:49

and what what would decide between these

103:51

two was how they forgave.

103:54

So,

103:55

they were puzzled by this. They said,

103:56

"Well, what Well, what then what

103:57

determines whether or not someone is

103:59

going to forgive conditionally versus

104:00

unconditionally?" So, they looked at a

104:02

bunch of factors and none of them stood

104:04

out except for one. And and the odds

104:06

ratio on this was like 2.5.

104:09

And and it boiled down to this one

104:10

question.

104:11

Do you believe

104:13

that God has forgiven you?

104:16

That was That was the major thing. If if

104:19

somebody believed that the God that they

104:21

had faith in had forgiven them, they

104:24

were two and a half times more likely

104:26

to to forgive somebody unconditionally.

104:29

Which then meant Which then was

104:31

associated with all of these other

104:33

things being low, like low less

104:35

depression, less anxiety.

104:38

So,

104:39

to me that that's an that's that's

104:40

fascinating that that in their minds

104:44

this is what's actually happening. And

104:46

so, there have been randomized

104:47

controlled trials where they have When

104:49

people are doing therapy, like let's you

104:51

say have you have anxiety. There is

104:53

cognitive behavioral therapy that we can

104:54

do for those people.

104:57

But, what has been shown in a randomized

104:59

placebo controlled fashion is that if

105:01

somebody is of a faith

105:04

and you inject into that cognitive

105:06

behavioral therapy aspects of that

105:09

faith,

105:10

the cognitive behavioral therapy is even

105:12

more effective.

105:16

So,

105:17

I guess I should preface this by saying

105:20

I don't believe that any of this stuff

105:23

should be placed on people without their

105:25

permission. So,

105:27

I'm working I work in a healthcare

105:28

environment. So,

105:30

do I go and pray for people

105:32

who don't believe? No. This is something

105:35

that that always has to be done

105:38

it has to be asked permission.

105:39

Do you think

105:40

>> Yeah.

105:41

people who believe in God are

105:44

healthier generally?

105:46

All other factors It would seem the data

105:48

would indicate that

105:51

people who

105:52

have a healthy relationship with their

105:54

church, who have a healthy relationship

105:56

in God, are associated with less

105:59

disease. Cuz from a causation point of

106:01

view, you could say well Causation,

106:02

yeah. So, this this is what we don't we

106:04

They probably have more friends. They

106:05

probably have Yeah. So, the question is

106:07

is whether or not people who are

106:11

healthier and have more friends are more

106:13

likely to be religious or is it the

106:16

other way around? And and sometimes it's

106:18

difficult to tell those things.

106:20

But I imagine there's a great calming

106:22

force that comes from believing in a

106:23

higher power.

106:24

Absolutely. And and the other thing that

106:26

the other thing that's interesting about

106:27

all of these like new start stuff is

106:29

when you look at other particular

106:31

religions,

106:33

how a lot of these things are actually

106:35

incorporate in this. Like for instance,

106:37

the Hindus are very famous for getting

106:39

up in the morning and welcoming the sun.

106:41

And we just talked about the benefits of

106:43

of sunlight. We didn't talk too much

106:45

about nutrition, but fasting is is an

106:46

important part of that and and and

106:48

Muslims are are obviously um

106:51

part of their religion is actually doing

106:52

fasting during Ramadan. So, religions

106:55

have a hot and cold practice as well,

106:56

don't they? Yes, yeah. At least through

106:58

history. And on the flip side of that, I

107:00

would say that it's also shown that if

107:02

you have a unhealthy relationship, like

107:04

if you have if you believe in a God who

107:06

is vindictive or who's out to get you or

107:08

who's going to do something to you

107:10

unless you do something else, that has

107:12

also been shown to be negatively

107:14

impactful.

107:16

So, it depends on the relationship that

107:18

you've got. What do you see in your

107:20

practice? Because you said something

107:21

earlier that you you're often there at

107:23

the end of people's lives. Yeah. Uh some

107:25

unfortunately sometimes I'm the last

107:27

person they see.

107:29

And and it's

107:32

you start to realize that you cannot I

107:34

mean that death is inevitable.

107:36

And all we do in medicine is delay the

107:39

inevitable.

107:40

So, what we try to do

107:43

and um and and I have a colleague uh

107:45

who's very philosophical about this is

107:48

we try to make sure that when these

107:50

things happen that they happen with

107:51

dignity and we celebrate the person's

107:53

life and and making sure that it's done

107:56

in the way that they would want to have

107:57

it done. What do people say as they're

108:00

about to die?

108:02

People become very

108:03

it's very different uh for for different

108:05

people, but uh they can become very

108:07

circumspect and and uh

108:10

I've seen such such contrasts. People

108:12

are ready to go. People feel like

108:14

they've they've done what they've come

108:16

to do. And uh they don't want anything

108:19

further to do. Like we're we're there to

108:21

to to to to to delay death, right? We're

108:23

there to put them on a ventilator or to

108:25

give them this medication. And you would

108:27

be surprised people who are look, you

108:31

know, relatively healthy, but and

108:32

something is happening that we could

108:33

easily correct. They're like, "No, I I

108:36

I don't want that. I I I choose not to

108:38

have that." And we have to respect

108:39

obviously what they

108:41

what they choose. Obviously, we have to

108:42

educate them to make sure that they're

108:44

making the right choice, but once once

108:46

they have given all the information

108:47

ultimately they're the one that makes

108:49

the decision. So, you see people choose

108:51

death.

108:52

When we can intervene in artificial

108:55

ways, they would rather not have that

108:57

and they would choose death, yes. Are

108:58

there any particular cases that changed

109:00

you?

109:01

Yeah.

109:02

Yeah, there's there's there's a case

109:03

that changed me. But not in the way that

109:06

we've just been talking about where it

109:07

was horrible. This is this is actually a

109:09

miracle.

109:10

I've actually seen a miracle happen. And

109:12

for me it happened early in my training.

109:15

So, it it it made me think twice about

109:18

being a prognosticating physician. A

109:20

prognosticating physician?

109:21

>> Yeah, saying oh

109:22

you're never going to walk again or or

109:24

you've only got 2 years to live. I I I

109:26

must have missed that day in med school.

109:28

I just didn't show up that day. This was

109:30

a young guy. He was he had he had

109:33

testicular cancer.

109:35

And he went to the operation. The

109:37

testicular cancer surgery was a success.

109:39

Unfortunately, during the operation

109:42

something happened. He didn't get enough

109:43

oxygen to the brain and he came out of

109:45

the operation with with anoxic injury of

109:48

the brain.

109:49

This guy must have been in his 20s. And

109:52

he had a young wife.

109:54

Um and uh

109:57

I had come onto the rotation as a as a

109:59

as a as a resident. And uh

110:02

we had attendings and you have to

110:03

realize in medicine you have attendings

110:05

above you and what they say is just, you

110:07

know, that's that's the word. That's

110:08

what happens and and the ICU attending

110:10

we were the ones that were sort of

110:11

taking care of the patient cuz he was on

110:13

a ventilator but there was the neuro

110:14

neurologist who looked at everything and

110:16

says, "Look, this guy is not waking up.

110:18

He has severe anoxic brain injury. We've

110:21

looked at the scans. This is what's

110:22

going to happen."

110:24

And so um

110:28

everyday we'd round on this guy and he

110:30

was just he was just a shaking mess. He

110:32

was just there and he was just kind of

110:33

shaking. His eyes were rolling. No

110:34

response. Nothing.

110:36

And everyday his wife would come in.

110:38

Young wife. And she uh

110:42

she just didn't believe that this guy

110:44

was going to be like this for the rest

110:45

of his life. He was going to wake up

110:46

eventually and so she he would she would

110:48

be at his bedside like attending to him

110:51

and making sure this that and the other.

110:53

And even asking us to put, you know,

110:55

some special concoction that she made at

110:57

home into his tube feeding so that this

110:59

could go and help him make him better.

111:01

And we would we would go along with her

111:03

but I was looking into my attendings and

111:04

they're like, "She's she does she

111:05

doesn't understand what's going on. She

111:07

doesn't understand that he's never going

111:08

to wake up."

111:10

So, this is this is what I'm seeing. Uh

111:12

one day she came in and she's and she

111:14

just had this

111:16

smile on her face. She was just uh so at

111:19

peace and a smile. And uh they they were

111:22

Hispanic couple, so we had to ask a

111:23

translator what was going on. And she

111:25

told us, she said, um

111:28

I had a dream last night.

111:30

I had a dream that he was going to come

111:32

home.

111:33

And she was convinced.

111:36

Just absolutely happy, beaming. And

111:38

we're like, man, this lady

111:40

is is crazy. She doesn't understand

111:42

what's going on.

111:44

Well,

111:45

days went on, weeks went on, and uh

111:48

one day uh and and where I was rounding,

111:50

it was it's just in this round nursing

111:52

The nursing station's in the middle, and

111:54

the doors to the rooms are all around in

111:55

a periphery.

111:57

And we were going around the circle

111:58

rounding on the patients, and I could

111:59

look in, and I saw him, and he was kind

112:02

of shaking, but he was kind of opening

112:03

his eyes.

112:04

And I said,

112:06

he seems to be focusing a little bit.

112:08

His eyes seem to be focusing a little

112:09

bit more than they would be.

112:11

And he kind of I I just said, I just

112:14

kind of put my hand up like this, and I

112:15

just kind of

112:16

and sure enough,

112:18

he put his hand up like this shaking.

112:20

And he put it back down again. I'm like,

112:22

what?

112:23

>> at you. Yeah.

112:24

What?

112:26

And I said,

112:27

let's go check this out again.

112:30

Long story short,

112:31

took months,

112:34

but that guy walked out of that

112:35

hospital.

112:37

6 months later, he and his wife came

112:40

back.

112:41

Walked onto the unit like like there was

112:43

nothing wrong with the guy.

112:44

And he gave us a big basket of flowers

112:47

to thank that unit for what they had

112:49

done for him. In my mind, I knew

112:52

that for most of that staff, for most of

112:54

that time,

112:56

they the people were just going through

112:57

the motions

112:58

keeping him alive, cuz that's what she

112:59

wanted.

113:01

Obviously, when when there were signs

113:02

that this guy was recovering, it was it

113:04

was complete shift. People were amazed.

113:06

And so, what that did to me in my career

113:11

was it made me think twice.

113:13

Like,

113:15

what why did this guy get better? Now,

113:17

he was 22, he was young. And And

113:19

typically, if something like that is

113:21

going to happen, it's going to happen in

113:22

someone who's very young, whose mind is

113:24

plastic, who can who can survive that

113:26

type of a situation.

113:29

But it really

113:30

it was a miracle.

113:32

I can't I can't say anything else. I

113:33

mean, it's not something that we would

113:34

know. All of the experts said that this

113:36

wasn't going to happen, but it happened.

113:38

What do you think happened?

113:40

I think he had a loving wife

113:42

who believed in him.

113:44

And something happened outside of the

113:46

physical and the mental.

113:48

Maybe the spiritual. I don't know.

113:52

It happens. It's very rare. And when it

113:55

does happen, it happens in young people.

113:56

That would be what the medical part of

113:58

my brain would say.

113:59

But the other part of my brain says,

114:01

"You know what? I only know about 10%,

114:04

maybe 5% of the world's knowl- No.

114:06

World's knowledge? 1% of the world's

114:08

knowledge. Maybe I have 5 to 10% of all

114:10

of the medical knowledge in this world.

114:12

And I would say probably the explanation

114:14

is somewhere in that other 80 to 90%

114:17

that I just don't know.

114:19

I think what it taught me was is that we

114:21

have to be humble

114:22

about what it is that we know.

114:24

There's things that we know we know, and

114:26

there's things that we don't know

114:28

that we don't know.

114:31

We talked a little bit about this

114:32

chemical earlier on, melatonin. Yeah. I

114:34

just wanted to close this off because I

114:35

had a question on it. Um a lot of people

114:37

take melatonin supplements

114:39

at night time to help them sleep. Yes.

114:42

Good, bad, and different?

114:44

I think it's good in certain situations.

114:46

So, if you're having difficulty falling

114:48

asleep, a little tiny dose of melatonin,

114:50

no more than 5 mg, can be actually very

114:53

beneficial. If you're wanting to shift

114:55

your circadian rhythm back instead of it

114:57

being pushed back, but you want it to be

114:59

advanced forward, melatonin can be very

115:02

beneficial. It's very beneficial for jet

115:04

lag. It's also beneficial for a few

115:06

sleep diseases, but I would not

115:08

recommend routinely for no other reason

115:10

taking large doses of melatonin. What's

115:12

the trade-off?

115:14

You said earlier on that everything has

115:15

side effects, right? And it impacts

115:16

another part of So, taking high doses of

115:18

melatonin can actually make you more

115:20

irritable.

115:21

And have irritable? Irritable, yeah.

115:23

>> In what regard? Just mentally irritable.

115:26

Yeah. What does that look like?

115:28

Things set you off,

115:30

um eat more easily. So, like a a mood

115:32

disorder?

115:32

>> Yeah, absolutely. Anything else with

115:34

melatonin that you're aware of?

115:36

Not that we have studies for. People

115:38

have concerns that sometimes taking too

115:40

much melatonin may actually affect the

115:44

melatonin secretion from the pineal

115:45

gland itself. I don't have evidence of

115:47

that as yet to see if that if that's

115:49

actually the case.

115:51

Dr. Rogers, is there anything else that

115:53

we haven't discussed that we should have

115:54

discussed?

115:56

We've discussed a lot.

115:58

Um

115:58

I think I think putting it all together

116:01

is

116:02

again, the links.

116:05

And if we have those links,

116:08

medications have their place, but the

116:10

way that they work is by breaking down

116:13

other parts of the chain to strengthen

116:15

the weak chain. That can have an effect,

116:17

especially at the end of life if you

116:19

want to sustain life. But if you're

116:21

interested in longevity, if you're

116:23

interested in making and living the best

116:25

life,

116:26

then you want to strengthen all of those

116:28

chains. And I believe the key to doing

116:30

that is something called NewStart.

116:33

We have a closing tradition on this

116:34

podcast where the last guest leaves a

116:36

question for the next guest not knowing

116:37

who they're leaving it for. And the

116:38

question that was left for you is what

116:41

is the area of your focus that you are

116:43

most dying to talk about which you are

116:46

almost never asked about?

116:50

This book, the Bible.

116:52

Uh which is my uh which is my tradition.

116:57

Evidence for

117:01

science

117:02

in the Bible.

117:04

What do you mean?

117:07

This we we what we've been talking about

117:09

is

117:11

the body

117:12

and the health of the body.

117:14

Um I'd like to put to to the test some

117:17

of the statements in the Bible to see if

117:19

they work scientifically.

117:21

Like turning water into wine?

117:23

No, well, potentially.

117:25

Uh that's a miracle.

117:26

What I was referring to is is this. And

117:29

this is what I've actually done and it's

117:31

actually quite interesting.

117:32

Is uh you know, Paul, who is one of the

117:35

New Testament writers in the Bible,

117:37

wrote to the Corinthians, "Don't you

117:39

don't you understand that your body

117:42

is the temple of the Holy Spirit?"

117:46

I said, "That's that's a really

117:47

interesting statement."

117:50

How would he come to that conclusion?

117:51

Like what does that mean, the temple of

117:53

the Holy Spirit? So,

117:55

the only temple at that time was the

117:57

temple in Jerusalem where they would

117:59

have the sacrifices and things. So, what

118:01

I did was I went back and this is and

118:04

this is answering the question is

118:06

I'm looking for evidence of scientific

118:09

truth

118:10

probably unknowingly

118:12

by some of the writers in the Bible to

118:14

see whether or not there is truth. Does

118:16

Does that make sense what I'm saying?

118:18

So,

118:19

if you look and most of Exodus 25

118:22

through 30

118:23

is this painstakingly detailed

118:26

description of the sanctuary that Moses

118:29

built in the wilderness that he

118:30

supposedly he got from God. This is what

118:32

he says.

118:33

So, here's a great way to see whether or

118:35

not this this all flashes out. Moses is

118:38

saying, "Here's the description of the

118:39

pattern that I got for the temple."

118:41

And Paul is saying,

118:43

"Your body is a temple." So, here's my

118:46

hypothesis.

118:47

If we look at the pattern in the temple,

118:50

should it match

118:52

the human body that they had no

118:54

understanding of at the time that Paul

118:56

wrote this. I We didn't know about cells

118:58

until Van Leeuwenhoek in the 1600s. We

119:00

didn't know about the circulation of the

119:02

heart

119:03

until the 1600s with um with

119:07

William Harvey, okay?

119:09

Yet,

119:11

if you look in the human body, you have

119:14

the blood system. You have blood

119:15

circulating around in the vascular

119:16

system. And then it And then it goes

119:18

into the interstitial fluid.

119:21

And then the interstitial fluid then

119:23

goes to the cell, which is There's a

119:25

There's a There's a plasma membrane on

119:27

the cell that you can't penetrate

119:28

through, but unless you actually have

119:29

the proteins to go. And then it goes

119:31

into the cell, which is a compartment

119:34

with two compartments within it, right?

119:35

You got the cell and the nucleus, right?

119:38

This is exactly the same structure that

119:41

Moses was given in in the wilderness.

119:43

And And by the way, uh Hindu temples are

119:45

actually similarly designed. They're

119:47

sort of this three-part situation. So,

119:50

you have this altar of sacrifice,

119:53

which is where the blood is.

119:55

That's That's the blood in the human

119:57

body. Next, you move to the laver,

120:00

which is this

120:01

container full of water.

120:03

And that's the interstitial space after

120:05

you go from the blood into the

120:06

interstitial space. Any pharmacologist

120:08

will know that this is the exactly the

120:10

pattern that you move to. The next thing

120:12

that comes is this structure

120:14

that has a veil that you can't penetrate

120:16

through unless you you go through it.

120:19

That's the cell cuz this this structure

120:21

is the building and it's got a room

120:23

within a room.

120:25

And that's exactly what the cell is. The

120:26

cell is this nucleus surrounded by the

120:28

cytoplasm. Well, in this room that you

120:31

go into it first,

120:33

there's pieces of furniture in there

120:34

that are very similar to the types of

120:37

organelles that you see in the in the in

120:40

the cytoplasm of the cell. For instance,

120:42

there's this seven-branch candlestick

120:44

that's in there that's burning olive oil

120:46

and producing energy. That's like beta

120:48

oxidation producing energy. That's

120:49

exactly what you see in the

120:50

mitochondria. But the final thing is you

120:53

move into the nucleus. And this temple

120:55

has something called the most holy place

120:58

where there's this altar of

121:01

where there's this ark of the covenant.

121:02

You've seen Indiana Jones, right?

121:03

>> Yeah. And and the first Raiders of the

121:05

Lost Ark, there's this ark and you open

121:08

it up and this is where the two tablets

121:10

of stone, the 10 Commandments, rested.

121:13

So, in

121:15

that area you have two tablets of stone

121:19

written by the hand of God

121:22

the code

121:24

of life.

121:25

And according to Jewish and and

121:27

Christian belief that this is the law

121:29

and if you break the law that's how sin

121:31

is and the consequences of sin is

121:33

disease and death.

121:35

Well, when we get to the nucleus of the

121:37

human body

121:39

you have two strands of DNA.

121:41

And on the strands of DNA is the code.

121:45

The nucleotides of which is the code of

121:47

life. If you manipulate that code that

121:49

leads to mutations which leads to

121:52

disease and death.

121:55

None of this was known until 1950 when

121:58

they discovered DNA. And yet

122:00

we have Paul

122:02

who's making this jump and saying your

122:05

body is the temple of the Holy Spirit.

122:07

I just I just find that fascinating.

122:10

Nobody ever asked me about that. But

122:11

that's what As soon as you ask that

122:13

question, that's the first thing that

122:15

By the way, there's so many other places

122:17

in in in the scriptures where I see that

122:20

alluded to. Um

122:22

it's fascinating. Paul Paul talks about

122:23

the body of Christ and how it's

122:26

one body, but it's made up of parts.

122:29

There's the hand, the foot. He didn't

122:30

know about cells, but that's exactly

122:32

what the human body is. The human body

122:33

is one body made up of many parts. Van

122:36

Leeuwenhoek didn't discover that until

122:37

the 1600s. We didn't have cell theory

122:40

until the 1800s.

122:42

So, this is it's interesting to me how

122:44

statements are made in ancient texts

122:47

which have scientific relevance far

122:49

below the surface.

122:51

I just find that interesting.

122:53

Thank you so much and I hope to speak to

122:54

you again very, very soon. And thank you

122:55

for all the work you're doing because

122:56

you've made some of these difficult

122:58

scientific subjects so unbelievably

123:00

accessible. You have a real art for

123:04

simplifying.

123:05

And simplifying in a way that means that

123:07

millions of people you've got millions

123:09

of subscribers on your YouTube channel.

123:11

Millions of people can access this

123:12

information which is often confined

123:14

within the walls of some academic study.

123:16

So, thank you for the work you're doing

123:17

because it's going to it's I'm sure it's

123:18

already had a profound impact on many,

123:20

many millions of people's lives and I'm

123:22

sure my audience are deeply

123:23

appreciative. So, thank you so much

123:24

Roger. I appreciate you. Thank you

123:25

Steven. Thank you for having me on and

123:26

having this opportunity.

123:29

Make sure you keep what I'm about to say

123:31

to yourself. I'm inviting 10,000 of you

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you.

Interactive Summary

Dr. Roger Seholt, a critical care physician, outlines eight pillars of health (NEWSTART: Nutrition, Exercise, Water, Sunlight, Temperance, Air, Rest, and Trust) that can strengthen organ systems and prevent chronic diseases. He emphasizes the importance of sunlight for mitochondrial health, explaining how infrared light penetrates deep into the body to support cellular energy production through melatonin regulation, contrasting this with simple Vitamin D supplementation. He further discusses how lifestyle interventions like saunas, time in nature, and managing sleep environments contribute to overall well-being and immune system resilience.

Suggested questions

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