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THIS Is How You Actually Lose Dangerous Visceral Fat

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THIS Is How You Actually Lose Dangerous Visceral Fat

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637 segments

0:00

It sounds like you train fasted.

0:02

>> I listen to how I feel. That's exactly

0:04

what I do. So, what what your podcast

0:07

guest and and the researchers are

0:08

talking about is exactly there are times

0:10

when I wake up in the morning and I'm

0:12

like, I need to eat something before I

0:14

work out.

0:15

>> And I do um but I often times do train

0:19

fasted one because I am practicing

0:21

intermittent fasting again. But I do it

0:23

I'm not like starving myself. And like I

0:25

said, the reason there's multiple

0:26

reasons I do it. One reason is because

0:28

it really did help me lose the belly fat

0:31

which is the visceral fat which is like

0:32

the worst kind of fat you can have and

0:34

we can talk more about that. Um but the

0:36

second reason is I love the cognitive

0:38

benefits I have in the morning with it

0:40

and it's the main reason I do it and so

0:42

there are many times when I do train

0:43

fasted but I am not out running 15

0:45

miles. Most of my my like I said my

0:48

sessions are about an hour long and am I

0:52

taking a little bit of a performance hit

0:53

with the high intensity? probably

0:55

probably yes but it's not much to matter

0:58

for me and you do burn a little bit more

1:01

fat if you train fasted I mean that's

1:02

known um you will if it is a longer

1:05

session you will take an import

1:06

performance enhancement hit that is also

1:08

known right so I think it really does

1:11

come down to like what is your goal how

1:13

do you feel and and then you kind of go

1:16

with that and I I completely agree like

1:18

there are times when you know I'm on my

1:20

cycle and I feel fine and I'm working

1:23

out just fine And there are time other

1:25

times that I'm like I don't feel good.

1:26

Like I'm going to I'm going to take it

1:27

easier. I still train. I just you listen

1:30

to your body. And I that's a pretty easy

1:33

>> I think rule of thumb. Sometimes people

1:35

like to complicate things, you know. I

1:37

don't there's lots of reasons why. I

1:39

don't we don't need to get into that.

1:40

>> I have theories. Yeah. I have theories,

1:42

but they're not important right now.

1:43

>> No, it's not important. Yeah. So, so

1:44

yeah, I do I do train fasted and it is

1:47

um for me it is has helped me

1:49

tremendously change my body composition.

1:51

Like I said, I'm in a different part of

1:53

my life than perhaps a 30-year-old woman

1:55

is, right? So, when I was 30, I mean,

1:57

like I I didn't have to train fasted. It

1:59

was it was it was easy to keep, you

2:01

know, the the belly fat, the visceral

2:03

fat lower. Um, hormonal changes are do

2:06

play a role in the way your body so

2:08

estrogen plays a role in telling your

2:11

body how to store fat. So, subcutaneous

2:13

fat would be the kind of fat that you

2:14

can just like pinch, right? the the fat

2:16

that we see, the visceral fat. That's

2:18

that deep fat that's lining your organs.

2:21

It's often belly fat, you know, and it's

2:24

lining the intestines, the liver, you

2:26

know, it's it's it's it's an it's almost

2:28

like an endocrine organ. I mean, because

2:30

it is secretreting hormones. It's

2:32

secretreting inflammatory factors. It's

2:35

metabolically active. It's constantly

2:36

breaking down triglycerides. It's

2:39

associated with double the increased

2:40

risk, double the risk of early death.

2:43

Um, people that have high visceral fat

2:44

have 44% higher chance of having cancer.

2:47

Many different types of cancers. Wow.

2:48

>> It's huge. It's huge, you know, and and

2:51

and of course insulin resistance is the

2:52

number one problem with visceral fat,

2:54

right? And I'd love to to talk about

2:56

that, but um if you want, I mean, we can

2:59

we can get into that. Yeah, it's it's so

3:01

>> with the visceral fat and like I said,

3:02

you know, visceral fat is something if

3:04

you really directly want to measure it,

3:06

you do a DEXA scan. But, you know, for

3:08

the average person that isn't going to

3:10

go out and do a DEXA scan, waist

3:12

circumference is a proxy. It's used in a

3:14

lot of studies. So, women that have a

3:16

waist circumference of 35 in or above

3:19

are considered to have a higher amount

3:20

of visceral fat. Men that have a waist

3:22

circumference of 40 in or above are

3:24

considered to have higher amount of

3:26

visceral fat. It's also that belly fat.

3:28

Like, you can you can you just know,

3:29

right? Um, interestingly, like 70% of

3:32

women over the age of 50 have high

3:35

visceral fat. 50% of men over the age of

3:37

50 do you know again coming down to

3:40

women go through menopause estrogen

3:43

plays an important role in telling the

3:45

body you know to store the fat

3:47

subcutaneously rather than viscerally

3:49

deep around organs and so as women

3:52

transition to pmenopause you know the

3:54

years before menopause and menopause it

3:57

their estrogen goes down and that does

3:59

change the way the body stores fat and

4:02

any woman that's going through either of

4:04

those stages knows And it's also why you

4:06

see often women over the age of 50 with

4:08

more belly fat, right? I mean, that's

4:10

something that I think it's it's hard to

4:13

deny, but um it's it's one of the

4:17

reasons why I kind of went back to

4:19

practicing intermittent fasting because

4:21

there's a there's a couple of ways that

4:23

you can really powerfully lose visceral

4:25

fat. And one of them is doing aerobic

4:28

exercise, high-intensity interval

4:29

training also really powerfully can do

4:31

it, but also being in a caloric deficit.

4:33

And I think when you start to get the

4:35

combination of both, that's what really

4:37

worked for me. It's crazy how quickly

4:39

you can gain it based on your diet as

4:42

well. So it is different from the

4:45

subcutaneous fat in many ways. I

4:47

mentioned it's secretreting these

4:49

inflammatory molecules. It's, you know,

4:51

hormones, but it's also constantly

4:54

breaking down triglycerides into free

4:56

fatty acids. And the location of it is

4:58

very dangerous because it's right

5:00

surrounding the liver, right? It's this

5:02

deep organ fat and that's very close to

5:05

the portal vein. And so you're

5:06

constantly getting this sort of

5:08

mainlinining free fatty acids to the

5:10

liver. And visceral fat is very

5:12

different from subcutaneous fat because

5:15

it doesn't respond to insulin like

5:17

subcutaneous fat does. In other words,

5:19

when you have a meal, you eat a

5:20

carbohydrate meal and you basically your

5:23

body increases insulin to help take it

5:25

up glucose into your liver uh muscle

5:28

atapost tissue lipolysis shuts down,

5:30

right? It's like, okay, no longer am I

5:31

going to break down these fats. It's

5:33

time to use this energy, right? Visceral

5:35

fat doesn't respond to insulin. So, it

5:37

just keeps going, right? And these free

5:39

fatty acids because they're going right

5:40

to the liver, uh it's it's essentially

5:43

antagonizing the insulin receptors. So,

5:46

it causes insulin receptors to become

5:49

more resistant to insulin. And this is

5:52

part of why people with high visceral

5:55

fat, by the way, you can gain visceral

5:57

fat without gaining a pound. And we can

6:00

talk about those studies like people

6:01

people are skinny and can have high

6:04

amounts of visceral fat. You've you've

6:06

heard of like lean metabolically

6:07

unhealthy but lean individuals. Those

6:10

people exist and so you can have a high

6:12

amount of visceral fat but not really

6:15

look like you do. So um when you know

6:18

obviously the insulin resistance is a

6:20

problem for many reasons but it also

6:23

plays a role in those energy crashes

6:26

that you experience right and that's

6:27

kind of like some of the first signs of

6:30

insulin resistance actually have to do

6:31

with what you're feeling. So we talked

6:32

about lethargy, right? So you know the

6:35

inflammation that's being generated from

6:37

these this visceral fat constantly

6:39

making these, you know, pro-inflammatory

6:42

compounds are an it's an energy sink,

6:44

right? So you do constantly feel tired,

6:47

but also because your cells are becoming

6:49

insulin resistant.

6:51

When you have a high glucose meal and

6:54

you're not responding, the body kind of

6:56

overcompensates and produces more

6:58

insulin. So it's like I we got to get

7:00

this blood glucose out of our system,

7:02

right? It can cause a lot of damage if

7:03

it sits around there. And so you make

7:05

more insulin and then what happens is

7:07

you your blood glucose goes way low

7:08

because it was like this

7:09

overcompensation, right? And then you

7:12

feel a crash. You feel like this crash

7:14

and that signals to the you know

7:15

hypothalamus part of the brain. Uh I

7:17

need energy, right? So then you you sort

7:20

of crave you get those cravings for

7:21

those calorically energetic dense foods.

7:24

What I'm talking about is like the

7:26

experience of like,

7:28

>> you know, insulin resistance. And what's

7:31

interesting is that you can cause

7:33

someone to gain visceral fat and and

7:35

their brain can become insulin

7:36

resistant. So we think a lot about

7:37

insulin resistance in the muscle, liver,

7:40

your brain also can become insulin

7:42

resistance quite quickly actually. So um

7:45

insulin is very important in the brain

7:47

for a lot of reasons as you know but you

7:49

know a couple of the things relative to

7:51

what we're relevant to what we're

7:53

talking about would be one is it does

7:55

act on the hypothalamus and help you

7:58

know tell it to basically um stop eating

8:02

be satiated like I took a meal in okay

8:04

like I'm going to be satiated but it

8:06

also plays a role in energy storage and

8:08

telling the the body how to store the

8:09

energy and so when your brain becomes

8:11

insulin resistant it's not doing that

8:14

and so you're not being satiated so you

8:16

eat more and you're storing the fat more

8:18

viscerally. And there was a study that

8:22

was published actually quite recently. I

8:24

covered this in a in a recent

8:26

newsletter. It was a really interesting

8:27

study because it was healthy young men

8:29

and researchers put them on a

8:33

little bit of a calorically dense. So it

8:35

was like they were eating 1,200 to 1500

8:37

more calories a day and it was high

8:39

saturated fat, high sugar. So it was a

8:41

processed foods, ultrarocessed foods

8:42

like you know ultimate right

8:44

>> that's a lot of extra calories

8:45

>> a lot of extra calories in five days it

8:47

is but what happened was their they did

8:51

cause their brain to become insulin

8:52

resistant and they didn't gain weight

8:55

but they gained visceral fat and they

8:57

started gaining fat around their liver

8:58

and that's something that happens as

8:59

well because visceral fat is surrounding

9:01

liver you're getting a lot of free fatty

9:02

acids and they're going right to the

9:04

liver so the liver has to store it right

9:05

so you get this non-alcoholic you know

9:07

fatty fatty liver but and that happened

9:10

after 5 days. I mean without gain

9:12

>> in otherwise young healthy

9:13

>> Yeah. but you know they were eating a

9:16

lot of calories extra calories.

9:17

>> Yeah. That's like an at 1,200 that's

9:19

like a half a pizza

9:22

extra above your maintenance calories.

9:24

>> That's probably what they were doing.

9:25

They were eating lots of Well, they were

9:26

eating like saturated fat and refined

9:28

sugar. So

9:28

>> burritos and French fries.

9:29

>> Yeah. I mean obviously if you're going

9:30

to do the study you want to kind of do

9:32

it to a degree where you're going to see

9:33

some change, right? So, so maybe like

9:36

maybe it's not going to happen in five

9:38

days if you're if you're only eating 500

9:40

more calories a day, but over time you

9:42

will be gaining visceral fat, right? So,

9:44

it's not going to be the same degree.

9:46

It's something to be concerned about.

9:48

It's something to think about and also

9:50

because you can gain it and not really

9:52

even know it like you know without

9:55

gaining a pound. And there are other

9:57

things that cause it not just you know

9:58

eating too many calories or diet

9:59

composition. I mean you mentioned

10:01

cortisol. I mean, chronic elevated

10:03

cortisol makes you store the fat around

10:05

around, you know, visceral fat. Sleep

10:07

loss. I mean, there's also studies

10:08

showing that you take healthy men, sleep

10:10

deprive them for a couple of weeks, I

10:12

think four hours, they're getting four

10:13

hours of sleep a night, they can start

10:15

gaining visceral fat, I mean, pretty

10:17

rapidly with only like a pound, gaining

10:19

a pound of weight. So, again, it's like

10:21

not necessarily something that you're

10:22

going to see on the scale, but it's

10:24

happening, right? And it's affecting

10:26

your short-term mood. I mean, how you

10:27

feel, your energy, it's affecting, you

10:30

know, the way you're eating. It's a

10:31

vicious cycle because you start to eat

10:32

more calories, right? And then it just

10:35

becomes this vicious cycle of that you

10:36

start to gain more visceral fat.

10:38

>> Could you tell us what the structure of

10:39

the intermittent fasting is for you?

10:40

Does that mean skipping breakfast,

10:42

skipping lunch, skipping dinner? Because

10:44

I know a number of people are sort of

10:46

getting drawn back to intermittent

10:48

fasting after a couple years of it

10:49

getting beat up on like not the best way

10:51

to lose fat or it is a I think it's a

10:53

terrific way to do the sorts of things

10:56

that you're describing and I'm learning

10:58

today more about the positive things it

10:59

can do. um for insulin sensitivity and

11:02

so forth. If you're on a bout of

11:05

intermittent fasting, are you doing it

11:07

by the clock? Are you doing it by feel?

11:09

What does it look like?

11:10

>> For me, it it it really does depend on

11:13

the day. And I really do try to stop

11:15

eating 3 hours before I go to bed. It

11:17

doesn't always happen, you know, with

11:19

family obligations, social obligations,

11:20

but it's the habit that's important,

11:22

right? So, intermittent fasting, you

11:25

know, it's more than just one

11:26

intervention. As I mentioned, it's a

11:27

behavioral tool that you can use to

11:30

limit your calorie intake with actually

11:32

without actually having to count all the

11:33

calories, which some people like to do,

11:35

some people don't, right? So, it's a

11:37

tool, but also it's really important for

11:39

a metabolic switch, as you mentioned,

11:41

insulin sensitivity. And the metabolic

11:43

switch is something that Dr. Mark Matson

11:46

coined, and I love it because I feel

11:48

like, you know, thinking about

11:50

intermittent fasting in that way makes

11:53

it a little more clear as to the

11:55

benefits of it. It depends on the meal

11:57

you have and how much exercise you do,

11:58

right? But on average, let's say 11 12

12:00

hours to deplete your liver glycogen

12:03

levels. And once that happens, you do

12:05

start to burn fat and use fatty acids as

12:09

fuel and make ketone body. So you go

12:11

into ketogenesis, right? And that's a

12:13

metabolic switch, metabolic flexibility.

12:16

You're not you're going from using

12:17

carbohydrates as fuel to using fatty

12:19

acids and making ketone bodies as fuel.

12:21

And that's something that, you know,

12:22

throughout human evolution was

12:24

ingrained, right? Like we didn't always

12:27

have access to Uber Eats and Instacart

12:29

and you just at a swipe you get food,

12:32

right? I mean there were many times when

12:34

you know people had to

12:36

>> not eat because they couldn't forge

12:38

their foods maybe the time of year or

12:40

they couldn't hunt their food because

12:41

they didn't get a win or whatever. I

12:44

don't know. So this metabolic

12:45

flexibility is something that's really

12:47

ingrained in in our in our our DNA in a

12:50

sense, right? I mean, one of the reasons

12:52

I like to do that is the ketone

12:54

production. And why is that? You know,

12:57

ketones are really clean. They're clean,

13:00

a clean way to burn energy. So, they

13:02

generate less oxidative stress, less

13:05

oxidative products, but they also are

13:09

energetically favorable in that it takes

13:11

less energy to use them to make energy

13:13

than glucose does. So, it takes more

13:15

energy to use glucose as energy than it

13:16

does ketones. But, they're also a

13:19

signaling molecule. So, it's a way for

13:21

the body, you know, to signal to other

13:23

parts of the body like, hey, this is a

13:26

stressful time. There's no food. I'm,

13:28

you know, burn. I'm I'm in ketosis. Um,

13:31

let's let's make you stronger, right?

13:34

Because that's kind of what evolution

13:35

wants. Like, if you're not able to find

13:37

or eat food, you have to be stronger to

13:39

be able to do it, right? And so, that's

13:41

that's kind of at the I say core of of

13:44

this metabolic switch and why it's

13:46

important. And I think that I really

13:48

like Mark Matson being the pioneer in

13:49

this and as a neuroscientist really

13:51

looking at the benefits in the brain as

13:53

well. You know these ketones like beta

13:55

hydroxybutyrate are activating you know

13:59

growth factors like brain derived

14:00

neurotro trophic factor in the brain as

14:02

you know it's very important for you

14:04

know learning memory you know synapse

14:07

formation and stuff that's hugely

14:09

important for um neuroplasticity. So,

14:12

it's activating beneficial compounds

14:14

like that. And again, um it it it's not

14:18

going to happen if you're never going

14:19

into this metabolic switch. And there's

14:21

other ways to get there, right? So, you

14:22

can limit your your food, go into this

14:24

ketosis, right? Where you're basically

14:26

depleting your liver glycogen or you can

14:29

exercise a lot, right? So, your energy

14:31

expenditure goes up. So, there's there's

14:33

different ways to get to this metabolic

14:34

switch. It doesn't necessarily have to

14:36

be intermittent fasting. Got And I'm

14:38

telling you this because you you I want

14:39

to tell you why I'm I'm sort of back

14:41

back on the intermittent fasting. And by

14:43

the way, it's not for everyone. Like I

14:45

said, I mean, I think that you can find

14:47

other ways to get this metabolic switch.

14:49

And um for me, intermittent fasting

14:51

works. And so what I do typically is I

14:54

will do most of the time my fast my

14:56

workouts will be fasted. Not all the

14:57

time. It it does depend on how I feel.

14:59

>> What time do you typically wake up?

15:01

>> So I wake up like between 6:00 and 7:00.

15:04

And if I didn't have a family, like, you

15:07

know, getting my son ready and I would

15:08

probably work out right away, but I

15:10

don't end up working out until like

15:12

8:30.

15:14

So, I'm, you know, and

15:15

>> still pretty early. What time do you go

15:16

to sleep, if you don't mind me asking?

15:18

>> I'm I'm asleep like I'm asleep by 10:00.

15:21

I usually in bed at 9:00 takes and an

15:23

hour of just like, you know, hanging out

15:25

and

15:26

>> yeah. So, so um or 9:30 sometimes, but

15:30

yeah, usually 10:00 is when I'm

15:31

sleeping. That's my my bedtime. And uh

15:34

so I do stop eating I try to stop eating

15:36

by 7 p.m. But typically my first meal on

15:40

it depends on the day, but it'll usually

15:42

be like around 11:00, maybe sometimes

15:44

12. If I'm doing a podcast, it'll be

15:46

later. And um I do like to be fasted in

15:49

the morning because the cognitive

15:51

benefits is is really what I'm

15:52

interested in with that metabolic

15:54

switch. And it does come down to ketones

15:56

and I know that u Mark Matson's talked a

15:58

lot about this. I had him on my podcast

16:00

a couple year a few years ago actually.

16:01

I learned so much. But the ketones like

16:03

beta hydroxybutyrate are increasing

16:05

GABA. They're like balancing the

16:07

glutamate, the you know excitatory

16:09

neurotransmitter with the inhibitory

16:11

one, GABA. And I think the increasing

16:14

GABA is what helps me and what I love

16:16

the most because it does help I think

16:19

quiet down some of the other I don't

16:21

know chitter chatter in my brain and

16:23

help me focus because it's like somehow

16:26

the GABA is calming in a way. I don't I

16:28

don't know exactly. You could probably

16:29

describe it better than I can. All I

16:30

know is that I really like it. And so in

16:33

the mornings, I like to be, you know, I

16:35

like to be fasted. I like to be

16:36

cognitively aware. It's when I get most

16:38

of my productive work done

16:40

>> and I feel smarter. So I'm doing it. And

16:43

that's why I typically like to shift my

16:46

breakfast to later. Now, I do I would

16:49

say some days I do only eat two meals

16:50

where I am on honestly I'm skipping the

16:53

quote unquote breakfast,

16:55

>> but uh some days I do have three meals

16:59

and you know oftent times they're like

17:01

different sized meals, right? And

17:03

usually my if I do have three meals, the

17:05

the the third one will be the in between

17:08

the first meal and the second meal will

17:10

be like a very like a half a or a three4

17:13

of a turkey burger or something. So it's

17:15

it's mostly protein and it's not heavy.

17:18

>> But so I would say I'm you know usually

17:20

11 to 7 probably is when I'm eating my

17:23

meals most most days. Some days it's you

17:26

know I I fast for a shorter period of

17:27

time.

17:28

>> Some days I wake up in the morning

17:30

>> because I I did too early of of like I

17:32

you know I stopped eating like like four

17:34

or five hours before bed and I'm like I

17:36

need to eat and I guess what I eat and

17:38

then I work out and it's like that's you

17:40

know you kind of just listen to to what

17:42

your body's doing. Actually, Mark Matson

17:43

just published a study very very

17:45

recently showing that he did a I think

17:48

he was doing a 52 intermittent fasting

17:50

protocol where it's like two days you're

17:52

getting pretty severe caloric

17:53

restriction like you're eating one meal

17:55

but it's like 500 calories you know for

17:58

that for the day and it's twice two days

18:00

two days out of the week the other five

18:01

days you're eating normal and compared

18:03

that to calorie like eating eating like

18:05

a healthy diet and they were somewhat

18:06

calorie restricted but not quite as much

18:08

as the people that were doing the

18:09

fasting and they had the fasting group

18:11

had massive cognitive benefit. It's like

18:12

20% improvement in a battery of tests

18:15

that were done.

18:16

>> He attributes that to the ketones and

18:17

the effect of ketones on

18:18

>> GABA. He attributes it to the ketones

18:21

and like can you get that to some degree

18:23

with caloric restriction? Probably.

18:24

Especially the more severe caloric

18:25

restriction you do. You can if we're

18:27

talking about weight loss, caloric

18:29

restriction is key, right? You have to

18:31

have that. If we're talking about the

18:33

cardiovascular reset, like caloric

18:35

restriction, if you're eating meals

18:37

right before bed, you're not going to

18:38

get that. Like that's those studies have

18:39

been done. And I think, you know,

18:40

Courtney Peterson was some of the one of

18:42

the first ones to really show that

18:43

effect on blood pressure, you know, like

18:46

really significant. In fact, if you if

18:48

you do early timerestricted eating and

18:50

stop eating, you know, I don't know how

18:52

early in the day it was, maybe 6 p.m.

18:54

Maybe it was 8:00 p.m. But, you know,

18:55

there was like a blood pressure drop

18:57

that was like 10, you know, 10 I think

19:00

was um uh points or something

19:03

millimeters of mercury.

19:05

>> Significant.

19:05

>> What is it? Is it

19:06

>> Yeah.

19:07

>> So, I mean, that's very significant.

19:08

It's on it's on magnitude of what you'd

19:10

see with like some of the firstline, you

19:12

know, drugs that are used to treat

19:14

hypertension. I would never say for

19:16

someone to do that, but I'm just saying

19:17

it's significant and that's not

19:19

something that you typically see if

19:20

you're eating, you know, fewer calories,

19:22

but you're eating constantly throughout

19:23

the day.

19:24

>> So, I do think there are special

19:26

benefits that can be had, but again,

19:28

it's also a tool that people use. I use

19:30

it as a tool as well. I don't like to

19:32

count calories. like I don't you know

19:33

some people do and that's fine then you

19:36

know that's that's the way you can you

19:37

can do it but I like it for the

19:40

metabolic switch as well. Um, if I were

19:42

an endurance athlete running, you know,

19:45

10, 14 miles a day, I wouldn't have to

19:47

do this. That would be my metabolic

19:49

switch. And it's not that I'm not

19:51

metabolically flexible. I mean, I do I

19:53

think you can just training itself, you

19:57

know, does to some degree help with

19:58

metabolic flexibility, right? The

20:00

ability to switch between burning

20:02

glucose and carbohydrates and and then

20:04

burning burn using fat, fat, and fatty

20:06

acids as your energy source. It's that I

20:10

really I want that. I want that real

20:12

switch to be on and I want it to be on

20:14

for a little bit of of time and then I

20:15

want to turn it off by eating. You know,

20:17

I'm not starving myself. And I think you

20:19

can go too far with exercise and with

20:22

fasting.

20:23

>> The problems with fasting and the people

20:24

that are like, "Oh, fasting is terrible

20:26

for you." I mean, it comes down to one,

20:28

they were all about it's like the weight

20:30

loss is not just due to intermittent

20:32

fasting itself. It's due to calorie

20:33

restriction. Guess what? They were

20:34

right. They were right. Number two,

20:36

they're afraid of losing muscle. And I

20:38

think we have enough data now that it's

20:40

if you're training, if you're doing

20:41

resistance training, you're not going to

20:43

be losing muscle. Now, maybe you won't

20:44

gain as much as if you were eating more

20:48

protein, but um you could you could just

20:50

eat more protein um within the window

20:53

that you're eating and be fine, right?

20:55

So, I think that's the other thing that

20:57

people are worried about is muscle loss,

21:00

including myself. And I train so much

21:02

now and it just works well for me and I

21:04

feel good and it's like this clean

21:06

feeling. you feel cognitively sharp and

21:09

it works. And I think that the metabolic

21:10

switch is something to not be scared of

21:13

as long as you're not again going to the

21:15

extreme, right? And if you don't want to

21:17

train while you're fast, don't train

21:18

while you're fasted. You don't have to.

21:20

You know, I I think there's a little bit

21:22

of an added benefit that works for me in

21:25

terms of burning fat, which is what I,

21:27

you know, particularly visceral fat. And

21:29

to me, that that's that's what works

21:31

well. But um you can obviously like if

21:34

you're training hard and a lot and

21:37

really fasting maybe that's too much.

21:38

It's too much of a stress. So you have

21:40

to kind of figure it out for

Interactive Summary

The video discusses the practice of intermittent fasting, focusing on its benefits for managing visceral fat, improving metabolic flexibility, and enhancing cognitive performance. The speaker explains the physiological process of the 'metabolic switch,' where the body shifts from burning glucose to burning stored fat and ketone bodies, and highlights the risks associated with visceral fat, including insulin resistance and inflammation. Practical advice is shared on implementing intermittent fasting, emphasizing listening to one's body, the importance of diet quality, and how factors like sleep and stress impact metabolic health.

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